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Ok, I've accumulated several studies or abstracts over the years on this

issue. Here they are.

The first one demonstrates that, at least in the African yam bean, that heat

treatment does indeed reduce the oxalate content and NOT BY LEACHING in the

case of auto-claving. And in this case, by 49%! When cooked in water, the

reduction increased to 72.2%. When I posted about this before, I didn't have

the abstract handy, but happened to dig it up from another list I posted it

to last year. I highlighted the pertinent part with " **** " .

The second study also found a reduction in oxalates with heat treatment.

Unfortunately, the link is now broken.

As for Heidi's contention that oxalates in food are only a problem for those

on antibiotics, I'd like to see some evidence of that before believing it.

There is no mention of antibiotics in some of the abstracts below in which

oxalates were found to be problematic for humans and animals. Heidi, do you

have any info on the studies that led you to believe there's a link to

antibiotic treatment?

Again, my feeling is that we are eating foods in a manner that was not done

by our species traditionally and just expecting to be OK with it. Just

because it's popular to eat raw high oxalate foods in our brief historical

moment of time doesn't mean it's healthy. My understanding is that early

Americans never ate raw or unfermented veggies. I could be wrong, but it's

also my understanding that raw green salads are a modern invention. I also

suspect that our soil depletion over the past 50 years or so is likely

contributing to the high level of oxalates in many varieties of veggies.

It's not *God* doing something wrong, IMHO - it's *us*.

----------------------------

http://tinyurl.com/4m6p9

Effect of heat treatment on the proximate composition, energy values, and

levels of some toxicants in African yam bean (Sphenostylis stenocarpa) seed

varieties.

Onyeike EN, Omubo-Dede TT.

Department of Biochemistry Nutrition and Toxicology Unit, Faculty of

Science, University of Port Harcourt, PMB 5323, Port Harcourt, Nigeria.

The effects of heat treatments on the proximate composition, energy content,

and levels of some antinutritional factors in brown and marble-colored

African yam bean (AYB) seed flours were investigated. In raw brown and

marble-colored AYB seed flours; moisture content, dry matter, crude protein,

crude fat, ash, total carbohydrate and caloric value did not differ

significantly at the 5% level. Autoclaving and cooking slightly increased

the moisture level. Crude protein, crude fat, and ash contents were

decreased by autoclaving and were further decreased by cooking. The decrease

was not, however, considerable for the AYB that is not eaten raw and whose

full nutritional potential as a legume can be derived only when heat

treated, as previous reports have indicated for legume seeds. ****The levels

of the toxicants were generally higher in the raw brown AYB compared to the

marble-colored, and were generally reduced by both autoclaving and cooking.

In the most commonly available and consumed marble-colored AYB, autoclaving

at 121 degrees C, 15 psi for 20 min decreased cyanogenic glycosides by 46%,

oxalate by 48.9%, ****tannin by 15.0%, saponin by 14.8% and trypsin

inhibitors by 61.3% while cooking for 3.5 hours in tap water decreased these

toxic factors by 66.5%, 70.3%, 72.2%, 48.7%, and 86.0%, respectively. The

results indicate that for raw samples, varietal difference did not

significantly affect nutrient composition though the toxicants were

generally higher in the brown AYB than the marble-colored. Autoclaving

decreased both nutrient value and the level of toxicants in the two seed

types; values were further reduced by cooking. Of the toxicants, trypsin

inhibitor was found to be the most heat-labile and of the heat treatment

methods, cooking to tenderness is recommendable.

----------------------------

http://147.46.94.112/e_journals/pdf_full/journal_p/p12_9953308.pdf

Removal of antinutrients like oxalic acid and phytic acid during the

blanching and cooking of these leaves might have asserted a beneficial

effect on extractability of calcium and zinc, as antinutrients are known to

have a hindering effect on mineral bioavailability [30].

----------------------------

http://www.sciencedaily.com/releases/1998/03/980327091009.htm

University Of Florida Researchers Test New Approach To Kidney Stone

Prevention

GAINESVILLE, Fla.---Aided by a new $1 million grant from the National

Institutes of Health, researchers at the University of Florida are

evaluating the feasibility of using a pill to prevent common calcium oxalate

kidney stones.

The grant will enable UF College of Medicine pathologist Ammon Peck and

colleagues to determine whether kidney stones can be prevented in laboratory

rats by giving them a coated pill to replenish a beneficial bacteria called

Oxalobacter formigenes. An absence of this intestinal bacteria has been

linked to the development of calcium oxalate kidney stones. (full article at

above link).

