Guest guest Posted May 10, 2003 Report Share Posted May 10, 2003 Most of the cat owning population is immune to toxoplasmosis as it is of feline origin. About 1/3 of the general population is immune whether they have a cat or not. if she had previously been handling raw meat (toxoplasmosis) or gardens (cat pooh=toxoplasmosis) I would be really surprised if she was not already immune. It is quite possible to get a sabin feldaman test (dye) and then an ELISA to check for toxoplasmosis IGm and IGg anti-bodies.infac ACOG recommends it. If the dye test is positive and the negative then she is immune to toxoplasmosis and the fetus is not at risk. Listeriosis (listeria) can be caught from certain animals but the dog is not one of them. Handling raw meat could be be asource althougnbh not the main source..this infection has also been traced to raw vegetables and mainly the issue is with ingesting uncooked or unpasteurised food . The current recommendation (medical ) is to wash ones hands after handling raw food. blessings Tania (not a midwife!) Quote Link to comment Share on other sites More sharing options...
Guest guest Posted May 10, 2003 Report Share Posted May 10, 2003 Listeriosis (listeria) can be caught from certain animals but the dog is not one of them. Handling raw meat could be be asource althougnbh not the main source..this infection has also been traced to raw vegetables and mainly the issue is with ingesting uncooked or unpasteurised food . The current recommendation (medical ) is to wash ones hands after handling raw food. you can get this from yogurt & ice cream too ....... pastuerized or not because it isn't killed by heat or cold (I'm 99% sure of this, I would have to hunt down the food handling book). in IL Quote Link to comment Share on other sites More sharing options...
Guest guest Posted May 13, 2003 Report Share Posted May 13, 2003 I've had cats all of my life and got tested during my third pregnancy for immunity to toxoplasmosis and was (not surprizingly) found immune. I've always washed my hands after messing with the litter box, in the garden, raw meats, etc. I think it just comes with the territory. )0(~~~)0(~~~)0( Mom to Brittany, born 8/31/93, dx'd-IDDM 5/28/01; , born 6/28/97; and Shayna, born 6/1/00. Before you vaccinate - http://www.vaccinetruth.org/ ICQ - 5368224 MSN - cerena_noir@... - jessica_anne010@... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted October 18, 2006 Report Share Posted October 18, 2006 I think they can make for attractive packaging, if that's the look you want. Quote Link to comment Share on other sites More sharing options...
Guest guest Posted October 18, 2006 Report Share Posted October 18, 2006 I use clamshells to package my soap petals. <xyz_akaine@...> wrote: What are your thoughts on using clamshells for packaging m & p soap as well as tarts? Quote Link to comment Share on other sites More sharing options...
Guest guest Posted October 18, 2006 Report Share Posted October 18, 2006 That's a neat idea. I like candles that use clamshells for the container. On 10/17/06, <xyz_akaine@...> wrote: > > What are your thoughts on using clamshells for packaging m & p soap as > well as tarts? > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted October 18, 2006 Report Share Posted October 18, 2006 I use the clamshells for the bars of glycerin soap the gentleman at work orders from me. He likes the fact that the soap can be popped out, used, put back in, etc. AND, he likes the ease of carrying them. I bought some a long while back, and haven't used many at all. Thought about using them to package small travel packs of BB items. Alice in SD Re: Your Thoughts I use clamshells to package my soap petals. <xyz_akaine@...> wrote: What are your thoughts on using clamshells for packaging m & p soap as well as tarts? Quote Link to comment Share on other sites More sharing options...
