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Andy,

>You still are trapped in the fallacy that a complicated

>literature is scientific, and are so overwhelmed with the complicated

>literature that you haven't made observations or talked to people.

Andy, I have been conversing with, learning from, and listening to parents

of autistic children for seven years, since my daughter's problems sent me

to internet lists to find resources. A lot of our children who were

toddlers then are getting close to puberty now, and we've " grown up "

together, so I'm one of the old folks. I went back to graduate school to

learn how to help my daughter, so my prejudices as a parent flavored my

approach to what I learned in school, not vice versa!

Almost everything I've learned (except what I learned in classes) began

from listening to parents, but I found it necessary to go went back into

the literature to listen some more: to see if I could find explanations for

what all these observant people had noticed. My presumption has been that

I would have a better crack at understanding things if I learned as much as

possible from MANY other people. That includes people who publish their

work in peer-reviewed journals. My only beef with them is that sometimes

they narrow the focus of their work too quickly, and don't know enough

about other fields to perceive their own work's usefulness.

Anyway, I didn't know about the problem of " unapplied findings " until I

started discovering this myself, (as you have regarding amalgam/mercury

literature) but it did not take me long in school to become shocked at what

was not being included in graduate classes in biology and neuroscience. I

decided to learn more about this whole research process in a different sort

of course I took in graduate school. In this class, I read a lot of

scholarly work about the process, politics and funding of medical research

and found that some " inside auditing " has determined that the vast majority

of the articles in peer-reviewed journals has been ignored. The quantity

of this lost information is vast and in the same quagmire as the

observations of parents and patients when their observations don't fit the

prevailing model. All of this lost information is an awful waste of human

effort.

The scholars who have studied this phenomenon say that the only literature

that gets noticed is the work of star scientists who set the curriculum for

what people outside their fields will learn. They are the ones who write

the textbooks that everyone else reads. The rest of the work, the work of

the majority of the scientists, is considered unimportant and is left

behind, as if it had never happened. I think it's pretty certain that this

" dismissal " of other people's work probably comes from people who feel they

are at the " top " of their fields, and believe their training is complete

and that there is nothing else very important to learn. They believe they

have seen enough of the whole picture that nothing else could be

important, or else they would have embraced it themselves..

I've just been " brought up " differently, since I approached all this FIRST

as a parent. My own experience has persuaded me that there will always be

something new to learn. Surely things look more complicated on this

" upside " of the learning curve, but i expect it will become simpler as we

learn more about how all the parts relate.

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, you have made an important observation. Proven test information and

results that have been rechecked by the " experts " and duplicated many times are

then published to gather dust and if noted will be in the next text book

printing in 7 to who knows how many years as it takes about 7 years for

information to be published.

Bottom line, those working hands on KNOW more about what is happening than the

professionals unless the professionals have been talking and working with such

cases and not let the text book get in the way.

C. W.

Re: [ ] Re: sulfur

Andy,

>You still are trapped in the fallacy that a complicated

>literature is scientific, and are so overwhelmed with the complicated

>literature that you haven't made observations or talked to people.

Andy, I have been conversing with, learning from, and listening to parents

of autistic children for seven years, since my daughter's problems sent me

to internet lists to find resources. A lot of our children who were

toddlers then are getting close to puberty now, and we've " grown up "

together, so I'm one of the old folks. I went back to

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I am sooooooooo NOT following this thread, which is probably why I

attended law school and not medical school LOL. But I have a few

comments.

> LOL he is

hyper anyway,

> so... I'm not sure how much is from these enzymes. He does seem more

in the

> " off " days, but it could very well be from something else, like for

example

> metals... or anything else I can't think about yet :)

If your son remains hyper or has other negative effects on the OFF

days [besides just the first off day, see my comments below], then you

should consider a yeast issue. Children should regress slightly

during the round, and then the first off day is difficult, but the

other off days should be improvement. If there is not noticeable

improvement on the other off days, then the first thing you should

consider, especially if you are using ALA, is yeast. I use GSE for my

son's yeast issues. It is available without rx from my health food

store.

