autism

[Guest guest]

I live in Ohio and grew up in " Amish country " . I don't know about

other Amish communities, but it was my observation that the Amish in my

area did not eat well. I was surprised to see what they had in their

carts at the grocery store. I would see processed junk food, Coke and

formula, just to name a few. ~

" Not that vaccines are the only cause of autism but the Amish also eat

their own fresh nonprocessed foods and have little or no exposure to

the toxins that we as a society are exposed to. "

le

[Guest guest]

How scary. I took antibiotics w/ both of my pregnancies for UTI's. I'm so

glad my mom convinced me never to take tylenol while preggo, my docs always

pushed it though.

Cat

In a message dated 1/10/2008 8:40:02 P.M. Eastern Standard Time,

vaccineinfo@... writes:

if unvaccinated often history of:

1. antibiotics to mom during pregnancy or while breastfeeding

2. antibiotics to infant

3. use of tylenol

4. amalgams in mom's mouth

5. mom vaccinated with rubella vaccine at some point before conception

6. rhogam shot given while pregnant (mercury in that)

7. strong emotional event for mom during pregnancy

**************Start the year off right. Easy ways to stay in shape.

http://body.aol.com/fitness/winter-exercise?NCID=aolcmp00300000002489

[Guest guest]

Hi All,

I have been seeing the posts on autism here and felt I needed to show

you a very interesting link. , one of " The Secret "

teachers has a device he calls the QED that is said to greatly

improve/heal austistic people and also Downs Syndrome. Each Thursday

he gets on to www.talkshoe.com (go to this link and type " EMC2 " in

the search box. You will get something like " EMC2 AIM Program of

Energetic Balancing " with Mike Connor) and gives an update on his

device anf the program.

I just sent my paperwork in to get on this device. I have been a

patient of the QXCI machine for about 4+ years now...and while it has

helped me...I wanted to take things a step further (while using the

QXCI machine to verify the results of the QED). Wayne Dyer,

, Dr. Rev. Bechwith, and Gray (from the TV show

Dallas) do this program and swear by it. I believe both the QXCI and

the QED can be used harmoniously.

and his colleagues say that for any illness to

manifest, it must first be in your consciousness and so the only way

to actually clear an illness is to clear it from your consciousness.

His device is a quantum physics device and he makes use or holograms

(your full head to toe picture). Previously being on his program was

only for the Hollywood elite who could pay $30,000 for 1 year on the

program. It is now $1000 for an individual for 1 year, or $2,000

for up to 5 family members (on the family plan). About 3 years ago

he made the hollographic breakthrough I am mentioning and that is why

the cost has dropped considerably.

He states that he has found that autism has " the frequency " of

heriditary syphillis and that this is the case also for many people

who have food allergies. People who have autism or downs syndrome

can get on his machine for free and I have heard many people so far

say I don't know what he's doing but they see their autistic child or

their downs syndrome child improve drastically on this QED.

I was wondering if any of you had discovered the frequency of

Syphillis in autistic children or among those with severe food

allergies with the QXCI machine. Anyone care to comment????

Anyways, if you are interested in checking this out with an open ear

then go to www.energeticmatrix.com and also www.talkshoe.com (for the

radio broadcast).

I'll let you know how this goes for me on the program while still on

the QXCI.

Janet

> Hi Everyone-

> I just got back from my beg. training and am wondering if

> someone can refer me to someone who works with the SCIO on autistic

> children? Also someone who works with severe depression? Where

might I

> have access to these types of protocols? -- Misty*

>

[Guest guest]

Hi Tina,Naltrexone is an FDA-approved medication for opium and heroin addiction. In very low doses, it is showing great promise in the treatment of autism. Dr. Jaquelyn McCandless is a neurologist and child psychiatrist who has researched this medication in the treatment of autism and has written a book about her experience which is described here: http://tinyurl.com/children-with-starving-brainsFor additional information about Low Dose Naltrexone, visithttp://tinyurl.com/intro-to-ldnWith best wishes, Dudley Delanydudley_delany

[Guest guest]

