Re: Fw: really scared now(was this all from lymes??)

[Guest guest]

oooh my the more i read the more scared i get dang i in 1995 ended up having my chest sawed in half basic open heart surgery but was not for my heart i supposedly had a my thumus gland that is productive for us as children then goes away approx of the age of 18 well mine was still with me and it developed a cyst on it.. well this is what i was told that i had a large mass that attatched to the right lung ,sternum and the superior venacava of the heart i was in intesive care for 3 days they thought i was dying my heart rate was 46 had tubes in my lungs and chest was a big big mess then about a year later my right lung started to feel like i was carrying a huge brick with me it was so heavy i had almost no lung capacity mine was 42 the avg lung capacity is 80% i cant remeber the rest of it but i was treated for severe asthma that i never never had in my life put on steroids 3 inhalers several pulmony function tests done my doctor made specials runs for me to inject me with steroids cause i could not breathe the last year my lung has not felt heavy i feel like i can breathe my doctor was really scared for me... but with his kind and gentle bedside manor we passed that i still see an alergist i am over due but i have not needed my inhalers is this all a lymes disease thing how terrifying i have been through so much and am still not well but i am getting better slowly

maybe i just opened a pandora box what do you MD's think??? am i way off line with this

[ ] Fw: [actionld] Spirochete Borrelia crocidurae

I did not send this whole citation because it is so long. (First link below.) It has many pictures of Borrelia in lungs, kidneys and barin of mice. But I hope some can view these slides of Borrelia in the Kidneys that are included in this e-mail. This article is really something. I don't know what kind of Borrelia we have in us, but if anything like what is in this citation, well no wonder some of us are so ill. I hope you have been able to go to site and get this information. Can anyone tell us we do have this kind of spirochete doing damage to our bodies?

Barb

--------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------

http://www.journals.uchicago.edu/JID/journal/issues/v180n6/990211/990211.html

The Journal of Infectious Diseases 1999;180:1929-1938© 1999 by the Infectious Diseases Society of America. All rights reserved.0022-1899/1999/18006-0023$02.00

Erythrocyte-Aggregating Relapsing Fever Spirochete Borrelia crocidurae Induces Formation of Microemboli

Alireza Shamaei-Tousi,1 Pierre ,1 Anders Bergh,2 Nils Burman,1 Brännström,2 and Sven Bergström1

Departments of 1Microbiology and 2Pathology, Umeå University, Umeå, Sweden

Received 22 February 1999; revised 16 August 1999; electronically published 29 October 1999.

The African relapsing fever spirochete Borrelia crocidurae forms aggregates with erythrocytes, resulting in a delayed immune response. Mice were infected with B. crocidurae and monitored during 50 days after infection. Spirochetes were observed extravascularly at day 2 after infection. Two days later, inflammatory responses, cell death, and tissue damage were evident. The pathologic responses in lungs and kidneys were similar, whereas the symptoms in the brains were delayed, with a less pronounced inflammatory response. Microemboli were found in the blood vessels, possibly a result of the erythrocyte aggregation. The B. crocidurae invasion emerged more rapidly than has been described for Lyme diseasecausing Borrelia species. In addition to erythrocyte rosetting, the presence of extravascular B. crocidurae indicates a novel route for these bacteria to propagate and cause damage in the mammalian host. The histopathologic findings in this study may explain the clinical manifestations of human relapsing fever.

http://www.journals.uchicago.edu/JID/journal/issues/v180n6/990211/990211.html

Figure 4. Histopathology in kidney of mice infected with Borrelia crocidurae. A, B, Spirochetes associated with erythrocytes in blood vessel of kidney at day 4 after infection detected by Cy3 fluorescence anti-flagellin antibody. Identification of blood vessels was done by light microscopy. C, Extravascular spirochetes outside of Bowman's capsule (arrows), day 3 after infection. D, Hemorrhagic medulla at day 7 after infection. E, Retention of erythrocytes in capillary of perfusion-fixed kidney, day 5 after infection. F, Fragmented parenchymatous cells in kidney (arrow) 7 days after infection. Stains: C, modified Dieterle's silver impregnation; E, F, hematoxylin-eosin; bars, 20 m.

