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Re: NSAIDS, Asprin

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In a message dated 6/21/2003 2:57:56 PM Central Daylight Time, rotghi@...

writes:

> Oh, does anyone know is Asprin is also not allowed for people with

> nephropathy? I would think it would not be allowed.

You're right - I've only had one doc tell me it's OK in a limited sense. The

only time I've ever taken it was when I was on dialysis, as an anticoagulant.

After transplant, I haven't been allowed to take it at any time for any

reason.

Robin s

http://www.bloggingnetwork.com/Blogs/Affil/?587

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> Dear ,

>

> That's interesting. I didn't know Clinoril was safer. I suppose

the new Celebrex, Vioxx, Mobic bunch are safer than NSAIDs but

nobody wants to take a chance and prescribe them to people with less

than perfect kidneys.

>

> Oh, does anyone know is Asprin is also not allowed for people with

nephropathy? I would think it would not be allowed. Also there is

a medication called Plavix that thins the blood but I don't think it

does anything to prostaglandins which is what the NSAIDS do.

>

> Rotghi

Thanks Rotghi,

Here is some info on it. Of course, one's neph should be involved in

any decision about prescribing it.

Sulindac and its sulfone metabolite undergo extensive enterohepatic

circulation relative to the sulfidemetabolite in animals. Studies in

man have also demonstrated that recirculation of the parent

drug,sulindac, and its sulfone metabolite, is more extensive than

that of the active sulfide metabolite. The activesulfide metabolite

accounts for less than six percent of the total intestinal exposure

to sulindac and itsmetabolites.The primary route of excretion in man

is via the urine as both sulindac and its sulfone metabolite

(freeand glucuronide conjugates). Approximately 50% of the

administered dose is excreted in the urine, withthe conjugated

sulfone metabolite accounting for the major portion. Less than 1% of

the administereddose of sulindac appears in the urine as the sulfide

metabolite. Approximately 25% is found in the feces,primarily as the

sulfone and sulfide metabolites.The bioavailability of sulindac, as

assessed by urinary excretion, was not changed by

concomitantadministration of an antacid containing magnesium

hydroxide 200 mg and aluminum hydroxide 225 mgper 5 mL.

Because CLINORIL is excreted in the urine primarily as biologically

inactive forms, it may possiblyaffect renal function to a lesser

extent than other non-steroidal anti-inflammatory drugs, however,

renaladverse experiences have been reported with CLINORIL (see

ADVERSE REACTIONS). In a study ofpatients with chronic glomerular

disease treated with therapeutic doses of CLINORIL, no effect

wasdemonstrated on renal blood flow, glomerular filtration rate, or

urinary excretion of prostaglandin E2andthe primary metabolite of

prostacyclin, 6-keto-PGF1. However, in other studies in healthy

volunteers andpatients with liver disease, CLINORIL was found to

blunt the renal responses to intravenous furosemide,i.e., the

diuresis, natriuresis, increments in plasma renin activity and

urinary excretion of prostaglandins.These observations may represent

a differentiation of the effects of CLINORIL on renal functions

basedon differences in pathogenesis of the renal prostaglandin

dependence associated with differing dose-response relationships of

different NSAIDs to the various renal functions influenced by

prostaglandins.

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And in Excedrin...

> I was told aspirin is especially harmful when it is combined with

> acetaminophen . You find this mainly in the powder form of pain

relievers.

>

>

>

>

>

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hmmmm..... maybe tell that nurse to go take a powder ????

Ooops, no, I did not say that.

Cy

Re: NSAIDS, Asprin

Loved those little powders to. They really worked.

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I have seen articles as well . Another thing mentioned were

things like tylonol #3 (with codeine).

> I don't know why. Perhaps someone else has the answer. I know I

have seen a

> few articles on it.

>

>

>

>

>

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