I have the book,by Dr.McAndless.I havn`t tried low dose

naltrexone,maybe I will,after I give some of the Cayce remedies a

try.Thanks for your response.Tina

>

> Hi Tina,

>

> Naltrexone is an FDA-approved medication for opium and heroin

addiction. In very low doses, it is showing great promise in the

treatment of autism

>

> Dr. Jaquelyn McCandless is a neurologist and child psychiatrist who

has researched this medication in the treatment of autism and has

written a book about her experience which is described here:

>

> http://tinyurl.com/children-with-starving-brains

>

> For additional information about Low Dose Naltrexone, visit

>

> http://tinyurl.com/intro-to-ldn

>

> With best wishes,

>

> Dudley Delany

>

> dudley_delany

>

[Guest guest]

Hi ,

See all this work here:- LINK

This isn't off-topic, it's all precisely on-topic

Sheila suggests getting vitamin D checked and making sure that this isn't a factor in low thyroid function.

Steroid/Hormone receptors interact with each other to form either homodimers or heterodimers. Two of the same kind or one of each type of receptor activated by its particular steroid/hormone.

These dimers then translate to the nucleus where they bind at the appropriate response element on the DNA and produce mRNA prior to output of the protein required for any of the usual jobs carried out by proteins (eg enzyme function).

Along the way, proteins have to be properly folded to make the correct shape for the function required of the protein.

When the energy available is insufficient to fold the protein, it languishes in the cell compartment ~ stuck, because it isn't folded properly, and then gets chopped up by the proteosome(s).

This total waste of effort causes further depletion of what little energy is available in a poorly or un ~ treated hypothyroid patient.

By the time the patient has tried to manufacture more protein to replace that lost by futile cycles of building and destruction, you can see that the entire medical profession hasn't got a clue about the intricacies of the process.

That's why they prefer to call it (failure to get well on demand) Functional Somatoform Disorder, or as we may properly describe that terminonology ~ an excuse cobbled together by ..............( OK, I censored it), too idle to find out what's going on.

CFS/ME/FMS call it what you like, the failure to recognise what is going on, leads them to label the patient as (derisorily) depressed/overweight/eating too much ~ actually it's failure to diagnose with a posh name......

so, there you are, times up, you've had your eight minutes....

Vitaimn D can help compensate for low thyroid hormone output, by using the heterodimerisation process, but can't substitute for extremes of deficiency in either case.

best wishes

Bob

>> Hi Bob,> > Sorry if we are off subject here but this is jaw dropping stuff. Does this potentially mean that the huge increase in autism in kids is , or could be due to their mothers being vitamin d deficient during pregnancy ?!? This is just awful......> > julia>

[Guest guest]

Sounds as if I should get a vitamin D supplement straight away (almost 13 weeks pregnant....). How much should I be taking Bob? Maybe I should get my vitamin D level checked at my booking appointment tomorrow (if they will!).

[Guest guest]

Very interesting stuff, Bob - thank you

One question ..... bearing in mind that natural sun light will make our bodies produce Vit. D (and presumably in sufficient numbers ??) - ignoring the dehydrating skin problem for a moment - would 10 minutes on an artificial sunbed once a week have the same effect ? Would that be a way to boost our Vit D during those long miserable winter months without popping the pills?

[Guest guest]

that is very intresting because my 3 year old has has autism and was only taking folic acid during my pregnancy

cheryl

thyroid treatment From: christina@...Date: Tue, 31 Mar 2009 09:41:01 +0000Subject: Re: Autism

Very interesting stuff, Bob - thank you

One question ..... bearing in mind that natural sun light will make our bodies produce Vit. D (and presumably in sufficient numbers ??) - ignoring the dehydrating skin problem for a moment - would 10 minutes on an artificial sunbed once a week have the same effect ? Would that be a way to boost our Vit D during those long miserable winter months without popping the pills?

Windows Live just got better. Find out more!

[Guest guest]

Hi ,

Sunbed may be faster, 10minutes per day perhaps, as an investment in your

immediate case

one, two or three 1000IU tabs/caps/gels (Solgar do them ~ £6.75/100) per day

integratd with your calcium intake, but be careful that you don't get cramp (may

need magnesium too).

best wishes

Bob

>

> Sounds as if I should get a vitamin D supplement straight away (almost 13

weeks pregnant....). How much should I be taking Bob? Maybe I should get my

vitamin D level checked at my booking appointment tomorrow (if they will!).

