Omega-3 Question

[Guest guest]

I have my 3 and 1/2 year old son on Omega -3 pills.I give him 2 pills in the

morning and 2 pills at night. Does it matter tha it's not Pro-Efa? Just as long

as he get the right amount of DHA and EPA. Or does it have to be Pro-Efa?

Haydee

[Guest guest]

Nick, in the case I was reffering to they said that the man excluded ALL

plant-based oils and was taking only fish and algae oild for omega-3. That was

my question - why was the flax oil excluded, why they said that plan-oils are

tumor-promoting. I would understand tumor promotion of safflower oil as it's

high in omega-6, but he was not taking flax as well.

> What made you worry that flax oil would be considered tumor promoting?

>

> In all the studies I recall reading it was the opposite - tumor growth

> inhibiting (as is the case for omega-3 fats in general).

>

[Guest guest]

Is lard good for cooking?

> It is the " normal " vegetable cooking oils which replaced coconut oil and

> lard which cause so many health problems.

>

[Guest guest]

It is possible that flaxseed oil was included because it is a phytoestrogen.

Some oncologists fail to understand that not all phytoestrogens are created

equally. So, they just lump everything into one category.

ar

>

> Nick, in the case I was reffering to they said that the man excluded ALL

plant-based oils and was taking only fish and algae oild for omega-3. That was

my question - why was the flax oil excluded, why they said that plan-oils are

tumor-promoting. I would understand tumor promotion of safflower oil as it's

high in omega-6, but he was not taking flax as well.

>

> > What made you worry that flax oil would be considered tumor promoting?

> >

> > In all the studies I recall reading it was the opposite - tumor growth

> > inhibiting (as is the case for omega-3 fats in general).

> >

>

[Guest guest]

No, lard is not good for cooking.

>

> Is lard good for cooking?

> > It is the " normal " vegetable cooking oils which replaced coconut oil and

> > lard which cause so many health problems.

> >

>

[Guest guest]

> Can someone please explain me if flax oil would be considered

tumor-promoting? It's a bit confusing because flax oil is also high in

omega-3.

You might want to think about using Flaxseed as well as Flaxseed Oil.

In this study:

http://www.ncbi.nlm.nih.gov/pubmed/15849746

They write:

Previous studies have shown that the n-3 fatty acid, alpha-linolenic

acid (ALA), as well as its metabolites, eicosapentaenoic (EPA) and

docosahexaenoic acid (DHA), and secoisolariciresinol diglycoside (SDG),

the precursor to the mammalian lignans, enterolactone (EL) and

enterodiol (ED), can inhibit the various stages of mammary

carcinogenesis. Flaxseed (FS) contains the highest level of SDG, with

values 75–800 times those in other plant

foods, and is also the richest source of ALA, suggesting that it may be

protective against mammary cancer.

Dietary supplementation of 5 or 10% FS indeed has been shown to inhibit

the initiation and early and late promotion stages of mammary

carcinogenesis in carcinogen-treated rats.

They conclude:

The anti-metastatic effect induced by Flaxseed may be attributed to both

the lignan and oil components.

[Guest guest]

Unfortunately, this study only talks about ER negative breast cancers. The fear

is still that the phytoestrogen properties of flaxseeds will cause ER positive

tumors to grow. My personal opinion is that it won't, but my personal opinion

doesn't account for much.

ar

>

> You might want to think about using Flaxseed as well as Flaxseed Oil.

>

> In this study:

> http://www.ncbi.nlm.nih.gov/pubmed/15849746

>

> They write:

>

> Previous studies have shown that the n-3 fatty acid, alpha-linolenic

> acid (ALA), as well as its metabolites, eicosapentaenoic (EPA) and

> docosahexaenoic acid (DHA), and secoisolariciresinol diglycoside (SDG),

> the precursor to the mammalian lignans, enterolactone (EL) and

> enterodiol (ED), can inhibit the various stages of mammary

> carcinogenesis. Flaxseed (FS) contains the highest level of SDG, with

> values 75–800 times those in other plant

> foods, and is also the richest source of ALA, suggesting that it may be

> protective against mammary cancer.

> Dietary supplementation of 5 or 10% FS indeed has been shown to inhibit

> the initiation and early and late promotion stages of mammary

> carcinogenesis in carcinogen-treated rats.

>

> They conclude:

>

> The anti-metastatic effect induced by Flaxseed may be attributed to both

> the lignan and oil components.

>

[Guest guest]

>

> Is lard good for cooking?

> > It is the " normal " vegetable cooking oils which replaced coconut oil

and

> > lard which cause so many health problems.

Lard is good for cooking in that it doesn't oxidize and go rancid at

high temperatures like most vegetables oils. It has always been a good

traditional cooking oil which didn't cause problems. However, animal

fats these days come from animals that are raised on hormones,

antibiotics and other chemicals, which probably concentrates in the fat.

The animals tend to be raised on grain, rather than grass-fed. So it's a

good idea to choose your source carefully.

But I would choose organic cold pressed virgin coconut oil for cooking

and other use. It won't go rancid when cooking and increases HDL (good)

cholesterol and strengthens the immune system.

Coconut Oil and Cancer

http://www.westonaprice.org/knowyourfats/coconut_oil.html

Lim-Sylianco (1987) has reviewed 50 years of literature showing

anticarcinogenic effects from dietary coconut oil. These animal studies

show quite clearly the nonpromotional effect of feeding coconut oil.