--------------------------------

http://www.nal.usda.gov/ttic/tektran/data/000008/44/0000084447.html

EVALUATING CHILDREN IN THE UKRAINE FOR COLONIZATION WITH THE INTESTINAL

BACTERIUM OXALOBACTER FORMIGENES, USING A POLYMERASE CHAIN REACTION-BASED

DETECTION SYSTEM

Author(s):

Interpretive Summary:

Oxalic acid (and its salts - oxalates) are widely distributed in diets of

man and animals. Dietary oxalates (as well as oxalate produced by tissue

metabolism) are potentially toxic because elevated concentrations of oxalate

in the urine increase the risk for formation of kidney stones composed of

calcium oxalate. Oxalobacter formigenes, a bacterium that degrades oxalate,

lives in the intestinal tract of animals and in some, but not all, humans.

Oxalate degradation by this bacterium can reduce the amount of oxalate that

is absorbed and thus reduce the risk for urinary stone disease. This paper

describes new methods to detect O. formigenes. The methods developed use

polymerase chain reaction (PCR) based amplification and then detection of

DNA fragments that are uniquely present in O. formigenes. The detection

system is more rapid than the cultural identification systems preivously

used. The new system was compared with the cultural system in a field test

using 100 fecal samples from children in the Ukraine (aged 0-12 yr). Data

collected indicate that most infants less than 1 yr old were not colonized

by O. formigenes but that all of the children became colonized by the age of

6-8 yr. The incidence of colonization then decreased in children who were

9-12 yr of age. These results provide the first insight into the time

sequence of colonization of humans by O. formigenes and further support the

concept that these bacteria are widely distributed in humans. The new

methods that are described will facilitate extensive surveys in man and

animals and will provide a basis for possible manipulations of the bacteria

to provide protection from kidney stones and other diseases caused by

oxalate.

---------------------

http://www.nal.usda.gov/ttic/tektran/data/000007/98/0000079894.html

DNA SEQUENCING AND EXPRESSION OF THE FORMYL COENZYME A TRANSFERASE GENE,

FRC, FROM OXALOBACTER FORMIGENES

Interpretive Summary:

Kidney stones caused by calcium oxalate are a continuing health problem to

farm animals and humans. The enzymes we found in Oxalobacter formigenes

degrade oxalate and make it nontoxic. In this study, the sequence of the

gene for formyl-CoA transferase was determined and its location on the

bacterial chromosome was found. This should lead to the cloning of this gene

into other bacteria found in the digestive tract of man and farm animals,

and ultimately to the alleviation of the problem of kidney stones by

oxalate. This would alleviate the pain, suffering, and the expense

associated with treating and removing kidney stones. This information would

be used by both the medical and veterinary profession.

----------------------

http://tinyurl.com/5hfzb

Oxalate content of foods and its effect on humans

SC Noonan BSc, MSc and GP Savage BSc(Hons), PhD, NZ Reg Nutr.

Food Group, Division of Animal and Food Sciences, Lincoln University,

Canterbury, New Zealand

Oxalic acid and its salts occur as end products of metabolism in a number of

plant tissues. When these plants are eaten they may have an adverse effect

because oxalates bind calcium and other minerals. While oxalic acid is a

normal end product of mammalian metabolism, the consumption of additional

oxalic acid may cause stone formation in the urinary tract when the acid is

excreted in the urine. Soaking and cooking of foodstuffs high in oxalate

will reduce the oxalate content by leaching. The mean daily intake of

oxalate in English diets has been calculated to be 70-150 mg, with tea

appearing to contribute the greatest proportion of oxalate in these diets;

rhubarb, spinach and beet are other common high oxalate-content foods.

Vegetarians who consume greater amounts of vegetables will have a higher

intake of oxalates, which may reduce calcium availability. This may be an

increased risk factor for women, who require greater amounts of calcium in

the diet. In humans, diets low in calcium and high in oxalates are not

recommended but the occasional consumption of high oxalate foods as part of

a nuritious diet does not pose any particular problem.

----------------------------------

http://tinyurl.com/63ken

The hole truth: intracrystalline proteins and calcium oxalate kidney stones.

Ryall RL, Fleming DE, Grover PK, Chauvet M, Dean CJ, Marshall VR.

Department of Surgery, Flinders University School of Medicine, Flinders

Medical Centre, Bedford Park, South Australia, Australia.

rose.ryall@...

