Re: Belly fat/ serosorting

[Guest guest]

I think it's interesting about your friend with diabetes and his hard belly,

since diabetes is another in a group of metabolic side effects that seem

related to lipodystrophy. I wonder about the similarities. Diabetes

sufferers also get neuropathy. I've been thinking about that lately. I don't

know the mechanism for HIV-related problems completely, much less diabetes.

May be coincidence, too? I know I'm also glucose intolerant and have to use

diet and lipitor to control lipids/cholesterol. These weird lipomas (small

fat globules under the skin) are increasing in number, too, 10 years after

starting protease inhibitors. It's a perplexing puzzle - HIV.

BG

Belly fat/ serosorting

personally i do not doubt the reality of 'crix belly', ie visceral

fat accumulation resulting from use of protease inhibitors...but i

will offer that my most frequent encounters with hard visceral fat

accumulation prior to moving to Chelsea 2 years ago were with an

obese childhood friend with diabetes. his belly is huge and has been

hard since he was a teen, he was dignosed diabetic around the age of

30. i've also known a number of big bearish types with hard bellies,

and most did not do oblique exercises so we're NOT talking muscular

development or the so-called " pitcher's body " .

it may well turn out that 'crix belly' was a red herring, that the

people getting it would have gotten it anyway but that the

mitochondrial toxicities of other drugs caused extremity bodyfat to

melt away, leaving the hard belly that much more obvious. i doubt it

tho, as most of the people i've known with big hard non-muscular

bellies got them relatively early in life, whereas people with P-

belly seem to manifest it over time after starting PIs, especially

after the age of 30. perhaps the normal accumulation of belly fat

that accompanies middle-age is being warped? I'm eager to know!

re: serosorting, the issues others have covered at length leave me

with the impression that *for people several years into their

infection and HAART therapy*, treatment adherence remains a far

bigger predictor of treatment failure and/or resistance-emergence. i

personally dont have a lot of treatment options left to me, and am as

obsessive about adherence as it is possible to be. i will take

whatever informed risks i and my partners deem acceptable to each of

us. i encourage others to have these kinds of discussions with their

partners.

[Guest guest]

I think it's interesting about your friend with diabetes and his hard belly,

since diabetes is another in a group of metabolic side effects that seem

related to lipodystrophy. I wonder about the similarities. Diabetes

sufferers also get neuropathy. I've been thinking about that lately. I don't

know the mechanism for HIV-related problems completely, much less diabetes.

May be coincidence, too? I know I'm also glucose intolerant and have to use

diet and lipitor to control lipids/cholesterol. These weird lipomas (small

fat globules under the skin) are increasing in number, too, 10 years after

starting protease inhibitors. It's a perplexing puzzle - HIV.

BG

Belly fat/ serosorting

personally i do not doubt the reality of 'crix belly', ie visceral

fat accumulation resulting from use of protease inhibitors...but i

will offer that my most frequent encounters with hard visceral fat

accumulation prior to moving to Chelsea 2 years ago were with an

obese childhood friend with diabetes. his belly is huge and has been

hard since he was a teen, he was dignosed diabetic around the age of

30. i've also known a number of big bearish types with hard bellies,

and most did not do oblique exercises so we're NOT talking muscular

development or the so-called " pitcher's body " .

it may well turn out that 'crix belly' was a red herring, that the

people getting it would have gotten it anyway but that the

mitochondrial toxicities of other drugs caused extremity bodyfat to

melt away, leaving the hard belly that much more obvious. i doubt it

tho, as most of the people i've known with big hard non-muscular

bellies got them relatively early in life, whereas people with P-

belly seem to manifest it over time after starting PIs, especially

after the age of 30. perhaps the normal accumulation of belly fat

that accompanies middle-age is being warped? I'm eager to know!

re: serosorting, the issues others have covered at length leave me

with the impression that *for people several years into their

infection and HAART therapy*, treatment adherence remains a far

bigger predictor of treatment failure and/or resistance-emergence. i

personally dont have a lot of treatment options left to me, and am as

obsessive about adherence as it is possible to be. i will take

whatever informed risks i and my partners deem acceptable to each of

us. i encourage others to have these kinds of discussions with their

partners.