> The first day after chelation, I see some

weird

> looking strings in his stool, I mean his digestion is affected a

little. But

> he's fine after that.

The first day after round, you should see rebind effect. This is my

son's worst day, altho not too bad any more. When my son has a yeast

issue, his bms look like they have melted stringy cheese in them, plus

they are " fluffy " like sponge cake, rather than firm and solid.

Here is also a comment from the a-m FAQ page.

" Bernie Windham has written a paper that describes how mercury

interferes with the enzyme that is needed to digest gluten and casein.

Many people who are mercury toxic are sensitive to food that are high

in sulfur, which includes all dairy products. "

Dana

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> If your child shows low sulfur in the plasma cycteine lab test, does

> this mean you should add the sulfur supplements such as Molybelenen,

> NAG & NAC during chelation?

Molybdenum and NAG are not sulfur supplements.

If he has low plasma cysteine, lots of sulfury supplements and foods

should be given at all times.

All useful supplements should always be given at all times regardless

of chelation.

> He is usually not good during chelation cycles. And we have not seen

> many gains after 18 rounds.

What protocol are you using?

If it is ALA chelation on an appropriate protocol and he truly is low

sulfur he should be BETTER on cycle.

> We are thinking that we don't have his

> sulfur levels under control. Should we continue giving the three

> sulfur supplements listed above

Only one of the supplements you listed is sulfury.

> during chelation? And, should we

> eliminate the mineral supplement during chelation?

You should use sulfury foods and supplements all the time if his

cysteine is low, and should give all supplements every day regardless

of chelation.

Andy

cw

cw

c

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Andy,

Which foods and supplements are sulphery?

--- andrewhallcutler <AndyCutler@...> wrote:

> > If your child shows low sulfur in the plasma

> cycteine lab test, does

> > this mean you should add the sulfur supplements

> such as Molybelenen,

> > NAG & NAC during chelation?

>

> Molybdenum and NAG are not sulfur supplements.

>

> If he has low plasma cysteine, lots of sulfury

> supplements and foods

> should be given at all times.

>

> All useful supplements should always be given at all

> times regardless

> of chelation.

>

> > He is usually not good during chelation cycles.

> And we have not seen

> > many gains after 18 rounds.

>

> What protocol are you using?

>

> If it is ALA chelation on an appropriate protocol

> and he truly is low

> sulfur he should be BETTER on cycle.

>

> > We are thinking that we don't have his

> > sulfur levels under control. Should we continue

> giving the three

> > sulfur supplements listed above

>

> Only one of the supplements you listed is sulfury.

>

> > during chelation? And, should we

> > eliminate the mineral supplement during chelation?

>

> You should use sulfury foods and supplements all the

> time if his

> cysteine is low, and should give all supplements

> every day regardless

> of chelation.

>

> Andy

>

> cw

> cw

>

> c

>

>

>

=====

Suzanne M. Schwarz

__________________________________________________

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> Which foods and supplements are sulphery?

In this context I use the word " sulfur " loosely. What I am really

talking about is thiols and thiol precursors. Sulfate is a form of

sulfur that doesn't convert into these and has an entirely separate

role in the body.

Foods: Eggs, dairy (including whey, which is often used in unexpected

places, e. g. store cooked chickens), cabbage, cauliflower broccoli

broccoflower onions garlic leeks turnips rutabagas brussels sprouts

radishes spinach kale jicama and most beans, especially soybeans,

which means tofu. Other things related to these.

Supplements: extracts of the above foods, cysteine, NAC

(n-acetylcysteine), glutathione, MSM, DMSO, whey, colostrum. Taurine

may or may not be OK for a given individual.

And medications: penicillamine, mucomyst.

