b12 methylates mercury - I hope not...

[Guest guest]

here is a link to a study:

adult-metal-chelation/message/29344

It's just the abstract but it says vitamin C and b12 will increase

brain mercury... I hope it's not true...

[Guest guest]

If you keep your eyes on the prize of recovery, you will find that many adults

and children with merc exposure, are doing extra well on MB12, some having

recovered completely. I wouldn't give it another thought.

mkarty2007 <mkarty2007@...> wrote:

here is a link to a study:

adult-metal-chelation/message/29344

It's just the abstract but it says vitamin C and b12 will increase

brain mercury... I hope it's not true...

---------------------------------

Never miss a thing. Make your homepage.

[Guest guest]

>

> here is a link to a study:

>

> adult-metal-chelation/message/29344

>

> It's just the abstract but it says vitamin C and b12 will increase

> brain mercury... I hope it's not true...

>

It's not true at physiological conditions.

If you find the whole study and not just the abstract you will

probably find that the doses of everything used are way, way higher

than would ever possibly be found in any person, even a seriously

toxic person.

J

[Guest guest]

,

You may know that Alzheimer's Desease is also caused by mercury. Vitamin B12

is a powerful scavenger of free radicals.

DK

Here's an article from Nexus Magazine:

High-Dose Vitamin B12 in

the Treatment of Dementia Few medical practitoners know that high doses of

vitamin B12 can prevent and even reverse the symptoms of Alzheimer's disease

and other mental illnesses, but new Codex regulations will restrict access

to such high-dosage vitamin therapy.

------------------------------

Extracted from Nexus Magazine, Volume 14, Number 6

<http://www.nexusmagazine.com/backissues/1405.conts.html> (October -

November 2007)

PO Box 30, Mapleton Qld 4560 Australia. editor@...

Telephone: +61 (0)7 5442 9280; Fax: +61 (0)7 5442 9381

From our web page at: www.nexusmagazine.com

© 2007 ph G. Hattersley, MA

2209 Craig Road SE

Olympia, WA 98501, USA

Telephone: +1 (360) 352 3688

Email: jghattersley@...

------------------------------

*IAT-HOME PREVENTION AND REVERSAL OF ALZHEIMER'S DEMENTIA AND SIX OTHER

MENTAL ILLNESSES*

*Introduction*

It is interesting to consider what proportion of Alzheimer's dementia (AD)

may result from under-nutrition,1 especially when it seems that an easy,

low-cost, perfectly safe nutritional way exists that may allow people to

avoid that misery of miseries which many consider worse than death. Some

people might say, " That's too good to be true! " However, an at-home

nutritional program using a high dosage of vitamin B12 may prevent and

virtually eliminate AD. An early launch of the treatment soon after first

warning symptoms start could even turn off the process.

Confusion, difficulty concentrating, loss of memory, marked changes in

personality that can lead to outbursts of violence, hallucinations,

wandering away and early death all characterise Alzheimer's dementia.

An estimated 2.3 million Americans now have AD. Prevalence doubles every

five years after the age of 60, increasing from one per cent among those 60

to 64 years old up to 40 per cent of those aged 85 years and older.

Nursing home care costs about US$47,000 per AD patient annually and this

figure is rising steadily, putting a huge burden on the health care system.

The disease is also terrible for the patients' caregivers. In what experts

are calling " a looming public health disaster " , statistics suggest there

will be between five and seven million Alzheimer's patients in the USA over

the next 10 years.2

Let's start with a little background. Mammals, including humans, are born

with serum levels of vitamin B12 at about 2,000 pg/mL (picograms, i.e.,

trillionths of a gram, per millilitre). The level declines throughout human

life owing to practices common in Western societies.4 Below 550 to 600

pg/mL, deficiencies start to appear in the cerebrospinal fluid.5, 6 US

clinical laboratories regard 200 pg/mL as the lower range of normal. That

low limit was set with haematologic criteria. But neuropsychiatric criteria,

which are much higher, have become more critical.