>

>

[Guest guest]

Careful! Ten minutes a day will cause damage and you definitely do not want to

start at 10 minutes. I don't know what a safe level is but I think it might be

as low as 3 minutes. Remember the news photo of the burned skin of that teenage

girl?

Tracey

>

>

> Hi ,

>

> Sunbed may be faster, 10minutes per day perhaps, as an investment in your

immediate case

>

> one, two or three 1000IU tabs/caps/gels (Solgar do them ~ £6.75/100) per day

integratd with your calcium intake, but be careful that you don't get cramp (may

need magnesium too).

>

> best wishes

> Bob

[Guest guest]

Hi Tracey,

Pre-treating the skin to be exposed (with lanolin) will both reduce the

intensity of the UV-B and add to the production of vit D3 (from lanolin).

At about £1/minute judge the cost/benefit.

The 1000IU cholecalciferol (vit D3) will shield areas of the skin as well, if

applied in an oil base (soft gel caps).

They can be used both internally and externally.

best wishes

Bob

>

> Careful! Ten minutes a day will cause damage and you definitely do not want to

start at 10 minutes. I don't know what a safe level is but I think it might be

as low as 3 minutes. Remember the news photo of the burned skin of that teenage

girl?

>

> Tracey

>

[Guest guest]

Its too late for me.. little one is 6mo, and the autistic one is 5 y.o...

thanks though Bob - hopefully we can nip this in the bud for future children

(not me specifically, we're done having kids...)

incidentally, my mom, who has been diagnosed with follicular thyroid cancer last

summer, also had a vitamin D level in the basement and has had injections and

pills to remedy it.

Hers is doubtless linked to her avoiding the sun for melanoma reasons (she had

melanoma aged 47, she is 58 in Sept this year, doesnt want it to reoccur!!) but

i wonder if all this sun block madness is related?

Loads of people i know slather that stuff on their children and themselves if

its just a sunny day... heaven forbid. I only use it on my two if we are going

out in the baking sun for more than an hour, but i wonder if the lack of sun

exposure for fear of skin cancer is related to the epidemic of vit-D

deficiencies and MS and etc?

At home the milk is fortified with vit A and D (the full fat is anyway) but my

mom drinks very little milk, and only 2% anyway, and eats little real dairy

products, only the fakey ones like I Can't Believe Its Not Butter, etc.

Jen

>

> Sounds as if I should get a vitamin D supplement straight away (almost 13

weeks pregnant....). How much should I be taking Bob? Maybe I should get my

vitamin D level checked at my booking appointment tomorrow (if they will!).

>

>

[Guest guest]

Hi Bob,

I recall that when I was at University, long, long ago, my biochemistry lecturer

said that skin ahd to be not too clean (like 24 hours since washed) to make Vit

D in sunlight. It was over 30 years ago, so I do not have a ref to hand, do you

know if this is still thought to be the case?

regards,

Kat

> Sunbed may be faster, 10minutes per day perhaps, as an investment in your

immediate case

[Guest guest]

HI,

Way too late for me and my 18 year old son who has Aspergers - a form of

autism. I kept asking if it was safe for me to get pregnant, and was told yes,

but as soon as my son was born I knew something was amiss.

Born with less than 50% hearing and several other problems too, he was allergic

to all dairy products, and I also have a low tolerance to dairy products. I

always said it was because of my hypothyroidism, this just makes me even more

sure. Lets hope they warn people that this could indeed be a problem to unborn

children of hypothyroid mothers.

Lynne

>

> Sorry if we are off subject here but this is jaw dropping stuff. Does this

potentially mean that the huge increase in autism in kids is , or could be due

to their mothers being vitamin d deficient during pregnancy ?!?  This is just

awful......

>

> julia

>

[Guest guest]

Do you mean that I should be taking Calcium too?

Thanks

one, two or three 1000IU tabs/caps/gels (Solgar do them ~ £6.75/100) per day integratd with your calcium intake

[Guest guest]

An example of a useful guideline:-

http://www.ncbi.nlm.nih.gov/pubmed/18053387

Pre-conceptional vitamin/folic acid supplementation 2007: the use of folic acid in combination with a multivitamin supplement for the prevention of neural tube defects and other congenital anomalies.

