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This is great news I feel your doing .5 mgs of arimidex is what is helping bring

down your E2 when taking HCG. HCG is a LH stim. and it is what converts or

makes a lot of E2 so I want you to try to cut back to 400 IU's see how you feel.

Do some tests to be sure your levels don't drop. Then is all is good lower it

to 300 IU's and so on. I did this and my levels never changed. You see your

testis can only make so much T with the use of HCG and the rest gets converted

into E2. Hell I am down to 250 IU's and my levels are stil the same. And E2 is

less of a problem.

Phil

ozzpat <ozzmed@...> wrote:

Meds…

Noverel 500iu 3X week

Androgel 5g once daily

Arimidex .5mg 3X week

Before:

ESTRADIOL 48 (10-50pg/ml)

SHBG 13 (5-49 nmol/L)

TTL Testosterone 722 (241-827 ng/dl)

I just started the Arimidex three weeks ago at .25mg 2X week and it

wasn’t having much affect I then went to .25 3X felt good for the

first day then the second I would feel like crap so I started taking

..5 every other day when taking my HCG and this seems to be working great.

I was taking the DIM every day with some results but nothing compared

to the Arimidex, I was experiencing lots of memory issues, severe

flushing and agitation even with my T’s up but now I’m seeing my

memory recall coming back, I use to have a severe problem with peoples

names, people who I have known for years now I can walk around work

and recall names easily that I would have had problems with a month ago.

My wife is extremely happy with my mood, I actually feel at peace now,

before I would get agitated very easily and was extremely moody, it is

great to actually start feeling better for a change.

I use to have issues with frequent flushing especially when stressed,

this is getting better. I had a bout a few days ago before increasing

my dose but seem to be doing well now we will see.

I have seen, and my wife has commented on a reduction in my breast

size, I have had somewhat large breasts that I have always attributed

to body fat, another issue that has plagued me for the past five plus

years, but now it looks like there has been some reduction.

Oh yea, I have had problems with arthritis in my left knee due to an

injury some time ago, this has been bothering me for the past six

months to the point I was thinking of going to see someone about it,

now it has gotten about seventy percent better over the last couple of

weeks, to the point I’m even thinking of taking a skiing trip,

something I haven’t felt good enough to do in five years.

Regarding morning erections as a measure of level, this has been hit

and miss for me, no problem getting one first thing in the morning but

I don’t always wake up with one, but I have had them during the day

for no reason, I feel like a teenager when this happens.

Before staring the DIM or Arimidex I was on TRT and felt like crap

after a few weeks with my numbers in the 700’s. Extreme fog, strange

taste in my mouth and just felt like crap, not to mention the extreme

ED issues that came with it as well. I tried to convey this to my

doctor and even went to see two other for more opinions and they where

willing to do even less about it and looked at me like I was nuts

regarding E2 being the issue.

The 1st Endo that I started with has been willing (but reluctant) to

work with me on this and has done everything I have asked for so I

guess I should feel fortunate, but if it wasn’t for this group and

learning about the condition I’m certain I would have stopped TRT

treatment a few months ago and would not be feeling the benefits I do

now, so thank you all.

---------------------------------

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>

> This is great news I feel your doing .5 mgs of arimidex is what is

helping bring down your E2 when taking HCG. HCG is a LH stim. and it

is what converts or makes a lot of E2 so I want you to try to cut back

to 400 IU's see how you feel. Do some tests to be sure your levels

don't drop. Then is all is good lower it to 300 IU's and so on. I

did this and my levels never changed. You see your testis can only

make so much T with the use of HCG and the rest gets converted into

E2. Hell I am down to 250 IU's and my levels are stil the same. And

E2 is less of a problem.

> Phil

> The only reason I’m taking the 500IU HCG is to try and improve the

fertility issues, so far I have almost doubled my numbers in three

months but am still low. I’m taking the Androgel because on the 500IU

3x HCG only my TTL Testosterone numbers where 350 (241-827 ng/dl) but

from what I understand the arimidex will also raise LH/FSH and

increase fertility not to mention raise testosterone levels.

http://cat.inist.fr/?aModele=afficheN & cpsidt=13469640

A total of 25 oligospermic men with semen analysis before and during

anastrozole treatment had an increase in semen volume (2.9 versus 3.5

ml., p <0.05), sperm concentration (5.5 versus 15.6 million sperm per

ml., p <0.001) and motility index (832.8 versus 2930.8 million motile

sperm per ejaculate, respectively, p <0.005)

http://www.maleinfertility.org/new-therapy.html#aromatase

Aromatase inhibitors block the conversion of testosterone to estrogen.