Andy

dq

d

ce

we

we

ewr

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Andy,

My son craves " Veggie Booty " , which are basically soy,

corn, kale and spinach. I mean he wants to eat the

entire bag of it even when he's hungry? Is there some

relationship here to what you crave and what is bad

for you(sulphur containing foods)?

--- andrewhallcutler <AndyCutler@...> wrote:

> > Which foods and supplements are sulphery?

>

> In this context I use the word " sulfur " loosely.

> What I am really

> talking about is thiols and thiol precursors.

> Sulfate is a form of

> sulfur that doesn't convert into these and has an

> entirely separate

> role in the body.

>

> Foods: Eggs, dairy (including whey, which is often

> used in unexpected

> places, e. g. store cooked chickens), cabbage,

> cauliflower broccoli

> broccoflower onions garlic leeks turnips rutabagas

> brussels sprouts

> radishes spinach kale jicama and most beans,

> especially soybeans,

> which means tofu. Other things related to these.

>

> Supplements: extracts of the above foods, cysteine,

> NAC

> (n-acetylcysteine), glutathione, MSM, DMSO, whey,

> colostrum. Taurine

> may or may not be OK for a given individual.

>

> And medications: penicillamine, mucomyst.

>

> Andy

>

> dq

> d

>

> ce

> we

> we

> ewr

>

>

=====

Suzanne M. Schwarz

__________________________________________________

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I've heard of others having problems with Veggie Bootie and that it may be cross

contaminated with gluten or casein. You may want to look into this, :)

Re: [ ] Re: Sulfur

Andy,

My son craves " Veggie Booty " , which are basically soy,

corn, kale and spinach. I mean he wants to eat the

entire bag of it even when he's hungry? Is there some

relationship here to what you crave and what is bad

for you(sulphur containing foods)?

--- andrewhallcutler <AndyCutler@...> wrote:

> > Which foods and supplements are sulphery?

>

> In this context I use the word " sulfur " loosely.

> What I am really

> talking about is thiols and thiol precursors.

> Sulfate is a form of

> sulfur that doesn't convert into these and has an

> entirely separate

> role in the body.

>

> Foods: Eggs, dairy (including whey, which is often

> used in unexpected

> places, e. g. store cooked chickens), cabbage,

> cauliflower broccoli

> broccoflower onions garlic leeks turnips rutabagas

> brussels sprouts

> radishes spinach kale jicama and most beans,

> especially soybeans,

> which means tofu. Other things related to these.

>

> Supplements: extracts of the above foods, cysteine,

> NAC

> (n-acetylcysteine), glutathione, MSM, DMSO, whey,

> colostrum. Taurine

> may or may not be OK for a given individual.

>

> And medications: penicillamine, mucomyst.

>

> Andy

>

> dq

> d

>

> ce

> we

> we

> ewr

>

>

=====

Suzanne M. Schwarz

__________________________________________________

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>Andy,

>My son craves " Veggie Booty " , which are basically soy,

>corn, kale and spinach. I mean he wants to eat the

>entire bag of it even when he's hungry? Is there some

>relationship here to what you crave and what is bad

>for you(sulphur containing foods)?

Dear Suzanne,

As you may note, I am not Andy---

Sulfur is NOT " bad for you " unless you happen to be

a " high sulfur " person. It varies. Your son may

crave sulfur foods because he is a " low sulfur " person.

It is very individual.

hope this is some help. you can read more here if you want:

/files/ANDY_INDEX

See section on sulfur and thiols.....

best wishes,

Moria

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What about epsom salts ? does that have anything to do with sulfur ? If my child

benefits from these, does that mean that he is " low sulfur " ?

Also, " sulfites " , is that something totally different ? I react to that stuff

big time, just curious.

Sharon

( the whole sulfur thing is also puzzling to me.......) :0(

[ ] Re: Sulfur

> Which foods and supplements are sulphery?

In this context I use the word " sulfur " loosely. What I am really

talking about is thiols and thiol precursors. Sulfate is a form of

sulfur that doesn't convert into these and has an entirely separate

role in the body.