RD, JA, Wyatt P, V, Gagnon A, Langlois S, Blight C, Audibert F, Désilets V, Brock JA, Koren G, Goh YI, Nguyen P, Kapur B; Genetics Committee of the Society of Obstetricians and Gynaecologists of Canada and The Motherrisk Program

Philadelphia PA, USA.

OBJECTIVE: To provide information regarding the use of folic acid in combination with a multivitamin supplement for the prevention of neural tube defects and other congenital anomalies, so that physicians, midwives, nurses, and other health care workers can assist in the education of women in the pre-conception phase of their health care.

OPTION: Supplementation with folic acid and vitamins is problematic, since 50% of pregnancies are unplanned, and women's health status may not be optimal when they conceive.

OUTCOMES: Folic acid in combination with a multivitamin supplement has been associated with a decrease in specific birth defects.

EVIDENCE: Medline, PubMed, and Cochrane Database were searched for relevant English language articles published between 1985 and 2007. The previous Society of Obstetricians and Gynaecologists of Canada (SOGC) Policy Statement of November 1993 and statements from the American College of Obstetrics and Gynecology and Canadian College of Medical Geneticists were also reviewed in developing this clinical practice guideline.

VALUES: The quality of evidence was rated using the criteria described in the Report of the Canadian Task Force on Preventive Health Care.

BENEFITS, HARMS, AND COSTS: Promoting the use of folic acid and a multivitamin supplement among women of reproductive age will reduce the incidence of birth defects. The costs are those of daily vitamin supplementation and eating a healthy diet. RECOMMENDATIONS: 1. Women in the reproductive age group should be advised about the benefits of folic acid in addition to a multivitamin supplement during wellness visits (birth control renewal, Pap testing, yearly examination) especially if pregnancy is contemplated. (III-A) 2. Women should be advised to maintain a healthy diet, as recommended in Eating Well With Canada's Food Guide (Health Canada). Foods containing excellent to good sources of folic acid are fortified grains, spinach, lentils, chick peas, asparagus, broccoli, peas, Brussels sprouts, corn, and oranges. However, it is unlikely that diet alone can provide levels similar to folate-multivitamin supplementation. (III-A) 3. Women taking a multivitamin containing folic acid should be advised not to take more than one daily dose of vitamin supplement, as indicated on the product label. (II-2-A) 4. Folic acid and multivitamin supplements should be widely available without financial or other barriers for women planning pregnancy to ensure the extra level of supplementation. (III- 5. Folic acid 5 mg supplementation will not mask vitamin B12 deficiency (pernicious anemia), and investigations (examination or laboratory) are not required prior to initiating supplementation. (II-2-A) 6. The recommended strategy to prevent recurrence of a congenital anomaly (anencephaly, myelomeningocele, meningocele, oral facial cleft, structural heart disease, limb defect, urinary tract anomaly, hydrocephalus) that has been reported to have a decreased incidence following preconception / first trimester folic acid +/- multivitamin oral supplementation is planned pregnancy +/- supplementation compliance. A folate-supplemented diet with additional daily supplementation of multivitamins with 5 mg folic acid should begin at least three months before conception and continue until 10 to 12 weeks post conception. From 12 weeks post-conception and continuing throughout pregnancy and the postpartum period (4-6 weeks or as long as breastfeeding continues), supplementation should consist of a multivitamin with folic acid (0.4-1.0 mg). (I-A) 7. The recommended strategy(ies) for primary prevention or to decrease the incidence of fetal congenital anomalies will include a number of options or treatment approaches depending on patient age, ethnicity, compliance, and genetic congenital anomaly risk status.

