Guest guest Posted December 16, 2005 Report Share Posted December 16, 2005 > Khepri wrote: > See this... > " Such antigenic mimicry might generate autoimmune inflammatory reactions > that could be responsible for arthritic as well as some neurological > symptoms associated with chronic Lyme disease. " > Typically cautious language -- " might " generate " autoimmune inflammatory reactions " Here's the quote in its context: <snip>...T cells from patients with chronic Lyme disease are reactive not only against B. burgdorferi-specific antigens but also against various host (self) antigens (Nature Medicine 5: 1375, 1999). Such antigenic mimicry might generate autoimmune inflammatory reactions that could be responsible for arthritic as well as some neurological symptoms associated with chronic Lyme disease. </snip> We've stopped autoimmune 'disorders' -- regardless of antigenic mimicry -- by correcting cell communication by addressing glyconutrient deficiency, by reducing toxin load, by normalizing the immune system with Transfer Factor, and by increasing cellular robustness with glutathione. What escapes the bureaucracy is the observation that an " autoimmune " isn't even a disease. Anything that can trigger chronic inflammation can have the effect. Duncan Quote Link to comment Share on other sites More sharing options...
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