ALA - direct inhibitor of retroviral replication

[Guest guest]

again unless this stuff is also addressed BEFORE being trapped, which is why kids UNDER 5 have an easier time recovering with out having to do chelation and fixing the body to do it's own job

..

Re: ALA - direct inhibitor of> retroviral replication> >> >> > "Could it be that the chelators work as antiretrovirals by> removing mercury? "> >> > , that is not the case, that is not how chelators> work here. They work by removing ions from cation-binding viral> proteins, causing unfolding/loss of protein functionality. For example> by opening zinc-finger proteins and similar. There are other mechanisms> observed, such as inhibiting viral DNA synthesis through RNA reductase> deactivation, see attached> >> > btw some of these studies are in vitro, no mercury in sight,> so the answer is NO they do not work as antivirals through chelating> mercury, at least not exclusively (meaning to say that who knows maybe> heavy metals are also directly utilised by some viruses)> >> > And whatever is observed to happen in HIV only is only> because HIV is studied lot more than others. There are no studies that> show other viral proteins would not be unfolding in the same way when> chelators added. Having said that there are studies on herpes/CMV and> others that indicate same things going on.> >> >> >> > The influence of divalent cations on the stability of human> rotavirus.> > The influence of divalent cations on the stability of human> rotavirus was investigated using the indirect immunofluorescence (FA)> technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity> was stabilized by calcium and strontium ions in solution but not by> magnesium ions. Rotavirus isolates were found to be much less stable at> 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus> morphology at intervals during the course of the experiment and> treatment with the chelating agents EDTA and EGTA suggests that loss of> infectivity coincides with the removal of the outer capsid layer and> that calcium may be required to maintain virus integrity. Arch Virol.> 1981;67(1):1-9. Shirley JA, Beards GM, Thouless ME, Flewett TH> >> >> > Biochemical studies of primate retroviruses.> > In the present paper, recent biochemical studies of> retroviruses carried out in our laboratory are summarized. Protein> compositions, peptide maps of internal structural proteins,> neighborhoods of major structural proteins, and serological properties> of reverse transcriptases of type D virus isolates from human cells> (including Graffi's isolate termed PMFV and also isolates from HeLa- and> HEp-2 cells) were compared with those of type D viruses from Old World> (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World> (squirrel monkey retrovirus) monkeys. The results provide various new> informations on, and further demonstrate the diversity of primate type D> viruses. Other studies showed that tumor promoting agents (phorbol ester> TPA, indole alkaloid teleocidin) are able to considerably increase, in a> transient manner, the production of type C and type D primate> retroviruses in persistently infected human cells. From experiments> demonstrating a disintegrating activity of chelating agents (EDTA, EGTA)> and certain psychoactive drugs (including trifluoperazine) on various> primate and nonprimate retroviruses it is concluded that divalent> cations, probably Ca2+ ions, and possibly also cation-binding proteins> are associated with retroviral membranes and that complexing of these> components results in loss of viral infectivity. Wunderlich Very, Uckert> W, Sydow G Arch Geschwulstforsch. 1983;53(3):267-78.> >>

[Guest guest]

again unless this stuff is also addressed BEFORE being trapped, which is why kids UNDER 5 have an easier time recovering with out having to do chelation and fixing the body to do it's own job

..

Re: ALA - direct inhibitor of> retroviral replication> >> >> > "Could it be that the chelators work as antiretrovirals by> removing mercury? "> >> > , that is not the case, that is not how chelators> work here. They work by removing ions from cation-binding viral> proteins, causing unfolding/loss of protein functionality. For example> by opening zinc-finger proteins and similar. There are other mechanisms> observed, such as inhibiting viral DNA synthesis through RNA reductase> deactivation, see attached> >> > btw some of these studies are in vitro, no mercury in sight,> so the answer is NO they do not work as antivirals through chelating> mercury, at least not exclusively (meaning to say that who knows maybe> heavy metals are also directly utilised by some viruses)> >> > And whatever is observed to happen in HIV only is only> because HIV is studied lot more than others. There are no studies that> show other viral proteins would not be unfolding in the same way when> chelators added. Having said that there are studies on herpes/CMV and> others that indicate same things going on.> >> >> >> > The influence of divalent cations on the stability of human> rotavirus.> > The influence of divalent cations on the stability of human> rotavirus was investigated using the indirect immunofluorescence (FA)> technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity> was stabilized by calcium and strontium ions in solution but not by> magnesium ions. Rotavirus isolates were found to be much less stable at> 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus> morphology at intervals during the course of the experiment and> treatment with the chelating agents EDTA and EGTA suggests that loss of> infectivity coincides with the removal of the outer capsid layer and> that calcium may be required to maintain virus integrity. Arch Virol.> 1981;67(1):1-9. Shirley JA, Beards GM, Thouless ME, Flewett TH> >> >> > Biochemical studies of primate retroviruses.> > In the present paper, recent biochemical studies of> retroviruses carried out in our laboratory are summarized. Protein> compositions, peptide maps of internal structural proteins,> neighborhoods of major structural proteins, and serological properties> of reverse transcriptases of type D virus isolates from human cells> (including Graffi's isolate termed PMFV and also isolates from HeLa- and> HEp-2 cells) were compared with those of type D viruses from Old World> (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World> (squirrel monkey retrovirus) monkeys. The results provide various new> informations on, and further demonstrate the diversity of primate type D> viruses. Other studies showed that tumor promoting agents (phorbol ester> TPA, indole alkaloid teleocidin) are able to considerably increase, in a> transient manner, the production of type C and type D primate> retroviruses in persistently infected human cells. From experiments> demonstrating a disintegrating activity of chelating agents (EDTA, EGTA)> and certain psychoactive drugs (including trifluoperazine) on various> primate and nonprimate retroviruses it is concluded that divalent> cations, probably Ca2+ ions, and possibly also cation-binding proteins> are associated with retroviral membranes and that complexing of these> components results in loss of viral infectivity. Wunderlich Very, Uckert> W, Sydow G Arch Geschwulstforsch. 1983;53(3):267-78.> >>