The ultimate aim of our research is to understand the role of macromolecules

in the formation of human kidney stones, particularly their interactions

with calcium oxalate (CaOx) crystals. The invariable association of stones

with proteins raises the possibility that proteins play a role in their

formation, similar to the role of proteins in healthy biomineralization. Do

these proteins induce mineralization? Are they merely a response to the

disease process? Or are they protective molecules that were overwhelmed by

mineral supersaturation? A protein of particular interest is fragment 1 (F1)

of prothrombin. We have shown that mRNA for prothrombin is present in the

kidney. Because the F1 fragment of prothrombin present in urine is slightly

different from that found in the blood, we refer to this protein as " urinary

prothrombin fragment 1 " (UPTF1). Available evidence suggests that the kidney

manufactures the protein for protection against stone disease and that the

protein has a directive role in stone formation. We now have evidence that

proteins are interred within CaOx crystals precipitated from human urine,

where it is distributed in continuous channels. These proteins could

facilitate crystal deconstruction and removal after attachment to the renal

epithelium and endocytosis. We suspect that the formation of CaOx crystals

in the urine is a normal process designed to permit harmless disposal of an

excess of calcium, oxalate, or both. The incorporation of proteins provides

a second line of defense against stone formation by enabling the destruction

and removal of retained crystals. Understanding the basic molecular

strategies by which plants produce protein-containing CaOx crystals may

provide insight into human CaOx stone formation.

Suze Fisher

Lapdog Design, Inc.

Web Design & Development

http://members.bellatlantic.net/~vze3shjg

Weston A. Price Foundation Chapter Leader, Mid Coast Maine

http://www.westonaprice.org

----------------------------

“The diet-heart idea (the idea that saturated fats and cholesterol cause

heart disease) is the greatest scientific deception of our times.” --

Mann, MD, former Professor of Medicine and Biochemistry at Vanderbilt

University, Tennessee; heart disease researcher.

The International Network of Cholesterol Skeptics

<http://www.thincs.org>

----------------------------

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In a message dated 10/12/04 9:26:53 AM Eastern Daylight Time,

s.fisher22@... writes:

I thought the steam *evaporates*?

______

~~~~> Steam can't evaporate, because it already *is* vapor. Liquids

evaporate, vapors don't. Did you mean you thought it disappears?

______

IF they are only *removed* from the bean flour (or other plant material) and

not destroyed or reduced by the heat while still IN the flour (like

goitrogens, trypsin inhibitors or heat-labile vitamins). Perhaps they are

reduced by both leaching AND heat?

_____

~~~~~>That's doubtful for two reasons. First, anti-trypsins are

glycoproteins, and proteins are easily denatured by heat. I don't know about

the others,

but that's probably the case or something similar to them. Oxalic acid does

not have a shape and function dependent on weak bonds. The second reason is

that the study I posted last year found that essentially all of the oxalate that

was missing from the vegetables was found in the cooking water. There was a

very, very, very tiny gap, but it wasn't significant, and it isn't clear that

it would be attributable to the heat anyway. So, it can be safely said that

heat has no significant effect on oxalates at all, IMO.

______

I just googled " heat labile oxalic acid "

and couldn't find anything though. I know other acids are indeed heat-labile

like ascorbic acid.

______

~~~~~> I haven't looked into the heat-lability of ascorbic acid, but one

thing I know is that the USDA shows a much higher vitamin C content for a baked

potato than for a boiled/mashed potato, even though baked potatoes are cooked at

twice the heat for four times as long. So I doubt that vitamin C is

extremely heat-labile and probably not very heat-labile in dry foods. It may be

somewhat heat-labile in milk and other wet foods (everything is destroyed more

easily in water when heated, to my understanding), but I'd guess that most

vitamin

C loss that occurrs in cooking is also attributable to leaching into cooking

water.

_______

But maybe *oxalic* acid isn't. It is interesting though

that *drying* may reduce it too, which involves no leaching into water or

steam. I wonder *what* causes the reduction then when no water or steam is

involved?

______

~~~~> I'm sorry, I think I missed this. Was there a reference to drying

reducing oxalates in your initial post?

______

I agree that they needed to have a dry heat control in order to determine it

was the heat and not leaching (exclusively) that reduced the oxalates so

drastically.

