Re: lowered pain threshold

[Guest guest]

I remember seeing this answer from Dr. Lowe about the heightened pain

sensitivity:

A subset of patients with thyroid hormone deficiency caused by Hashimoto's

has a lowered pain threshold. The susceptible patient perceives as painful

stimuli that aren't painful to other people. The pain results from too little

thyroid hormone regulation of certain nerve cells. Some of the cells, mainly in

her

spinal cord, when under-regulated by thyroid hormone, release excess amounts

of " substance P. " The excess substance P then amplifies the transmission of

" pain " impulses in the central nervous system.

Too little thyroid hormone regulation of other cells in the brain stem

decreases the release in the spinal cord of a nerve transmitting substance

called

" noradrenaline. " The decreased noradrenaline in turn reduces the amount of

opiates (morphine-like chemicals) released into the spinal cord. These opiates

normally reduce the number of sensory impulses that enter the spinal cord and

brain stem. When too few of the opiates are released, more sensory impulses make

their way into the spinal cord and brain stem. As a result, the patient's

perception of pain is heightened. 

The combination of high substance P and low noradrenaline (and hence low

opiates) causes the patient to perceive pain in the absence of painful stimuli.

For example, the patient might perceive as painful the mere movement of some

joints. She might experience pain from the pressure on her underside when she is

sits or lies on a well-padded surface. And her pain threshold might be so low

that she experiences aches and pains despite no apparent stimulus such as

movement or pressure.

My impression is that most doctors and researchers don’t know that too little

thyroid hormone regulation of cells in the brain stem and spinal cord can

induce and sustain pain. When a hypothyroid patient is under-treated or denied

treatment with thyroid hormone (the standard provisions of conventional

medicine), and her main hypothyroid symptom is chronic, widespread pain, her

doctor is

likely to diagnose her pain as " fibromyalgia. "

After the fibromyalgia diagnosis, conventional treatment will follow. This

will entail various medications that don’t correct the underlying cause of her

pain (hypothyroidism) and that are largely ineffective. Through conventional

care, her health is likely to deteriorate further over time—partly from her

continuing hypothyroidism and partly from the adverse effects of conventional

medications. To avert this from happening, I suggest that you and your daughter

promptly abandon conventional medical care, and at the same time, get her under

the care of an alternative doctor who’ll competently treat her for her

hypothyroidism. I wish her the very best.

[Guest guest]

hmmm when I had my wisdom teeth pulled (one at a time with just a

local) my oral surgeon prescribed 800 mg ibuprofin for the pain but

300 was enough for me.

Guess it depends on the person.

Louise

PS Looking back on it - even with a serious head injury I wasn't in

that much pain. The headaches were more annoying.

> I remember seeing this answer from Dr. Lowe about the heightened

pain

> sensitivity:

>

>

>

> A subset of patients with thyroid hormone deficiency caused by

Hashimoto's

> has a lowered pain threshold. The susceptible patient perceives as

painful

> stimuli that aren't painful to other people. The pain results from

too little

> thyroid hormone regulation of certain nerve cells. Some of the

cells, mainly in her

> spinal cord, when under-regulated by thyroid hormone, release

excess amounts

> of " substance P. " The excess substance P then amplifies the

transmission of

> " pain " impulses in the central nervous system.

>

> Too little thyroid hormone regulation of other cells in the brain

stem

> decreases the release in the spinal cord of a nerve transmitting

substance called

> " noradrenaline. " The decreased noradrenaline in turn reduces the

amount of

> opiates (morphine-like chemicals) released into the spinal cord.

These opiates

> normally reduce the number of sensory impulses that enter the

spinal cord and

> brain stem. When too few of the opiates are released, more sensory

impulses make

> their way into the spinal cord and brain stem. As a result, the

patient's

> perception of pain is heightened. 

>

> The combination of high substance P and low noradrenaline (and

hence low

> opiates) causes the patient to perceive pain in the absence of

painful stimuli.

> For example, the patient might perceive as painful the mere

movement of some

> joints. She might experience pain from the pressure on her

underside when she is

> sits or lies on a well-padded surface. And her pain threshold might

be so low

> that she experiences aches and pains despite no apparent stimulus

such as

> movement or pressure.

>

> My impression is that most doctors and researchers don’t know

that too little

> thyroid hormone regulation of cells in the brain stem and spinal

cord can

> induce and sustain pain. When a hypothyroid patient is under-

treated or denied

> treatment with thyroid hormone (the standard provisions of

conventional

> medicine), and her main hypothyroid symptom is chronic, widespread

pain, her doctor is

> likely to diagnose her pain as " fibromyalgia. "

>

> After the fibromyalgia diagnosis, conventional treatment will

follow. This

> will entail various medications that don’t correct the underlying

cause of her

> pain (hypothyroidism) and that are largely ineffective. Through

conventional

> care, her health is likely to deteriorate further over

time†" partly from her

> continuing hypothyroidism and partly from the adverse effects of

conventional

> medications. To avert this from happening, I suggest that you and

your daughter

> promptly abandon conventional medical care, and at the same time,

get her under

> the care of an alternative doctor who’ll competently treat her

for her

> hypothyroidism. I wish her the very best.

>

>

>

>

>

>

>

>

[Guest guest]

hmmm when I had my wisdom teeth pulled (one at a time with just a

local) my oral surgeon prescribed 800 mg ibuprofin for the pain but

300 was enough for me.

