Guest guest Posted May 29, 2001 Report Share Posted May 29, 2001 No can wait,. But it wasn't the surgeon who prescribed the fosamax. It was the primary care dr who I'm going to have to have a talk with, and I didn't take the med. Believe me I will be in touch with you all and I hope it isn't to find that surgeon in Texas who specializes in reconstructive surgery. But what the heck...what is his name and where is he? Do you know who I am talking about? Joyce --- Racine <lindaracine@...> wrote: > Hi Joyce... > > I know you really don't want to hear this, but > you've sent up so many > red flags, that I've begun to be seriously > concerned. It's possible > that you'll have the surgery and have a great > outcome. And, I hope > that's the case, but what if you don't? I know it > seems like you don't > have any alternatives at this moment. I'm afraid > that, if you have a > bad outcome, you'll hate yourself for rushing into > this revision surgery > so fast without investigating the alternatives. > Isn't there some way > that your husband can have his knee surgeries first > and recover while > you do your research? > > Regards, > > __________________________________________________ Quote Link to comment Share on other sites More sharing options...
Guest guest Posted May 30, 2001 Report Share Posted May 30, 2001 Joyce... You're probably thinking of Mike LaGrone. He's in Amarillo. -- Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 19, 2007 Report Share Posted December 19, 2007 So much help. Reviewing histiory here is our deal: 1) Too much eye contact Mrs. , not autistic, not sure about the language, it will come. No one could ever explain why it came and left! 2) Started the signs of walking and then regressed for 3 months. 3) Intermittent loss of strength early on...milk I believe as this happened when that was his only food. 4) Petit Mal siezures as a baby. 5) YES! 6) Peekaboo progress when on allergen. More steady progress now. 7) Celiac sister (though it took his stuff for us to find out). 8) Lost weight last year. > > I found this on another group--It talks about autism, but I thought > it had good info that might help some with apraxia also. > Tina > > Dr. Marvin Natowicz is a neurogeneticist previously practicing at > Mass General Hosp., Boston and the Eunice Kennedy Shriver Center in > Waltham, MA where he was the Medical Director of Genetics. He is now > a member of the metabolic team at the Cleveland Clinic. Natowicz is > specifically interested in metabolic disorders in autism and, in a > 1999 Boston based " LADDERS " lecture, enumerated a number of " red > flags " which invite investigation into underlying metabolic > (including mito) disease in autism: > > Red flags requiring further scrutiny by metabolic clinicians: > > 1. The autism is not classic and/or the diagnosis is not > straightforward when observed by credible specialists. Examples of > this are children who may score as autistic or PDD-NOS by DSM-IV > criteria because they have language, social and behavioral deficits. > However, professionals often say that they have " too much eye > contact " or a certain " eye quality " or are " too social " even though > their social skills are below expectations for developmental age. > Diagnosticians use terms like " atypical autism " or " features of > atypical autism, " or they may say, it's " not quite autism " but we're > not sure what it is either. This is a " squishy " diagnosis. > > 2. Developmental regression: Because some 25-33% of autism is > regressive in the first year of life, some clinicians discard these > kids as unworthy of further scrutiny. Loss of previously attained > skills is always significant and should be carefully regarded by > medical professionals. Video documentation is very helpful. > > 3. Neurological regression: This might manifest as loss of > muscle strength or physical ability, easy fatigue or lethargy. Be on > the look out for intermittent loss. > > 4. Seizures: Some 33% or more of children with autism are > expected to show EEG abnormality or seizure activity in their > lifetime so many clinicians discard this very important marker for > metabolic stress. > > 5. Food intolerances or avoidance: If foods cause changes in > neurological status, this is significant for metabolic disorder. A > child who has typical or near typical muscle skills but becomes > frankly ataxic upon eating a certain food, may have a " leaky form " or > partial defect associated with a given metabolic disorder. For > example, children with less advanced maple syrup urine disease (MSUD) > can become clumsy after eating foods high in branched chain amino > acids (generally proteins). The disorder may be more apparent under > circumstances where there is a greater catabolic demand on the body > such as during fasting (i.e. overnight) or infection. For this > reason, first in the AM urine is often preferred for analysis. This > underscores the need to collect urine samples during times of obvious > unbalance or muscle loss. > > 6. Given the proper educational, behavioral and therapeutic > supports, children with autism are capable of learning. When > children do not learn (or lose cognitive skills), one may first > question whether the child is being taught appropriately. If the > answer is " yes " or if the educational piece is corrected and the > child still does not make progress, metabolic scrutiny is often > appropriate. When observed together with one or more other " red > flags, " lack of learning in autism demands scrutiny. > > 7. Family history: a second affected sibling cries out for > metabolic scrutiny. I would venture to add here that families who > have a history of miscarriage along with an affected child, should > demand further metabolic work up in their child. > > 8. Unusual findings on physical examination including: > *growth retardation or excessive growth > *small head circumference esp. if this declines over time > relative to over-all-size > *significant motor dysfunction > *atypical biochemical findings [examples include but not limited > to low blood CO2, high blood ammonia, liver function abnormalities, > creatine phosphokinase (CPK) abnormalities indicative of muscle > injury, etc.. Some clinicians feel that values must be at least 2 > standard deviations from the mean in order to be significant. Most > agree that flagged values (i.e. any value outside the normal > reference range) warrent a repeat blood draw for validation.] > Quote Link to comment Share on other sites More sharing options...
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