Guest guest Posted October 19, 2006 Report Share Posted October 19, 2006 > > What are your thoughts on using clamshells for packaging m & p soap as well as tarts? > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 26, 2007 Report Share Posted April 26, 2007 In a message dated 4/26/2007 4:39:14 P.M. Eastern Daylight Time, d.klaver@... writes: What questions should you ask someone who wants to make a 12 lb batch of soap? (Answer this before reading down farther) Sure, why not? If she has a formula and is using an eo/fo that she has success with, and calculates the amts. properly for her lye amt., then go for it. Of course, she'd need to be sure she had the equipment for that size batch to mix the soap in and so on. My biggest concern would be in selling soap for someone who's only been making it for a few months. There is a learning curve to making soap, and I can't imagine anyone being ready to sell it so soon. I know I'm overly cautious and such, but that's how it seems to me. Beth _www.soapandgarden.com_ (http://www.soapandgarden.com/) www.saponifier.com ************************************** See what's free at http://www.aol.com. Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 26, 2007 Report Share Posted April 26, 2007 Well, just run the formula thru the lye calculator, get your molds ready, in fact try to be ready for whatever situation comes along. Research whatever troubleshooting you need to know for whatever situation could come across, basically just be prepared. Sometimes soap can be finicky. Sometimes it seems each batch has its own mind. Everyone has had a first " larger " batch. It all adds to the experience we learn along the way. If you are going to scent this batch, I'd be sure to use a fo or eo that is not known to accelerate trace or cause problems. And be prepared that if this large batch of soap does not turn out well, be prepared to not sell it. If you sell someone a questionalbe bar of soap, do you think they will come back for a second ? To me that is just common sence. KWIM? Shaye your thoughts What questions should you ask someone who wants to make a 12 lb batch of soap? (Answer this before reading down farther) Note, the largest batch ever made by this person was 6 lbs, She researches her oils and fragrances she is however new to soap making. making soap for 3 months now. She is making soap and going to be selling it.. What are your thoughts and questions? D'Ann Quote Link to comment Share on other sites More sharing options...
Guest guest Posted April 26, 2007 Report Share Posted April 26, 2007 lol, nope cause I wouldn't come back if I got bad lotion from someone or anything else! Thank you., D'Ann On 4/26/07, Shaye <shaye@...> wrote: > > Well, just run the formula thru the lye calculator, get your molds ready, > in > fact try to be ready for whatever situation comes along. Research > whatever > troubleshooting you need to know for whatever situation could come across, > basically just be prepared. Sometimes soap can be finicky. Sometimes it > seems each batch has its own mind. Everyone has had a first " larger " > batch. It all adds to the experience we learn along the way. If you are > going to scent this batch, I'd be sure to use a fo or eo that is not known > to accelerate trace or cause problems. And be prepared that if this large > batch of soap does not turn out well, be prepared to not sell it. If you > sell someone a questionalbe bar of soap, do you think they will come back > for a second ? To me that is just common sence. KWIM? > > Shaye > > > > > > your thoughts > > > > > What questions should you ask someone who wants to make a 12 lb batch of > soap? (Answer this before reading down farther) > > Note, the largest batch ever made by this person was 6 lbs, > > She researches her oils and fragrances > > she is however new to soap making. making soap for 3 months now. She > is making soap and going to be selling it.. > > What are your thoughts and questions? > > D'Ann > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 17, 2008 Report Share Posted August 17, 2008 You're thoughts are not morbid to me.......my thoughts have run side by side with your thoughts just like you have said in your email. This "thing" has to be something new. I am a strong person, I'm not even allergic to poison ivy. I have slept in barns to wait for baby calves to be born. Never even knew I had skin until this mess hit. The little animals are being stricken with something and now it has passed from them to us. I am mad..............mad as hell............we can't get help....being told we are crazy..........like I have said before....just as soon as I pick up on this train of thought from a person I'm trying to get help from I sit in their chair and I shake their hand as long as I can. I can understand why a person without this on their skin can't understand what it is, but to turn people away who are in pain, who are visually suffering and send them home with nothing more than a pill to ease their nerves..........well..........it's in human treatment. A doctor who lacks doubt is not a doctor, he is an executioner. Rita Oh, By The Way-> > > Has anybody written to Dr. Phil to educate him and his staff about the> actual, physical aspects of this affliction? And to let him know that> it is increasing to epidemic proportions at warp speed?