OPTION A: Patients with no personal health risks, planned pregnancy, and good compliance require a good diet of folate-rich foods and daily supplementation with a multivitamin with folic acid (0.4-1.0 mg) for at least two to three months before conception and throughout pregnancy and the postpartum period (4-6 weeks and as long as breastfeeding continues). (II-2-A)

OPTION B: Patients with health risks, including epilepsy, insulin dependent diabetes, obesity with BMI >35 kg/m2, family history of neural tube defect, belonging to a high-risk ethnic group (e.g., Sikh) require increased dietary intake of folate-rich foods and daily supplementation, with multivitamins with 5 mg folic acid, beginning at least three months before conception and continuing until 10 to 12 weeks post conception. From 12 weeks post-conception and continuing throughout pregnancy and the postpartum period (4-6 weeks or as long as breastfeeding continues), supplementation should consist of a multivitamin with folic acid (0.4-1.0 mg). (II-2-A)

OPTION C: Patients who have a history of poor compliance with medications and additional lifestyle issues of variable diet, no consistent birth control, and possible teratogenic substance use (alcohol, tobacco, recreational non-prescription drugs) require counselling about the prevention of birth defects and health problems with folic acid and multivitamin supplementation. The higher dose folic acid strategy (5 mg) with multivitamin should be used, as it may obtain a more adequate serum red blood cell folate level with irregular vitamin / folic acid intake but with a minimal additional health risk. (III- 8.The Canadian Federal Government could consider an evaluation process for the benefit/risk of increasing the level of national folic acid flour fortification to 300 mg/100 g (present level 140 mg/100 g). (III- 9.The Canadian Federal Government could consider an evaluation process for the benefit/risk of additional flour fortification with multivitamins other than folic acid. (III- 10.The Society of Obstetricians and Gynaecologists of Canada will explore the possibility of a Canadian Consensus conference on the use of folic acid and multivitamins for the primary prevention of specific congenital anomalies. The conference would include Health Canada/Congenital Anomalies Surveillance, Canadian College of Medical Geneticists, Canadian Paediatric Society, Motherisk, and pharmaceutical industry representatives.

VALIDATION: This is a revision of a previous guideline and information from other consensus reviews from medical and government publications has been used. SPONSOR: The Society of Obstetricians and Gynaecologists of Canada.

J Obstet Gynaecol Can. 2007 Dec;29(12):1003-26

PMID: 18053387 [PubMed - indexed for MEDLINE]

http://www.ncbi.nlm.nih.gov/pubmed/18053387

>> Do you mean that I should be taking Calcium too?> Thanks> > > one, two or three 1000IU tabs/caps/gels (Solgar do them ~ £6.75/100) per day integratd with your calcium intake>

[Guest guest]

How the thyroid function might go wrong:-

http://www.ncbi.nlm.nih.gov/pubmed/18279780

Thyroid gland development and defects.

Kratzsch J, Pulzer F.

Institute of Laboratory Medicine, Clinical Chemistry and Molecular Diagnostics, University Hospital, -List-Str. 13-15, D-04103 Leipzig, Germany. kraj@...

During the functional ontogenesis of the thyroid gland an increasing number of transcription factors play fundamental roles in thyroid-cell differentiation, maintenance of the differentiated state, and thyroid-cell proliferation. The early growth and development of the fetal thyroid appears to be generally independent of thyroid-stimulating hormone (TSH). TSH and thyroxine (T4) levels increase from the 12th week of gestation until delivery, whereas triiodothyronine (T3) levels remain relatively low. At birth, a cold-stimulated short-lived TSH surge is observed, followed by a TSH decrease until day 3 or 4 of life by T4 feedback inhibition. Disorders of thyroid gland development and/or function are relatively common, affecting approximately one newborn infant in 2000-4000. The most prevalent disease, congenital hypothyroidism, is frequently caused by genetic defects of transcription factors involved in the development of the thyroid or pituitary gland. A major cause of congenital hyperthyroidism is the transplacental passage of stimulating thyrotropin antibodies from the mother to the fetus. Hypothyroxinaemia or hypotriiodthyroninaemia is frequently observed in preterm infants with or without severe non-thyroidal illness. Whereas congenital hypo- and hyperthyroidism may be treated successfully with T4 or thyrostatic drugs, there is still insufficient evidence on whether the use of T4 for treatment of the latter condition results in changes in neonatal morbidity or reductions in neurodevelopmental impairment.