In a study by Reddy et al (1984) straight coconut oil was more

inhibitory than MCT oil to induction of colon tumors by azoxymethane.

Chemically induced adenocarcinomas differed 10-fold between corn oil

(32%) and coconut oil (3%) in the colon. Both olive oil and coconut oil

developed the low levels (3%) of the adenocarcinomas in the colon, but

in the small intestine animals fed coconut oil did not develop any

tumors while 7% of animals fed olive oil did.

Studies by Cohen et al (1986) showed that the nonpromotional effects of

coconut oil were also seen in chemically induced breast cancer. In this

model, the slight elevation of serum cholesterol in the animals fed

coconut oil was protective as the animals fed the more polyunsaturated

oil had reduced serum cholesterol and more tumors. The authors noted

that " ...an overall inverse trend was observed between total serum

lipids and tumor incidence for the 4 [high fat] groups. "

[Guest guest]

I believe that the reason fish and algae oils were used was because the

subject of the article decided that EPA and DHA rich oils were his best

bet - very likely due to the fact that he lived next door to Prof.

Pardini who was and is doing research on Omega-3 fats and cancer. Prof.

Pardini's focus was on fish and algae oils, so I'm guessing that's why

the focus was not on flax oil.

As a general comment: I have read many articles on the effectiveness of

the various omega-3 fats on tumor growth and at this point, the evidence

is somewhat mixed in terms of which are the most effective for

inhibiting or regressing tumor growth. If my memory serves me, EPA and

DHA have on balance appeared to show a stronger effect, but it should be

noted that ALA, EPA and DHA may have specific and possibly different

mechanisms by which they act on tumor growth. There may also be

co-factors present in the sources of these fatty acids that also come

into play.

Kind regards,

Nick

On Tue, 22 Sep 2009, katerinka70 wrote:

> Nick, in the case I was reffering to they said that the man excluded

> ALL plant-based oils and was taking only fish and algae oild for

> omega-3. That was my question - why was the flax oil excluded, why

> they said that plan-oils are tumor-promoting. I would understand tumor

> promotion of safflower oil as it's high in omega-6, but he was not

> taking flax as well.

>

> > What made you worry that flax oil would be considered tumor promoting?

> >

> > In all the studies I recall reading it was the opposite - tumor growth

> > inhibiting (as is the case for omega-3 fats in general).

[Guest guest]

" arlynsg " wrote:

> Unfortunately, this study only talks about ER negative breast cancers. The

fear is still that the phytoestrogen properties of flaxseeds will cause ER

positive tumors to grow. My personal opinion is that it won't, but my personal

opinion doesn't account for much.

>

If you search Google Scholar, you'll find most of the studies seem to be

on ER+ bc with flaxseed. So I think this one was particularly useful for

people with ER- bc who may wonder whether FS will benefit them or not.

Here's one I found previously on ER+ bc:

Dietary Flaxseed Enhances the Inhibitory Effect of Tamoxifen on the

Growth of Estrogen-Dependent Human Breast Cancer (MCF-7) in Nude Mice

http://clincancerres.aacrjournals.org/content/10/22/7703.full.pdf

Conclusions: FS inhibited the growth of human estrogen- dependent breast

cancer and strengthened the tumorinhibitory effect of TAM at both low

and high E2 levels.

Discussion:

This study showed for the first time that dietary FS can inhibit the

growth of human ER+ breast cancer, and enhance rather than antagonize

the anticancer effect of TAM in nude mice in the presence of low or high

E2 level, which mimics the hormone status of post- and premenopausal

women, respectively.

It also agrees with the results from a double-blinded placebocontrolled

clinical trial, which showed that 25 g of FS reduces the tumor cell

proliferation of preoperative breast cancer in postmenopausal patients.

The mammalian lignans enterodiol and enterolactone, derived from

flaxseed SDG, possess weak and/or antiestrogenic activities, which may

be ER-mediated.

Although lignans promoted MCF-7 tumor cell growth in vitro with E2

deprivation because of weak-estrogen activity (24), this was not

observed in ovariectomized athymic mice in this study. In fact, the

present study showed that FS regressed the tumors to a similar extent as

the negative control, and failed to stimulate cell proliferation and

uterus development in mice after E2 pellet withdrawal, suggesting that

FS does not elicit an estrogenic effect on the tumor and uterus.

In addition to ER-mediated effects, lignans can modulate the activities

of enzymes involved in estrogen biosynthesis and metabolism. Lignans

have been shown to moderately inhibit the activity of aromatase, an

estrogen synthetase converting androgen to estrogen, in human tissues

and MCF-7 cells.

Continues....

I'd say flaxseed might be good enough without Tamoxifen, if that's what

one wants to do.

Ketogenic flaxseed and coconut cookie

------------------------------------------------

2 tbsp coconut oil

15 g butter

4 tbsp flaxseed, ground

1 tsp cinnamon

Pure Stevia

Mix and form into a cookie shape, place on baking paper on top of a

small plate. Put in fridge until formed.

" This cookie packs it all. Coconut oil (MCT), Flax Seed (Omega-3), and

Cinnamon as a glucose disposal agent. Eat one of these and you will be

humming along all day. Great as a meal replacement. "

Modified from this recipe, which contains almond, also probably great.

http://forum.lowcarber.org/showthread.php?t=288667 & page=1 & pp=15