> > I have gone to his website, www.drphil.com and have sent him three> different emails of educational material on the subject.> > It would help a whole lot if everyone on this list would send him > their own three messages illuminating his to this horrible blight.> > Willow> Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 17, 2008 Report Share Posted August 17, 2008 I just sent two of those sites to Dr. Phil. I use a different choice for who to contact, each time. That's five now. And Oprah is next on my list! w. > > > > I will send info to him also. In the summer of 2006 I contacted > ever environmental group I could think of. I have a background in > working on environmental issues and have even worked with Greenpeace. > Not one group that I contacted had heard about this and even with " my > pull " I couldn't get any of them interested in looking into this > since we are seeing so many birds just dropping out of the sky (which > happened in Austin Texas a year ago) with no obvious signs of health > problems other than a heavy load of parasites. Rita > > > > http://cbs11tv.com/national/Austin.dead.birds.2.278736.html > > > > http://www.news.com.au/comments/0,23600,21035741-2,00.html. > > > > > > http://onlynow99.newsvine.com/_news/2007/12/22/1181344-flock-of- > birds-mysteriously-die-in-staten-island > > > > Squirrels loosing hair: http://www.phototour.minneapolis.mn.us/2571 > > > > Hair loss on deer from sub-genus of lice from Africa: > http://www.phototour.minneapolis.mn.us/2571 > > > > Bears loosing hair: http://www.usatoday.com/news/nation/2002-12-11- > bald-bears_x.htm > > > > What on earth is going on?????? > > > > > > > > > > > > > > Oh, By The Way- > > > > > > Has anybody written to Dr. Phil to educate him and his staff > about the > > actual, physical aspects of this affliction? And to let him know > that > > it is increasing to epidemic proportions at warp speed? > > > > I have gone to his website, www.drphil.com and have sent him three > > different emails of educational material on the subject. > > > > It would help a whole lot if everyone on this list would send him > > their own three messages illuminating his to this horrible blight. > > > > Willow > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted July 13, 2009 Report Share Posted July 13, 2009 What are these MRI's saying? Surgery?  Neck  The bone marrow signal is normal. There is evidence of prior fusion at C6-C7. The signal though out the spinal cord is normal. The brain stem and and cerebellum do not demonstrate any significant abnormalities. There are at least two lesions of high T2 signal within the posterior nasopharynx midline consistent with Thornwaldt cysts.  C2, C3, and C4 are normal  C4-C5- Bilateral uncoarthrosis is present with accompanying disc bulging. There is complete attenuation of the anterior subarachnoid space and mild effacement of the spinal cord. There is mild to moderate narrowing of the right neural foramina. There is no significant narrowing seen on the left.  C5-C6- There is a small central disc protrusion present with attenuation of the anterior subaracanoid space. There is no central cord compression. The neural foramina are widely patent.  C6-C7- There has been a prior fusion. There is posterior convexity to the bone at the level of the fusion with attenuation of the anterior subarachnoid space but no cord compression.   C7-T1- Broad-based disc extrusion is present at this level extending partially posterior to the T1 vertebral body. There is also bilateral uncoarthrosis, right more than left. There is mild attenuation of the anterior subarachnoid space but no cord compression. There is no significant left-sided neural foraminal stenosis. Mild right-sided neural foraminal stenosis is present.  Multilevel spondylitic changes as described above. Mild effacement of the anterior spinal cord is present at C4-C5 due to uncovertebral hypertrophy and disc bulging. Multlevel right-sided neural foraminal narrowing is also present.  Thornwald cysts within the posterior nasopharynx.   Upper Back   * C7-T1- Diffuse annular bulging, mild endplate spondylosis, mild ventral thecal sac contour deformity without cord impingement or compressive foraminal narrowing  T1-2, T2-3, and T3-4- minimal facet arthrosis  * T4-5 Minor annular bulging and endplate spondylosis. Moderate bilateral facet arthrosis with a low signal intensity structure in the posterior midline spinal canal potentially representing focally prominent facet-ligamentous complex spur, intruding into the spinal canal with a mild deformity of the posterior thecal sac contour, without direct cord impingement or significant overall canal stenosis,  T5-6 and T6-7- Minimal annular bulging, endplate spondylosis and facet arthrosis without neural impingement.  