Thinking out loud

[Guest guest]

When the hints are there, but not sure how to interpret, you can rely on the Linking portion of the Test matrix. Take an area of concern the client has. ie there big toe and type it in to the search. Now take the highest number of relevance in the test matrix it gives you on the toe. Drag it over to the hold box and drop it in. Take the second area of concern they have, ie their shoulder pain, type that into the search. Now take the highest number you feel relevant from the test matrix on this. Drop it into the second hold box. Now on the new program there is a button that is to the right of these to link the two together. Put your test matrix back on the "main" test matrix information and then click that link button. What does it tell you now below your "go to number box" bottom right of page. You can also put your main panel on a different item like worms and then click the link button to see the possible links.

You'll be surprised what this will tell you.

If you don't have the link button yet, then just do this on one hold button at a time. Check that "hold" item to your choice of sub categories. Worms, virus, bio-warfare, etc. The machine gives you all kinds of help, we just have to find out where it is.

Yours in health,

Kathy jas_barnes <jas_barnes@...> wrote:

Hi group,As a QX user it has struck me on several occasions when dealing with people where nothing obvious seems to stand out except occasionally a hormone, phenol, sugar or amino acid or whatever. When this happens I refer to the books I have and although I have been told they are pretty good they don't have the detail to explain what the significance could be. I realise we are lucky in that the QX is very sensitive but unless we understand the relevance of the hints we may lose the adantage the QX should give us in helping people with their health.It strikes me that it would be good if there was an online site loaded with this type of information per QX item. it would explain what the item was, what its significance might indicate, contraindications, if a remedy where it can be found eg diet, if a toxin what likley

sources. It would also enable us to check out something quickly if in posession of another pc.Now if this already exists someone please let me know. If it does not I'm sure I'm not the only one that would be interested in subscribing to such a database for a moderate fee if anyone feels the inclination and has the knowledge to do something like this.Regards to all.............................................

[Guest guest]

This is a great idea! Anyone have the time to set it up.......?

love Jayne xxx

Thinking out loud

Hi group,

As a QX user it has struck me on several occasions when dealing with

people where nothing obvious seems to stand out except occasionally

a hormone, phenol, sugar or amino acid or whatever. When this

happens I refer to the books I have and although I have been told

they are pretty good they don't have the detail to explain what the

significance could be. I realise we are lucky in that the QX is very

sensitive but unless we understand the relevance of the hints we may

lose the adantage the QX should give us in helping people with their

health.

It strikes me that it would be good if there was an online site

loaded with this type of information per QX item. it would explain

what the item was, what its significance might indicate,

contraindications, if a remedy where it can be found eg diet, if a

toxin what likley sources. It would also enable us to check out

something quickly if in posession of another pc.

Now if this already exists someone please let me know. If it does

not I'm sure I'm not the only one that would be interested in

subscribing to such a database for a moderate fee if anyone feels

the inclination and has the knowledge to do something like this.

Regards to all.

.............................................

[Guest guest]

Okay, so my previously mentioned online guru, andrew, tells me that

calcium loads up in the nerves and triggered by excess glutamate

causing a cascade of calcium toxicity (for those with seizures).

The article I posted also mentions calcium toxicity, as well.

Interestingly, I know my daughters med's (depakene) works by

blocking glutamines and is known to deplete calcium. It also does a

lot of other things, depletes other nutrients/vitamins. These are

the only two that I know *why* the meds are doing this.

Too much calcium,supplementing calcium must not be good for those

with seizures, then.

I read eons ago something about sodium ion channels and epilepsy.

I am wondering how calcium is related to sodium (if at all). What

calcium ions have to do with sodium ions (if at all). How these

apply to sugars, sugar control. (We know glutamines are the trigger

for this calcium cascade). Why do people with seizures do this and

others don't?

What regulates calcium nerve loading? (magnesium???)

What regulates the sodium ions?

Is there a magnesium " regulation " disorder/deficiency/problem?

Are there amino acids or enzymes responsible for this?

Okay, this is what I'm going to be checking out. If anyone knows

the answers to these things...please share info. Or if you have

questions to add that would be worth looking into, please set me on

the trail.

Mindy

[Guest guest]

I would really be interested in the answers to these questions.