Foods: Eggs, dairy (including whey, which is often used in unexpected

places, e. g. store cooked chickens), cabbage, cauliflower broccoli

broccoflower onions garlic leeks turnips rutabagas brussels sprouts

radishes spinach kale jicama and most beans, especially soybeans,

which means tofu. Other things related to these.

Supplements: extracts of the above foods, cysteine, NAC

(n-acetylcysteine), glutathione, MSM, DMSO, whey, colostrum. Taurine

may or may not be OK for a given individual.

And medications: penicillamine, mucomyst.

Andy

dq

d

ce

we

we

ewr

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> What about epsom salts ? does that have anything to do with sulfur ?

If my child benefits from these, does that mean that he is " low

sulfur " ?

> Also, " sulfites " , is that something totally different ? I react to

that stuff big time, just curious.

> Sharon

> ( the whole sulfur thing is also puzzling to me.......) :0(

To repeat:

> In this context I use the word " sulfur " loosely. What I am really

> talking about is thiols and thiol precursors. Sulfate is a form

of

> sulfur that doesn't convert into these and has an entirely

separate

> role in the body.

Thiol and other reduced type sulfur -----> sulfite which is REALLY

toxic ------> sulfate which is good for you.

These reactions are irreversible. They only go one way.

There are many types of more reduced sulfur, and most of them do seem

to interconvert (taurine seems to be an exception but this isn't

totally clear).

Sulfite is converted to sulfate rapidly by an enzyme requiring

molydbdenum. This often gets messed up by mercury, making you sulfite

sensitive (or more so) and also making you get problems from your own

production of sulfite.

Many people with " sulfur problems " in having too MUCH of the thiol

type actually don't have enough sulfate. So sulfate being good for

you doesn't mean anything about the thiol type sulfur problem.

Andy

c

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> What about epsom salts ? does that have anything to do with sulfur ? If my

child benefits from these, does that mean that he is " low sulfur " ?

Hi Sharon,

Epsom salts are magnesium sulfate (you probably know that already).

This " sulfur " takes in the body all kinds of forms. Some of these forms can be

used and " recycled " and transformed into something else (specialized

enzymes are attaching other molecules to them) and used again in the new form,

and other forms cannot be transformed anymore after they are used.

The body either stores them to use them later or excretes them.

Sulfate is one of the forms of sulfur (SO4) that cannot be transformed into

something else anymore. It is made up of sulfur, but the body cannot

transform sulfate back into cysteine for example (cysteine is another form of

sulfur that sulfate is made from).

Sulfate is used in the body to detoxify some toxins (some drugs, phenols,

alcohols, steroid hormones) and for many other needs. So it is very

important. But if your body cannot produce enough sulfate (it happens many times

in mercury poisoned people) your liver and brain will suffer,

because those toxins cannot be taken out, so they poison you. So it is important

to support your liver with additional sulfate (in the form of Epsom

salt).

The reason why sulfate is not important when you talk about " sulfur level " is

exactly this: the fact that your body cannot use sulfate for other

" needs " other than for sulfation. So it's not relevant for the rest of the

sulfur chemistry that takes place in the body.

It's my understanding that cysteine is the most important form of sulfur. This

is the chemical that is used in the body to produce most of the other

forms of sulfur molecules (like glutathion for example, which is important in

detoxification of heavy metals. Glutathion is made from cysteine).

This is why Andy recommends the *plasma cysteine* test. Because it tells you the

level of this form of sulfur that *can* be usually transformed in

the body in the most of the other forms of sulfur. If it's low, you will need to

add more in the form of NAC (n-acetyl cysteine) or MSM (some people

react bad to this); if it's high you will need to reduce your intake of " sulfury

foods " , because cysteine is toxic when it's high and will make you

feel bad. Chelation (especially ALA) usually will increase your cysteine level,

so that's why it's useful to know where you stand

Hope this helps...