T7-8 Mild disc space narrowing with annular bulging and endplate spondylosis with right paracentral disc and spur complex which contacts and very minimally deforms the right ventral cord contour. Thecal sac posterior to the spinal cord remains capacious without overall central canal stenosis. There is no substantial facet arthrosis or intervertebral neural foraninal narrowing.  T8-9 Mild circumferential annular bulging and endplate spondylosis with a shallow posterior central disc and spur complex which results in minimal flattening of the ventral cord contour, without overall central canal stenosis.  T9-10 Mild disc space narrowing with predominantly ventral bulging and endplate spondylosis with mild facet arthrosis, without neural impingement.  T10-11- Minimal ventral bulging and endplate spondylosis with mild to moderate facet arthrosis, without neural impingement.  T11-12 and T12-L1- Bilateral facet arthrosis, without additional significant abnormality, allowing for suspected minimal artifact propagating across the thoracic spinal cord, no definite thoracic intramedullary cord pathology is seen. Evident on the sagittal loclizing sequence of the cervical spine is loss of the normal cervical lordosis with either postsurgical or congenital fusion at C6-7. This could be further evaluated with cervical spine MRI if clinically indicated. There is potential borderline cerebellar tonsillar ectopia. (Also known as Chiari) http://www.asap.org/chiari-malformation.html  Sagittal sequences disclose adequate alignment of thoracic vertebral bodies. There are mild multilevel discogenic endplate changes most pronounce at T8-9 and T9-10 where there is mild alteration of anterior endplate signal intensity with minimal fatty infiltration as well as mild wedge deformities ot the T8 and T9 vertebrae.  Mild thoracic scolosis convex to the left.  Multilevel thoracic degenerative disc disease and spondylosis is present as outline dequentially above, with disc bulges and or shallow protrusions in conjunction with endplate spondylosis and varying degrees of facet arthrosis noted to result in mild flattening of the ventral cord contour at the T7-8 and T8-9. Presumed posterior midline facet/ligamentous complex hypertrophy at T4-5.  Lower back Mild lumbar scoliosis L1-2 Minimal ventral endplate spondylosis Suspected mild facet arthrosis primarily on the left at T 11-12 without the evidence of neural impingement. L2-3 Minimal ventral annular bulging and endplate spondylosis L3-4 Reduced disc hydration, minor annular bulging, shallow noncompressive right lateral disc protusion L4-5 Reduced disc hydration, mild circumferential annular bulging with curvilinear T2 signal hyperintensity along the right posterolateral disc margin compatible with a small annular tear. Minimal endplate spondylosis and mild facet arthrosis. Very mild bilateral foraminal comprimise L5-S1-Smoothly marginated rounded water signal intensity structure measuring approximately 2 cm in diameter situated in the right hemipelvis, most compatible with a small physiologic adnexal cyst.  Mild multilevel noncompressive lumbar degenerative disc disease and spondylosis as present as outlined sequentially above, with mild lumbar levoscoliosis. Shallow noncompressive right lateral disc protusion is noted at L3-4, and there is an annular tear along the right posterolateral L4-5 disc margin. Central canal diameter remains capacious and there is no evidence of lateral recess stenosis or compressive intervertebral neural foraminal narrowing.   Quote Link to comment Share on other sites More sharing options...
Guest guest Posted July 13, 2009 Report Share Posted July 13, 2009 I don't only care about fucking...but thats all you give me. And shoot me for having a fucking sex drive! ________________________________ From: IAMTHEKEY() <shiningeyes3@...> spinal problems Sent: Sunday, July 12, 2009 10:17:39 PM Subject: Your thoughts What are these MRI's saying? Surgery?  Neck  The bone marrow signal is normal. There is evidence of prior fusion at C6-C7. The signal though out the spinal cord is normal. The brain stem and and cerebellum do not demonstrate any significant abnormalities. There are at least two lesions of high T2 signal within the posterior nasopharynx midline consistent with Thornwaldt cysts.  C2, C3, and C4 are normal  C4-C5- Bilateral uncoarthrosis is present with accompanying disc bulging. There is complete attenuation of the anterior subarachnoid space and mild effacement of the spinal cord. There is mild to moderate narrowing of the right neural foramina. There is no significant narrowing seen on the left.  C5-C6- There is a small central disc protrusion present with attenuation of the anterior subaracanoid space. There is no central cord compression. The neural foramina are widely patent.  C6-C7- There has been a prior fusion. There is posterior convexity to the bone at the level of the fusion with attenuation of the anterior subarachnoid space but no cord compression.   