Best Pract Res Clin Endocrinol Metab. 2008 Feb;22(1):57-75

PMID: 18279780 [PubMed - indexed for MEDLINE]

http://www.ncbi.nlm.nih.gov/pubmed/18279780

http://dx.doi.org/10.1016/j.beem.2007.08.006

>> Do you mean that I should be taking Calcium too?> Thanks> > > one, two or three 1000IU tabs/caps/gels (Solgar do them ~ £6.75/100) per day integratd with your calcium intake>

[Guest guest]

As usual, the Finns got there twenty years ago.

http://www.ncbi.nlm.nih.gov/pubmed/3488981

Serum levels of vitamin D metabolites, calcium, phosphorus, magnesium and alkaline phosphatase in Finnish women throughout pregnancy and in cord serum at delivery.

Kuoppala T, Tuimala R, Parviainen M, Koskinen T, Ala-Houhala M

Serum concentrations of 25(OH)D, 24,25(OH)2D, 1,25(OH)2D, total calcium, protein, phosphorus, magnesium and alkaline phosphatase were measured in two groups of Finnish women throughout pregnancy and in cord serum at delivery. The autumn group delivered in August-September and the spring group in February-March.

There was strong seasonal variation in the 25(OH)D concentrations in both groups. Maternal values (mean +/- s.d.) at delivery were 44.3 +/- 20.8 nmol/l in autumn and 26.0 +/- 13.0 nmol/l in spring. Fetal concentrations were 28.8 +/- 14.3 and 18.3 +/- 11.3 nmol/l, respectively. In both mothers and infants low 25(OH)D values were measured in winter. In the autumn group 7 out of 21 mothers (33 per cent) and in the spring group 17 out of 36 mothers (47 per cent) had values below 17 nmol/l, which is the lowest winter reference value recorded in our laboratory. No significant seasonal variation was observed in dihydroxylated vitamin D metabolites, although 24,25(OH)2D values were a little higher in summer than in winter. Concentrations of 1,25(OH)2D tended to rise towards delivery. Corrected calcium, magnesium and phosphorus concentrations did not change during pregnancy. Fetal calcium and phosphorus concentrations were significantly (P less than 0.001) higher than maternal ones.

The data indicate that many mothers and infants have poor vitamin D status in the latitude of Finland. Our results support the concept that vitamin D supplementation should be considered in Finland for pregnant women at least in winter.

Hum Nutr Clin Nut. 1986 Jul;40(4):287-93.

PMID: 3488981 [PubMed - indexed for MEDLINE]>> Do you mean that I should be taking Calcium too?> Thanks> > > one, two or three 1000IU tabs/caps/gels (Solgar do them ~ £6.75/100) per day integratd with your calcium intake>

[Guest guest]

Effectiveness and safety of vitamin D in relation to bone health.

Cranney A, Horsley T, O'Donnell S, Weiler H, Puil L, Ooi D, Atkinson S, Ward L, Moher D, Hanley D, Fang M, Yazdi F, Garritty C, Sampson M, Barrowman N, Tsertsvadze A, Mamaladze V.

OBJECTIVES: To review and synthesize the literature in the following areas: the association of specific circulating 25(OH)D concentrations with bone health outcomes in children, women of reproductive age, postmenopausal women and elderly men; the effect of dietary intakes (foods fortified with vitamin D and/or vitamin D supplementation) and sun exposure on serum 25(OH)D; the effect of vitamin D on bone mineral density (BMD) and fracture or fall risk; and the identification of potential harms of vitamin D above current reference intakes.

DATA SOURCES: MEDLINE® (1966-June Week 3 2006); Embase (2002-2006 Week 25); CINAHL (1982-June Week 4, 2006); AMED (1985 to June 2006); Biological Abstracts (1990-February 2005); and the Cochrane Central Register of Controlled Trials (2nd Quarter 2006).

REVIEW METHODS: Two independent reviewers completed a multi-level process of screening the literature to identify eligible studies (title and abstract, followed by full text review, and categorization of study design per key question). To minimize bias, study design was limited to randomized controlled trials (RCTs) wherever possible. Study criteria for question one were broadened to include observational studies due to a paucity of available RCTs, and question four was restricted to systematic reviews to limit scope. Data were abstracted in duplicate and study quality assessed. Differences in opinion were resolved through consensus or adjudication. If clinically relevant and statistically feasible, meta-analyses of RCTs on vitamin D supplementation and bone health outcomes were conducted, with exploration of heterogeneity. When meta-analysis was not feasible, a qualitative systematic review of eligible studies was conducted.