[Guest guest]

oooh my the more i read the more scared i get dang i in 1995 ended up having my chest sawed in half basic open heart surgery but was not for my heart i supposedly had a my thumus gland that is productive for us as children then goes away approx of the age of 18 well mine was still with me and it developed a cyst on it.. well this is what i was told that i had a large mass that attatched to the right lung ,sternum and the superior venacava of the heart i was in intesive care for 3 days they thought i was dying my heart rate was 46 had tubes in my lungs and chest was a big big mess then about a year later my right lung started to feel like i was carrying a huge brick with me it was so heavy i had almost no lung capacity mine was 42 the avg lung capacity is 80% i cant remeber the rest of it but i was treated for severe asthma that i never never had in my life put on steroids 3 inhalers several pulmony function tests done my doctor made specials runs for me to inject me with steroids cause i could not breathe the last year my lung has not felt heavy i feel like i can breathe my doctor was really scared for me... but with his kind and gentle bedside manor we passed that i still see an alergist i am over due but i have not needed my inhalers is this all a lymes disease thing how terrifying i have been through so much and am still not well but i am getting better slowly

maybe i just opened a pandora box what do you MD's think??? am i way off line with this

cindy in mich

[ ] Fw: [actionld] Spirochete Borrelia crocidurae

I did not send this whole citation because it is so long. (First link below.) It has many pictures of Borrelia in lungs, kidneys and barin of mice. But I hope some can view these slides of Borrelia in the Kidneys that are included in this e-mail. This article is really something. I don't know what kind of Borrelia we have in us, but if anything like what is in this citation, well no wonder some of us are so ill. I hope you have been able to go to site and get this information. Can anyone tell us we do have this kind of spirochete doing damage to our bodies?

Barb

--------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------

http://www.journals.uchicago.edu/JID/journal/issues/v180n6/990211/990211.html

The Journal of Infectious Diseases 1999;180:1929-1938© 1999 by the Infectious Diseases Society of America. All rights reserved.0022-1899/1999/18006-0023$02.00

Erythrocyte-Aggregating Relapsing Fever Spirochete Borrelia crocidurae Induces Formation of Microemboli

Alireza Shamaei-Tousi,1 Pierre ,1 Anders Bergh,2 Nils Burman,1 Brännström,2 and Sven Bergström1

Departments of 1Microbiology and 2Pathology, Umeå University, Umeå, Sweden

Received 22 February 1999; revised 16 August 1999; electronically published 29 October 1999.

The African relapsing fever spirochete Borrelia crocidurae forms aggregates with erythrocytes, resulting in a delayed immune response. Mice were infected with B. crocidurae and monitored during 50 days after infection. Spirochetes were observed extravascularly at day 2 after infection. Two days later, inflammatory responses, cell death, and tissue damage were evident. The pathologic responses in lungs and kidneys were similar, whereas the symptoms in the brains were delayed, with a less pronounced inflammatory response. Microemboli were found in the blood vessels, possibly a result of the erythrocyte aggregation. The B. crocidurae invasion emerged more rapidly than has been described for Lyme diseasecausing Borrelia species. In addition to erythrocyte rosetting, the presence of extravascular B. crocidurae indicates a novel route for these bacteria to propagate and cause damage in the mammalian host. The histopathologic findings in this study may explain the clinical manifestations of human relapsing fever.

http://www.journals.uchicago.edu/JID/journal/issues/v180n6/990211/990211.html

Figure 4. Histopathology in kidney of mice infected with Borrelia crocidurae. A, B, Spirochetes associated with erythrocytes in blood vessel of kidney at day 4 after infection detected by Cy3 fluorescence anti-flagellin antibody. Identification of blood vessels was done by light microscopy. C, Extravascular spirochetes outside of Bowman's capsule (arrows), day 3 after infection. D, Hemorrhagic medulla at day 7 after infection. E, Retention of erythrocytes in capillary of perfusion-fixed kidney, day 5 after infection. F, Fragmented parenchymatous cells in kidney (arrow) 7 days after infection. Stains: C, modified Dieterle's silver impregnation; E, F, hematoxylin-eosin; bars, 20 m.

