Recent articles (long)

[Guest guest]

How about some discussion on those recent articles that give us new

insight into what ails us here.

Why don't we as a group discuss each article and see if we can help

each other understand these complex articles and find relevance

for each particular battle with chiari.

started with his response to Dr Ellebogans " Towards a

rational approach to Chiari malformation and Syringomelia " , he

found this a positive source of information and it gave him and

Ina some hope.

I have heaps of questions that I would love some help and

discussion about. Through the list would be the most beneficial

I think, okay here goes.

Interesting things in relation to children.. higher CSF flow

velocities in pediatric patients..most dramatic at the forman

magnum and magendies foramen.

I wonder how this impacts on children as they get older, could it be

a positive thing? As Rox's gets older and the velocity becomes less,

will the impact on her CCJ be less also, with each rhythmic contraction

(systole flow) her tonsils impact, is the impaction less if the velocity is

less?

Could anyone suggest what greater intercranial compliance in childrens

means?

It also talk about how in normal patients caudal CSF pulsations are

greatest is systole and cranial pulsations greatest in diastole. I

am presuming and would like help if I am wrong in my mothers

understanding of this, that this means during the contraction (systole)

CSF pulsations are greatest in the tail end, ie.spinal area whilst

during relaxation (diastole) CFS pulsations are greatest in cranial

or head.????

Assuming I have half a grasp on this, what are the implications for

syrinx formation or growth, when during the systole

flow the tonsils are impacting, the tail end pulsations are at their

greatest, and with the impaction of the tonsils, CSF is trapped?

I also read in another article that a elongated systolic flow was a

more likely scenario for syrinx formation, than a shortened systolic

flow. What causes a elongated systolic flow vrs a shortened one?

Restoration of CSF flow, addressing adhesions to restore CFS flow

are big factors. I wonder about the adhesions alot, impaction of

the tonsils but how much does the surgery contribute to more

adhesions? Lots of talk re opening the dura vrs not, the need to

get in there and address the adhesions. Rox is still quite tight

how predisposed are we to more adhesions forming, especially since

we have messed about in there already three times?

Thats alot of question marks for one post, any thoughts, theories,

speculation would make for some interesting reading and discussion.

Kym

Mother to Roxanne

[Guest guest]

> Restoration of CSF flow, addressing adhesions to restore CFS flow

> are big factors. I wonder about the adhesions alot, impaction of

> the tonsils but how much does the surgery contribute to more

> adhesions? Lots of talk re opening the dura vrs not, the need to

> get in there and address the adhesions. Rox is still quite tight

> how predisposed are we to more adhesions forming, especially since

> we have messed about in there already three times?

This concerns me as well. I've had 2 decompressions, with adhesions

removed both times. The second surgery the adhesions were worse- tonsils

adhered to 4th ventrical, part of tonsils removed.

What causes the adhesions to form? Is it the pressure or the friction or

something else? I'm not doing as well as I'd like to be, and I'm

concerned that they will reform and cause things to get even worse.

in VA