Valentina

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At 07:48 PM 4/2/2002 -0500, you wrote:

>What about epsom salts ? does that have anything to do with sulfur ? If my

child benefits from these, does that mean that he is " low sulfur " ?

>Also, " sulfites " , is that something totally different ? I react to that

stuff big time, just curious.

>Sharon

>( the whole sulfur thing is also puzzling to me.......) :0(

I'm with you in the " puzzled " camp. I do understand that the SORT of

stuff one gets from epsom salts (possibly sulfite) is NOT THE SAME and

not related to the stuff one gets from " sulfury " foods and supplements

(NAC, glutathione, cabbage, cauliflower, garlic......etc). This stuff

could (maybe LOL) be sulfate. But don't quote me on it LOL. I am

clear that the 2 are not related though--- that is, if you are high

sulfur (high plasma cysteine) you may still get a lot of benefit

from epsom salts.

best wishes,

Moria

p.s. just in case you don't know, you can also read about both sorts

of sulfur here:

/files/ANDY_INDEX

see section on sulfur and thiols. I'm not sure where epsom salts

are (which section). I am NOT trying to be " a broken record " about

this file ;) -- rather I hope to tell you this IN CASE you are interested.

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Thanks, I guess this answers my question, just because he benefits from epson

salts, doesn't mean he's low sulfur.

I guess I will run that plasma cysteine after all.

Sharon

Re: [ ] Re: Sulfur

At 07:48 PM 4/2/2002 -0500, you wrote:

>What about epsom salts ? does that have anything to do with sulfur ? If my

child benefits from these, does that mean that he is " low sulfur " ?

>Also, " sulfites " , is that something totally different ? I react to that

stuff big time, just curious.

>Sharon

>( the whole sulfur thing is also puzzling to me.......) :0(

I'm with you in the " puzzled " camp. I do understand that the SORT of

stuff one gets from epsom salts (possibly sulfite) is NOT THE SAME and

not related to the stuff one gets from " sulfury " foods and supplements

(NAC, glutathione, cabbage, cauliflower, garlic......etc). This stuff

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ok..........thanks................Now I know why I flunked chemistry !!!!!!!!!!!

I suspect I'm mercury toxic too, since I just had 10 fillings replaced, some of

them were 30 yeasr old, some were cracked.

oh well, I guess I will look into that as well. Makes me wonder if my son was

already born mercury toxic, and that's why he's been so hard to chelate.

Sharon

[ ] Re: Sulfur

> What about epsom salts ? does that have anything to do with sulfur ?

If my child benefits from these, does that mean that he is " low

sulfur " ?

> Also, " sulfites " , is that something totally different ? I react to

that stuff big time, just curious.

> Sharon

> ( the whole sulfur thing is also puzzling to me.......) :0(

To repeat:

> In this context I use the word " sulfur " loosely. What I am really

> talking about is thiols and thiol precursors. Sulfate is a form

of

> sulfur that doesn't convert into these and has an entirely

separate

> role in the body.

Thiol and other reduced type sulfur -----> sulfite which is REALLY

toxic ------> sulfate which is good for you.

These reactions are irreversible. They only go one way.

There are many types of more reduced sulfur, and most of them do seem

to interconvert (taurine seems to be an exception but this isn't

totally clear).

Sulfite is converted to sulfate rapidly by an enzyme requiring

molydbdenum. This often gets messed up by mercury, making you sulfite

sensitive (or more so) and also making you get problems from your own

production of sulfite.

Many people with " sulfur problems " in having too MUCH of the thiol

type actually don't have enough sulfate. So sulfate being good for

you doesn't mean anything about the thiol type sulfur problem.

Andy

c

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In a message dated 5/21/03 5:23:00 PM Eastern Daylight Time,

writes:

> It is possible that some of the sulfate in epsom salts might be turned into

>

> other forms of sulfur, but if that happens, it probably involves some help

> from our gut flora.