_____

~~~~~> I think that would be best, but it would be sufficient in the case of

boiling where all the heating water, or most of it, is recovered, to measure

the oxalate content of the water, to give a rough idea.

_____

However, they do mention that the moisture content increased

slightly *after* the treatment. It seems to me if something were being

leached *from* a food into water or steam, then the moisture content of the

food would *decrease* not *increase* after being cooked or autoclaved.

______

~~~~~> Why? That doesn't make sense to me. The oxalate, not water, is

leaching from the foods.

______

One interesting side not here, notice that the trypsin inhibitors were the

most heat-labile. I wonder how much of the trypsin inhibitors are reduced in

legumes, nuts, etc when they are cooked on medium or low heat *without*

pre-soaking as so many *non*-traditional peoples do? Maybe the auto-claving

at the temp and pressure that these researchers used served as a sort of

" pre-soak " , or maybe not. But it's interesting to see how heat affects

trypsin-inhibitors. Maybe we don't always need to pre-soak foods with

trypsin inhibitors in order to reduce them significantly?

______

~~~~~> Considering that Sally recommends cooking, not soaking, egg whites,

I'd guess that cooking should generally do the trick. But anti-trypsin is a

protein, so I would guess it would be pretty heat-labile (though I generally

object to using that term with reference to the study you posted, since the

study

doesn't study heat-lability per se).

____

>>I don't remember it. Do you still have it? I'd like to see it, if so.

____

~~~~~> It's in the archives <weg>

I don't have it saved on my comp.

_____

>I agree. That's why I said (in so many words) they could be wrong in their

conclusion and we'd need the full study to determine if they were or not.

____

~~~~> Unless they are more incompetent in writing abstracts, I don't think we

do. If they had a dry-heat control or measured the oxalate content of the

water, it would be unambiguously abstract-worthy, and obviously much more

important than the quantitative data present in the abstract.

_____

>>You are *supposed to* throw out the water :-) That goes for anything you

soak - legumes, rice, nuts etc, for the very reason that several

anti-nutrients are leached out into the water.

_____

~~~~~> I've never thrown out oatmeal water. I thought that was only for

certain foods like beans and nuts.

Chris

With boiling, the water is usually thrown

>out, so that would

>be helpful. But the water will contain all of the soluble oxalates, so

>consuming it would be no different than consuming the unprocessed grain.

I don't think we can conclude based on that *one* study on one particular

food item (or was it multiple varieties of foods?) that ALL insoluble

oxalates remain intact in all foods regardless of soaking and regardless of

time and temp cooked.

Suze Fisher

Lapdog Design, Inc.

Web Design & Development

http://members.bellatlantic.net/~vze3shjg

Weston A. Price Foundation Chapter Leader, Mid Coast Maine

http://www.westonaprice.org

----------------------------

" The diet-heart idea (the idea that saturated fats and cholesterol cause

heart disease) is the greatest scientific deception of our times. " --

Mann, MD, former Professor of Medicine and Biochemistry at Vanderbilt

University, Tennessee; heart disease researcher.

The International Network of Cholesterol Skeptics

<http://www.thincs.org>

----------------------------

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> Re: OXALATES

>

>

>

>In a message dated 10/12/04 9:26:53 AM Eastern Daylight Time,

>s.fisher22@... writes:

>I thought the steam *evaporates*?

>______

>

>~~~~> Steam can't evaporate, because it already *is* vapor. Liquids

>evaporate, vapors don't. Did you mean you thought it disappears?

Ah yes, you are right. I meant that it dissipates... disappears. I don't

know where to. It's been a long time since I saw an autoclave in action.

>______

>IF they are only *removed* from the bean flour (or other plant

>material) and

>not destroyed or reduced by the heat while still IN the flour (like

>goitrogens, trypsin inhibitors or heat-labile vitamins). Perhaps they are

>reduced by both leaching AND heat?

>_____

>

>~~~~~>That's doubtful for two reasons. First, anti-trypsins are

>glycoproteins, and proteins are easily denatured by heat. I don't

>know about the others,

>but that's probably the case or something similar to them. Oxalic

>acid does

>not have a shape and function dependent on weak bonds. The second

>reason is

>that the study I posted last year found that essentially all of

>the oxalate that

>was missing from the vegetables was found in the cooking water.

What vegetables did they cook?

>There was a

>very, very, very tiny gap, but it wasn't significant, and it isn't

>clear that

>it would be attributable to the heat anyway. So, it can be safely

>said that

>heat has no significant effect on oxalates at all, IMO.