" Most cases of Alzheimer's dementia are actually missed B12 deficiency

cases, because of the too-low normal range for B12, " wrote V. Dommisse,

MD, in 1991 in *Medical Hypotheses*.3 Dommisse, who practises medicine in

Tucson, Arizona, has confirmed that Alzheimer's disease appears to result

from too-low serum vitamin B12, and repletion of the vitamin succeeds

despite other risk factors.7, 8, 9 Replenishing B12, according to Dommisse,

can reverse 75 per cent of B12 deficiency dementias when discovered early

enough.10

As mentioned above, B12 therapy is perfectly safe; in other words, the risk

of overdose is virtually nil. Here's the proof... Patients of Dr H. L.

Newbold in New York City injected themselves three times daily with

triple-strength doses of B12 (9,000 micrograms/day as hydroxycobalamin, the

natural form) indefinitely. Their serum B12 levels reached 200,000 pg/mL

(100 times the normal level found in newborn babies and higher). But none

had any significant side effects.3

Other aspects of the therapy should be noted: The neurological and cerebral

manifestations of B12 deficiency require dosages larger,11 and extending

over a longer time,12 than those needed to reverse haematologic effects;13

and there is no reason to run the risk of not catching deficiency in time or

to go to any unnecessary expense and inconvenience. To put it bluntly: try

the harmless therapy and see if you, the patient, benefit(s).

*Other Conditions Benefit From B12 Replenishment

* Besides Alzheimer's disease, B12 ( " cobalamin " *) deficiency can also cause

the following conditions. And when started early, replenishing B12 (i.e.,

restoring it to or near to levels found at birth) may often significantly

improve these afflictions as well:

* *Depression*. Often in mild form, depression can be one of the first clues

foretelling dementia.14-19 Reading, BSc, DipAgSc, MBBS, of Australia

concurs: " ... in most cases of [not only mild but also] 'intractable

depression', a subtle B12, or other nutrient, or thyroid hormone, deficiency

has been missed. " 20, 21 Psychotic depression has been particularly

associated with B12 deficiency.22

As I'll discuss later, success of B12 therapy against depression in its

various forms is " probable " .

* *Paranoid psychosis*. This nutritional deficit has also been linked to

paranoid psychosis, characterised by over-suspiciousness and delusions of

grandeur or persecution.23

* *Bipolar-1 disorder* (manic depression), marked by alternating periods of

elation and depression; and more commonly bipolar-2 disorder (cyclothymic

personality), marked by swings of mood but within normal limits.24, 25, 26

* *Chronic fatigue syndrome*.27, 28, 29

* *Weakened immunity*. Weakened immunity can lead to susceptibility to

recurrent infections and cancer, as well as increasing the risk of

cardiovascular disease, cancer30 and much more by a second pathway:

hindering remethylation of the toxic sulphur amino acid homocysteine back

into the nontoxic essential amino acid methionine.31

* *Asthma*. Incomplete digestion of foods due to hypochlorhydria and low

pepsin production (see below) can be involved in a subsequent allergic

response in asthma.32

* *Disrupted sleeping/waking rhythms*.33, 34

* *Environmental illness*.35

* *Low stress tolerance*.36

* *Osteoporosis*.37

* *AIDS* (acquired immuno-deficiency syndrome).38

* *Premature ageing*.39

* *Multiple sclerosis*.40, 41 Symptoms of MS have been noted in persons with

a vitamin B12 deficiency prior to evidence of megaloblastic anaemia. *There

is a remarkable epidemiologic similarity between MS and pernicious anaemia *,

and similar HLA (human lymphocyte antigens) are suggested for the

association of the two conditions.