Grace

littlroses <littlroses@...> wrote:

Okay, so my previously mentioned online guru, andrew, tells me that

calcium loads up in the nerves and triggered by excess glutamate

causing a cascade of calcium toxicity (for those with seizures).

The article I posted also mentions calcium toxicity, as well.

Interestingly, I know my daughters med's (depakene) works by

blocking glutamines and is known to deplete calcium. It also does a

lot of other things, depletes other nutrients/vitamins. These are

the only two that I know *why* the meds are doing this.

Too much calcium,supplementing calcium must not be good for those

with seizures, then.

I read eons ago something about sodium ion channels and epilepsy.

I am wondering how calcium is related to sodium (if at all). What

calcium ions have to do with sodium ions (if at all). How these

apply to sugars, sugar control. (We know glutamines are the trigger

for this calcium cascade). Why do people with seizures do this and

others don't?

What regulates calcium nerve loading? (magnesium???)

What regulates the sodium ions?

Is there a magnesium " regulation " disorder/deficiency/problem?

Are there amino acids or enzymes responsible for this?

Okay, this is what I'm going to be checking out. If anyone knows

the answers to these things...please share info. Or if you have

questions to add that would be worth looking into, please set me on

the trail.

Mindy

[Guest guest]

Remember the old junior high school frog's leg experiments? Sodium make the

leg contract potassium makes it relax and calcium regulates the two of them.

Now there is another relationship between magnesium and phosphorus and

calcium as the work in balance as well. Calcium isn't always evil with

epileptics; it is the imbalance of calcium, sodium, potassium, magnesium as

well as chlorides, bicarbonates, and sulfates that causes the problems. To

further complicate things, calcium can also reduce iron but that isn't

always a bad thing as excessive iron can be oxidative and that increases

inflammatory processes which lower the seizure threshold.

As for the amino acids, glycine, GABA and taurine have involvement as

counterbalances to the excitatory amino acids. But, and it's a big but,

those excitatory aren't all bad either. Aspartic acid, very excitatory is

important in removing ammonia from the system and hyperammonemia can lower

the seizure threshold as well. Zinc depletion, which some of the meds can

do, will reduce the effectiveness of the neuroinhibitory amino acids.

Honestly, it gets more complex from here. This is why most neurologists

won't go down this path. Too hard to control all of these issues on a

patient to patient basis. This is why we all have to do it ourselves.

Mark Schauss, MBA, DB

www.carbonbased.com

Re: [ ] Thinking out loud

I would really be interested in the answers to these questions.

Grace

littlroses <littlroses@...> wrote:

Okay, so my previously mentioned online guru, andrew, tells me that

calcium loads up in the nerves and triggered by excess glutamate

causing a cascade of calcium toxicity (for those with seizures).

The article I posted also mentions calcium toxicity, as well.

Interestingly, I know my daughters med's (depakene) works by

blocking glutamines and is known to deplete calcium. It also does a

lot of other things, depletes other nutrients/vitamins. These are

the only two that I know *why* the meds are doing this.

Too much calcium,supplementing calcium must not be good for those

with seizures, then.

I read eons ago something about sodium ion channels and epilepsy.

I am wondering how calcium is related to sodium (if at all). What

calcium ions have to do with sodium ions (if at all). How these

apply to sugars, sugar control. (We know glutamines are the trigger

for this calcium cascade). Why do people with seizures do this and

others don't?

What regulates calcium nerve loading? (magnesium???)

What regulates the sodium ions?

Is there a magnesium " regulation " disorder/deficiency/problem?

Are there amino acids or enzymes responsible for this?

Okay, this is what I'm going to be checking out. If anyone knows

the answers to these things...please share info. Or if you have

questions to add that would be worth looking into, please set me on

the trail.

Mindy

[Guest guest]

Unfortunately, I was a slacker in science and math back then. I

took biology, but my dad was U.S. army and juuuuust about the time

we were getting into the frog thing, we packed up and moved to

another locale where they had just finished off the frog thing. I

thought I was pretty lucky then! Talk about kicking myself in the

pants. I thought English and history were the way to go. LOL

Thanks for explaining the sodium,potassium, calcium relationship in

easy to understand terms.