RESULTS: 167 studies met our eligibility criteria (112 RCTs, 19 prospective cohorts, 30 case-controls and six before-after studies). The largest body of evidence on vitamin D status and bone health was in older adults with a lack of studies in premenopausal women and infants, children and adolescents. The quality of RCTs was highest in the vitamin D efficacy trials for prevention of falls and/or fractures in older adults. There was fair evidence of an association between low circulating 25(OH)D concentrations and established rickets. However, the specific 25(OH)D concentrations associated with rickets is uncertain, given the lack of studies in populations with dietary calcium intakes similar to North American diets and the different methods used to determine 25(OH)D concentrations. There was inconsistent evidence of an association of circulating 25(OH)D with bone mineral content in infants, and fair evidence that serum 25(OH)D is inversely associated with serum PTH.

In adolescents, there was fair evidence for an association between 25(OH)D levels and changes in BMD. There were very few studies in pregnant and lactating women, and insufficient evidence for an association between serum 25(OH)D and changes in BMD during lactation, and fair evidence of an inverse correlation with PTH. In older adults, there was fair evidence that serum 25(OH)D is inversely associated with falls, fair evidence for a positive association with BMD, and inconsistent evidence for an association with fractures. The imprecision of 25(OH)D assays may have contributed to the variable thresholds of 25(OH)D below which the risk of fractures, falls or bone loss was increased. There was good evidence that intakes from vitamin D-fortified foods (11 RCTs) consistently increased serum 25(OH)D in both young and older adults.

Eight randomized trials of ultraviolet (UV)-B radiation (artificial and solar exposure) were small and heterogeneous with respect to determination of the exact UV-B dose and 25(OH)D assay but there was a positive effect on serum 25(OH)D concentrations. It was not possible to determine how 25(OH)D levels varied by ethnicity, sunscreen use or latitude. Seventy-four trials examined the effect of vitamin D(3) or D(2) on 25(OH)D concentrations. Most trials used vitamin D(3), and the majority enrolled older adults. In three trials, there was a greater response of serum 25(OH)D concentrations to vitamin D(3) compared to vitamin D(2), which may have been due to more rapid clearance of vitamin D(2) in addition to other mechanisms. Meta-analysis of 16 trials of vitamin D(3) was consistent with a dose-response effect on serum 25(OH)D when comparing daily doses of <400 IU to doses >/= 400 IU.

An exploratory analysis of the heterogeneity demonstrated a significant positive association comparable to an increase of 1 - 2 nmol/L in serum 25(OH)D for every 100 additional units of vitamin D although heterogeneity remained after adjusting for dose. Vitamin D(3) in combination with calcium results in small increases in BMD compared to placebo in older adults although quantitative synthesis was limited due to variable treatment durations and BMD sites. The evidence for fracture reduction with vitamin D supplementation was inconsistent across 15 trials. The combined results of trials using vitamin D(3) (700 - 800 IU daily) with calcium (500 - 1,200 mg) was consistent with a benefit on fractures although in a subgroup analysis by setting, benefit was primarily in elderly institutionalized women (fair evidence from two trials).

There was inconsistent evidence across 14 RCTs of a benefit on fall risk. However, a subgroup analysis showed a benefit of vitamin D in postmenopausal women, and in trials that used vitamin D(3) plus calcium. In addition, there was a reduction in fall risk with vitamin D when six trials that adequately ascertained falls were combined. Limitations of the fall and fracture trials included poor compliance with vitamin D supplementation, incomplete assessment of vitamin D status and large losses to follow-up. We did not find any systematic reviews that addressed the question on the level of sunlight exposure that is sufficient to maintain serum 25(OH)D concentrations but minimizes risk of melanoma and non-melanoma skin cancer.

