Re: Early puberty becoming more common

[Guest guest]

> Regarding soy and estrogen...

Thanks for the information on soy. I know about all that. But there

is so much conflicting information out there, it is confusing.

I eat more soy than my sisters. Yet it appears that my fibroids were

bigger than theirs. I don't remember them having a big tummy like

me. Did the soy make my fibroids grow instead of inhibiting them?

I eat brown rice at home. If white rice inhibits estrogen, does

brown rice make them grow or is brown rice neutral?

At home, I drink Lipton tea, herbal tea and green tea. But if I am

in a good restaurant, I will have a cup of real coffee to go with the

real cheesecake. :-) My company offers free tea and coffee. That is

hard to resist when I am sleepy. Why can't they offer free juice?

They just want to drug us so that we will keep working there just for

the fix.

Some of you eat very healthy and exercise, yet you have fibroids.

What gives?

[Guest guest]

I found this article that looks interesting on PubMed:

Influence of exogenous estrogen receptor ligands on uterine

leiomyoma: evidence from an in Vitro/in vivo animal model for uterine

fibroids

Hunter DS, Hodges LC, Eagon PK, Vonier PM, Fuchs-Young R, Bergerson

JS, CL

Department of Carcinogenesis, University of Texas M.D.

Cancer Center, ville, Texas, USA.

[Record supplied by publisher]

The remarkable frequency of uterine leiomyoma in the human population

calls into question the potential for the participation of

environmental factors in tumor etiology. Having been implicated in

the dramatic rise in hormone-related cancers in recent years,

endocrine disruptors are salient suspects in this pathogenesis,

although the mechanism by which they might participate is unclear.

Investigations using the Eker rat model show that uterine leiomyoma

may have an enhanced sensitivity to modulation via the estrogen

receptor. This sensitivity could make these tumors a target for

disruption by exogenous estrogen receptor ligands. Direct evidence

for a pathogenic role of exogenous compounds in leiomyomas is

lacking; however, it can be demonstrated that such diverse agents as

organochlorine pesticides, dietary flavonoids, botanical extracts,

and therapeutic antiestrogens have either estrogen agonist or

antagonist function in myometrial tissues. The use of this model will

help define the impact of exogenous estrogen receptor modulators on

uterine leiomyoma and will permit the evaluation of strategies for

therapeutic intervention.

> > >Some of you eat very healthy and exercise, yet you have fibroids.

> > >What gives?

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