Guest guest Posted May 23, 2008 Report Share Posted May 23, 2008 http://www.ncbi.nlm.nih.gov/pubmed/8289586 http://www.psychosomaticmedicine.org/cgi/reprint/51/4/441.pdf Glucose (fasting and area under the curve) was not modified significantly after naltrexone administration. Baseline hormone levels were similar in the two groups, and they did not change after long-term treatment with naltrexone or placebo. In conclusion, these results support the hypothesis of elevated opioid tonus and increased insulin secretion as a possible mechanism of hyperinsulinism in a group of hyperandrogenic women of ovarian origin. This alteration could act as an additional factor in the pathogenesis of insulin resistance found in an important proportion of these patients. see - http://cat.inist.fr/?aModele=afficheN & cpsidt=2445124 Celia, Scotland.celia@... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted May 27, 2008 Report Share Posted May 27, 2008 could someone explain what this is saying in plain english please? http://www.ncbi.nlm.nih.gov/pubmed/8289586 http://www.psychosomaticmedicine.org/cgi/reprint/51/4/441.pdf Glucose (fasting and area under the curve) was not modified significantly after naltrexone administration. Baseline hormone levels were similar in the two groups, and they did not change after long-term treatment with naltrexone or placebo. In conclusion, these results support the hypothesis of elevated opioid tonus and increased insulin secretion as a possible mechanism of hyperinsulinism in a group of hyperandrogenic women of ovarian origin. This alteration could act as an additional factor in the pathogenesis of insulin resistance found in an important proportion of these patients. see - http://cat.inist.fr/?aModele=afficheN & cpsidt=2445124 thanks monique Quote Link to comment Share on other sites More sharing options...
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