* *Alzheimer's mimicking and non-Alzheimer's dementia*.42

Intramuscular injection of B12 also has yielded seeming miracle cures in

still other desperate illnesses. Further, in numerous cases of patients with

violent behaviour, when B12 was replenished (with or without other changes

in life), violent behaviour disappeared.45-49

" The only question now, " writes Dommisse, " is, what proportion of cases of

mood-disorder is caused by B12-deficiency and what percentage is

idiopathic. " Almost all of his uni- and bipolar patients have had B12 levels

in the lowest one-third of the so-called normal (to prevent pernicious

anaemia) range--levels that he now regards as deficient for adequate

affective, cognitive and other mental functions. When their levels have been

raised to the highest one-third of that " normal " range, every one of those

patients felt better. For some patients who came out of their depression or

mood-swing disorder, this was the only new or different treatment they

received. In subsequent instances when their affective disorder worsened,

B12 levels had again dropped.

So, would restoring ample serum B12 levels prevent many or most of those

adverse conditions? Evidence shown below, Dr Dommisse cautiously suggests,

means, " Yes, at least in the case of depression " .

There is no maximum allowable age to begin B12 therapy. Everyone loves an

anecdote. A friend told me her 90-year-old live-in mother was beginning to

think less clearly than in the past and to feel a bit depressed. I

suggested: " If your dear mother would like to stop her incipient downward

slide, let her start the therapy. " God never wrote on tablets of stone that

90 years of age is too old to turn one's health around and begin to make

life fun again. After a few weeks on high-dose B12 she was out in the woods

with her daughter gathering items of interest. But three years later she

" felt terrible " for want of enough water (F. Batmanghelidj, MD's

authoritative study, *Your Body's Many Cries for Water,* states that chronic

dehydration is the cause of AD).

Having a quality laboratory measure serum B12 is an essential part of the

replenishment process. Simply supplementing B12 " in the dark " could miss the

mark badly. And to really know the patient's whole picture, Sherry A.

, MD, suggests the ION (Individualised Optimum Nutrition) Panel is

well worth its cost (currently, about US$600; in the USA, contact NEEDS on

1800 634 1380).50

The normal range for serum B12, states Dr Dommisse, should be defined as 600

to 2,000 pg/mL. Japan's " normal " range is 500-1,300.51 According to

Dommisse, this may explain why Japan has such a low rate of Alzheimer's

dementia52 compared to the USA.53 By some estimates, as many as 80 per cent

of elderly American patients may share hidden B12 insufficiency.54, 55, 56

Also, B12 deficiency is common with folate deficiency in dementia57, 58, 59

and worsens over time as the deficiency increases.60 The impact was seen

first on neuropsychiatric measures; augmentation of B12 and folate

materially improved scores on cognitive performance tests.61, 62

(* The name " cobalamin " has been advocated because the large B12 molecule

includes a cobalt molecule; see W. S. Beck, " Cobalamin and the nervous

system " [editorial], *New Eng J Med* 1988; 318:1752-54. However, about

one-third of this vitamin's molecule is not cobalamin, but metabolically

inactive crinoids which may be effective for bacteria but do not serve as

haemopoietic agents; see J. S. Bland, *Preventive Medicine Update*, October

1994. See also Addendum.)

*Methods of Treatment*

The most direct method for adding these megadoses of B12 is through

intramuscular (IM) injection, which requires a doctor's prescription and a

doctor's or nurse's instruction. It is about as difficult technically as

pushing a pin into a ripe orange63 and can be economical if a patient can

self-administer or a companion can administer.

Another feasible approach uses inexpensive sublingual B12 at 2,500-5,000 mcg

(2.5 to 5 mg), which anyone can buy at a quality health food store. Taken in

that way, evidence indicates that most of the vitamin goes via the lymphatic

system (see Sherry A. , MD, *Detoxify or Die*, 2005 ed., p. 270).