>

>

>

Thanks for posting that. Really interesting.

Nell

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Sulfate is not converted to other forms of sulfur under

physiologically relevant conditions.

Andy .. . . . . .

> > It is possible that some of the sulfate in epsom salts might be

turned into

> >

> > other forms of sulfur, but if that happens, it probably involves

some help

> > from our gut flora.

> >

> >

> >

>

> Thanks for posting that. Really interesting.

>

> Nell

>

>

> [Non-text portions of this message have been remov

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Andy,

At 02:12 AM 5/22/2003 +0000, you wrote:

>Sulfate is not converted to other forms of sulfur under

>physiologically relevant conditions.

I think you have overlooked something important. There are lots of critters

in our gut that convert sulfate to other things. We just don't know with

much precision how much that contributes to our overall sulfur budget, or

from what source primarily they are getting the sulfate these critters

eat. Shedding of sulfated molecules from the intestinal wall is a very

normal process, so the sulfate that reaches the colon doesn't need to come

from diet. It seems that a lot of the sulfate the liver produces probably

comes from this alternative bacteria-requiring pathway, as that is the

pathway for making sulfate that is most likely to fail in liver disease.

I have just put some quick examples below. The second article makes it

sound that in their case, an oral dose of sulfate was probably NOT the

sulfate that ended up processed by intestinal bacteria in the mice in the

study. The last abstract shows that the products of these sulfate-eating

bacteria is converted back to sulfate through the intermediate compound

thiosulfate through the cooperation of colonic cells and the liver. When

you are talking biochemistry, you have to account for all potential

contributors to the system...even the microbes! Biological systems are

trickier than other chemistries. The chemistry of humans actually includes

lots of other critters that share our internal ecology and contribute to it

and have genes that are very different from ours. Scientists have shown

that animals raised in microbe free environments are not healthy at all.

Dr. Waring in Birmingham England found that pooled together, 232 children

with autism had seven times the thiosulfate in their urine as controls, and

it is in the context of the colon that you hear about thiosulfate. For

that reason, Dr. Waring's data may really be pointing to an importance of

this microbe-related issue/pathway to children with autism. That shouldn't

be a surprise in consideration of how many children with autism have

dysbiosis. We still have much to learn about this.

FEMS Microbiol Ecol 2000 Dec 1;34(2):147-155 Related Articles, Links

Screening of sulfate-reducing bacteria in colonoscopy samples from

healthy and colitic human gut mucosa.

Zinkevich V V, Beech IB.

University of Portsmouth, School of Pharmacy and Biomedical Sciences,

St 's Building, White Swan Road, PO1 2DT, Portsmouth, UK

A PCR-based approach combined with microbiological cultivation methods

was employed to determine the occurrence of sulfate-reducing bacteria (SRB)

in colon biopsy samples from ulcerative colitis patients and from

non-colitic controls. The detection of mucosa-associated SRB was carried

out by digoxigenin-dUTP-labelled PCR amplification, in liquid Postgate

medium B and in a new liquid medium, termed VM medium I. Using Postgate

medium B, the growth of SRB was confirmed in 92% of the colitic specimens

and in 52% of non-colitic samples. However, PCR analysis and incubation in

VM medium I detected SRB in 100% of biopsy material indicating ubiquitous

presence of SRB in human colon mucosa.

PMID: 11102692 [PubMed - as supplied by publisher]

Exp Biol Med (Maywood) 2003 Apr;228(4):424-33 Related Articles, Links

[Click here to read]

Gastrointestinal and microbial responses to sulfate-supplemented

drinking water in mice.

Deplancke B, Finster K, Graham WV, Collier CT, Thurmond JE, Gaskins HR.

Division of Nutritional Sciences, and Department of Animal Sciences,

University of Illinois, Urbana, Illinois 61801, USA.