Guess it depends on the person.

Louise

PS Looking back on it - even with a serious head injury I wasn't in

that much pain. The headaches were more annoying.

> I remember seeing this answer from Dr. Lowe about the heightened

pain

> sensitivity:

>

>

>

> A subset of patients with thyroid hormone deficiency caused by

Hashimoto's

> has a lowered pain threshold. The susceptible patient perceives as

painful

> stimuli that aren't painful to other people. The pain results from

too little

> thyroid hormone regulation of certain nerve cells. Some of the

cells, mainly in her

> spinal cord, when under-regulated by thyroid hormone, release

excess amounts

> of " substance P. " The excess substance P then amplifies the

transmission of

> " pain " impulses in the central nervous system.

>

> Too little thyroid hormone regulation of other cells in the brain

stem

> decreases the release in the spinal cord of a nerve transmitting

substance called

> " noradrenaline. " The decreased noradrenaline in turn reduces the

amount of

> opiates (morphine-like chemicals) released into the spinal cord.

These opiates

> normally reduce the number of sensory impulses that enter the

spinal cord and

> brain stem. When too few of the opiates are released, more sensory

impulses make

> their way into the spinal cord and brain stem. As a result, the

patient's

> perception of pain is heightened. 

>

> The combination of high substance P and low noradrenaline (and

hence low

> opiates) causes the patient to perceive pain in the absence of

painful stimuli.

> For example, the patient might perceive as painful the mere

movement of some

> joints. She might experience pain from the pressure on her

underside when she is

> sits or lies on a well-padded surface. And her pain threshold might

be so low

> that she experiences aches and pains despite no apparent stimulus

such as

> movement or pressure.

>

> My impression is that most doctors and researchers don’t know

that too little

> thyroid hormone regulation of cells in the brain stem and spinal

cord can

> induce and sustain pain. When a hypothyroid patient is under-

treated or denied

> treatment with thyroid hormone (the standard provisions of

conventional

> medicine), and her main hypothyroid symptom is chronic, widespread

pain, her doctor is

> likely to diagnose her pain as " fibromyalgia. "

>

> After the fibromyalgia diagnosis, conventional treatment will

follow. This

> will entail various medications that don’t correct the underlying

cause of her

> pain (hypothyroidism) and that are largely ineffective. Through

conventional

> care, her health is likely to deteriorate further over

time†" partly from her

> continuing hypothyroidism and partly from the adverse effects of

conventional

> medications. To avert this from happening, I suggest that you and

your daughter

> promptly abandon conventional medical care, and at the same time,

get her under

> the care of an alternative doctor who’ll competently treat her

for her

> hypothyroidism. I wish her the very best.

>

>

>

>

>

>

>

>

[Guest guest]

Here I am suddenly thinking about that poor man who gave a talk in our

Medical Terminology class at the local college. This poor man had such

severe ongoing pain from unknown causes, but was diagnosed with

Fibromyalgia. He had such severe pain for several yrs that he had one of

those pain med pumps inserted around the spinal area. What a horrible

thought that someone actually had hypothyroid that noone would diagnose, and

I KNOW now that that was what was wrong with that man. What a shame!!

Fibromyalgia wasn't " invented " until the advent of the almighty god like TSH

test and it's position as the diagnostic tool for thyroid treatment!

Re: lowered pain threshold

>

> I remember seeing this answer from Dr. Lowe about the heightened pain

> sensitivity:

>

>

>

> A subset of patients with thyroid hormone deficiency caused by Hashimoto's

> has a lowered pain threshold. The susceptible patient perceives as painful

> stimuli that aren't painful to other people. The pain results from too

little

> thyroid hormone regulation of certain nerve cells. Some of the cells,

mainly in her

> spinal cord, when under-regulated by thyroid hormone, release excess

amounts

> of " substance P. " The excess substance P then amplifies the transmission

of

> " pain " impulses in the central nervous system.

>

> Too little thyroid hormone regulation of other cells in the brain stem

> decreases the release in the spinal cord of a nerve transmitting substance

called

> " noradrenaline. " The decreased noradrenaline in turn reduces the amount of

> opiates (morphine-like chemicals) released into the spinal cord. These

opiates

> normally reduce the number of sensory impulses that enter the spinal cord

and

> brain stem. When too few of the opiates are released, more sensory

impulses make

> their way into the spinal cord and brain stem. As a result, the patient's

> perception of pain is heightened.

>

> The combination of high substance P and low noradrenaline (and hence low

> opiates) causes the patient to perceive pain in the absence of painful

stimuli.

> For example, the patient might perceive as painful the mere movement of

some

> joints. She might experience pain from the pressure on her underside when

she is

> sits or lies on a well-padded surface. And her pain threshold might be so

low

> that she experiences aches and pains despite no apparent stimulus such as

> movement or pressure.

>

> My impression is that most doctors and researchers don’t know that too

little

> thyroid hormone regulation of cells in the brain stem and spinal cord can

> induce and sustain pain. When a hypothyroid patient is under-treated or

denied

> treatment with thyroid hormone (the standard provisions of conventional

> medicine), and her main hypothyroid symptom is chronic, widespread pain,

her doctor is

> likely to diagnose her pain as " fibromyalgia. "