[Guest guest]

vit A protical helped our son, we pulled titers and his were more then 10 times the expected range. tetuns was also at cronic levels in him for titers, one of them was 42 times higher then expected levels just not sure what one it was meseals or tetnus

Re: ALA > > - direct inhibitor of retroviral replication> > > > â€Å"Could it be that the chelators work as > > antiretrovirals by removing mercury? â€Å"> > > > , that is not > > the case, that is not how chelators work here. They work by removing > > ions from cation-binding viral proteins, causing unfolding/loss of > > protein functionality. For example by opening zinc-finger proteins > > and similar. There are other mechanisms observed, such as inhibiting > > viral DNA synthesis through RNA reductase deactivation, see > > attached> > > > btw some of these studies are in vitro, no mercury > > in sight, so the answer is NO they do not work as antivirals through > > chelating mercury, at least not exclusively (meaning to say that who > > knows maybe heavy metals are also directly utilised by some > > viruses)> > > > And whatever is observed to happen in HIV only is > > only because HIV is studied lot more than others. There are no > > studies that show other viral proteins would not be unfolding in the > > same way when chelators added. Having said that there are studies on > > herpes/CMV and others that indicate same things going on. > > > > > > > >  > > The influence of divalent cations on the > > stability of human rotavirus.> > The influence of divalent > > cations on the stability of human rotavirus was investigated using > > the indirect immunofluorescence (FA) technique in LLC-MK2 cells to > > titrate infectivity. Rotavirus infectivity was stabilized by calcium > > and strontium ions in solution but not by magnesium ions. Rotavirus > > isolates were found to be much less stable at 37 degrees C than at + > > 4 degrees C or 20 degrees. A study of virus morphology at intervals > > during the course of the experiment and treatment with the chelating > > agents EDTA and EGTA suggests that loss of infectivity coincides > > with the removal of the outer capsid layer and that calcium may be > > required to maintain virus integrity. Arch Virol. > > 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett > > TH> > > > > > Biochemical studies of primate > > retroviruses.> > In the present paper, recent biochemical > > studies of retroviruses carried out in our laboratory are > > summarized. Protein compositions, peptide maps of internal > > structural proteins, neighborhoods of major structural proteins, and > > serological properties of reverse transcriptases of type D virus > > isolates from human cells (including Graffi's isolate termed PMFV > > and also isolates from HeLa- and HEp-2 cells) were compared with > > those of type D viruses from Old World (Mason-Pfizer virus of rhesus > > monkeys, langur virus) and New World (squirrel monkey retrovirus) > > monkeys. The results provide various new informations on, and > > further demonstrate the diversity of primate type D viruses. Other > > studies showed that tumor promoting agents (phorbol ester TPA, > > indole alkaloid teleocidin) are able to considerably increase, in a > > transient manner, the production of type C and type D primate > > retroviruses in persistently infected human cells. From experiments > > demonstrating a disintegrating activity of chelating agents (EDTA, > > EGTA) and certain psychoactive drugs (including trifluoperazine) on > > various primate and nonprimate retroviruses it is concluded that > > divalent cations, probably Ca2+ ions, and possibly also > > cation-binding proteins are associated with retroviral membranes and > > that complexing of these components results in loss of viral > > infectivity. Wunderlich Very, Uckert W, Sydow G Arch > > Geschwulstforsch. > > 1983;53(3):267- 78.> >>

[Guest guest]

rebecca ok NOW we have hit common ground....yes if you have metal issues you DO need to address them, there are ways to do that

Re: ALA - direct inhibitor of> > > > retroviral replication> > > > >> > > > >> > > > > "Could it be that the chelators work as antiretrovirals by> > > > removing mercury? "> > > > >> > > > > , that is not the case, that is not how chelators> > > > work here. They work by removing ions from cation-binding viral> > > > proteins, causing unfolding/loss of protein functionality. For example> > > > by opening zinc-finger proteins and similar. There are other mechanisms> > > > observed, such as inhibiting viral DNA synthesis through RNA reductase> > > > deactivation, see attached> > > > >> > > > > btw some of these studies are in vitro, no mercury in sight,> > > > so the answer is NO they do not work as antivirals through chelating> > > > mercury, at least not exclusively (meaning to say that who knows maybe> > > > heavy metals are also directly utilised by some viruses)> > > > >> > > > > And whatever is observed to happen in HIV only is only> > > > because HIV is studied lot more than others. There are no studies that> > > > show other viral proteins would not be unfolding in the same way when> > > > chelators added. Having said that there are studies on herpes/CMV and> > > > others that indicate same things going on.> > > > >> > > > >> > > > >> > > > > The influence of divalent cations on the stability of human> > > > rotavirus.> > > > > The influence of divalent cations on the stability of human> > > > rotavirus was investigated using the indirect immunofluorescence (FA)> > > > technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity> > > > was stabilized by calcium and strontium ions in solution but not by> > > > magnesium ions. Rotavirus isolates were found to be much less stable at> > > > 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus> > > > morphology at intervals during the course of the experiment and> > > > treatment with the chelating agents EDTA and EGTA suggests that loss of> > > > infectivity coincides with the removal of the outer capsid layer and> > > > that calcium may be required to maintain virus integrity. Arch Virol.> > > > 1981;67(1):1-9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > >> > > > >> > > > > Biochemical studies of primate retroviruses.> > > > > In the present paper, recent biochemical studies of> > > > retroviruses carried out in our laboratory are summarized. Protein> > > > compositions, peptide maps of internal structural proteins,> > > > neighborhoods of major structural proteins, and serological properties> > > > of reverse transcriptases of type D virus isolates from human cells> > > > (including Graffi's isolate termed PMFV and also isolates from HeLa- and> > > > HEp-2 cells) were compared with those of type D viruses from Old World> > > > (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World> > > > (squirrel monkey retrovirus) monkeys. The results provide various new> > > > informations on, and further demonstrate the diversity of primate type D> > > > viruses. Other studies showed that tumor promoting agents (phorbol ester> > > > TPA, indole alkaloid teleocidin) are able to considerably increase, in a> > > > transient manner, the production of type C and type D primate> > > > retroviruses in persistently infected human cells. From experiments> > > > demonstrating a disintegrating activity of chelating agents (EDTA, EGTA)> > > > and certain psychoactive drugs (including trifluoperazine) on various> > > > primate and nonprimate retroviruses it is concluded that divalent> > > > cations, probably Ca2+ ions, and possibly also cation-binding proteins> > > > are associated with retroviral membranes and that complexing of these> > > > components results in loss of viral infectivity. Wunderlich Very, Uckert> > > > W, Sydow G Arch Geschwulstforsch. 1983;53(3):267-78.> > > > >> > > >> > >> >>

