Guest guest Posted January 5, 2007 Report Share Posted January 5, 2007 1. Can ALA actually take mercury all the way out of the body? I understood until I joined this group a couple weeks ago that ALA could take mercury in and out of the brain/cns but couldn't get it out of the body, so ALA alone would essentially guarantee redistribution of the mercury without another " DM " chelator present to hand off to and go out urine or poop. I mentioned in last post perhaps the metals hand off was ALA to glutathione then out the poop, but last response indicated this was not the case. I now understand that people use ALA and it somehow gets the metals out of the body. How? Your last post seemed to say that most chelators - ALA, DMs, etc. take it out urine. Is this accurate? Are any more likely to go through urine than poop, as I understood DMSA took metals more out through poop than DMPS and this is why DMPS is " safer " in a withholding child. 2. Can ALA or other chelators (DM's) safely chelate a child with very low (out of normal range) circulating glutathione? ie with gluathione gene deletion without supplementation? I mentioned in last post that we have glutathione gene (liver kidney only - not brain form) missing, and as a result have TD glutathione to slather on our daughter every day. My understanding based on the EWG.org (environmental working group website) is that the substances in the TD cream that " unzip " the skin cells and " force " the glutatathione/nac in are all either carcinogenic or endocrine disrupting (confirmed by our compounding pharmacist). Oral glutathione liposomal is nearly inedible /unhideable. If we can eliminate oral and transdermal glutathione without significantly raising our risk of redistribution or just chelation not working, we want to. (Not to mention the extra cost of course). Your last post stated that glutathione is not a " hand off " for mercury via ALA or any other chelator. Does this mean that we can stop worrying about glutathione supplementation, since up to now it appears it has had NO significant effect (when I stop I notice no difference from when we use it)? In other words, can we use ALA alone (or DM's plus ALA) without " needing " glutathione there in normal levels? 3. Does any of the above change if the child is not pooping for 5-7 days at a time? 4. Does any of the above change if you use " TTFD " (modified B-1/thiamine) as a chelator? Just curious as I see it in the DAN literature and wonder if its behavior is consistent with the DMs or ALA, since I haven't noticed it getting mentioned in this group yet. Is TTFD something useful for chelation esp. in an irregularly pooping child? 5. Does your book still contain current info on your current positions/understandings? I just purchased it and wanted to review dose, schedule, chelator info - what would you change since it was published esp. pertaining to dosages, timing, practical chelating info. Quote Link to comment Share on other sites More sharing options...
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