Therefore, this treatment mode may yield more benefit by avoiding the

digestive system.65

*Causes of B12 Insufficiency*

Several common features of modern life accelerate the decline of

vitamin B12in serum through life, including the following:

* *Microwave ovens*. In one test, microwaving milk degenerated 30-40 per

cent of its vitamin B12 content in six minutes; with conventional heating,

25 minutes of boiling was needed to depress B12 that much.67 More

importantly, the heat of microwaving destroys all the enzymes in ingested

food, which are required to enable absorption and utilisation of food. And

so by eating microwaved food, both at home and in restaurant and take-out

meals, tens of millions of Americans are making themselves increasingly

vulnerable to AD as well as to cancer. One further note: microwaving,

invented by the Nazis, is wisely forbidden in Russia.67

* *The Western diet*. B12 ingestion and stores tend to be insufficient among

millions who have for decades eaten RDA-fortified, yet vitamin- and

mineral-depleted, processed Western diets, which are also big sources of

disease-creating free radicals.69 Too low levels of omega-3 essential fatty

acids in Western diets, harmful on their own in many ways, must also

contribute to insufficient B12 levels.70 Omega-3 supplementation may yield

its benefits largely through augmenting vitamin B12. Too-low levels of

acetyl-carnitine and folic acid also appear to worsen the risk of the

condition.71, 72

It's worth noting that in an Alzheimer's disease mouse model, a diet rich in

omega-3 essential fatty acids, specifically DHA (docosahexaenoic acid), has

been shown potentially to slow or even to prevent Alzheimer's disease.73 At

modest cost, we can easily ingest DHA and EPA (eicosapentaenoic acid) in

fish oil or cod liver oil (I find the Carlson brand to be the best). And how

about trans-fatty acids found in products labelled " zero trans-fats " with

EPA approval? In a study of over 800 seniors, those with high TFA intake

were twice as likely to suffer with Alzheimer's disease as those with the

lowest intake (hsiresearch@..., 17 February 2006).

* *Hypochlorhydria*. Most commonly, B12 insufficiency results directly from

hypochlorhydria (insufficient hydrochloric acid [HCl] in the stomach) or by

achlorhydria (no HCl at all). The acid should be concentrated enough, in

middle age, to dissolve a nail in an hour.77 Hypochlorhydria is likely

caused by zinc or vitamin B6 deficiency78 and a shortage of ionised

calcium.79, 80 (Both deficiencies are typically present in older people.)

Lack of enough pepsin or HCl in the stomach to generate the bond

between B12and its carrier protein typically shows with atrophic

gastritis.81, 82 Both are also risk factors for gastric cancer.83 Incomplete

digestion of foods due to hypochlorhydria and low pepsin production also can

be involved in subsequent allergic response in asthma.84

* *Intrinsic Factor, Bacteria, Cobalamid, Failure of Absorption*.

B12deficiency can also result from inadequate stomach secretion of the

tiny

open-ended protein capsule known as " intrinsic factor " ; from presence in the

gut of bacterial overgrowth;85 from ingestion of cobalamid, a

B12antagonist;86 or failure of absorption for other

reasons.87

* *Antacids and Antibiotics*. Chronic overuse of antacids, both prescribed

and over the counter (OTC), by tens of millions of elderly people may also

be responsible. When all the acid is mopped up daily by antacids, the B

vitamins never even get to first base.88 For example, B12 absorption is

dramatically reduced when the drug Prilosec (omeprazole), which has recently

been made available OTC in the USA, is used.89, 90 " A significant percentage

of patients taking omeprazole are also being treated for or are at high risk

of heart disease, and therefore almost all are instructed to eat a diet low

in red meat (or devoid of it completely) and of animal products, which are

the best source of vitamin B-12 " .91 Also, omeprazole reduces gastric

(stomach) levels of multi-protective ascorbic acid (vitamin C),92 still

another route to cancer (see J. G. Hattersley, " Alzheimer's dementia,

vitamins B12 and B6, lithium, gingko biloba, dental mercury, genetic risk,

and drinking water fluoridation " , *J Appl Nutr* 2005).