C7-T1- Broad-based disc extrusion is present at this level extending partially posterior to the T1 vertebral body. There is also bilateral uncoarthrosis, right more than left. There is mild attenuation of the anterior subarachnoid space but no cord compression. There is no significant left-sided neural foraminal stenosis. Mild right-sided neural foraminal stenosis is present.  Multilevel spondylitic changes as described above. Mild effacement of the anterior spinal cord is present at C4-C5 due to uncovertebral hypertrophy and disc bulging. Multlevel right-sided neural foraminal narrowing is also present.  Thornwald cysts within the posterior nasopharynx.   Upper Back   * C7-T1- Diffuse annular bulging, mild endplate spondylosis, mild ventral thecal sac contour deformity without cord impingement or compressive foraminal narrowing  T1-2, T2-3, and T3-4- minimal facet arthrosis  * T4-5 Minor annular bulging and endplate spondylosis. Moderate bilateral facet arthrosis with a low signal intensity structure in the posterior midline spinal canal potentially representing focally prominent facet-ligamentous complex spur, intruding into the spinal canal with a mild deformity of the posterior thecal sac contour, without direct cord impingement or significant overall canal stenosis,  T5-6 and T6-7- Minimal annular bulging, endplate spondylosis and facet arthrosis without neural impingement.  T7-8 Mild disc space narrowing with annular bulging and endplate spondylosis with right paracentral disc and spur complex which contacts and very minimally deforms the right ventral cord contour. Thecal sac posterior to the spinal cord remains capacious without overall central canal stenosis. There is no substantial facet arthrosis or intervertebral neural foraninal narrowing.  T8-9 Mild circumferential annular bulging and endplate spondylosis with a shallow posterior central disc and spur complex which results in minimal flattening of the ventral cord contour, without overall central canal stenosis.  T9-10 Mild disc space narrowing with predominantly ventral bulging and endplate spondylosis with mild facet arthrosis, without neural impingement.  T10-11- Minimal ventral bulging and endplate spondylosis with mild to moderate facet arthrosis, without neural impingement.  T11-12 and T12-L1- Bilateral facet arthrosis, without additional significant abnormality, allowing for suspected minimal artifact propagating across the thoracic spinal cord, no definite thoracic intramedullary cord pathology is seen. Evident on the sagittal loclizing sequence of the cervical spine is loss of the normal cervical lordosis with either postsurgical or congenital fusion at C6-7. This could be further evaluated with cervical spine MRI if clinically indicated. There is potential borderline cerebellar tonsillar ectopia. (Also known as Chiari) http://www.asap. org/chiari- malformation. html  Sagittal sequences disclose adequate alignment of thoracic vertebral bodies. There are mild multilevel discogenic endplate changes most pronounce at T8-9 and T9-10 where there is mild alteration of anterior endplate signal intensity with minimal fatty infiltration as well as mild wedge deformities ot the T8 and T9 vertebrae.  Mild thoracic scolosis convex to the left.  Multilevel thoracic degenerative disc disease and spondylosis is present as outline dequentially above, with disc bulges and or shallow protrusions in conjunction with endplate spondylosis and varying degrees of facet arthrosis noted to result in mild flattening of the ventral cord contour at the T7-8 and T8-9. Presumed posterior midline facet/ligamentous complex hypertrophy at T4-5.  Lower back Mild lumbar scoliosis L1-2 Minimal ventral endplate spondylosis Suspected mild facet arthrosis primarily on the left at T 11-12 without the evidence of neural impingement. L2-3 Minimal ventral annular bulging and endplate spondylosis L3-4 Reduced disc hydration, minor annular bulging, shallow noncompressive right lateral disc protusion L4-5 Reduced disc hydration, mild circumferential annular bulging with curvilinear T2 signal hyperintensity along the right posterolateral disc margin compatible with a small annular tear. Minimal endplate spondylosis and mild facet arthrosis. Very mild bilateral foraminal comprimise L5-S1-Smoothly marginated rounded water signal intensity structure measuring approximately 2 cm in diameter situated in the right hemipelvis, most compatible with a small physiologic adnexal cyst.  Mild multilevel noncompressive lumbar degenerative disc disease and spondylosis as present as outlined sequentially above, with mild lumbar levoscoliosis. Shallow noncompressive right lateral disc protusion is noted at L3-4, and there is an annular tear along the right posterolateral L4-5 disc margin. Central canal diameter remains capacious and there is no evidence of lateral recess stenosis or compressive intervertebral neural foraminal narrowing.   Quote Link to comment Share on other sites More sharing options...