There is increasing evidence that hydrogen sulfide (H2S), produced by

intestinal sulfate-reducing bacteria (SRB), may be involved in the

etiopathogenesis of chronic diseases such as ulcerative colitis and

colorectal cancer. The activity of SRB, and thus H2S production, is likely

determined by the availability of sulfur-containing compounds in the

intestine. However, little is known about the impact of dietary or

inorganic sulfate on intestinal sulfate and SRB-derived H2S concentrations.

In this study, the effects of short-term (7 day) and long-term (1 year)

inorganic sulfate supplementation of the drinking water on gastrointestinal

(GI) sulfate and H2S concentrations (and thus activity of resident SRBs),

and the density of large intestinal sulfomucin-containing goblet cells,

were examined in C3H/HeJBir mice. Additionally, a PCR-denaturing gradient

gel electrophoresis (DGGE)-based molecular ecology technique was used to

examine the impact of sulfate-amended drinking water on microbial community

structure throughout the GI tract. Average H2S concentrations ranged from

0.1 mM (stomach) to 1 mM (cecum). A sulfate reduction assay demonstrated in

situ production of H2S throughout the GI tract, confirming the presence of

SRB. However, H2S generation and concentrations were greatest in the cecum

and colon. Sulfate supplementation of drinking water did not significantly

increase intestinal sulfate or H2S concentrations, suggesting that

inorganic sulfate is not an important modulator of intestinal H2S

concentrations, although it altered the bacterial profiles of the stomach

and distal colon of 1-year-old mice. This change in colonic bacterial

profiles may reflect a corresponding increase in the density of

sulfomucin-containing goblet cells in sulfate-supplemented compared with

control mice.

PMID: 12671187 [PubMed - indexed for MEDLINE]

Curr Microbiol 1997 Nov;35(5):294-8 Related Articles, Links

[Click here to read]

Nutritional aspects of dissimilatory sulfate reduction in the human

large intestine.

Willis CL, Cummings JH, Neale G, Gibson GR.

Institute of Food Research, Earley Gate, Reading, UK.

In contrast to other anaerobic ecosystems, such as marine and

estuarine sediments, there is a lack of information on the nutritional

requirements of human gut sulfate-reducing bacteria (SRB). Various

substrates stimulated sulfate reduction in mixed culture, including

short-chain fatty acids and other organic acids, alcohols, and amino acids

(but not sugars or aromatic compounds). However, the use of sodium

molybdate as a specific inhibitor of sulfate reduction caused an

accumulation of ethanol and malonate only, and reduced the rate of

utilization of lactate. This indicates the importance of these electron

donors for sulfate reduction. Since ethanol and lactate are primarily

utilized by members of the Desulfovibrio genus, the results suggest a

physiologically important role for this group. Experiments with two strains

of Desulfovibrio desulfuricans isolated from human feces demonstrated that

both were able to reduce sulfite, thiosulfate or nitrate in the absence of

sulfate. In addition, one strain (DsvUC1) was able to grow by fermentative

metabolism, although the second strain (DsvFD1) showed more restricted

fermentative growth. The data indicate that desulfovibrios are ecologically

the most significant group of SRB in the human colon, and that colonic

isolates belonging to this genus are versatile, in terms of both the

electron acceptors and donors that they are able to utilize.

PMID: 9462959 [PubMed - indexed for MEDLINE]

Biochem Pharmacol 2001 Jul 15;62(2):255-9 Related Articles, Links

[Click here to read]

Oxidation of hydrogen sulfide and methanethiol to thiosulfate by rat

tissues: a specialized function of the colonic mucosa.

Furne J, Springfield J, Koenig T, DeMaster E, Levitt MD.

Research Service, Minneapolis VA Medical Center, 1 Veterans Drive,

Minneapolis, MN 55417, USA.