This is an egregious example of iatrogenic disease, created by

tunnel-visioned one-organ specialists (who seldom if ever communicate with

each other) using a " band-aid " approach to treatment of a symptom or test

reading, oblivious to the possibly disastrous long-term effect on the

patient. Other causes of B12 deficiency include excessive long-term use of

antibiotics and other drugs to mask symptoms without learning and correcting

their cause; oral antibiotics destroy the trillions of " good " bugs in the

gut as well as the bad, thus ruining absorption.93

* *Vegan Diets*. Many vegan (total) vegetarians have for decades consumed

few, if any, foods containing B12. As a result, their body stores of the

vitamin have gradually diminished. Forty-seven of 78 adult vegans had levels

below 200 pg/mL; when they chewed a 100-microgram B12 tablet once a week,

their levels promptly rose to normal.94 Some vegans depend on sea vegetables

such as arame, wakame and some varieties of kombu, or on algae.95 The B12 in

these, although absorbed, may not be fully bio-available.96 A study

published in the May 2003 *Townsend Letter for Doctors and Patients* (TLfDP)

provided strong evidence that a commonly consumed seaweed known as nori

does, in fact, contain bio-available forms of B12.97 But whether that

substance is available to large numbers of vegans and whether its use would

lift serum B12 levels enough is not known. And in a recent study from India

published in Neurology India,1 most of the B12-deficient people studied were

" vegetarian " .

*Vitamin B12 Repletion Succeeds Despite Risk Factors*

In his study, Dr Dommisse does not reveal, or need to know, the proportion

of his patients who experience other AD risks: for example, how many are

thyroid deficient, drink fluoridated water, have extensive dental amalgams,

take Ibuprofen98 (some non-steroidal anti-inflammatory drugs, along with

their famous multitude of adverse effects, may lower AD risk by about 50 per

cent99), etc. He doesn't consider the number of patients who have been

exposed occupationally to electro-magnetic fields, which promote the

formation of beta amyloid, a protein common in the brains of Alzheimer's

patients.100, 101 Nor does he present any numbers showing high content of

aluminium in AD patients' brains102 from consumption of aluminium-treated

drinking water103, 104 and/or from consuming a variety of everyday sources

(refer also to the combination of aluminium-containing alum with fluoridated

water in J. G. Hattersley, *J Appl Nutr* 2005). Dommisse's study also

doesn't consider the extent of use of melatonin as a brain antioxidant to

counteract accumulation of free-radical-creating iron,105 nor does it look

at potentially brain-damaging homocysteine in patients' serum.106, 107

[As a side note, possible mechanisms for spinal cord and peripheral nerve

effects of vitamin B12 deficiency include axonal degeneration and

demyelination of insulating nerve sheaths.108 Deana et al. found low levels

of neurotransmitters in the brains of B12-deficient rats.109 A University of

Kentucky study found impaired G-protein signalling and proposed a

feed-forward cycle of progressive neuronal dysfunction, related to

phosphoinositide signalling.110 Spector et al. have hypothesised that

idiopathic (Alzheimer's) dementia is a brain-vitamin-deficiency state due to

inadequate transport of vitamins from the blood across the choroid plexus

(the " blood brain barrier " ) and into the cerebrospinal fluid--the only

source of these nutrients for the brain.111]

Dr Dommisse prefers vitamin B12 as hydroxycobalamin. A person taking

cyanocobalamin from a pharmacy might over a long period of years accumulate

a toxic amount of cyanide and possibly damage vision.112 (Many patients in

Britain using cyanocobalamin from a pharmacy did go blind due to damage to

the optic nerve, and few physicians knew of it [letter 9/3/05 from Wayne

, *TLfDP* contributing writer, Fairhope, Alabama]).

