Re: interesting article from National Post

[Guest guest]

This is very interesting. You know Prof. Nicholson is

now studying Autism and mycoplasma. Can you imagine

the list of diseases that are possibly caused by a

bacteria. I feel 99.9% sure that my daughter's

Vitiligo (skin whitening disease - body is killing

pigments) is caused from the mycoplasma hominis that

she has tested positive for.

--- Bryarwoode@... wrote:

> January 25, 2001

>

>

> Schizophrenia linked to cat feces

>

>

> Phil Surguy

> National Post

> Researchers in the United States have found evidence

> that cats really do

> drive people mad.

>

> Dr. Yolken, a pediatrician at s Hopkins

> University in Baltimore,

> and Dr. Fuller Torrey, director of the Stanley

> Foundation Research Programs

> in Bethesda, Md., believe schizophrenia and possibly

> bipolar disorders such

> as manic depressive illness are caused by an

> infectious agent acting on the

> brain. And one of their prime suspects is a parasite

> called toxoplasma

> gondii, which is spread to humans by cat feces.

>

> The parasite can cause toxoplasmosis, a generally

> mild illness. Most people's

> immune systems will cut short a T. gondii invasion

> without their ever knowing

> it. However, pregnant women who contract the

> parasite can transmit it to the

> fetus, with devastating effects on brain

> development.

>

> Dr. Torrey and Dr. Yolken believe the parasite can

> enter the fetal brain, lie

> dormant for 15 to 30 years, then activate and induce

> schizophrenia.

>

> Pathologists at the Stanley Foundation Brain Bank in

> Bethesda are searching

> for signs of T. gondii infection in the brains of

> people who suffered

> schizophrenia, bipolar disorder or severe

> depression.

>

> They are about to begin clinical trials in which

> schizophrenics will be given

> drugs normally used to treat the infection.

>

> Dr. Torrey and Dr. Yolken began to suspect a link

> between schizophrenia and

> the cat-borne infection because people with the

> disease are more likely to

> have been born in late winter or early spring than

> the general population --

> and cats stay inside and use their litter boxes more

> in winter.

>

> They also noted bipolar disorder and schizophrenia

> were relatively rare in

> Europe until the late 19th century, when cats became

> popular as pets.

>

> Also, they found in one test that children who went

> on to develop

> schizophrenia were more likely to come from homes

> with cats.

>

> Blood samples taken between 1959 and 1966 from 2,500

> pregnant women in

> Providence, R.I., have revealed the mothers of

> children who later exhibited

> psychoses were more than four times more likely to

> have antibodies to

> toxoplasmosis, one of the diseases that can have

> schizophrenia-like symptoms.

>

> But, Dr. Torrey cautions: " Don't get rid of your

> cats yet. We haven't proven

> anything. "

>

> He and Dr. Yolken are also considering other

> possible agents, particularly

> the herpes viruses, which can lie dormant for long

> periods, and which have

> symptoms that wax and wane like those of

> schizophrenia.

>

> " They all have a strong affinity for brain tissue, "

> Dr. Torrey adds. He says

> the herpes virus HSV1 " in particular has a strong

> affinity for the same part

> of the brain that we're looking at in

> schizophrenia. "

>

> In one of the upcoming clinical trials,

> schizophrenia patients will be given

> acyclovir, an anti-viral drug used to treat herpes.

>

> In the other trial, patients will be given

> antibiotics used in the treatment

> of toxoplasmosis.

>

> Dr. Seeman, a University of Toronto psychiatry

> professor and the first

> holder of the Tapscott Chair for Studies in

> Schizophrenia, says Dr. Torrey's

> ideas are " just as plausible as any other theory of

> schizophrenia, since the

> causes are completely unknown, other than that genes

> are involved.

>

> " These days, infections have been shown to be

> associated with peptic ulcers

> and heart attacks, so why not schizophrenia? "

>

> She adds a bacterium has been identified as a cause

> of obsessive compulsive

> disorders, " which many people used to think was

> caused for psychological

> reasons. "

>

>

>

>

------------------------------------------------------------------------------

>

> --

>

>

>

>

__________________________________________________

[Guest guest]

That is very interesting, , and thanks for posting it. If you see

anything about mycoplasma and autism, could you please let us know? I saw

something on an arthritis newsgroup a couple of weeks ago about a cats and

RA study at Royal Adelaide Hospital. Anyone know anything about that one?

Wouldn't you know if you were pregnant and contracted toxoplasmosis? I mean,

wouldn't there be obvious symptoms? I had cats through all my pregnancies,

but did *not* change the litter box myself, because of the birth defect

warning they give about that.

And how is Adia doing? Liz G.

[Guest guest]

Adia is doing fine. She seems to be healthy except

for the Vitiligo. She turned 1 last Thursday.