Treatment with an aromatase inhibitor decreases estrogen levels, which

leads to increased LH and FSH release from the pituitary, with a

subsequent increase in testicular stimulation and serum testosterone

levels without the usual increase in estrogen levels seen for

anti-estrogens. Although one uncontrolled study suggested an increase

in sperm concentration for nearly all patients treated (89%), other

well-designed placebo-controlled studies have demonstrated no

significant improvement in pregnancy rates for treated patients.

Although many patients will have increased sperm concentration, no

improvement in sperm motility was seen in these studies.

http://jcem.endojournals.org/cgi/content/full/89/3/1174

bioavailable testosterone levels increased from 99 ± 31 to 207 ± 65

ng/dl in group 1 (anastrozole 1 mg daily) and from 115 ± 37 to 178 ±

55 ng/dl in group 2 (anastrozole 1 mg twice weekly)

I’m taking the minimum dose for Hypogonadtropic Hypogonadism for

fertility.

http://www.ferringusa.com/fertility_products/insert_novarel.htm

Selected Cases Of Hypogonadotropic Hypogonadism In Males:

(1) 500 to 1,000 USP Units three times a week for three weeks,

followed by the same dose twice a week for three weeks.

(2) 4,000 USP Units three times weekly for six to nine months,

following which the dosage may be reduced to 2,000 USP Units three

times weekly for an additional three months.

I don’t want to mess with what is working already but also don’t want

to go high, the doctor would rather I used more HCG but from all the

reading I have done 500IU is the max anything else risks LH

insensitivity and I don’t want that. And I also want

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Ok I read so many posts I can't remember.

Sorry Phil

ozzpat <ozzmed@...> wrote:

>

> This is great news I feel your doing .5 mgs of arimidex is what is

helping bring down your E2 when taking HCG. HCG is a LH stim. and it

is what converts or makes a lot of E2 so I want you to try to cut back

to 400 IU's see how you feel. Do some tests to be sure your levels

don't drop. Then is all is good lower it to 300 IU's and so on. I

did this and my levels never changed. You see your testis can only

make so much T with the use of HCG and the rest gets converted into

E2. Hell I am down to 250 IU's and my levels are stil the same. And

E2 is less of a problem.

> Phil

> The only reason I’m taking the 500IU HCG is to try and improve the

fertility issues, so far I have almost doubled my numbers in three

months but am still low. I’m taking the Androgel because on the 500IU

3x HCG only my TTL Testosterone numbers where 350 (241-827 ng/dl) but

from what I understand the arimidex will also raise LH/FSH and

increase fertility not to mention raise testosterone levels.

http://cat.inist.fr/?aModele=afficheN & cpsidt=13469640

A total of 25 oligospermic men with semen analysis before and during

anastrozole treatment had an increase in semen volume (2.9 versus 3.5

ml., p <0.05), sperm concentration (5.5 versus 15.6 million sperm per

ml., p <0.001) and motility index (832.8 versus 2930.8 million motile

sperm per ejaculate, respectively, p <0.005)

http://www.maleinfertility.org/new-therapy.html#aromatase

Aromatase inhibitors block the conversion of testosterone to estrogen.

Treatment with an aromatase inhibitor decreases estrogen levels, which

leads to increased LH and FSH release from the pituitary, with a

subsequent increase in testicular stimulation and serum testosterone

levels without the usual increase in estrogen levels seen for

anti-estrogens. Although one uncontrolled study suggested an increase

in sperm concentration for nearly all patients treated (89%), other

well-designed placebo-controlled studies have demonstrated no

significant improvement in pregnancy rates for treated patients.

Although many patients will have increased sperm concentration, no

improvement in sperm motility was seen in these studies.

http://jcem.endojournals.org/cgi/content/full/89/3/1174

bioavailable testosterone levels increased from 99 ± 31 to 207 ± 65

ng/dl in group 1 (anastrozole 1 mg daily) and from 115 ± 37 to 178 ±

55 ng/dl in group 2 (anastrozole 1 mg twice weekly)

I’m taking the minimum dose for Hypogonadtropic Hypogonadism for

fertility.

http://www.ferringusa.com/fertility_products/insert_novarel.htm

Selected Cases Of Hypogonadotropic Hypogonadism In Males:

(1) 500 to 1,000 USP Units three times a week for three weeks,

followed by the same dose twice a week for three weeks.

(2) 4,000 USP Units three times weekly for six to nine months,

following which the dosage may be reduced to 2,000 USP Units three

times weekly for an additional three months.

I don’t want to mess with what is working already but also don’t want

to go high, the doctor would rather I used more HCG but from all the

reading I have done 500IU is the max anything else risks LH

insensitivity and I don’t want that. And I also want

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