[Guest guest]

rebecca ok NOW we have hit common ground....yes if you have metal issues you DO need to address them, there are ways to do that

Re: ALA - direct inhibitor of> > > > retroviral replication> > > > >> > > > >> > > > > "Could it be that the chelators work as antiretrovirals by> > > > removing mercury? "> > > > >> > > > > , that is not the case, that is not how chelators> > > > work here. They work by removing ions from cation-binding viral> > > > proteins, causing unfolding/loss of protein functionality. For example> > > > by opening zinc-finger proteins and similar. There are other mechanisms> > > > observed, such as inhibiting viral DNA synthesis through RNA reductase> > > > deactivation, see attached> > > > >> > > > > btw some of these studies are in vitro, no mercury in sight,> > > > so the answer is NO they do not work as antivirals through chelating> > > > mercury, at least not exclusively (meaning to say that who knows maybe> > > > heavy metals are also directly utilised by some viruses)> > > > >> > > > > And whatever is observed to happen in HIV only is only> > > > because HIV is studied lot more than others. There are no studies that> > > > show other viral proteins would not be unfolding in the same way when> > > > chelators added. Having said that there are studies on herpes/CMV and> > > > others that indicate same things going on.> > > > >> > > > >> > > > >> > > > > The influence of divalent cations on the stability of human> > > > rotavirus.> > > > > The influence of divalent cations on the stability of human> > > > rotavirus was investigated using the indirect immunofluorescence (FA)> > > > technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity> > > > was stabilized by calcium and strontium ions in solution but not by> > > > magnesium ions. Rotavirus isolates were found to be much less stable at> > > > 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus> > > > morphology at intervals during the course of the experiment and> > > > treatment with the chelating agents EDTA and EGTA suggests that loss of> > > > infectivity coincides with the removal of the outer capsid layer and> > > > that calcium may be required to maintain virus integrity. Arch Virol.> > > > 1981;67(1):1-9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > >> > > > >> > > > > Biochemical studies of primate retroviruses.> > > > > In the present paper, recent biochemical studies of> > > > retroviruses carried out in our laboratory are summarized. Protein> > > > compositions, peptide maps of internal structural proteins,> > > > neighborhoods of major structural proteins, and serological properties> > > > of reverse transcriptases of type D virus isolates from human cells> > > > (including Graffi's isolate termed PMFV and also isolates from HeLa- and> > > > HEp-2 cells) were compared with those of type D viruses from Old World> > > > (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World> > > > (squirrel monkey retrovirus) monkeys. The results provide various new> > > > informations on, and further demonstrate the diversity of primate type D> > > > viruses. Other studies showed that tumor promoting agents (phorbol ester> > > > TPA, indole alkaloid teleocidin) are able to considerably increase, in a> > > > transient manner, the production of type C and type D primate> > > > retroviruses in persistently infected human cells. From experiments> > > > demonstrating a disintegrating activity of chelating agents (EDTA, EGTA)> > > > and certain psychoactive drugs (including trifluoperazine) on various> > > > primate and nonprimate retroviruses it is concluded that divalent> > > > cations, probably Ca2+ ions, and possibly also cation-binding proteins> > > > are associated with retroviral membranes and that complexing of these> > > > components results in loss of viral infectivity. Wunderlich Very, Uckert> > > > W, Sydow G Arch Geschwulstforsch. 1983;53(3):267-78.> > > > >> > > >> > >> >>

[Guest guest]

I just have a different opinion than you.

> >

> >

> > From: Christel King <christelking1@>

> > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > To: mb12 valtrex

> > Date: Friday, November 20, 2009, 8:59 AM

> >

> >

> >

> >

> > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate! not ALL autistic kids have mercury

issues! my son didn't become autistic because he had metals, he became autistic

when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> >

> > Re: ALA - direct inhibitor of retroviral

replication

> >

> >

> >  " Could it be that the chelators work as antiretrovirals by removing

mercury?  "

> >

> > , that is not the case, that is not how chelators work here. They

work by removing ions from cation-binding viral proteins, causing unfolding/loss

of protein functionality. For example by opening zinc-finger proteins and

similar. There are other mechanisms observed, such as inhibiting viral DNA

synthesis through RNA reductase deactivation, see attached

> >

> > btw some of these studies are in vitro, no mercury in sight, so the answer

is NO they do not work as antivirals through chelating mercury, at least not

exclusively (meaning to say that who knows maybe heavy metals are also directly

utilised by some viruses)

> >

> > And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> >

> >

> >

> > The influence of divalent cations on the stability of human rotavirus.