Also, methylcobalamin, widely used in Japan, is increasingly popular in the

USA, in part because it is reputed to be better absorbed. Certain OTC

sublingual preparations provide methylcobalamin, which in the absence of

alcoholic and other liver damage is the only version of B12 that penetrates

the blood-brain barrier (the choroid plexus) and reaches the brain and

spinal cord (Sherry A. , MD, *Detoxify or Die*, p. 270).

In *Preventive Medicine Update* (May 1995), Bland, PhD, reported:

" Five clinician/subscribers have sent clinical case histories. On high-dose

folate/B12/B6, homocysteine levels dropped... Also, a number of reports have

come... about patients suffering with presenile dementia or Alzheimer-like

symptoms. On IM B12, their MMA (methyl malonic acid levels, an indication of

deficient B12 status) came down to normal range, and their walking, balance,

gait, and perception improved. I've had reports of individuals who had not

been able to read, start to read, people who had not been able to look at

video screens now comfortable looking at them, and two reports of people who

had movement dystonia, who after vitamin B12 therapy were able to get in the

car and be transported without fear of being unable to accommodate passing

scenery. So there is a wide range of very important clinical outcomes from

improving folate/B12/B6 status, cutting across neurological and arterial

functional status. " 113

Because the typical environmentally ill (EI) patient often has low zinc, B6

and thyroid hormone levels, low chromium glucose tolerance factor (GTF) and

high candida/low bifidus as well as low B12, to suggest B12 alone as the

sole treatment would not be consistent with holistic thinking, suggested Dr

Earl Conroy.114 So, if high-dose B12 doesn't do the trick pretty fast,

consider additional measures, guided by the results of the ION Panel test,

if given.115

Little research has been published about B12 therapy for AD and other

neurological diseases, Dommisse writes, because of the " ...heavy

pharmaceutical industry sponsorship of research and teaching in medical

schools. Career-track academicians have realised that, if they want to

fulfill their ambitions, they have to eschew nutritional research for that

of drugs. " 3 The volume of published research on drugs to fight Alzheimer's

disease is overwhelming. To continue their careers, the authors of these

studies have a powerful financial incentive to report positive results,

whether truthful or not. Yet, the best that Alzheimer's drugs can do is to

conceal the symptoms for a while. The underlying cause--notably, deficiency

of vitamin B12--continues to worsen unabated.

In that vein, I offer a caution on cholesterol-lowering statin drugs. Ten

years ago I exposed these high-margin products, along with others, as

potential patient-killers.116 Big Pharma is waking up belatedly to that

fact. Now pharmaceutical companies would like to promote statins as

preventives for Alzheimer's dementia. Drug therapies reported up to

September 2005 showed no success (*Acres USA*, September 2005).

A recent CBS Evening News report quoted a University of California medical

professor who is conducting new government-funded (not drug-maker-funded)

research on statin drugs' effect on Alzheimer's-susceptible patients: " We

have people who have lost thinking ability so rapidly that within the course

of a couple of months they went from being heads of major divisions of

companies to not being able to balance a checkbook and being fired from

their companies. " 117, 118

Vitamin B12 therapy still faces a very real obstacle: Codex regulations,

likely to go into effect in the near future, will prohibit any dose of any

vitamin to be sold at much above the Recommended Dietary Allowance (RDA).

For B12, the adult RDA dosage is only 3.02 micrograms. Millions will suffer

and die from this terrible B12-preventable disease if that Codex regulation

is enforced. And a new bill in Congress, cited in *TLfDP* (November 2005) by

Musnick, MD, will reinforce Codex restrictions.