I've been trying to understand the inflammatory issues and

prostaglandins. Sugar and grains are inflammatory, but of course,

can't be the total problem. I guess a good question is, what's at

work causing the inflammatory response? Virus? Heavy metals? All

of the above?

> Okay, so my previously mentioned online guru, andrew, tells me

that

> calcium loads up in the nerves and triggered by excess glutamate

> causing a cascade of calcium toxicity (for those with seizures).

>

> The article I posted also mentions calcium toxicity, as well.

>

> Interestingly, I know my daughters med's (depakene) works by

> blocking glutamines and is known to deplete calcium. It also does

a

> lot of other things, depletes other nutrients/vitamins. These are

> the only two that I know *why* the meds are doing this.

>

> Too much calcium,supplementing calcium must not be good for those

> with seizures, then.

>

> I read eons ago something about sodium ion channels and epilepsy.

>

> I am wondering how calcium is related to sodium (if at all). What

> calcium ions have to do with sodium ions (if at all). How these

> apply to sugars, sugar control. (We know glutamines are the

trigger

> for this calcium cascade). Why do people with seizures do this

and

> others don't?

>

> What regulates calcium nerve loading? (magnesium???)

>

> What regulates the sodium ions?

>

> Is there a magnesium " regulation " disorder/deficiency/problem?

>

> Are there amino acids or enzymes responsible for this?

>

> Okay, this is what I'm going to be checking out. If anyone knows

> the answers to these things...please share info. Or if you have

> questions to add that would be worth looking into, please set me

on

> the trail.

>

> Mindy

>

>

>

>

>

>

[Guest guest]

Mindy,

You ask some really good and difficult to find snappy answers for,

questions. All i can contribute at the moment is this link to an

article on mineral toxicity.

Zoe

http://www.chclibrary.org/micromed/00056760.html

> Okay, so my previously mentioned online guru, andrew, tells me that

> calcium loads up in the nerves and triggered by excess glutamate

> causing a cascade of calcium toxicity (for those with seizures).

>

> The article I posted also mentions calcium toxicity, as well.

>

> Interestingly, I know my daughters med's (depakene) works by

> blocking glutamines and is known to deplete calcium. It also does

a

> lot of other things, depletes other nutrients/vitamins. These are

> the only two that I know *why* the meds are doing this.

>

> Too much calcium,supplementing calcium must not be good for those

> with seizures, then.

>

> I read eons ago something about sodium ion channels and epilepsy.

>

> I am wondering how calcium is related to sodium (if at all). What

> calcium ions have to do with sodium ions (if at all). How these

> apply to sugars, sugar control. (We know glutamines are the

trigger

> for this calcium cascade). Why do people with seizures do this and

> others don't?

>

> What regulates calcium nerve loading? (magnesium???)

>

> What regulates the sodium ions?

>

> Is there a magnesium " regulation " disorder/deficiency/problem?

>

> Are there amino acids or enzymes responsible for this?

>

> Okay, this is what I'm going to be checking out. If anyone knows

> the answers to these things...please share info. Or if you have

> questions to add that would be worth looking into, please set me on

> the trail.

>

> Mindy

[Guest guest]

Mindy,

You ask some really good and difficult to find snappy answers for,

questions. All i can contribute at the moment is this link to an

article on mineral toxicity.

Zoe

http://www.chclibrary.org/micromed/00056760.html

> Okay, so my previously mentioned online guru, andrew, tells me that

> calcium loads up in the nerves and triggered by excess glutamate

> causing a cascade of calcium toxicity (for those with seizures).

>

> The article I posted also mentions calcium toxicity, as well.

>

> Interestingly, I know my daughters med's (depakene) works by

> blocking glutamines and is known to deplete calcium. It also does

a

> lot of other things, depletes other nutrients/vitamins. These are

> the only two that I know *why* the meds are doing this.

>

> Too much calcium,supplementing calcium must not be good for those

> with seizures, then.

>

> I read eons ago something about sodium ion channels and epilepsy.

>

> I am wondering how calcium is related to sodium (if at all). What

> calcium ions have to do with sodium ions (if at all). How these

> apply to sugars, sugar control. (We know glutamines are the

trigger

> for this calcium cascade). Why do people with seizures do this and

> others don't?

