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Re: Autoimmune vs Retro

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I have a question in this very interesting discussion going on, which does

provide a lot of food for thought so to speak.

What exactly defines an autoimmune disease?

What exactly defines a Retro Virus?

Thanks for any answers.

Jody

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Hi Jody-

These from Stedman's medical dictionary.

Autoimmunity - To denote the condition in which one's own tissues are

subject to deleterious effects of the immunological system.

Retrovirus - Any of the family Retroviridae; characterized by the presence

of reverse transcriptase ; they possess RNA-dependent DNA polymerase

(reverse transcriptase).

The retroviruses transfer information in the form of RNA which is then

transformed into DNA in the host cell.

Cancer can be caused by a retrovirus. The retroviruses can cause our own

cells to proliferate abnormally probably by gene control. They can also

quickly mutate. Both of these characteristics could cause them to go

undetected by our immune system which normally wipes out invaders.

Take care,

Re: Autoimmune vs Retro

> I have a question in this very interesting discussion going on, which does

> provide a lot of food for thought so to speak.

>

> What exactly defines an autoimmune disease?

>

> What exactly defines a Retro Virus?

>

> Thanks for any answers.

> Jody

> _________________________________________________________________

> Get your FREE download of MSN Explorer at http://explorer.msn.com

>

>

>

> -------------------------------------

> The Graves' list is intended for informational purposes only and is not

intended to replace expert medical care.

> Please consult your doctor before changing or trying new treatments.

> ----------------------------------------

>

>

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Thanks ,

Besides that I think I am looking for something more...not quite sure what

*I* even mean...but how is ANY autoimmune disease determined to be

autoimmune. More and more diseases are falling into this category and I

guess what I am asking is who determines what is autoimmune?

Before long I expect to see Parkinson's or Huntington's diseases as well as

other lumped in with the 80+ autoimmune diseases that already exist.

In the 1950's when my cousin was diagnosed with SLE they didn't have a clue

what it was. It wasn't until joining this group and the aard (? for

autoimmune diseases) that I found out so many of them are now in this

category. I'm confused.

Thanks again,

Jody

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Hi Jody,

There are autoantibodies in both MS and vitiligo. MS has myelin basic protein

autoantibodies, oligoclonal immunoglobulins in spinal fluid, and they have an

altered IgA index.

In vitiligo, there are autoantibodies to melanin and possibly dermal cells.

Although most autoimmune diseases have several related autoantibodies, there

are a few that don't. However, they have characteristic signs of autoimmunity

when the cells are examined on biopsy samples. The thyroid in Graves'

disease, for instance, is way different from normal thyroid tissue. The

immune involvement is evident with increased cell size and cell growth,

different granules in the cytoplasm, etc.

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Hi Jody-

What they use to determine autoimmune disease is an abnormal immune response

against self.

For example, in Graves' disease we are actually making antibodies that

attach to a portion of the TSH receptor in thyroid cells. The antibodies

are reacting to the TSH receptor as if it's a foreign invader.

In Lupus, there is tissue damage caused by autoantibodies and other immune

reactions.

In MS, there is inflammation that is destroying myelin of nerve cells. They

are not positive that it's an autoimmune disease but indirect evidence

(inflammation and the presence of mononuclear cells that are mostly T-cells

and macrophages) leads experts to think it is.

Does that help? In us, the presence of autoantibodies determines that it's

an autoimmune disease. For other diseases, that may or may not be true.

But it seems like inflammation, which is usually the body's way of attacking

foreign antigens, seems to be the common theme. In these cases of

autoimmune disease, the inflammation is destroying the own patients tissues.

Take care,

Re: Autoimmune vs Retro

> Thanks ,

> Besides that I think I am looking for something more...not quite sure what

> *I* even mean...but how is ANY autoimmune disease determined to be

> autoimmune. More and more diseases are falling into this category and I

> guess what I am asking is who determines what is autoimmune?

>

> Before long I expect to see Parkinson's or Huntington's diseases as well

as

> other lumped in with the 80+ autoimmune diseases that already exist.

>

> In the 1950's when my cousin was diagnosed with SLE they didn't have a

clue

> what it was. It wasn't until joining this group and the aard (? for

> autoimmune diseases) that I found out so many of them are now in this

> category. I'm confused.

> Thanks again,

> Jody

> _________________________________________________________________

> Get your FREE download of MSN Explorer at http://explorer.msn.com

>

>

>

> -------------------------------------

> The Graves' list is intended for informational purposes only and is not

intended to replace expert medical care.