Guest guest Posted July 13, 2009 Report Share Posted July 13, 2009 oops...big boo boo....someone please delete this for me! I dont know how! m: IAMTHEKEY() <shiningeyes3@...> > spinal problems > Sent: Sunday, July 12, 2009 10:17:39 PM > Subject: Your thoughts > > > > > > What are these MRI's saying? Surgery? >  > Neck >  > The bone marrow signal is normal. There is evidence of prior fusion at C6-C7. The signal though out the spinal cord is normal. The brain stem and and cerebellum do not demonstrate any significant abnormalities. There are at least two lesions of high T2 signal within the posterior nasopharynx midline consistent with Thornwaldt cysts. >  > C2, C3, and C4 are normal >  > C4-C5- Bilateral uncoarthrosis is present with accompanying disc bulging. There is complete attenuation of the anterior subarachnoid space and mild effacement of the spinal cord. There is mild to moderate narrowing of the right neural foramina. There is no significant narrowing seen on the left. >  > C5-C6- There is a small central disc protrusion present with attenuation of the anterior subaracanoid space. There is no central cord compression. The neural foramina are widely patent. >  > C6-C7- There has been a prior fusion. There is posterior convexity to the bone at the level of the fusion with attenuation of the anterior subarachnoid space but no cord compression. >  >  > C7-T1- Broad-based disc extrusion is present at this level extending partially posterior to the T1 vertebral body. There is also bilateral uncoarthrosis, right more than left. There is mild attenuation of the anterior subarachnoid space but no cord compression. There is no significant left-sided neural foraminal stenosis. Mild right-sided neural foraminal stenosis is present. >  > Multilevel spondylitic changes as described above. Mild effacement of the anterior spinal cord is present at C4-C5 due to uncovertebral hypertrophy and disc bulging. Multlevel right-sided neural foraminal narrowing is also present. >  > Thornwald cysts within the posterior nasopharynx. >  >  > Upper Back >  >  > * C7-T1- Diffuse annular bulging, mild endplate spondylosis, mild ventral thecal sac contour deformity without cord impingement or compressive foraminal narrowing >  > T1-2, T2-3, and T3-4- minimal facet arthrosis >  > * T4-5 Minor annular bulging and endplate spondylosis. Moderate bilateral facet arthrosis with a low signal intensity structure in the posterior midline spinal canal potentially representing focally prominent facet-ligamentous complex spur, intruding into the spinal canal with a mild deformity of the posterior thecal sac contour, without direct cord impingement or significant overall canal stenosis, >  > T5-6 and T6-7- Minimal annular bulging, endplate spondylosis and facet arthrosis without neural impingement. >  > T7-8 Mild disc space narrowing with annular bulging and endplate spondylosis with right paracentral disc and spur complex which contacts and very minimally deforms the right ventral cord contour. Thecal sac posterior to the spinal cord remains capacious without overall central canal stenosis. There is no substantial facet arthrosis or intervertebral neural foraninal narrowing. >  > T8-9 Mild circumferential annular bulging and endplate spondylosis with a shallow posterior central disc and spur complex which results in minimal flattening of the ventral cord contour, without overall central canal stenosis. >  > T9-10 Mild disc space narrowing with predominantly ventral bulging and endplate spondylosis with mild facet arthrosis, without neural impingement. >  > T10-11- Minimal ventral bulging and endplate spondylosis with mild to moderate facet arthrosis, without neural impingement. >  > > T11-12 and T12-L1- Bilateral facet arthrosis, without additional significant abnormality, allowing for suspected minimal artifact propagating across the thoracic spinal cord, no definite thoracic intramedullary cord pathology is seen. Evident on the sagittal loclizing sequence of the cervical spine is loss of the normal cervical lordosis with either postsurgical or congenital fusion at C6-7. This could be further evaluated with cervical spine MRI if clinically indicated. There is potential borderline cerebellar tonsillar ectopia. > (Also known as Chiari) > http://www.asap. org/chiari- malformation. html >  > Sagittal sequences disclose adequate alignment of thoracic vertebral bodies. There are mild multilevel discogenic endplate changes most pronounce at T8-9 and T9-10 where there is mild alteration of anterior endplate signal intensity with minimal fatty infiltration as well as mild wedge deformities ot the T8 and T9 vertebrae.  