Prof. Nicholson is researching autism and mycoplasma.

He has some information on his web-site -

www.immed.org.

--- " Liz G. " <pioneer@...> wrote:

> That is very interesting, , and thanks for

> posting it. If you see

> anything about mycoplasma and autism, could you

> please let us know? I saw

> something on an arthritis newsgroup a couple of

> weeks ago about a cats and

> RA study at Royal Adelaide Hospital. Anyone know

> anything about that one?

>

> Wouldn't you know if you were pregnant and

> contracted toxoplasmosis? I mean,

> wouldn't there be obvious symptoms? I had cats

> through all my pregnancies,

> but did *not* change the litter box myself, because

> of the birth defect

> warning they give about that.

>

> And how is Adia doing? Liz G.

>

>

__________________________________________________

[Guest guest]

Hi Liz,

The following site should have the article on cats as a possible

cause or carrier of RA.

http://www.docguide.com/news/content.nsf/news/5555F262DC9CD4CD852569B3

00712BD7?

OpenDocument & id=5753362958dfd5e9852569d000691221 & c=Rheumatoid%

20Arthritis & count=10

It is supposed to read as one long line.....looks like you might have

to cut and paste...if you can't get it to work, let me know and I can

send it from my Outlook files.

take care and thanks for all of your posts,

Connie

> I saw

> something on an arthritis newsgroup a couple of weeks ago about a

cats and

> RA study at Royal Adelaide Hospital. Anyone know anything about

that one?

>

[Guest guest]

I want to read the article but I only get a page that says there was an

error from the docguide page.

Thanks,

Gloria

[Guest guest]

The cat connection is interesting to me because I read somewhere that a

person can aquire " Ascaris Lumbricoides " nematode from cat fur where the

eggs may lie. Ascaris is a worm that lives in the lung and hatches eggs

every night in its host it migrates up to the throat and then when host

swallows the eggs (I believe) ar redeposited into the small intestine where

they migrate to liver and then again to lungs. when the eggs hatch they

also release a bunch of toxins, bacterial, viral and fungal (I believe) and

thus our immune system would have a daily assault from numerous sources if

infected.

I had a darkfield microscopy (electron microscope) of my live blood and

daughters live blood...we saw an ascaris Lumbricordes. Another patient who

has Ankylosing also had the ascaris in her blood as well. And....its

difficult to eradicate the eggs...not by heat or cold, therefore difficult

to cure this infection. Some sources say that its difficult to kill the

adult worms even with medicines (antiparasitic). What I find even more

fascinating is that some German researchers found that a different type of

nematode called Filariasis (also a worm, but it lives in the lymphatic

primarily) they found that by treating patients with Doxcycline, the doxy

rendered the nematode sterile, ie it wasn't able to kill the mature worm, ie

the worms were no longer able to lay thousands of eggs nightly that the

immune system would have to cope with (as well as the toxins associated

with the hatching of the egg.) Could one of the benefits of the Minocycline

or Doxy be acting on a hidden nematode infection that causes RA. Another

interesting point is that the symptoms of these infections overlap symptoms

of RA....skin itchiness, lung involvement, bone spurs (in case of

filariasis) eye involvement (in the case of filariasis) endema, arthalgias,

skin depigmentation (symptom of filariasis) GI involvement....and more. So

many symptoms overlap. This may not make much sense to many people but I

believe that RA is a parasitic infection of one kind or another and that

Doxy may render the bug sterile and thus take a huge burden off our system.

I also believe that L-form bacteria and spirochetes and mycoplasmas also

play a role and some may be the residue or result of the parasite and others

may be infections acquired along the way, for example Lyme disease being

passed either congenitally or by a tick would not be generated from a

nematode infection.

I have Lyme (positive urine antigen test) but I have seen filariasis in my

live blood and ascaris in my daughters....when you read the symptoms of

filariasis ...so many things overlap with RA, and there exists a role for

antibiotic protocal in RA, Lyme, Filariasis and perhaps in Ascaris, and

mycoplasms.

last point....how could we possibly NOT know if we had a hug worm in

habiting our body? I believe that we are not looking for a large nematode

and that doctors don't think parasites are endemic in north america, but we

travel the world and can be infected by a mosequito or a black fly.

finally filariasis is the disease of the " elephant man " causes

elephantitis....however that is in the most extreme cases....I believe there

are many unknown cases that manifest more like RA and perhaps there is a

genetic co-factor....Its a hunch, intuition, perhaps I am mad and may undo

this theory later, but I wanted to share my thoughts....below are some

article excerpts.