> Please consult your doctor before changing or trying new treatments.

> ----------------------------------------

>

>

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,

That helps a lot! I still have some questions regarding how drs. or

researchers determine if a disease is autoimmune...though I do understand

what you are saying. I guess if ALL diseases had auto antibodies then it

would make sense, but if MS or Vitaligo don't, then how can the be

classified as autoimmune.

Don't worry about trying to pound it into my head...your explanation has

helped a lot...some days though I have to question everything, you know,

like, why is a hand a hand and not a foot kind of thing <grin>

Maybe this is just one of them. I want to understand their logic with how

they classify things.

Thanks again,

Jody

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Thanks Elaine!

What would similar characteristics be in autoimmune diseases that don't have

antibodies...or could it just be that those with no known antibodies just

haven't had them identified yet? If that is the case, will the Genome

Project come into play here also?

Just being really curious on all of this and trying hard to understand,

which isn't easy without the backgrounds so many of you have...so thanks for

all the information, patience with questions and explanations!

TTYL,

Jody

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Hi Jody-

That's it! For instance the definition of MS is plaques that cause

demyelinization of nerve cells. Later they found an inflammation response

and (I wasn't sure about the antibodies yet because of their reluctance to

classify it as autoimmune) they must have found antibodies. I think that

they are reluctant to say that it's autoimmune for sure because they haven't

found that the antibodies cause the inflammation which causes the plaques

resulting in demyelinization. They have, however, found an association and

think it's autoimmune.

Take care,

Re: Autoimmune vs Retro

> Thanks Elaine!

> What would similar characteristics be in autoimmune diseases that don't

have

> antibodies...or could it just be that those with no known antibodies just

> haven't had them identified yet? If that is the case, will the Genome

> Project come into play here also?

>

> Just being really curious on all of this and trying hard to understand,

> which isn't easy without the backgrounds so many of you have...so thanks

for

> all the information, patience with questions and explanations!

> TTYL,

> Jody

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Hi Jody,

It's likely, there will be autoantibodies found for the other autoimmune

diseases, but maybe not... since there are two separate processes that cause

autoantibodies to develop. One is a cell-mediated process where white cells

aggregate in the affected area, releasing chemicals called cytokines that

also contribute to the inflammatory process. The other mechanism is humoral

immunity where autoantibodies do their thing. Autoantibodies work in

different ways. Like in GD, they stimulate the TSH receptor, and in HT they

block it. In SLE and some other diseases with a renal component, they form

complexes, lattice like formations where antigens and antibodies link

together. These immune complexes, which are also a bad sign, impede blood

flow in blood vessels. And in many autoimmune diseases, cell-mediated and

humoral autoimmunity both contribute to symptoms.

In some diseases, like MS and hypoparathyroidism, the cause may be autoimmune

and it may not be. They think there are probably separate subtypes of these

disorders.

The human genome will help with some of the diagnostic issues. Right now, the

focus has been on immune system genes, like the HLA antigens. But eventually,

more organ specific genes will wend their way into the picture. Like the TSH

receptor gene, which is on I think chromosome 21 or 14, is also referred to

as the Graves' disease gene.

Back to the immune system involvement... one of the standard classifications

for considering a disease autoimmune is a favorable response with

immunosuppressive drugs. In Graves' disease, this doesn't work because it's a

diminished level of suppressor T cells (the ones that nip autoreactive cells

before they develop) that largely contributes to disease development.

Immunosuppressants don't work in the autoimmune pemphigoid disorders either.

But the fact that something that suppresses the immune system can diminish

autoimmune disease symptoms so remarkably well, is considered proof that the

immune system is the underlying cause.

As mentioned, the physiology of the stress mechanism causes immune

system changes that lead to the production of autoantibodies. Stress is a key

player in autoimmune disease development, which also explains why symptoms

flare up in times of stress. And certain other environmental triggers are

associated with very specific types of autoimmune disease. Like mercury--it's

known to cause autoimmune kidney disorders.

Unlike accidents, where you know the cause of the ailment, autoimmune

diseases are difficult to link to environmental agents because of the time it

takes for certain disorders to develop. But with all the research that's been

going on, with immunologists from all over the world gathering to compare

notes, a very clear picture is unfolding.

Enough of my rambling, I ate too much sugar driving home from work.

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