You may be right, although I'm still curious about the Andean Indians

reducing oxalates simply by drying yams in the *sun*. So far, most or all of

the pubmed abstracts I've looked at discuss it being leached out into water

(except the one mentioned above, in which the oxalates DID leach into the

water, but it was attributed to the heat). Of course they're not interested

in ancient or traditional food preparation, so it's not likely they'd be

testing oxalate content after sun drying. LOL

>______

>I just googled " heat labile oxalic acid "

>and couldn't find anything though. I know other acids are indeed

>heat-labile

>like ascorbic acid.

>______

>

>~~~~~> I haven't looked into the heat-lability of ascorbic acid, but one

>thing I know is that the USDA shows a much higher vitamin C

>content for a baked

>potato than for a boiled/mashed potato, even though baked potatoes

>are cooked at

>twice the heat for four times as long. So I doubt that vitamin C is

>extremely heat-labile and probably not very heat-labile in dry

>foods. It may be

>somewhat heat-labile in milk and other wet foods (everything is

>destroyed more

>easily in water when heated, to my understanding), but I'd guess

>that most vitamin

>C loss that occurrs in cooking is also attributable to leaching

>into cooking

>water.

>

Possibly. But this brings up an interesting point. Remember that the USDA

data is VERY unreliable when it comes to determining nutrient values in

foods for several reasons. Chief among them is that the nutrient content of

a crop grown in one field as opposed to another can vary as high as several

hundred percent! Even those grown across the street from each other can vary

significantly in their nutrient content. So unless they were testing a

boiled and baked potato grown very close together in the same field, the

numbers should be taken with a grain of salt when compared to one another.

Secondly, and my first point makes this moot for the most part, what was

their sample size? Sometimes, it's just a few specimens. Basing conclusions

on just a few data points is not very useful.

Regarding comparing the vit C content of vegetables - here is something

interesting. I just compared the vit. C content of RAW sweet potato and

baked sweet potato (skin on). Both used only 4 test samples. The raw sweet

potato had 2.4 mgs vitamin C per 100 grams edible portion and the baked

sweet potato had 19.6 mgs per the same portion! How do you suppose *baking*

a sweet potato could increase the vitamin C content 8 TIMES?? I have two

guesses. One - these sweet potatoes were grown in different soils and their

nutrient content reflects that. Two, the ascorbic acid in the *raw* sweet

potato is bound to something...maybe calcium? And the heat (dry heat in this

case) unbinds them, thus increasing the ascorbic acid content (although it

could possibly be simultaneously destroying *some* of the ascorbic acid

content, as well).

As for " regular " potatoes, here are the ascorbic acid amounts listed on the

USDA database per 100 grams:

raw: 19.7 mgs

boiled (w/skin): 5.2 mgs

baked (w/skin): 13.5 mgs

How do we interpret these data? First, the raw potato was listed as a " red "

potato and the other two weren't. So that muddies the already muddy waters.

The trend among these potatoes of unknown origins *seems* to be that baking

reduces *some* of the ascorbic acid (although it had the *opposite* effect

in the *sweet* potatoes *assuming* [probably erroneously] that the sweet

potatoes had an equivalent amount of ascorbic acid to begin with) and

boiling seems to reduce it even more. However as I mentioned before, I

hesitate to draw any conclusions based on 3 data points of potatoes that

might have had extreme nutrient level differences in their virgin state from

the others I'm comparing them to.

In any case, it's fun to play games with USDA data, but it's probably fairly

useless to base any conclusions by comparing a few data points from

vegetables that might have started out with drastically different nutrient

values than each other in the raw state.

>_______

>But maybe *oxalic* acid isn't. It is interesting though

>that *drying* may reduce it too, which involves no leaching into water or

>steam. I wonder *what* causes the reduction then when no water or steam is

>involved?

>______

>

>~~~~> I'm sorry, I think I missed this. Was there a reference to drying

>reducing oxalates in your initial post?

No, *Deb* posted about it. She said that when she was researching yams,

she'd read that the Andean Indians' traditional method of reducing oxalate

content in their " oca " (yams) was to leave them out in the sun for several

days before eating them either raw or cooked. That's actually the first time

I've heard of potatoes eaten *raw* by an ancient culture. Perhaps she has a

URL or source where we can look into that interesting tidbit further...

>>I agree. That's why I said (in so many words) they could be wrong in their

>conclusion and we'd need the full study to determine if they were or not.

>____

>

>~~~~> Unless they are more incompetent in writing abstracts, I

>don't think we

>do. If they had a dry-heat control or measured the oxalate content of the

>water, it would be unambiguously abstract-worthy, and obviously much more

>important than the quantitative data present in the abstract.

