Re: SchaferAutismReport: Researchers Identify Critical Gene for Brain Development, Mental Retardation [7 Attachments]

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Does anyone have a problem with their ASD child chewing on EVERYTHING? My 7 year old bites and chews on wood, leather, metal, hard plastics (that splinter when he bites them into pieces). Has anyone come up with remedies for chewing? Are there supplements, meds, etc. that have worked, to prevent excessive chewing?

Sara

From: schafer <schafersprynet (DOT) com>Subject: Researchers Identify Critical Gene for Brain Development, Mental RetardationTo: "Schafer Autism Report " <sarnetslists (DOT) igc.org>Date: Friday, September 4, 2009, 2:28 PM

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RESEARCHResearchers Identify Critical Gene for Brain Development, Mental RetardationFragile Period of Childhood Brain Development Could Underlie EpilepsyPUBLIC HEALTHCDC Says Special Needs Children Should Be First In Line For H1N1 VaccineSwine Flu Vaccine Doses To Contain Mercury AdditiveEDUCATIONEmbattled Teacher Will Retain Her JobPEOPLECaregiver Guilty of Assault, Deprivation of LibertyMetro Transit To Apologize To Autistic BoyMEDIA McCarthy Will Have Own Talk ShowEVENTSFinding the TruthCOMMENTARYWhy We Must Ban BPA From Products Made For Children

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Hundreds of Local Autism EventsRESEARCHResearchers Identify Critical Gene for Brain Development, Mental Retardation From sflorg.com is.gd/2STHb In laying down the neural circuitry of the developing brain, billions of neurons must first migrate to their correct destinations and then form complex synaptic connections with their new neighbors. When the process goes awry, neurodevelopmental disorders such as mental retardation, dyslexia or autism may result. Researchers at the University of North Carolina at Chapel Hill School of Medicine have now discovered that establishing the neural wiring necessary to function normally depends on the ability of neurons to make finger-like projections of their membrane called filopodia. The finding, published as the cover story of the Sept. 4 issue of the journal Cell, indicates that the current notion regarding how cells change shape, migrate or differentiate needs to be revisited. Scientists

have thought that the only way for a cell to morph and move is through the action of the cytoskeleton or the scaffold inside the cell, pushing membrane forward or sucking it in, said senior study investigator Franck Polleux, Ph.D., associate professor of pharmacology at the UNC School of Medicine. But Polleux’s study shows that the brain protein srGAP2 can also impose cell shape by directly bending membranes, forming filopodia as a mean to control the migration and branching of neurons during brain development. Interestingly, srGAP2 is one of a family of proteins that have been implicated in a severe mental retardation syndrome called the 3p- syndrome. Therefore this research could also yield important insights into the underlying causes of this and other forms of mental retardation. Polleux and his colleagues began looking at srGAP2 because the gene

was almost exclusively “turned on†or expressed during brain development. The brain protein contains a unique combination of

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