> > The influence of divalent cations on the stability of human rotavirus was

investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> >

> >

> > Biochemical studies of primate retroviruses.

> > In the present paper, recent biochemical studies of retroviruses carried

out in our laboratory are summarized. Protein compositions, peptide maps of

internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> >

>

[Guest guest]

accept I can back that opinon with data of kids with out metal issues who still have autism. you can't show all kids with metal issues have autism, and all kids with autism have metal issues...and not all kids who chelate recover

Re: ALA - direct inhibitor of retroviral replication> > > > > > â€Å"Could it be that the chelators work as antiretrovirals by removing mercury? â€Å"> > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > Biochemical studies of primate retroviruses.> > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> >>

[Guest guest]

Christel,

Maybe all kids with autism have liver issues.

> > >

> > >

> > > From: Christel King <christelking1@>

> > > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > > To: mb12 valtrex

> > > Date: Friday, November 20, 2009, 8:59 AM

> > >

> > >

> > >

> > >

> > > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate! not ALL autistic kids have mercury

issues! my son didn't become autistic because he had metals, he became autistic

when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > >

> > > Re: ALA - direct inhibitor of retroviral

replication

> > >

> > >

> > >  " Could it be that the chelators work as antiretrovirals by

removing mercury?  "

> > >

> > > , that is not the case, that is not how chelators work here. They

work by removing ions from cation-binding viral proteins, causing unfolding/loss

of protein functionality. For example by opening zinc-finger proteins and

similar. There are other mechanisms observed, such as inhibiting viral DNA

synthesis through RNA reductase deactivation, see attached

> > >

> > > btw some of these studies are in vitro, no mercury in sight, so the

answer is NO they do not work as antivirals through chelating mercury, at least

not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > >

> > > And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > >

> > >

> > >

> > > The influence of divalent cations on the stability of human rotavirus.

> > > The influence of divalent cations on the stability of human rotavirus

was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > >

> > >

> > > Biochemical studies of primate retroviruses.

> > > In the present paper, recent biochemical studies of retroviruses carried

out in our laboratory are summarized. Protein compositions, peptide maps of

internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > >

> >

>

[Guest guest]

i have seen kids with out that as well.....see that is the problem, many dr's have exception to the rules of what we "think" may be the cause. it's the difference in figureing out the cause verses symptoms that makes it hard to put a finger on what came first. to find that cure and answer for all our kids,. I know kanners kids who have recovered which is thought to not happen since they are "born" that way and nothing caused them to regress they just never developed, I know kids with celiac who have been able to even reverse that part of their damage (3 cases) I know kids with no metals, no allergies, no yeast, no bacteria yet have autism, I know families that have done it all with no gains, I know kids who do nothing and have had gains....it's never just THIS Is what is wrong and if all kids did it all kids would recover...I wish it WAS that way, we could save generations of our kids....so we are all left for now until we figure out the genie in the bottle that made our kids this way to find other kids like our kids (since all are different) and find what worked for them, try it and hope for the same good chance.....we should NEVER discourage ANY parent from looking for others like them, or from trying this or that to get their kids back, and at the same time tell them the ONLY way to help there kid is to do this or that. I am a HUGE diet freak and for 100% and have seen it make a HUGE differenece in 1000s of kids. I advocage ALL kids try it, and as much as i would LOVE to say you can never recover a kid with out it, I know that's not true....some can....

Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > â€Å"Could it be that the chelators work as antiretrovirals by removing mercury? â€Å"> > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > > > Biochemical studies of primate retroviruses.> > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > >> >>

[Guest guest]

Come on Christel, we have a difference of opinion, the argument is fruitless. I

got a porphyrins test showing is completely within normal limits which

is completely wrong according to his Doctor's Data Hair Elements Test, Toxic

Urine Elements Test, and Toxic Hair Elements Tests. Testing just isn't very

accurate, so neither of us can prove anything, we have a difference of opinion.

> > >

> > >

> > > From: Christel King <christelking1@>

> > > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > > To: mb12 valtrex

> > > Date: Friday, November 20, 2009, 8:59 AM

> > >

> > >

> > >

> > >

> > > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate! not ALL autistic kids have mercury

issues! my son didn't become autistic because he had metals, he became autistic

when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > >

> > > Re: ALA - direct inhibitor of retroviral

replication

> > >

> > >

> > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > >

> > > , that is not the case, that is not how chelators work here. They

work by removing ions from cation-binding viral proteins, causing unfolding/loss

of protein functionality. For example by opening zinc-finger proteins and

similar. There are other mechanisms observed, such as inhibiting viral DNA

synthesis through RNA reductase deactivation, see attached

> > >

> > > btw some of these studies are in vitro, no mercury in sight, so the

answer is NO they do not work as antivirals through chelating mercury, at least

not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > >

> > > And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > >

> > >

> > >

> > > The influence of divalent cations on the stability of human rotavirus.

> > > The influence of divalent cations on the stability of human rotavirus

was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > >

> > >

> > > Biochemical studies of primate retroviruses.

