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http://www.charcot-marie-tooth.org/hnpp.htm

What is HNPP?

By Maureen Horton

Edited by Dr. Chance, the CMTA Medical Advisory Board

(Editor's note: The CMTA will be working with Maureen to keep our readership

updated on HNPP. She has HNPP and first noticed problems during her

pregnancies. She was finally diagnosed with HNPP and because information was

so difficult to find, she began to teach herself about the disorder. As a

nurse, Maureen offers our readers both the personal side of the disorder and

medical expertise on the subject.)

Hereditary neuropathy with liability to pressure palsies or HNPP is

associated with a deletion on chromosome 17p11.2-12. This is the same

chromosomal site where CMT1A has been found to have a duplication, and it

contains an important myelin gene, peripheral myelin protein-22 gene (PMP22).

Therefore, while CMT1A results from having an extra copy of PMP22, HNPP

results from the loss of a copy of PMP22.

HNPP is caused by an autosomal dominant gene. HNPP is also a demyelinating

form of neuropathy like CMT1A. However, the affected nerves in HNPP are

described as having focal regions with " tomaculous " changes or sausage shaped

figures, due to the irregular thickening of the myelin sheath.

HNPP causes episodes or periods of numbness and weakness, similar to an arm

or leg going to sleep. But, instead of lasting only a few seconds, as it does

in a normal person, this numbness and weakness can last from several minutes

to months. These episodes may be associated with fairly mild trauma

(stretching or pressure on the nerves) which would not ordinarily affect a

normal person, such as: leaning on elbows, crossing legs, cutting with

scissors, knitting, kneeling, sleeping in the wrong position, etc. It has

usually been described as a painless disorder, although some people do have

pain, which is probably a subjective sense of numbness. Most individuals with

HNPP have normal strength and sensation, unless experiencing an attack.

Typically, persons who have inherited HNPP will develop symptoms in their

teens or twenties, but individuals may be first symptomatic during childhood

or late adulthood. Like CMT, there is a wide range in how severely people are

affected. Many are so mild that they are unaware that they have a problem.

Others develop the symptoms of a more generalized hereditary motor and

sensory neuropathy, which on occasion may be misdiagnosed as a form of CMT.

Carpal tunnel syndrome is frequently seen in patients with HNPP.

As a hereditary neuropathy it is progressive, though to what extent it will

progress in any individual is unknown. There is no treatment or cure for

HNPP. People are usually advised to avoid activities which they have learned

cause symptoms to develop. Leg crossing and leaning on elbows is discouraged.

AFOs can be used during periods of acute peroneal nerve palsy or for

permanent foot drop and/or balance problems.

For more information on HNPP, please visit www.hnpp.org.

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In a message dated 2/11/01 11:06:16 AM, rmax@... writes:

<< >>>>>>>Kat so this says that HNPP is not a " form " of CMT? >>

" Therefore, while CMT1A results from having an extra copy of PMP22, HNPP

results from the loss of a copy of PMP22. "

They are genetic opposites but result in many symptoms in common. I've met

many HNPPers in the CMT chat room. As with CMT, we have many similar problems

as well as differences.

Some people think CMT types 1 and 2 may not actually be properly called CMT

for various reasons. For example: I don't have the usual atrophied calves or

extremely high arches, but I still have CMT type 2. Some HNPPers DO have

atrophied calves and/or extremely high arches.

Diagnosing is still in a state of flux and learning.

Kat

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In a message dated 2/11/2001 2:12:39 PM Eastern Standard Time,

lisarich.com@... writes:

<< I'm grateful you posted this, because I didn't really know what the

differences were. Thanks.

>>

As far as symptoms go, there aren't that many differences. Right?

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In a message dated 2/11/01 12:46:49 PM, lls@... writes:

<< One thing is certain with CMT, HNPP, HMSN, or whatever-- " We are all

connected " >>

I agree completely, but I don't think the medical community is finished

fine-tuning the labels. Some types of CMT were formerly called other names,

for example. Dorothy Gosling, RN, is the person who got me thinking about

this. As with your family, they are still learning, and last I heard she

didn't fit just one catagory either.

I actually was trying to say we are indeed all connected, but there is more

to learn.