Mild thoracic scolosis convex to the left. >  > Multilevel thoracic degenerative disc disease and spondylosis is present as outline dequentially above, with disc bulges and or shallow protrusions in conjunction with endplate spondylosis and varying degrees of facet arthrosis noted to result in mild flattening of the ventral cord contour at the T7-8 and T8-9. Presumed posterior midline facet/ligamentous complex hypertrophy at T4-5. >  > Lower back > Mild lumbar scoliosis > L1-2 Minimal ventral endplate spondylosis > Suspected mild facet arthrosis primarily on the left at T 11-12 without the evidence of neural impingement. > L2-3 Minimal ventral annular bulging and endplate spondylosis > L3-4 Reduced disc hydration, minor annular bulging, shallow noncompressive right lateral disc protusion > L4-5 Reduced disc hydration, mild circumferential annular bulging with curvilinear T2 signal hyperintensity along the right posterolateral disc margin compatible with a small annular tear. Minimal endplate spondylosis and mild facet arthrosis. Very mild bilateral foraminal comprimise > L5-S1-Smoothly marginated rounded water signal intensity structure measuring approximately 2 cm in diameter situated in the right hemipelvis, most compatible with a small physiologic adnexal cyst. >  > Mild multilevel noncompressive lumbar degenerative disc disease and spondylosis as present as outlined sequentially above, with mild lumbar levoscoliosis. Shallow noncompressive right lateral disc protusion is noted at L3-4, and there is an annular tear along the right posterolateral L4-5 disc margin. Central canal diameter remains capacious and there is no evidence of lateral recess stenosis or compressive intervertebral neural foraminal narrowing. >  > >  > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted July 15, 2009 Report Share Posted July 15, 2009 Ha ha...you're awesome Deb! We should chat about our problem sometime... ________________________________ From: Deb <i_ownaberner@...> spinal problems Sent: Tuesday, July 14, 2009 12:37:51 PM Subject: RE: Re: Your thoughts . I got the same problem you have..wink wink..so shoot both of us.nothing to be embaressed about..we all f up sometimes.. Deb From: spinedisorderssuppo rtgroup@gro ups.com [mailto:spinedisorderssuppo rtgroup@gro ups.com] On Behalf Of Sent: Sunday, July 12, 2009 11:10 PM spinedisorderssuppo rtgroup@gro ups.com Subject: Re: Your thoughts oops...big boo boo....someone please delete this for me! I dont know how! m: IAMTHEKEY( ) <shiningeyes3@ ...> > spinedisorderssuppo rtgroup@gro ups.com <mailto:spinedisord erssupportgroup% 40groups. com> > Sent: Sunday, July 12, 2009 10:17:39 PM > Subject: Your thoughts > > > > > > What are these MRI's saying? Surgery? > > Neck > > The bone marrow signal is normal. There is evidence of prior fusion at C6-C7. The signal though out the spinal cord is normal. The brain stem and and cerebellum do not demonstrate any significant abnormalities. There are at least two lesions of high T2 signal within the posterior nasopharynx midline consistent with Thornwaldt cysts. > > C2, C3, and C4 are normal > > C4-C5- Bilateral uncoarthrosis is present with accompanying disc bulging. There is complete attenuation of the anterior subarachnoid space and mild effacement of the spinal cord. There is mild to moderate narrowing of the right neural foramina. There is no significant narrowing seen on the left. > > C5-C6- There is a small central disc protrusion present with attenuation of the anterior subaracanoid space. There is no central cord compression. The neural foramina are widely patent. > > C6-C7- There has been a prior fusion. There is posterior convexity to the bone at the level of the fusion with attenuation of the anterior subarachnoid space but no cord compression. > > > C7-T1- Broad-based disc extrusion is present at this level extending partially posterior to the T1 vertebral body. There is also bilateral uncoarthrosis, right more than left. There is mild attenuation of the anterior subarachnoid space but no cord compression. There is no significant left-sided neural foraminal stenosis. Mild right-sided neural foraminal stenosis is present. > > Multilevel spondylitic changes as described above. Mild effacement of the anterior spinal cord is present at C4-C5 due to uncovertebral hypertrophy and disc bulging. Multlevel right-sided neural foraminal narrowing is also