Common Helminth Infections:

Battling Wormlike Parasites in Primary Care

A. VandeWaa, PhD, D. , PhD, RPh, L. White,

Jr, PA-C, MSPH, PhD, J. Nowatzke, PA-S

[Clinician Reviews 8(5):75-77, 81-82, 85-90, 92, 1998. © 1998 Clinicians

Publishing Group and & Wilkins.]

Abstract

The rising prevalence of helminth infection in the United States is likely a

result of increased travel and immigration, particularly to and from

underdeveloped countries. Helminths invade the digestive, circulatory, and

lymphatic systems, causing side effects that range from uncomfortable rectal

itching and skin rash to neurologic manifestations. Common intestinal

helminths include pinworm, whipworm, roundworm, hookworm, threadworm, and

tapeworm. Helminths that invade tissue include Trichinella spiralis, Brugia

malayi, Wuchereria bancrofti, and Onchocerca volvulus. Blood helminths

include Schistosoma species. Occasionally, humans contract such zoonotic

agents as Toxocara canis and Ancylostoma braziliense. Diagnosis is based on

stool, blood, or tissue sampling. Treatment depends on the specific helminth

identified.

Onchocerciasis -- Onchocerca volvulus

This disease is seen primarily in parts of Africa and Central and South

America. Because it may affect the host's eyes and is transmitted by the

blackfly, which breeds in fast-flowing rivers, it often is called " river

blindness. " Once inside the host, the infective larvae develop into adult

parasites that wall themselves off in subcutaneous fibrous nodules, from

which they produce microfilariae. The microfilariae are deposited in the

skin where they may cause itching and inflammation, or in the eye where they

may lead to punctate keratitis and corneal fibrosis. Ivermectin effectively

kills microfilariae in a single dose, but repeated doses are required to

maintain suppression of dermal and ocular microfilariae.[14]

Diagnosis is confirmed by skin biopsy or eye exam for microfilariae.

Avoiding blackfly bites is the chief means of prevention. In order to

eradicate adult worms, treatment may be required for 10 years or longer;

several regimens have been tried.[10]

other Web enhancements, go to:

http://www.medscape.com/CPG/ClinReviews/1998/v08.n05/c0805.02.vand/c0805.02.

vand-01.html.

Terms related to this article: Filariasis / Care and treatment ,

Onchocerciasis / Drug therapy , Worms, Intestinal and parasitic / Effect of

drugs on , Doxycycline / Therapeutic use

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Endosymbiotic bacteria in worms as targets for a novel chemotherapy in

filariasis.(Research Letters)(Brief Article)(Statistical Data Included)

Author/s: Achim Hoerauf

Issue: April 8, 2000

Endosymbiotic bacteria living in plasmodia or worm parasites are required

for the homoeostasis of their host and should be excellent targets for

chemotherapy of certain parasitic diseases. We show that targeting of

Wolbachia spp bacteria in Onchocerca volvulus filariae by doxycycline leads

to sterility of adult worms to an extent not seen with drugs used against

onchocerciasis, a leading cause of blindness in African countries.

Filariae are responsible for devastating diseases in man, including

blindness and elephantiasis, with 150 million infections worldwide. The

world community had made it a goal to interrupt transmission and to

eliminate these diseases.(1) However, present chemotherapy(1) such as

ivermectin (drug of first choice) are mainly targeted at mature

microfilariae, and not at adult worms or early embryos, leading to a

reappearance of skin microfilariae several months after treatment. Since

adult worms have a long lifespan (up to 15 years), mass treatment will have

to be maintained for many years if transmission is to be interrupted.(1)

Computer simulation shows tremendous risks in these programmes with present

drugs alone.(2) There is thus a pressing need for new antifilarial drugs

that have microfilaricidal efficacy or that show total and longlasting

suppression of embryo production, to complement microfilaricides such as

ivermectin.(1)

Evidence from work in animals shows that Wolbachia spp (order Rickettsiales)

endobacteria in filariae are targets for chemotherapy, since their depletion

by tetracycline led to degeneration and sterility of adult worms.(3,4) This

approach has not been examined in human filariasis. Therefore, we

investigated the effectiveness of targeting wolbachia in human

onchocerciasis with respect to worm fertility and survival.

In an area of Ghana outside the onchocerciasis control programme, volunteer

onchocerciasis patients aged 18-50 years who had not had ivermectin were

assigned, after informed consent, to a control group or to treatment with

doxycycline (Vibramycin, Pfizer; 100 mg orally per day) for 6 weeks. Daily

tablet intake was supervised. 4 months after the end of treatment, which was

well tolerated in all cases, onchocercomata (nodules containing one to six

female worms) were excised and coded for blinded examinations by two

independent examiners. One part of each nodule was analysed by

immunohistology for the presence of wolbachia and for morphological

alterations (ie, adult worm degeneration, disturbance of embryogenesis,

table; methods as described earlier(3)). The other part was processed for

semiquantitative PCR to quantify bacterial versus nematode DNA. Two series

of PCR reactions per nodule sample were undertaken: one in which 16S rDNA

was amplified with endobacterial primers and serial dilutions of an

endobacterial competitor plasmid against a fixed amount of sample; and the

other in which 5S rDNA was amplified with nematode primers and serial

dilutions of nematode competitor. An index was calculated between the

dilutions at equivalence of the competitor plasmids for bacterial versus

nematode DNA.

continued ...