Kat

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It is my understanding that HNPP is the genetic opposite of only one

very specific type of CMT, that being CMT1A. Because there are so

many forms of CMT caused by so many identified AND unidentified

genes, and because so many CMTers CANNOT have their specific CMT type

identified because gene tests are not available for all types, why

not include HNPP in our discussion as well??? The problems and

coping solutions for HNPP are similar to the problems and coping

solutions for the very many different types of CMT.

Ruth Warren

> Just to clarify for anyone new to - this group is for

people

> and their families with CMT. CMT and HNPP are NOT the same. They

are

> exact genetic opposites. Anyone wanting HNPP info can go to

> http://www.hnpp.org or hnpp://www..groups.com/group/HNPP

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-----Original Message-----

From: KathleenLS@... <KathleenLS@...>

< >

Date: Sunday, February 11, 2001 12:21 PM

Subject: Re: [] CMT not HNPP

>http://www.charcot-marie-tooth.org/hnpp.htm

>What is HNPP?

>By Maureen Horton

>

others develop the symptoms of a more generalized hereditary motor and

>sensory neuropathy, which on occasion may be misdiagnosed as a form of CMT.

>

www.hnpp.org.

>

>>>>>>>Kat so this says that HNPP is not a " form " of CMT? ~>Becky M.

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I'm grateful you posted this, because I didn't really know what the

differences were. Thanks.

----- Original Message -----

From: <KathleenLS@...>

< >

Sent: Sunday, February 11, 2001 12:23 PM

Subject: Re: [] CMT not HNPP

> http://www.charcot-marie-tooth.org/hnpp.htm

> What is HNPP?

> By Maureen Horton

>

> Edited by Dr. Chance, the CMTA Medical Advisory Board

>

> (Editor's note: The CMTA will be working with Maureen to keep our

readership

> updated on HNPP. She has HNPP and first noticed problems during her

> pregnancies. She was finally diagnosed with HNPP and because information

was

> so difficult to find, she began to teach herself about the disorder. As a

> nurse, Maureen offers our readers both the personal side of the disorder

and

> medical expertise on the subject.)

>

> Hereditary neuropathy with liability to pressure palsies or HNPP is

> associated with a deletion on chromosome 17p11.2-12. This is the same

> chromosomal site where CMT1A has been found to have a duplication, and it

> contains an important myelin gene, peripheral myelin protein-22 gene

(PMP22).

> Therefore, while CMT1A results from having an extra copy of PMP22, HNPP

> results from the loss of a copy of PMP22.

>

> HNPP is caused by an autosomal dominant gene. HNPP is also a demyelinating

> form of neuropathy like CMT1A. However, the affected nerves in HNPP are

> described as having focal regions with " tomaculous " changes or sausage

shaped

> figures, due to the irregular thickening of the myelin sheath.

>

> HNPP causes episodes or periods of numbness and weakness, similar to an

arm

> or leg going to sleep. But, instead of lasting only a few seconds, as it

does

> in a normal person, this numbness and weakness can last from several

minutes

> to months. These episodes may be associated with fairly mild trauma

> (stretching or pressure on the nerves) which would not ordinarily affect a

> normal person, such as: leaning on elbows, crossing legs, cutting with

> scissors, knitting, kneeling, sleeping in the wrong position, etc. It has

> usually been described as a painless disorder, although some people do

have

> pain, which is probably a subjective sense of numbness. Most individuals

with

> HNPP have normal strength and sensation, unless experiencing an attack.

>

> Typically, persons who have inherited HNPP will develop symptoms in their

> teens or twenties, but individuals may be first symptomatic during

childhood

> or late adulthood. Like CMT, there is a wide range in how severely people

are

> affected. Many are so mild that they are unaware that they have a problem.

> Others develop the symptoms of a more generalized hereditary motor and

> sensory neuropathy, which on occasion may be misdiagnosed as a form of

CMT.

> Carpal tunnel syndrome is frequently seen in patients with HNPP.

>

> As a hereditary neuropathy it is progressive, though to what extent it

will

> progress in any individual is unknown. There is no treatment or cure for

> HNPP. People are usually advised to avoid activities which they have

learned

> cause symptoms to develop. Leg crossing and leaning on elbows is

discouraged.