> > > In the present paper, recent biochemical studies of retroviruses carried

out in our laboratory are summarized. Protein compositions, peptide maps of

internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > >

> >

>

[Guest guest]

Burn!!!! (think Kelso, that 70's show)

> > >

> > >

> > > From: Christel King <christelking1@>

> > > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > > To: mb12 valtrex

> > > Date: Friday, November 20, 2009, 8:59 AM

> > >

> > >

> > >

> > >

> > > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate! not ALL autistic kids have mercury

issues! my son didn't become autistic because he had metals, he became autistic

when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > >

> > > Re: ALA - direct inhibitor of retroviral

replication

> > >

> > >

> > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > >

> > > , that is not the case, that is not how chelators work here. They

work by removing ions from cation-binding viral proteins, causing unfolding/loss

of protein functionality. For example by opening zinc-finger proteins and

similar. There are other mechanisms observed, such as inhibiting viral DNA

synthesis through RNA reductase deactivation, see attached

> > >

> > > btw some of these studies are in vitro, no mercury in sight, so the

answer is NO they do not work as antivirals through chelating mercury, at least

not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > >

> > > And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > >

> > >

> > >

> > > The influence of divalent cations on the stability of human rotavirus.

> > > The influence of divalent cations on the stability of human rotavirus

was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > >

> > >

> > > Biochemical studies of primate retroviruses.

> > > In the present paper, recent biochemical studies of retroviruses carried

out in our laboratory are summarized. Protein compositions, peptide maps of

internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > >

> >

>

[Guest guest]

> > > > > > > > > From: Christel King <christelking1@>> > > Subject: Re: Re: ALA - direct inhibitor of retroviral replication> > > To: mb12 valtrex > > > Date: Friday, November 20, 2009, 8:59 AM> > > > > > > > > > > > > > > then I guess we don't have mercury in the brain because our son isn't autistic anymore....and we didn't chelate! not ALL autistic kids have mercury issues! my son didn't become autistic because he had metals, he became autistic when he got an MMR....in HOURS after it!n we had tested 2 weeks prior developmental testing and he was 6 months advanced. after 7 hours after his MMR he became autistic, lost lang, no eye contact, started seizering, ect. we tested him 2 weeks after this because I had no idea what had happened to my son, he then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have become autistic when the thermitor exploded at 6 months old in our dishwasher and he didn't then!!!! it would also mean that my son would STILL be autistic since we haven't chelated with your therory and he is not!> > > > > > Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > ââ,¬Å"Could it be that the chelators work as antiretrovirals by removing mercury? ââ,¬Å"> > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > > > Biochemical studies of primate retroviruses.> > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > >> >>

[Guest guest]

Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > ââ,¬Å"Could it be that the chelators work as antiretrovirals by removing mercury? ââ,¬Å"> > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > > > Biochemical studies of primate retroviruses.> > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > >> >>

[Guest guest]

Re: ALA - direct inhibitor of> retroviral replication> >> >> > "Could it be that the chelators work as antiretrovirals by> removing mercury? "> >> > , that is not the case, that is not how chelators> work here. They work by removing ions from cation-binding viral> proteins, causing unfolding/loss of protein functionality. For example> by opening zinc-finger proteins and similar. There are other mechanisms> observed, such as inhibiting viral DNA synthesis through RNA reductase> deactivation, see attached> >> > btw some of these studies are in vitro, no mercury in sight,> so the answer is NO they do not work as antivirals through chelating> mercury, at least not exclusively (meaning to say that who knows maybe> heavy metals are also directly utilised by some viruses)> >> > And whatever is observed to happen in HIV only is only> because HIV is studied lot more than others. There are no studies that> show other viral proteins would not be unfolding in the same way when> chelators added. Having said that there are studies on herpes/CMV and> others that indicate same things going on.> >> >> >> > The influence of divalent cations on the stability of human> rotavirus.> > The influence of divalent cations on the stability of human> rotavirus was investigated using the indirect immunofluorescence (FA)> technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity> was stabilized by calcium and strontium ions in solution but not by> magnesium ions. Rotavirus isolates were found to be much less stable at> 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus> morphology at intervals during the course of the experiment and> treatment with the chelating agents EDTA and EGTA suggests that loss of> infectivity coincides with the removal of the outer capsid layer and> that calcium may be required to maintain virus integrity. Arch Virol.> 1981;67(1):1-9. Shirley JA, Beards GM, Thouless ME, Flewett TH> >> >> > Biochemical studies of primate retroviruses.> > In the present paper, recent biochemical studies of> retroviruses carried out in our laboratory are summarized. Protein> compositions, peptide maps of internal structural proteins,> neighborhoods of major structural proteins, and serological properties> of reverse transcriptases of type D virus isolates from human cells> (including Graffi's isolate termed PMFV and also isolates from HeLa- and> HEp-2 cells) were compared with those of type D viruses from Old World> (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World> (squirrel monkey retrovirus) monkeys. The results provide various new> informations on, and further demonstrate the diversity of primate type D> viruses. Other studies showed that tumor promoting agents (phorbol ester> TPA, indole alkaloid teleocidin) are able to considerably increase, in a> transient manner, the production of type C and type D primate> retroviruses in persistently infected human cells. From experiments> demonstrating a disintegrating activity of chelating agents (EDTA, EGTA)> and certain psychoactive drugs (including trifluoperazine) on various> primate and nonprimate retroviruses it is concluded that divalent> cations, probably Ca2+ ions, and possibly also cation-binding proteins> are associated with retroviral membranes and that complexing of these> components results in loss of viral infectivity. Wunderlich Very, Uckert> W, Sydow G Arch Geschwulstforsch. 1983;53(3):267-78.> >>

[Guest guest]

so is that why you had to jump in and say something with no data? yes there is of course data showing autism kids with metals I am not saying there is not, but there are for SURE NO studies showing where they have tested a slew of kids that ALL had metals issues which is what would be required to prove your point, or a study to show that kids that were ALL chelated ALL recovered and healed. again which would prove your point,. there IS no such study which is exactly what I am arguing, there are also lots of studies showing people with metals who again have OTHER issues (again not disagreeing that metals cause issues, we all agree there, just that it doesn't make everyone autistic) fibro, thyroid issues, I even know people with Lou gerigs disease who have metal issues and improve with chelation. please if there is study showing ALL Kids tested with autism had metal issues post it! again MY middle has metal issues and is not delayed in ANY area......she had growth issuse from low carnintine as a child and some allergies and IGG issues...but not autistic.... I have metal issues and I am not on specturm either. again which breaks your therory!

Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > ââ,¬Å"Could it be that the chelators work as antiretrovirals by removing mercury? ââ,¬Å"> > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > > > Biochemical studies of primate retroviruses.> > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > >> >>

[Guest guest]

so is that why you had to jump in and say something with no data? yes there is of course data showing autism kids with metals I am not saying there is not, but there are for SURE NO studies showing where they have tested a slew of kids that ALL had metals issues which is what would be required to prove your point, or a study to show that kids that were ALL chelated ALL recovered and healed. again which would prove your point,. there IS no such study which is exactly what I am arguing, there are also lots of studies showing people with metals who again have OTHER issues (again not disagreeing that metals cause issues, we all agree there, just that it doesn't make everyone autistic) fibro, thyroid issues, I even know people with Lou gerigs disease who have metal issues and improve with chelation. please if there is study showing ALL Kids tested with autism had metal issues post it! again MY middle has metal issues and is not delayed in ANY area......she had growth issuse from low carnintine as a child and some allergies and IGG issues...but not autistic.... I have metal issues and I am not on specturm either. again which breaks your therory!

Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > ââ,¬Å"Could it be that the chelators work as antiretrovirals by removing mercury? ââ,¬Å"> > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > > > Biochemical studies of primate retroviruses.> > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > >> >>

[Guest guest]

well I don't need to prove anything, it's common knowledge that the BBB doesn't form till that age, meaning things can be moved at any time out. if the body can take cafe of itself to detox and those pathways aren't broken then it wouldn't be an issue. it's about fixing those pathways, Yasko has all kinds of great stuff on her site with lots of families that are fixing those pathways and doing regular urine testing to show them dumping stuff...I believe on her vidoe she even shows actualy kids testing and shows how it is dumping things

Re: ALA - direct inhibitor of> retroviral replication> >> >> > "Could it be that the chelators work as antiretrovirals by> removing mercury? "> >> > , that is not the case, that is not how chelators> work here. They work by removing ions from cation-binding viral> proteins, causing unfolding/loss of protein functionality. For example> by opening zinc-finger proteins and similar. There are other mechanisms> observed, such as inhibiting viral DNA synthesis through RNA reductase> deactivation, see attached> >> > btw some of these studies are in vitro, no mercury in sight,> so the answer is NO they do not work as antivirals through chelating> mercury, at least not exclusively (meaning to say that who knows maybe> heavy metals are also directly utilised by some viruses)> >> > And whatever is observed to happen in HIV only is only> because HIV is studied lot more than others. There are no studies that> show other viral proteins would not be unfolding in the same way when> chelators added. Having said that there are studies on herpes/CMV and> others that indicate same things going on.> >> >> >> > The influence of divalent cations on the stability of human> rotavirus.> > The influence of divalent cations on the stability of human> rotavirus was investigated using the indirect immunofluorescence (FA)> technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity> was stabilized by calcium and strontium ions in solution but not by> magnesium ions. Rotavirus isolates were found to be much less stable at> 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus> morphology at intervals during the course of the experiment and> treatment with the chelating agents EDTA and EGTA suggests that loss of> infectivity coincides with the removal of the outer capsid layer and> that calcium may be required to maintain virus integrity. Arch Virol.> 1981;67(1):1-9. Shirley JA, Beards GM, Thouless ME, Flewett TH> >> >> > Biochemical studies of primate retroviruses.> > In the present paper, recent biochemical studies of> retroviruses carried out in our laboratory are summarized. Protein> compositions, peptide maps of internal structural proteins,> neighborhoods of major structural proteins, and serological properties> of reverse transcriptases of type D virus isolates from human cells> (including Graffi's isolate termed PMFV and also isolates from HeLa- and> HEp-2 cells) were compared with those of type D viruses from Old World> (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World> (squirrel monkey retrovirus) monkeys. The results provide various new> informations on, and further demonstrate the diversity of primate type D> viruses. Other studies showed that tumor promoting agents (phorbol ester> TPA, indole alkaloid teleocidin) are able to considerably increase, in a> transient manner, the production of type C and type D primate> retroviruses in persistently infected human cells. From experiments> demonstrating a disintegrating activity of chelating agents (EDTA, EGTA)> and certain psychoactive drugs (including trifluoperazine) on various> primate and nonprimate retroviruses it is concluded that divalent> cations, probably Ca2+ ions, and possibly also cation-binding proteins> are associated with retroviral membranes and that complexing of these> components results in loss of viral infectivity. Wunderlich Very, Uckert> W, Sydow G Arch Geschwulstforsch. 1983;53(3):267-78.> >>

[Guest guest]

well I don't need to prove anything, it's common knowledge that the BBB doesn't form till that age, meaning things can be moved at any time out. if the body can take cafe of itself to detox and those pathways aren't broken then it wouldn't be an issue. it's about fixing those pathways, Yasko has all kinds of great stuff on her site with lots of families that are fixing those pathways and doing regular urine testing to show them dumping stuff...I believe on her vidoe she even shows actualy kids testing and shows how it is dumping things