Colonic bacteria release large quantities of the highly toxic thiols

hydrogen sulfide (H(2)S) and methanethiol (CH(3)SH). These gases rapidly

permeate the colonic mucosa, and tissue damage would be expected if the

mucosa could not detoxify these compounds rapidly. We previously showed

that rat cecal mucosa metabolizes these thiols via conversion to

thiosulfate. The purpose of the present study in rats was to determine if

this conversion of thiols to thiosulfate is (a) a generalized function of

many tissues, or (B) a specialized function of the colonic mucosa. The

tissues studied were mucosa from the cecum, right colon, mid-colon, ileum,

and stomach; liver; muscle; erythrocytes; and plasma. The metabolic rate

was determined by incubating homogenates of the various tissues with

H(2)(35)S and CH(3)(35)SH and measuring the rate of incorporation of (35)S

into thiosulfate and sulfate. The detoxification activity of H(2)S

(expressed as nmol/mg per min) that resulted in thiosulfate production was

at least eight times greater for cecal and right colonic mucosa than for

the non-colonic tissues. Thiosulfate production from CH(3)SH was at least

five times more rapid for cecal and right colonic mucosa than for the

non-colonic tissues. We conclude that colonic mucosa possesses a

specialized detoxification system that allows this tissue to rapidly

metabolize H(2)S and CH(3)SH to thiosulfate. Presumably, this highly

developed system protects the colon from what otherwise might be injurious

concentrations of H(2)S and CH(3)SH. Defects in this detoxification pathway

possibly could play a role in the pathogenesis of various forms of colitis.

PMID: 11389886 [PubMed - indexed for MEDLINE]

>Andy .. . . . . .

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After reading Andy's info about high sulfur and high cysteine and

sensitivity to sulfury foods I checked his metametrix results which

showed plasma homocysteine to be about average or a little above

(just at the 3rd-4th quintile juncture). Value 4 in a ref range of

<8. Just wanted to add that to my post

Thanks

Karin

>

> still pouring over my son's hair results. he was high in sulfur.

> dont know if this is meaningful, but i know there is a lot of

> discussion about tolerance of sulfury foods. Since my son cant

> tolerate a darn thing I was wondering if this might be something to

> look into. Could this high sulfur value mean that I should limit

the

> sulfur in his diet? Thanks

>

> Karin

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>

> still pouring over my son's hair results. he was high in sulfur.

> dont know if this is meaningful,

It is completely and totally irrelevant.

> but i know there is a lot of

> discussion about tolerance of sulfury foods. Since my son cant

> tolerate a darn thing I was wondering if this might be something to

> look into. Could this high sulfur value mean that I should limit the

> sulfur in his diet? Thanks

>

> Karin

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yep, someone kindly sent me that information from your book after I

posted it. Love rendering information irrelevant!

Karin

> >

> > still pouring over my son's hair results. he was high in

sulfur.

> > dont know if this is meaningful,

>

> It is completely and totally irrelevant.

>

> > but i know there is a lot of

> > discussion about tolerance of sulfury foods. Since my son cant

> > tolerate a darn thing I was wondering if this might be something

to

> > look into. Could this high sulfur value mean that I should limit

the

> > sulfur in his diet? Thanks

> >

> > Karin

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my daughter's breath or diaper often smells like sulfur, does this mean

anything?

[ ] Digest Number 7701

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>

>

> my daughter's breath or diaper often smells like sulfur, does this

mean anything?

Does she eat a lot of high sulfur foods?

Dana

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SULFUR his mineral helps skin and hair. Fights bacterial infection. Aids liver function. Disinfects blood. Protects against toxic substances. HERBAL SOURCES: Horsetail. Suzi What is a weed? A plant whose virtues have not yet been discovered. health/ http://suziesgoats.wholefoodfarmacy.com/ http://360./suziesgoats __________________________________________________

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Not Dana,

Epsom salts and No Fenol enzymes..and if you are on MB 12, that helps

here

Bridget

>

> Dana,

>

> Is there an enzyme that helps with the digestion of sulfur foods?

>

> Carol

>

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