One final note. It is important to cultivate a positive, optimistic outlook

to maximise the prospect for success against such mental diseases. One

should avoid the scenario in which the attending physician tells the cancer

patient, " You have x months to live " . Classes and groups for

Alzheimer's-fearing people often move in that direction. In fact, I have

heard of no AD awareness classes that even mention vitamin B12 therapy in a

positive way. Physicians, whose medical education omitted or put a negative

spin on anything using nutrition, may be behind the structure of many such

classes. What a different story it might be if instruction and awareness

emphasised the usually successful measures brought out in this article. One

has to ask: why don't doctors at least tell the public about this seemingly

magical therapy, which is available to all at trivial cost? Just think about

that, and the answer becomes obvious. °

*About the Author:

* ph G. Hattersley has an MA in Economics from the University of

California-Berkeley. In 1953, he completed all requirements for a PhD except

the dissertation. In 1976, at age 54, a seeming nutritional miracle launched

his career of writing on a wide range of health topics. Mr Hattersley has

previously contributed two articles to NEXUS: " Soybean Products: A Recipe

for Disaster <http://www.nexusmagazine.com/articles/soya.html> " , in vol. 4,

no. 3, and " The Healing Power of Full-spectrum Light " in vol. 8, no. 4.

Mr Hattersley can be contacted by post at 2209 Craig Road SE, Olympia, WA

98501, USA, by telephone on +1 (360) 352 3688 and by email on

jghattersley@....

*Endnotes:*

1. Bland JS. *Funct Med Update* 2002; August.

2. Centers for Disease Control. Mortality from Alzheimer's disease - United

States 1979-1987. *JAMA*. 1991; 265(3):313-317.

3. Dommisse JV. *Med Hypotheses*. 1989.

4. Dommisse JV. Subtle vitamin-B12 deficiency and psychiatry: A largely

unnoticed but devastating relationship? *Med Hypotheses*. 1991; 34:131-140.

5. Regland B. *Vitamin-B12 Deficiency in Dementia

Disorders*(Monograph/doctoral thesis, comprising six papers, with

co-authors). Dept.

of Psych. and Neurochem, University of Goteborg, Sweden. January 1991.

6. Ikeda T, Furuwaka Y, Mashimoto S et al. Vitamin B12 levels in serum and

cerebrospinal fluid of people with Alzheimer's disease. *Acta Scand

Psychiatrica *1990; 82; 4:327-329.

7. Dommisse JV. Psychiatry and vitamin B12 deficiency. Kirk Hamilton,

PAC, *Clinical

Pearls News*. March 1998: Interview.

8. Dommisse JV. *Subtle vitamin-B12 deficiency and psychiatry*. Op. cit.

9. Spector R, Cancilla P, Damasio AR. Is idiopathic dementia a regional

vitamin-deficiency state? Op. cit.

10. Roos D. Neurological complications in patients with impaired

vitamin B12absorption following partial gastrectomy (monograph/D Med

Sci thesis).

Supplement 69 to *Acta Neurologica Scandinavica*. 19, Minksgaard,

Copenhagen, 1978.

11. Roos D. Neurological complications in patients with impaired

vitamin B12absorption.

*Idem*.

12. Whitehead JA, Chosen MM. Paraphrenia and pernicious anaemia. Geriatrics.

1972; May: 148-158.

13. ADM. Megaloblastic madness. *Brit Med Jour*. 1960; 2:1840.

14. Ferrara A, Arieti S, English WH. Cerebral changes in the course of

pernicious anaemia, and their relationship to psychic symptoms. *Jour of

Neuropathology & Experimental Neurology*. 1945; 4:217.

15. Burvill PW, JM, WG. Psychiatric symptoms due to vitamin-

B12 deficiency without anaemia. *Med Jour Australia*. 1969; Aug 23:388-390.

16. Levitt AJ, Joffe RT. Vitamin B12 in psychotic depression. *Brit Jour

Psychiatry*. 1988; 153:266-267.

17. SL, Kahamer KP. An unusual presentation of

vitamin-B12deficiency (letter).

*Amer Jour Psychiatry*. 1983; 145; 4:529.

18. DL, Edelsohn GA, Golden RN. Organic psychosis without anaemia or

spinal cord symptoms in patients with vitamin-B12 deficiency. *Amer Jour

Psychiatry*. 1983; 140; 2:218-221.