There is little evidence from existing trials that vitamin D above current reference intakes is harmful. In most trials, reports of hypercalcemia and hypercalciuria were not associated with clinically relevant events. The Women's Health Initiative study did report a small increase in kidney stones in postmenopausal women aged 50 to 79 years whose daily vitamin D(3) intake was 400 IU (the reference intake for 50 to 70 years, and below the reference intake for > 70 years) combined with 1000 mg calcium. The increase in renal stones corresponded to 5.7 events per 10,000 person-years of exposure. The women in this trial had higher calcium intakes than is seen in most post-menopausal women. CONCLUSIONS: The results highlight the need for additional high quality studies in infants, children, premenopausal women, and diverse racial or ethnic groups.

There was fair evidence from studies of an association between circulating 25(OH)D concentrations with some bone health outcomes (established rickets, PTH, falls, BMD). However, the evidence for an association was inconsistent for other outcomes (e.g., BMC in infants and fractures in adults). It was difficult to define specific thresholds of circulating 25(OH)D for optimal bone health due to the imprecision of different 25(OH)D assays. Standard reference preparations are needed so that serum 25(OH)D can be accurately and reliably measured, and validated. In most trials, the effects of vitamin D and calcium could not be separated. Vitamin D(3) (>700 IU/day) with calcium supplementation compared to placebo has a small beneficial effect on BMD, and reduces the risk of fractures and falls although benefit may be confined to specific subgroups. Vitamin D intake above current dietary reference intakes was not reported to be associated with an increased risk of adverse events. However, most trials of higher doses of vitamin D were not adequately designed to assess long-term harms.

Evid Rep Technol Assess (Full Rep). 2007 Aug;(158):1-235.

PMID: 18088161 [PubMed - indexed for MEDLINE]

http://www.ncbi.nlm.nih.gov/pubmed/18088161

>> Do you mean that I should be taking Calcium too?> Thanks> > > one, two or three 1000IU tabs/caps/gels (Solgar do them ~ £6.75/100) per day integratd with your calcium intake>

[Guest guest]

http://journals.cambridge.org/action/displayAbstract?fromPage=online & aid=1590728

What do we currently know about nutrition and bone health in relation to United Kingdom public health policy with particular reference to calcium and vitamin D?

>> Hi Bob,> > Sorry if we are off subject here but this is jaw dropping stuff. Does this potentially mean that the huge increase in autism in kids is , or could be due to their mothers being vitamin d deficient during pregnancy ?!? This is just awful......> > julia>

[Guest guest]

Hi ,

This may be even more jaw-dropping .....

Problems with vitamin D arise if there is obvious inflammatory disease and/or

any of the conditions mentioned within this video ~

http://bacteriality.com/2008/05/07/mpintro/

best wishes

Bob

>

> Hi Bob,

>

> Sorry if we are off subject here but this is jaw dropping stuff. Does this

potentially mean that the huge increase in autism in kids is , or could be due

to their mothers being vitamin d deficient during pregnancy ?!?  This is just

awful......

>

> julia

>

[Guest guest]

Although it wasn't you that Manda was referring to , I

think you two would have an awful lot in common.

luv - Sheila

Hi Manda,

I wasn't at Sheilas house but I

do have an autistic son....... not sure if it's me your'e talking about.

julia

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06:15:00

[Guest guest]

Hi Lorraine

I have found children with Autism, anxiety, or attention deficit are better off in subspace for he first session. It seems to prime them and they are more able to have the sit down session in the future. I also use electro magnetic protection for them prior to the session( and there after). I like Bio Pro or Phyllis light products, and there are several choices from both suppliers. I do not over load a person(child or otherwise) with too much the first session. Lay down the basics (like Levi's protocol), along with being sure to open the organs of elimination and work on spiritual healing.  I find the NLP to be most helpful and clearing or becoming aware of miasms a must.  Kim

On Wed, Jun 10, 2009 at 4:06 PM, bumsteadl <labumstead@...> wrote:

Hi groupI just had a very distraught mother of a 3 year old approach me at the health food store. Her 3 year old has been 'diagnosed' with autism spectrum disorder. She requested a biofeedback session. I need advise about how to work with this young lad. I have worked with children before but no one as young as this.

Specifically:how long should the session be?what are the most important panels to run?Any information would be humbly appreciated. I don't have the 'new' version yet.You can email me at labumstead@... or post here.

ThanksLorraine

[Guest guest]

I searched the archived responses regarding Autism from this group and

found lots of information. Thanks to those who have responded.

Lorraine