Re: CDC Acknowledges Inhaled Mycotoxins Can Cause Death

[Guest guest]

As I understand it, this is from March 2005. However it was just put up on

their website as of Jan 18, 2006. Go figure!

_http://www.bt.cdc.gov/agent/trichothecene/pdf/trichothecenecasedef.pdf_

(http://www.bt.cdc.gov/agent/trichothecene/pdf/trichothecenecasedef.pdf)

Somebody should really tell Dr. Redd of the CDC about this...Ya

know. The guy who is in charge of this issue. Because on Sept 28, 2005, while

presenting a telecast meant to alert the citizen of New Orleans of the health

hazards caused by mold, Dr. Redd stated,

" The third category is that of toxin-mediated disease. Some molds are capable

of producing toxins. They won't produce these toxins at all times but under

certain circumstances, like the nutrient supply is getting short or some

environmental issue, they may start producing toxins and those can be dangerous

if they're eaten or if they're touched.

There's up to now not been evidence that airborne mold toxins have produced

disease. "

The presentation was entitled, " Update on Health Issues Related to Mold,

Mildew and Mud in Hurricane and Flood Affected Areas " Wednesday, September

28,

2005

Looks to me like Dr. Redd is the one who needs to be updated!

Tell me why it is that the CDC produced this document in March of 2005, but

Redd was telling the public there is no evidence that airborne mold toxins

have produced disease in September of 2005.

I guess death is not caused by disease, huh?

Sharon

[Guest guest]

Hello!

Do you have a CDC URL for this datasheet? And is it new news (*how did

you hear about it*)

Thank you!

On 1/24/06, snk1955@... <snk1955@...> wrote:

> CDC Chemical Emergencies

>

> CASE DEFINITION

> Trichothecene Mycotoxins

> Clinical description

>

> Trichothecene mycotoxins might be weaponized and dispersed through the air

> or mixed in food or beverages. Initially, route-specific effects are typically

> prominent. Dermal exposure leads to burning pain, redness, and blisters, and

> oral exposure leads to vomiting and diarrhea. Ocular exposure might result in

> blurred vision, and inhalational exposure might cause nasal irritation and

> cough. Systemic symptoms can develop with all routes of

> exposure and might include weakness, ataxia, hypotension, coagulopathy, and

> death (1).

>

> Laboratory criteria for diagnosis

>

> • Biologic: Selected commercial laboratories are offering immunoassays to

> identify trichothecenes or trichothecene-specific antibodies in human blood or

> urine (2, 3). However, these procedures have not been analytically validated

> and are not recommended.

>

> • Environmental: Detection of trichothecene mycotoxins in environmental

> samples, as determined by FDA.

>

> As a result of indoor air-quality investigations involving mold and

> potentially mold-related health effects, mycotoxin analyses of bulk

environmental

> samples are now commercially available through environmental microbiology

> laboratories in the United States (4). Studies have not been done to

determine the

> background level of trichothecenes in non-moldy homes and office buildings or

> nonagricultural outdoor environments. Therefore, the simple detection of

> trichothecenes in environmental samples does not invariably indicate an

> intentional

> contamination.

>

> Case classification

>

> • Suspected: A case in which a potentially exposed person is being evaluated

> by health-care workers or public health officials for poisoning by a

> particular chemical agent, but no specific credible threat exists.

>

> • Probable: A clinically compatible case in which a high index of suspicion

> (credible threat or patient history regarding location and time) exists for

> trichothecene mycotoxins exposure, or an epidemiologic link exists between

this

> case and a laboratory-confirmed case.

>

> • Confirmed: A clinically compatible case in which laboratory tests of

> environmental samples have confirmed exposure.

>

>

>

>

>

> The case can be confirmed if laboratory testing was not performed because

> either a predominant amount of

> clinical and nonspecific laboratory evidence of a particular chemical was

> present or a 100% certainty of the

> etiology of the agent is known.

>

>

>

>

> Additional resources

>

>

>

> 1. Wannemacher RW Jr, Wiener SL. Trichothecene mycotoxins. In: Zajtchuk R,

> Bellamy RF, eds.

> Textbook of military medicine: medical aspects of chemical and biologic

> warfare. Washington, DC:

> Office of the Surgeon General at TMM Publications, Borden Institute, Walter

> Army Medical

> Center; 1997:655-77.

> 2. Croft WA, Jastromski BM, Croft AL, s HA. Clinical confirmation of

> trichothecene mycotoxicosis

> in patient urine. J Environ Biol 2002;23:301-20.

> 3. Vojdani A, Thrasher HD, Madison RA, Gray MR, Heuser G, AW.

> Antibodies to molds and

> satratoxin in individuals exposed in water-damaged buildings. Arch Environ

> Health. 2003;58:421-32.

> 4. Tuomi T, Reijula K, sson T, et al. Mycotoxins in crude building

> materials from water-damaged

> buildings. Appl Environ Microbiol 2000;66:1899-904.

>

>

>

>

>

>

[Guest guest]

Answered my own question.. Thank you..

Here is the web page:

http://www.bt.cdc.gov/agent/trichothecene/casedef.asp

[Guest guest]

Perhaps death is caused by ... POLITICS...