> AFOs can be used during periods of acute peroneal nerve palsy or for

> permanent foot drop and/or balance problems.

> For more information on HNPP, please visit www.hnpp.org.

>

>

>

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Kat,

Over 15 neurologists have diagnosed my various family members with CMT.

All genetic tests are negative. Nerve biopsy indicates type 1, nerve

conduction indicated Type-2 . In compiling medical records for the research

on my family, I have encountered three reports that state " both axonal and

demyelinative " . One thing is certain with CMT, HNPP, HMSN, or

whatever-- " We are all connected "

----- Original Message -----

From: KathleenLS@...

Sent: Sunday, February 11, 2001 02:22 PM

Subject: Re: [] CMT not HNPP

In a message dated 2/11/01 11:06:16 AM, rmax@... writes:

<< >>>>>>>Kat so this says that HNPP is not a " form " of CMT? >>

" Therefore, while CMT1A results from having an extra copy of PMP22, HNPP

results from the loss of a copy of PMP22. "

They are genetic opposites but result in many symptoms in common. I've met

many HNPPers in the CMT chat room. As with CMT, we have many similar

problems

as well as differences.

Some people think CMT types 1 and 2 may not actually be properly called

CMT

for various reasons. For example: I don't have the usual atrophied calves

or

extremely high arches, but I still have CMT type 2. Some HNPPers DO have

atrophied calves and/or extremely high arches.

Diagnosing is still in a state of flux and learning.

Kat

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They sound awfully close to me. But, I'm no CMT genius either....still

learning.

----- Original Message -----

From: <ktbugg54@...>

< >

Sent: Sunday, February 11, 2001 2:13 PM

Subject: Re: [] CMT not HNPP

> In a message dated 2/11/2001 2:12:39 PM Eastern Standard Time,

> lisarich.com@... writes:

>

> << I'm grateful you posted this, because I didn't really know what the

> differences were. Thanks.

> >>

>

> As far as symptoms go, there aren't that many differences. Right?

>

>

>

>

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>Gretchen,

>You make me feel genetically discriminated against by this statement. CMT

is

>actually a multiplicity of genetic defects most of which we do not have a

>clear relationship between symptoms (phenotype) and causes (genotype).

Their

>is a long way to go to sort that out. In the meantime, as several have

>stated, we have similar problems and flock together to help resolve them in

>our lives.

>

>As for me, I know that I have HNPP thanks due to the Athena Labs DNA tests

>and am very thankful that is so. The old view that HNPP is only a minor

>numbness problem is now dead these past couple of years. Personally, I have

>most of the physical deformity and pain problems displayed by CMT1A

patients

>and strongly consider myself to be under the aegis of CMT in general. In my

>book, duplication vs. deletion is not a discriminating factor and only

>different forms of genetic defects. The treatments and solutions to the

>disease's problems are entirely common to both. When (and not if) we

resolve

>the phenotype-genotype puzzle, I believe that more deletions and

>duplications will be found in the family of CMT diseases and have no

>problems with them being identified with the people who discovered them -

>Messrs Charcot, Marie, and Tooth. So, what's in a name ? A disease by any

>other name would be its same debilitating self. EdM

>

>

>-----Original Message-----

>From: Gretchen Glick <liliwigg@...>

> < >

>Date: Sunday, February 11, 2001 1:08 PM

>Subject: [] CMT not HNPP

>

>

>>Just to clarify for anyone new to - this group is for people

>>and their families with CMT. CMT and HNPP are NOT the same. They are

>>exact genetic opposites. Anyone wanting HNPP info can go to

>>http://www.hnpp.org or hnpp://www..groups.com/group/HNPP

>>

>>

>>

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Kat,

Great point. Some CMT types affect the myelin and others affect the

axon. In the end, the nerve is still damaged and subsequently causes

a wide range of problems.

I like the analogy about the boat!

Ruth Warren

> This also reminds me that the medical difference between the

several type 1

> CMTs and the several type 2 CMTs is another opposite. With type 1

the damage

> is on the outside of the nerve and with type 2 the damage is on the

inside.

> But both are called CMT. Both have similar symptoms.

>

> We are all in the same boat, just in some of the different sections

of it.

>

> Kat

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