Re: ALA - direct inhibitor of> retroviral replication> >> >> > "Could it be that the chelators work as antiretrovirals by> removing mercury? "> >> > , that is not the case, that is not how chelators> work here. They work by removing ions from cation-binding viral> proteins, causing unfolding/loss of protein functionality. For example> by opening zinc-finger proteins and similar. There are other mechanisms> observed, such as inhibiting viral DNA synthesis through RNA reductase> deactivation, see attached> >> > btw some of these studies are in vitro, no mercury in sight,> so the answer is NO they do not work as antivirals through chelating> mercury, at least not exclusively (meaning to say that who knows maybe> heavy metals are also directly utilised by some viruses)> >> > And whatever is observed to happen in HIV only is only> because HIV is studied lot more than others. There are no studies that> show other viral proteins would not be unfolding in the same way when> chelators added. Having said that there are studies on herpes/CMV and> others that indicate same things going on.> >> >> >> > The influence of divalent cations on the stability of human> rotavirus.> > The influence of divalent cations on the stability of human> rotavirus was investigated using the indirect immunofluorescence (FA)> technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity> was stabilized by calcium and strontium ions in solution but not by> magnesium ions. Rotavirus isolates were found to be much less stable at> 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus> morphology at intervals during the course of the experiment and> treatment with the chelating agents EDTA and EGTA suggests that loss of> infectivity coincides with the removal of the outer capsid layer and> that calcium may be required to maintain virus integrity. Arch Virol.> 1981;67(1):1-9. Shirley JA, Beards GM, Thouless ME, Flewett TH> >> >> > Biochemical studies of primate retroviruses.> > In the present paper, recent biochemical studies of> retroviruses carried out in our laboratory are summarized. Protein> compositions, peptide maps of internal structural proteins,> neighborhoods of major structural proteins, and serological properties> of reverse transcriptases of type D virus isolates from human cells> (including Graffi's isolate termed PMFV and also isolates from HeLa- and> HEp-2 cells) were compared with those of type D viruses from Old World> (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World> (squirrel monkey retrovirus) monkeys. The results provide various new> informations on, and further demonstrate the diversity of primate type D> viruses. Other studies showed that tumor promoting agents (phorbol ester> TPA, indole alkaloid teleocidin) are able to considerably increase, in a> transient manner, the production of type C and type D primate> retroviruses in persistently infected human cells. From experiments> demonstrating a disintegrating activity of chelating agents (EDTA, EGTA)> and certain psychoactive drugs (including trifluoperazine) on various> primate and nonprimate retroviruses it is concluded that divalent> cations, probably Ca2+ ions, and possibly also cation-binding proteins> are associated with retroviral membranes and that complexing of these> components results in loss of viral infectivity. Wunderlich Very, Uckert> W, Sydow G Arch Geschwulstforsch. 1983;53(3):267-78.> >>

[Guest guest]

It was meant to be funny. The discussion is getting old. You guys are just

going in circles. A lot of the same info is being repeated and it would seem

everyone just wants the last word.

> > > >

> > > >

> > > > From: Christel King <christelking1@>

> > > > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > > > To: mb12 valtrex

> > > > Date: Friday, November 20, 2009, 8:59 AM

> > > >

> > > >

> > > >

> > > >

> > > > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate! not ALL autistic kids have mercury

issues! my son didn't become autistic because he had metals, he became autistic

when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > >

> > > > Re: ALA - direct inhibitor of retroviral

replication

> > > >

> > > >

> > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > > >

> > > > , that is not the case, that is not how chelators work here.

They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > >

> > > > btw some of these studies are in vitro, no mercury in sight, so the

answer is NO they do not work as antivirals through chelating mercury, at least

not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > >

> > > > And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > >

> > > >

> > > >

> > > > The influence of divalent cations on the stability of human rotavirus.

> > > > The influence of divalent cations on the stability of human rotavirus

was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > >

> > > >

> > > > Biochemical studies of primate retroviruses.

> > > > In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > >

> > >

> >

>

[Guest guest]

It was meant to be funny. The discussion is getting old. You guys are just

going in circles. A lot of the same info is being repeated and it would seem

everyone just wants the last word.

> > > >

> > > >

> > > > From: Christel King <christelking1@>

> > > > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > > > To: mb12 valtrex

> > > > Date: Friday, November 20, 2009, 8:59 AM

> > > >

> > > >

> > > >

> > > >

> > > > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate! not ALL autistic kids have mercury

issues! my son didn't become autistic because he had metals, he became autistic

when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > >

> > > > Re: ALA - direct inhibitor of retroviral

replication

> > > >

> > > >

> > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > > >

> > > > , that is not the case, that is not how chelators work here.

They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > >

> > > > btw some of these studies are in vitro, no mercury in sight, so the

answer is NO they do not work as antivirals through chelating mercury, at least

not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > >

> > > > And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > >

> > > >

> > > >

> > > > The influence of divalent cations on the stability of human rotavirus.

> > > > The influence of divalent cations on the stability of human rotavirus

was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > >

> > > >

> > > > Biochemical studies of primate retroviruses.

> > > > In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > >

> > >

> >

>

[Guest guest]

Come on people, we are all entitled to have an opinion. We dont have to think a

like, thats what the group is for...Happy Thanksgiving!!!!

> > >

> > >

> > > From: Christel King <christelking1@>

> > > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > > To: mb12 valtrex

> > > Date: Friday, November 20, 2009, 8:59 AM

> > >

> > >

> > >

> > >

> > > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate! not ALL autistic kids have mercury

issues! my son didn't become autistic because he had metals, he became autistic

when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > >

> > > Re: ALA - direct inhibitor of retroviral

replication

> > >

> > >

> > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > >

> > > , that is not the case, that is not how chelators work here. They

work by removing ions from cation-binding viral proteins, causing unfolding/loss

of protein functionality. For example by opening zinc-finger proteins and

similar. There are other mechanisms observed, such as inhibiting viral DNA

synthesis through RNA reductase deactivation, see attached

> > >

> > > btw some of these studies are in vitro, no mercury in sight, so the

answer is NO they do not work as antivirals through chelating mercury, at least

not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > >

> > > And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > >

> > >

> > >

> > > The influence of divalent cations on the stability of human rotavirus.