>

> What regulates calcium nerve loading? (magnesium???)

>

> What regulates the sodium ions?

>

> Is there a magnesium " regulation " disorder/deficiency/problem?

>

> Are there amino acids or enzymes responsible for this?

>

> Okay, this is what I'm going to be checking out. If anyone knows

> the answers to these things...please share info. Or if you have

> questions to add that would be worth looking into, please set me on

> the trail.

>

> Mindy

[Guest guest]

Andre wrote:

>

> It helps me to dream of ways that science and technology could one day

> fix A, or at least help make it much more manageable. I am not a

> doctor, and some of this is probably in the far future, but I thought

> it wouldn't hurt to discuss the topic since having hope is important.

>

I also enjoy science and tech and how they may lead to something for

achalasia. I also like hope but I know that false hope in this context

is can be damaging.

>

> 1) Stem cells. Possibly the ultimate solution?

>

The problem here is first that it could be many years away if it can

even be done for achalasia. In achalasia there is ongoing destruction of

nerves. Can the stem cells produce nerve cells in an environment that is

destroying them? If it can will the nerves survive the destructive

process or for how long.

> True, even the massive opposition to stem cell research from some

> quarters pales into insignificance compared to the medical and

> technical obstacles, but we've all probably read the articles about

> stem cells already demonstrating some neuron-like abilities in tests.

>

The opposition is to fetal stem cells, not stem cells, and the political

debate was about government funding of fetal stem cell research not

about banning fetal stem cell research and certainly not about banning

stem cell research.

Neural stem cells for the treatment of disorders of the enteric nervous

system: Strategies and challenges

http://onlinelibrary.wiley.com/doi/10.1002/dvdy.20975/full

That study ends with statements that stem cell therapy may be over rated

but that it appears to offer hope. So, we have some hope but we need to

remember that it is not a promise.

It should be noted that this stem cell research was done at: Enteric

Neuromuscular Disorders and Pain Laboratory, Division of

Gastroenterology and Hepatology, University of Texas Medical Branch,

Galveston, Texas. Some people don't think we do stem cell research in

the US. Not that you said that. Also, this research relates to

achalasia. Some people don't think scientist are interested in doing

research for achalasia. Not that you said any such thing either.

> 2) A bionic E, fully peristaltic, even though it is a mechanical piece

> of kit.

>

That much work would probably be equivalent to having an esophagectomy.

I think in that case I would rather just have a gastric pull-up.

>

> 4) An implanted device to electically stimulate the muscles of the

> real LES - a clump of artificial neurons. I gather the problem is more

> mechanical than electrical, in the sense that electric currents close

> muscles, not open them, but perhaps the implanted bio-neurons could be

> made to secrete the chemicals needed to open the (non-HM) LES. This

> feels like a very remote possibility, even by the standards of this list.

>

For those that like images:

Parallel inhibitory and excitatory innervation of the esophageal smooth

muscle.

http://www.nature.com/gimo/contents/pt1/fig_tab/gimo3_F5.html

Which is part of:

Physiology of esophageal motility

http://www.nature.com/gimo/contents/pt1/full/gimo3.html

The neurons in question are like the green postganglionic neuron in the

image. The innervation of the esophagus is actually much more

complicated than that image but the images is good for this discussion.

Doing what you suggest looks simple on that image, just replace the

short green neuron. There are lots of these microscopic little neurons

though. Not that you necessarily have to replace all of them, but you

would have to distribute the chemicals around enough of the LES to be

effective. Some how you would have to control it and somehow you would

have to pass chemicals through it.

> Any others?

>

There is work on growing organs such as the esophagus from one's own

cells. Again, I see problems with this kind of thing being connected up

the nerves. If it can not be innervated then it is just an inactive

tube. You would still have to have your esophagus removed to do that so

it may just make more sense to have a gastric pull-up.

I think there is hope long down the road to come. The problem I see is

that some people would put off treatment that exists today in hope that

some new treatment will be here tomorrow. In the meantime they may be

suffering and doing damage to their esophagi needlessly.

Lets be hopeful but not give in to false hope. Sometimes the best hope

now is on things you can actually do today.

notan