[Guest guest]

oooh my the more i read the more scared i get dang i in 1995 ended up having my chest sawed in half basic open heart surgery but was not for my heart i supposedly had a my thumus gland that is productive for us as children then goes away approx of the age of 18 well mine was still with me and it developed a cyst on it.. well this is what i was told that i had a large mass that attatched to the right lung ,sternum and the superior venacava of the heart i was in intesive care for 3 days they thought i was dying my heart rate was 46 had tubes in my lungs and chest was a big big mess then about a year later my right lung started to feel like i was carrying a huge brick with me it was so heavy i had almost no lung capacity mine was 42 the avg lung capacity is 80% i cant remeber the rest of it but i was treated for severe asthma that i never never had in my life put on steroids 3 inhalers several pulmony function tests done my doctor made specials runs for me to inject me with steroids cause i could not breathe the last year my lung has not felt heavy i feel like i can breathe my doctor was really scared for me... but with his kind and gentle bedside manor we passed that i still see an alergist i am over due but i have not needed my inhalers is this all a lymes disease thing how terrifying i have been through so much and am still not well but i am getting better slowly

maybe i just opened a pandora box what do you MD's think??? am i way off line with this

[ ] Fw: [actionld] Spirochete Borrelia crocidurae

I did not send this whole citation because it is so long. (First link below.) It has many pictures of Borrelia in lungs, kidneys and barin of mice. But I hope some can view these slides of Borrelia in the Kidneys that are included in this e-mail. This article is really something. I don't know what kind of Borrelia we have in us, but if anything like what is in this citation, well no wonder some of us are so ill. I hope you have been able to go to site and get this information. Can anyone tell us we do have this kind of spirochete doing damage to our bodies?

Barb

--------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------

http://www.journals.uchicago.edu/JID/journal/issues/v180n6/990211/990211.html

The Journal of Infectious Diseases 1999;180:1929-1938© 1999 by the Infectious Diseases Society of America. All rights reserved.0022-1899/1999/18006-0023$02.00

Erythrocyte-Aggregating Relapsing Fever Spirochete Borrelia crocidurae Induces Formation of Microemboli

Alireza Shamaei-Tousi,1 Pierre ,1 Anders Bergh,2 Nils Burman,1 Brännström,2 and Sven Bergström1

Departments of 1Microbiology and 2Pathology, Umeå University, Umeå, Sweden

Received 22 February 1999; revised 16 August 1999; electronically published 29 October 1999.

The African relapsing fever spirochete Borrelia crocidurae forms aggregates with erythrocytes, resulting in a delayed immune response. Mice were infected with B. crocidurae and monitored during 50 days after infection. Spirochetes were observed extravascularly at day 2 after infection. Two days later, inflammatory responses, cell death, and tissue damage were evident. The pathologic responses in lungs and kidneys were similar, whereas the symptoms in the brains were delayed, with a less pronounced inflammatory response. Microemboli were found in the blood vessels, possibly a result of the erythrocyte aggregation. The B. crocidurae invasion emerged more rapidly than has been described for Lyme diseasecausing Borrelia species. In addition to erythrocyte rosetting, the presence of extravascular B. crocidurae indicates a novel route for these bacteria to propagate and cause damage in the mammalian host. The histopathologic findings in this study may explain the clinical manifestations of human relapsing fever.

http://www.journals.uchicago.edu/JID/journal/issues/v180n6/990211/990211.html

Figure 4. Histopathology in kidney of mice infected with Borrelia crocidurae. A, B, Spirochetes associated with erythrocytes in blood vessel of kidney at day 4 after infection detected by Cy3 fluorescence anti-flagellin antibody. Identification of blood vessels was done by light microscopy. C, Extravascular spirochetes outside of Bowman's capsule (arrows), day 3 after infection. D, Hemorrhagic medulla at day 7 after infection. E, Retention of erythrocytes in capillary of perfusion-fixed kidney, day 5 after infection. F, Fragmented parenchymatous cells in kidney (arrow) 7 days after infection. Stains: C, modified Dieterle's silver impregnation; E, F, hematoxylin-eosin; bars, 20 m.

