Re: My experiences as AFIB sufferer for 12 years

[Guest guest]

> In the case of AF, and here is a bit of speculation, it is

> conceivable that the infection/inflammation interferes with the

> electrical signals sent out by a group of cells called the

> sino-atrial node /SA node in the heart, that is electrically

> controlling the beating of the heart. The end result is S A, instead

> of sending signals for the normal 72 beats per minute is sending

> chaotic signals to the upper heart atria, causing it to beat up to

> 350 times per minute.

>

Hi Alfred, welcome to the group. I'm a fan of inflammation playing a

role in some people and there's already some research on this. e.g.

C-reactive protein and atrial fibrillation. Is inflammation a

consequence or a cause of atrial

fibrillation?http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubme\

d & dopt=Abstract & list_uids=15240964

Inflammation as a risk factor for atrial fibrillation.

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstra\

ct & list_uids=14623805

C-reactive protein and paroxysmal atrial fibrillation: evidence of the

implication of an inflammatory process in paroxysmal atrial

fibrillation.

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstra\

ct & list_uids=11791805

(and much more can be found at PubMed)

Though I confess I don't recall reading about Cardiac-CRP and am a

little surprised the research dealing with heart inflammation doesn't

mention it.

Once inflammation is there though is it not possible that something other than

the SA node starts the

process of AF off? For example the inflammation might produce some conduction

block and make it

more likely for an ectopic to trigger AF. I don't believe there is much evidence

that the SA node

'drives' AF in many cases.

All the best

--

D

[Guest guest]

snip........

> I was an AFIB sufferer for twelve years. As a doctor and scientist,

> when I discovered I had AFIB, I began an intensive study into the

> origins, behavior, and treatment of the disease. What I have found

> along the way, I hope will be able to help many others, as it has

> helped me.

>

> The following is information regarding & #65279;THE CARDIAC C-

REACTIVE PROTEIN

> (C-CRP)......snip

Dr. Sapse, thank you very, very much for this information about C-CRP, and

your additional insights. I will continue to explore this rapidly evolving

subject.

I do have a quick question, though. Pulmonary Vein Ablation procedures are

increasingly effective in bringing AF sufferers back into nsr, particularly in

the

hands of such experienced physicians as Dr. Natale at the Cleveland Clinic,

who has helped hundreds of AF patients return to nsr via PVA. Could you help

me understand why there is a connection between infection-induced alterations

in the electrically excitable cells of the SA nodal region, and correction by

PVA

electrical isolation of the openings of the pulmonary veins into the ventricle?

Thank you very much once again. Kathleen (24/7, atenolol, digoxin,

coumadin).