present. > > Thornwald cysts within the posterior nasopharynx. > > > Upper Back > > > * C7-T1- Diffuse annular bulging, mild endplate spondylosis, mild ventral thecal sac contour deformity without cord impingement or compressive foraminal narrowing > > T1-2, T2-3, and T3-4- minimal facet arthrosis > > * T4-5 Minor annular bulging and endplate spondylosis. Moderate bilateral facet arthrosis with a low signal intensity structure in the posterior midline spinal canal potentially representing focally prominent facet-ligamentous complex spur, intruding into the spinal canal with a mild deformity of the posterior thecal sac contour, without direct cord impingement or significant overall canal stenosis, > > T5-6 and T6-7- Minimal annular bulging, endplate spondylosis and facet arthrosis without neural impingement. > > T7-8 Mild disc space narrowing with annular bulging and endplate spondylosis with right paracentral disc and spur complex which contacts and very minimally deforms the right ventral cord contour. Thecal sac posterior to the spinal cord remains capacious without overall central canal stenosis. There is no substantial facet arthrosis or intervertebral neural foraninal narrowing. > > T8-9 Mild circumferential annular bulging and endplate spondylosis with a shallow posterior central disc and spur complex which results in minimal flattening of the ventral cord contour, without overall central canal stenosis. > > T9-10 Mild disc space narrowing with predominantly ventral bulging and endplate spondylosis with mild facet arthrosis, without neural impingement. > > T10-11- Minimal ventral bulging and endplate spondylosis with mild to moderate facet arthrosis, without neural impingement. > > > T11-12 and T12-L1- Bilateral facet arthrosis, without additional significant abnormality, allowing for suspected minimal artifact propagating across the thoracic spinal cord, no definite thoracic intramedullary cord pathology is seen. Evident on the sagittal loclizing sequence of the cervical spine is loss of the normal cervical lordosis with either postsurgical or congenital fusion at C6-7. This could be further evaluated with cervical spine MRI if clinically indicated. There is potential borderline cerebellar tonsillar ectopia. > (Also known as Chiari) > http://www.asap. org/chiari- malformation. html > > Sagittal sequences disclose adequate alignment of thoracic vertebral bodies. There are mild multilevel discogenic endplate changes most pronounce at T8-9 and T9-10 where there is mild alteration of anterior endplate signal intensity with minimal fatty infiltration as well as mild wedge deformities ot the T8 and T9 vertebrae. Mild thoracic scolosis convex to the left. > > Multilevel thoracic degenerative disc disease and spondylosis is present as outline dequentially above, with disc bulges and or shallow protrusions in conjunction with endplate spondylosis and varying degrees of facet arthrosis noted to result in mild flattening of the ventral cord contour at the T7-8 and T8-9. Presumed posterior midline facet/ligamentous complex hypertrophy at T4-5. > > Lower back > Mild lumbar scoliosis > L1-2 Minimal ventral endplate spondylosis > Suspected mild facet arthrosis primarily on the left at T 11-12 without the evidence of neural impingement. > L2-3 Minimal ventral annular bulging and endplate spondylosis > L3-4 Reduced disc hydration, minor annular bulging, shallow noncompressive right lateral disc protusion > L4-5 Reduced disc hydration, mild circumferential annular bulging with curvilinear T2 signal hyperintensity along the right posterolateral disc margin compatible with a small annular tear. Minimal endplate spondylosis and mild facet arthrosis. Very mild bilateral foraminal comprimise > L5-S1-Smoothly marginated rounded water signal intensity structure measuring approximately 2 cm in diameter situated in the right hemipelvis, most compatible with a small physiologic adnexal cyst. > > Mild multilevel noncompressive lumbar degenerative disc disease and spondylosis as present as outlined sequentially above, with mild lumbar levoscoliosis. Shallow noncompressive right lateral disc protusion is noted at L3-4, and there is an annular tear along the right posterolateral L4-5 disc margin. Central canal diameter remains capacious and there is no evidence of lateral recess stenosis or compressive intervertebral neural foraminal narrowing. > > > > > Quote Link to comment Share on other sites More sharing options...
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