[Guest guest]

oooh my the more i read the more scared i get dang i in 1995 ended up having my chest sawed in half basic open heart surgery but was not for my heart i supposedly had a my thumus gland that is productive for us as children then goes away approx of the age of 18 well mine was still with me and it developed a cyst on it.. well this is what i was told that i had a large mass that attatched to the right lung ,sternum and the superior venacava of the heart i was in intesive care for 3 days they thought i was dying my heart rate was 46 had tubes in my lungs and chest was a big big mess then about a year later my right lung started to feel like i was carrying a huge brick with me it was so heavy i had almost no lung capacity mine was 42 the avg lung capacity is 80% i cant remeber the rest of it but i was treated for severe asthma that i never never had in my life put on steroids 3 inhalers several pulmony function tests done my doctor made specials runs for me to inject me with steroids cause i could not breathe the last year my lung has not felt heavy i feel like i can breathe my doctor was really scared for me... but with his kind and gentle bedside manor we passed that i still see an alergist i am over due but i have not needed my inhalers is this all a lymes disease thing how terrifying i have been through so much and am still not well but i am getting better slowly

maybe i just opened a pandora box what do you MD's think??? am i way off line with this

cindy in mich

[ ] Fw: [actionld] Spirochete Borrelia crocidurae

I did not send this whole citation because it is so long. (First link below.) It has many pictures of Borrelia in lungs, kidneys and barin of mice. But I hope some can view these slides of Borrelia in the Kidneys that are included in this e-mail. This article is really something. I don't know what kind of Borrelia we have in us, but if anything like what is in this citation, well no wonder some of us are so ill. I hope you have been able to go to site and get this information. Can anyone tell us we do have this kind of spirochete doing damage to our bodies?

Barb

--------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------

http://www.journals.uchicago.edu/JID/journal/issues/v180n6/990211/990211.html

The Journal of Infectious Diseases 1999;180:1929-1938© 1999 by the Infectious Diseases Society of America. All rights reserved.0022-1899/1999/18006-0023$02.00

Erythrocyte-Aggregating Relapsing Fever Spirochete Borrelia crocidurae Induces Formation of Microemboli

Alireza Shamaei-Tousi,1 Pierre ,1 Anders Bergh,2 Nils Burman,1 Brännström,2 and Sven Bergström1

Departments of 1Microbiology and 2Pathology, Umeå University, Umeå, Sweden

Received 22 February 1999; revised 16 August 1999; electronically published 29 October 1999.

The African relapsing fever spirochete Borrelia crocidurae forms aggregates with erythrocytes, resulting in a delayed immune response. Mice were infected with B. crocidurae and monitored during 50 days after infection. Spirochetes were observed extravascularly at day 2 after infection. Two days later, inflammatory responses, cell death, and tissue damage were evident. The pathologic responses in lungs and kidneys were similar, whereas the symptoms in the brains were delayed, with a less pronounced inflammatory response. Microemboli were found in the blood vessels, possibly a result of the erythrocyte aggregation. The B. crocidurae invasion emerged more rapidly than has been described for Lyme diseasecausing Borrelia species. In addition to erythrocyte rosetting, the presence of extravascular B. crocidurae indicates a novel route for these bacteria to propagate and cause damage in the mammalian host. The histopathologic findings in this study may explain the clinical manifestations of human relapsing fever.

http://www.journals.uchicago.edu/JID/journal/issues/v180n6/990211/990211.html

Figure 4. Histopathology in kidney of mice infected with Borrelia crocidurae. A, B, Spirochetes associated with erythrocytes in blood vessel of kidney at day 4 after infection detected by Cy3 fluorescence anti-flagellin antibody. Identification of blood vessels was done by light microscopy. C, Extravascular spirochetes outside of Bowman's capsule (arrows), day 3 after infection. D, Hemorrhagic medulla at day 7 after infection. E, Retention of erythrocytes in capillary of perfusion-fixed kidney, day 5 after infection. F, Fragmented parenchymatous cells in kidney (arrow) 7 days after infection. Stains: C, modified Dieterle's silver impregnation; E, F, hematoxylin-eosin; bars, 20 m.