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Terms related to this article: Filariasis / Care and treatment ,

Onchocerciasis / Drug therapy , Worms, Intestinal and parasitic / Effect of

drugs on , Doxycycline / Therapeutic use

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Lymphatic filariasis

Up to 130 million people worldwide may be infected with one of the three

lymph dwelling filariae, Wuchereria bancrofti, Brugia malayi, and B timori.

In lymphatic filariasis, infective larvae are inoculated by mosquitoes;

adult worms are found in lymph nodes or adjacent lymphatics, and offspring

(microfilariae) circulate in the blood, often only at night.

The variable clinical manifestations of lymphatic filariasis include

subclinical microfilaraemia or asymptomatic carriage of adult worms, acute

adenolymphangitis, and lymphatic obstruction (for example, hydrocoele, or

elephantiasis). The pathogenesis of adenolymphangitis and obstruction is

complex and includes immune mediated inflammatory processes; secondary

bacterial infection superimposed on lymphatic dysfunction is an important

contributing factor.[17]

In brugian filariasis, diagnosis rests on microscopic detection of the

parasite, most commonly the microfilariae. In bancroftian filariasis,

detection of circulating antigen by enzyme linked immunosorbent assay

(ELISA) or rapid immunochromatographic testing has replaced microscopy.[18]

Filarial DNA can also be detected by polymerase chain reaction.

Ultrasonography identifies adult worms in situ (commonly seen in scrotal

lymphatics as the " filaria dance sign " [19]); lymphoscintigraphy helps to

define the nature and extent of lymphatic damage or dysfunction.

Oral diethylcarbamazine kills both macrofilariae and microfilariae and

remains the treatment of choice in all forms of lymphatic filariasis

including subclinical infection. Repeated courses may be necessary. In

subclinical infection, alternative treatments include diethvlcarbamazine

plus oral albendazole or albendazole plus oral ivermectin. Tools to assess

infection status (ultrasonography, circulating antigenaemia) are allowing

more rational use of antifilarial drugs.[20] In adenolymphangitis,

antipyretics and analgesics are recommended along with diethylcarbamazine;

antibiotics are also useful if secondary infection is likely. For chronic

manifestations of lymphatic filariasis, adjunctive regimens to

diethylcarbamazine which emphasise hygiene and skin care, limb elevation,

prophylactic antibiotics to reduce secondary bacterial infections, and

physiotherapy have gained wide acceptance. Hydrocoeles can be drained

repeatedly or managed surgically.

Programmes are being established to eliminate lymphatic filariasis

worldwide.[21] These efforts will be similar to the integrated control

programmes for onchocerciasis in Africa and the Americas, which are based on

interrupting transmission. Long term microfilarial suppression is envisaged

from the use of mass, annual distribution of single dose combinations of

albendazole plus diethylcarbamazine or ivermectin; these regimens are known

to suppress microfilariae for over a year. An added benefit of these

regimens is their effect on gastrointestinal helminths.[22] Vaccine

development is in its infancy.

rheumatic Re: interesting article from National Post

>Hi Liz,

>

>The following site should have the article on cats as a possible

>cause or carrier of RA.

>

>http://www.docguide.com/news/content.nsf/news/5555F262DC9CD4CD852569B3

>00712BD7?

>OpenDocument & id=5753362958dfd5e9852569d000691221 & c=Rheumatoid%

>20Arthritis & count=10

>

>It is supposed to read as one long line.....looks like you might have

>to cut and paste...if you can't get it to work, let me know and I can

>send it from my Outlook files.

>

>take care and thanks for all of your posts,

>

>Connie

>

>

>

>> I saw

>> something on an arthritis newsgroup a couple of weeks ago about a

>cats and

>> RA study at Royal Adelaide Hospital. Anyone know anything about

>that one?

>>

>

>

>

>To unsubscribe, email: rheumatic-unsubscribeegroups

>

>

>

[Guest guest]

Hi,

Can people who do not travel outside the US get the rickettsia and

filariae, etc.? Hulda also thinks that most diseases are caused by

parasites. Yuk! Is there a way to find them in blood tests without the

darkfield microscope?

What did they do to treat the ascaris in your daughters? Hulda thinks

that ascaris causes cancer.

Thanks,

Gloria