ALA, glutathione rqts if any, not pooping& Andy's book - Qs for Andy

[Guest guest]

1. Can ALA actually take mercury all the way out of the body?

I understood until I joined this group a couple weeks ago that ALA

could take mercury in and out of the brain/cns but couldn't get it out

of the body, so ALA alone would essentially guarantee redistribution

of the mercury without another " DM " chelator present to hand off to and

go out urine or poop. I mentioned in last post perhaps the metals hand

off was ALA to glutathione then out the poop, but last response

indicated this was not the case. I now understand that people use ALA

and it somehow gets the metals out of the body. How?

Your last post seemed to say that most chelators - ALA, DMs, etc. take

it out urine. Is this accurate? Are any more likely to go through

urine than poop, as I understood DMSA took metals more out through

poop than DMPS and this is why DMPS is " safer " in a withholding child.

2. Can ALA or other chelators (DM's) safely chelate a child with very

low (out of normal range) circulating glutathione? ie with gluathione

gene deletion without supplementation?

I mentioned in last post that we have glutathione gene (liver kidney

only - not brain form) missing, and as a result have TD glutathione to

slather on our daughter every day. My understanding based on the

EWG.org (environmental working group website) is that the substances

in the TD cream that " unzip " the skin cells and " force " the

glutatathione/nac in are all either carcinogenic or endocrine

disrupting (confirmed by our compounding pharmacist). Oral

glutathione liposomal is nearly inedible /unhideable. If we can

eliminate oral and transdermal glutathione without significantly

raising our risk of redistribution or just chelation not working, we

want to. (Not to mention the extra cost of course).

Your last post stated that glutathione is not a " hand off " for mercury

via ALA or any other chelator. Does this mean that we can stop

worrying about glutathione supplementation, since up to now it appears

it has had NO significant effect (when I stop I notice no difference

from when we use it)? In other words, can we use ALA alone (or DM's

plus ALA) without " needing " glutathione there in normal levels?

3. Does any of the above change if the child is not pooping for 5-7

days at a time?

4. Does any of the above change if you use " TTFD " (modified

B-1/thiamine) as a chelator? Just curious as I see it in the DAN

literature and wonder if its behavior is consistent with the DMs or

ALA, since I haven't noticed it getting mentioned in this group yet.

Is TTFD something useful for chelation esp. in an irregularly pooping

child?

5. Does your book still contain current info on your current

positions/understandings? I just purchased it and wanted to review

dose, schedule, chelator info - what would you change since it was

published esp. pertaining to dosages, timing, practical chelating info.