19. Elsborg L, Hansen T, son OJ. Vitamin-B12 concentrations in

psychiatric patients. *Acta Psychiatrica Scandinavica*. 1979; 59:145-152.

20. Reading CM. X-linked dominant manic-depressive illness: Op. cit.

21. Reading CM. Latent pernicious anaemia: A preliminary report. *Med Jour

Australia*. 1975; (Jan. 25) 1:91-94.

22. Levitt AJ, Joffe RT. Vitamin B12 in psychotic depression. *Brit Jour

Psychiatry*. 1988; 153:266-267.

23. Lindenbaum J, Healton EB, Savage DG et al. *Neuropsychiatric disorders*.

Op. cit.

24. Reading CM. Latent pernicious anaemia (letter to the editor). *Med Jour

Australia*. 1975; March 29:430-431.

25. Zucker DK, Livingston RL, Narka R, Clayton P. B12-deficiency and

psychiatric disorders: Case-report and literature review. *Biological

Psychiatry*. 1981; 16; 2:197-205.

26. Van Tiggelen CJM, Peperkamp JPC, TerToolen JFW. Assessment of vitamin-

B12 status in CSF. *Amer Jour Psychiatry*. 1984; 141; 1:136-137.

27. Ellis FR, Nasser S. A pilot study of vitamin-B12 in the treatment of

tiredness. *British Jour Nutrition*. 1973; April.

28. Lindenbaum J, Healton EB, Savage DG et al. Neuropsychiatric disorders.

Op. cit.

29. Reading CM. X-linked dominant manic-depressive illness: Linkage with Xg

blood--group, red-green color-blindness and vitamin-B12 deficiency.

*Orthomolecular

Psychiatry.* 1979; 8; 2:68-77.

30. Bland JS. *Functional Medicine Update*. 2004; Jan.

31. McCully KS. Homocysteine theory. Development and current status.

*Atherosclerosis

Rev*. 1983; 11:157-246.

32. JV. Treatment of childhood asthma with parenteral vitamin B12,

gastric re-acidification, and attention to food allergy, magnesium and

pyridoxine: Three case reports with background and an integrated hypothesis.

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205. ADDENDA.

206. (1) Details on Cobalamin. Cobalamin for Soil & Animal Health, by Jerry

Brunetti. *Acres-USA*, Sept. 2005: pp 11, 33; reprinted from Weston A. Price

Foundation, PMB 106-380, 4200 Wisconsin Ave NW, Washington, DC 20016. The

paper is essential to understanding of our material. A few copies are

available from me on request.

(2) Background on Dommissee, MD. Emphasize the first syllable, viz.

DOMmissee. He is of Dutch-Africaans origin, earned his first MD degree at

the very advanced University of Johannesburg, South Africa and flies down

there occasionally. He earned a second, nutritionally oriented MD degree at

University of Toronto in 1967.

Dr Dommisse works out of an office at 1840 E. River Road, Suite 210, in

Tucson, AZ 85718. (520) 577-1940. john@... He specializes in

treatment of subclinical hypothyroid conditions, and maintains contact with

his patients worldwide by email. He told me his treatment of that condition

is " the best in the world. "

Medical societies have tried for the past eight, and especially the last

four years to deprive Dr Dommissee of his license to practice

medicine--presumably because his successful high-dose B12 therapy deprives

many physicians of ongoing patients' visits and sales of medications.

On 12/15/07, <lindajaytee@...> wrote:

>

>

> >

> > here is a link to a study:

> >

> > adult-metal-chelation/message/29344

> >

> > It's just the abstract but it says vitamin C and b12 will increase

> > brain mercury... I hope it's not true...

> >

>

> It's not true at physiological conditions.

>

> If you find the whole study and not just the abstract you will

> probably find that the doses of everything used are way, way higher

> than would ever possibly be found in any person, even a seriously

> toxic person.

>

> J

>

>

>