> > > The influence of divalent cations on the stability of human rotavirus

was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > >

> > >

> > > Biochemical studies of primate retroviruses.

> > > In the present paper, recent biochemical studies of retroviruses carried

out in our laboratory are summarized. Protein compositions, peptide maps of

internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > >

> >

>

[Guest guest]

Come on people, we are all entitled to have an opinion. We dont have to think a

like, thats what the group is for...Happy Thanksgiving!!!!

> > >

> > >

> > > From: Christel King <christelking1@>

> > > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > > To: mb12 valtrex

> > > Date: Friday, November 20, 2009, 8:59 AM

> > >

> > >

> > >

> > >

> > > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate! not ALL autistic kids have mercury

issues! my son didn't become autistic because he had metals, he became autistic

when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > >

> > > Re: ALA - direct inhibitor of retroviral

replication

> > >

> > >

> > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > >

> > > , that is not the case, that is not how chelators work here. They

work by removing ions from cation-binding viral proteins, causing unfolding/loss

of protein functionality. For example by opening zinc-finger proteins and

similar. There are other mechanisms observed, such as inhibiting viral DNA

synthesis through RNA reductase deactivation, see attached

> > >

> > > btw some of these studies are in vitro, no mercury in sight, so the

answer is NO they do not work as antivirals through chelating mercury, at least

not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > >

> > > And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > >

> > >

> > >

> > > The influence of divalent cations on the stability of human rotavirus.

> > > The influence of divalent cations on the stability of human rotavirus

was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > >

> > >

> > > Biochemical studies of primate retroviruses.

> > > In the present paper, recent biochemical studies of retroviruses carried

out in our laboratory are summarized. Protein compositions, peptide maps of

internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > >

> >

>

[Guest guest]

Christel,

My daughter has the same story with immediate regression after MMR.

How did you recover your son?

Thanks,

> > > >

> > > >

> > > > From: Christel King <christelking1@>

> > > > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > > > To: mb12 valtrex

> > > > Date: Friday, November 20, 2009, 8:59 AM

> > > >

> > > >

> > > >

> > > >

> > > > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate! not ALL autistic kids have mercury

issues! my son didn't become autistic because he had metals, he became autistic

when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > >

> > > > Re: ALA - direct inhibitor of retroviral

replication

> > > >

> > > >

> > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > > >

> > > > , that is not the case, that is not how chelators work here.

They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > >

> > > > btw some of these studies are in vitro, no mercury in sight, so the

answer is NO they do not work as antivirals through chelating mercury, at least

not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > >

> > > > And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > >

> > > >

> > > >

> > > > The influence of divalent cations on the stability of human rotavirus.

> > > > The influence of divalent cations on the stability of human rotavirus

was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > >

> > > >

> > > > Biochemical studies of primate retroviruses.

> > > > In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > >

> > >

> >

>

[Guest guest]

Christel,

My daughter has the same story with immediate regression after MMR.

How did you recover your son?

Thanks,

> > > >

> > > >

> > > > From: Christel King <christelking1@>

> > > > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > > > To: mb12 valtrex

> > > > Date: Friday, November 20, 2009, 8:59 AM

> > > >

> > > >

> > > >

> > > >

> > > > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate! not ALL autistic kids have mercury

issues! my son didn't become autistic because he had metals, he became autistic

when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > >

> > > > Re: ALA - direct inhibitor of retroviral

replication

> > > >

> > > >

> > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > > >

> > > > , that is not the case, that is not how chelators work here.

They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > >

> > > > btw some of these studies are in vitro, no mercury in sight, so the

answer is NO they do not work as antivirals through chelating mercury, at least

not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > >

> > > > And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > >

> > > >

> > > >

> > > > The influence of divalent cations on the stability of human rotavirus.

> > > > The influence of divalent cations on the stability of human rotavirus

was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > >

> > > >

> > > > Biochemical studies of primate retroviruses.

> > > > In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > >

> > >

> >

>

[Guest guest]

OK I had never heard of anyone who still has metals,but has lost their autism diagnosis.

From: Christel King <christelking1@ myfairpoint. net>Subject: Re: Re: ALA - direct inhibitor of retroviral replicationTo: mb12 valtrex@ yahoogroups. comDate: Friday, November 20, 2009, 8:59 AM

then I guess we don't have mercury in the brain because our son isn't autistic anymore....and we didn't chelate! not ALL autistic kids have mercury issues! my son didn't become autistic because he had metals, he became autistic when he got an MMR....in HOURS after it!n we had tested 2 weeks prior developmental testing and he was 6 months advanced. after 7 hours after his MMR he became autistic, lost lang, no eye contact, started seizering, ect. we tested him 2 weeks after this because I had no idea what had happened to my son, he then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have become autistic when the thermitor exploded at 6 months old in our dishwasher and he didn't then!!!! it would also mean that my son would STILL be autistic since we haven't chelated with

your therory and he is not!

Re: ALA - direct inhibitor of retroviral replication

“Could it be that the chelators work as antiretrovirals by removing mercury? “, that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attachedbtw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would

not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. The influence of divalent cations on the stability of human rotavirus.The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA,

Beards GM, Thouless ME, Flewett THBiochemical studies of primate retroviruses.In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and

type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.