Yes, you are right. Although I sometimes wonder if significant points of

studies are *indeed* left out of abstracts. Although in this case, it would

seem pretty absurd.

>_____

>

>>>You are *supposed to* throw out the water :-) That goes for anything you

>soak - legumes, rice, nuts etc, for the very reason that several

>anti-nutrients are leached out into the water.

>_____

>

>~~~~~> I've never thrown out oatmeal water. I thought that was only for

>certain foods like beans and nuts.

I haven't had oatmeal in a while, but I generally throw out the water of

everything I soak. I'd read before that there are several things, like

saponins or tannins on the *surface* of some (or maybe all?) grains and that

it's traditional practice to toss out the soaking water. So I do.

Also, in your response you didn't address my comments below. Do you have any

comment?

>With boiling, the water is usually thrown

>>out, so that would

>>be helpful. But the water will contain all of the soluble oxalates, so

>>consuming it would be no different than consuming the unprocessed grain.

>

>I don't think we can conclude based on that *one* study on one particular

>food item (or was it multiple varieties of foods?) that ALL insoluble

>oxalates remain intact in all foods regardless of soaking and regardless of

>time and temp cooked.

>

Suze Fisher

Lapdog Design, Inc.

Web Design & Development

http://members.bellatlantic.net/~vze3shjg

Weston A. Price Foundation Chapter Leader, Mid Coast Maine

http://www.westonaprice.org

----------------------------

" The diet-heart idea (the idea that saturated fats and cholesterol cause

heart disease) is the greatest scientific deception of our times. " --

Mann, MD, former Professor of Medicine and Biochemistry at Vanderbilt

University, Tennessee; heart disease researcher.

The International Network of Cholesterol Skeptics

<http://www.thincs.org>

----------------------------

>

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>

> Oxalobacter formigenes. An absence of this intestinal bacteria has

been

> linked to the development of calcium oxalate kidney stones. (full

article at

> above link).

>

> The link between oxalobacter and kidney stones was first noted in

> humans ... folks who had bladder infections were more likely to get

> kidney stones, and the connection seems to be that a person with

> bladder infections gets antibiotics.

Oxalobacter formigenes. That's the critter I was reading about that

was associated with low incidence of kidney stones.

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At 08:04 PM 10/13/04 -0000, you wrote:

>Oxalobacter formigenes. That's the critter I was reading about that

>was associated with low incidence of kidney stones.

>

>

At the risk of having almost every single post of mine today be completely

facetious ...

Can anyone say " oxalobacter formigenes " six times really fast???? Try

it. If you're not laughing at the end, I've not done my job.

*See run. See run fast.*

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> Re: OXALATES

>

>

>

>In a message dated 10/18/04 9:45:20 AM Eastern Daylight Time,

>s.fisher22@... writes:

>Well, what's interesting is that the vitamin C content in regular potatoes

>*decreases* with baking, which contradicts what happens with the *sweet*

>potato. At first I hadn't seen regular raw potatoes listed, but just found

>it now. It has 11.4 mgs/100 grams raw, and 13.5 mgs/100 grams

>baked. You see

>why I think the USDA database is fairly useless?

>_____

>

>~~~~> No, you've confused me. Did you type that sentence wrong? You're

>showing the vitamin C increasing with baking as in the other example, not

>decreasing, which fits the explanation offered by me and Mike.

Oops! I don't know where I got that figure from. It's not correct. I think I

scanned the entries too quickly and selected *just* skin, assuming it meant

WITH skin. Here are the correct data for ascorbic acid content per 100

grams.

raw: 19.7 mgs

baked: 9.6 mgs (w/salt)

boiled: 13 mgs (w/salt)

boiled: 13 mgs (without/salt)

So *baking* a potato (according to these data) decreases the ascorbic acid

content approx. 20% more than boiling does in *potatoes*. Would salt

interfere with ascorbic acid loss in baking? Apparently it doesn't in

boiling.

>(although if it

>didn't, it wouldn't mean the data were useless, it would seem to

>suggest that there

>is a large heat-induced loss of vitamin C).

That's what it looks like, n'est pas?

Here's the sweet potato data:

raw: 2.4

baked: 19.6 (without salt)

boiled: 12.8 mgs (without SKIN). no data for boiled WITH skin.)

So we're right back to what I was fussing about re the usefuleness of these

data earlier - these data totally contradict each other. While the sweet

potato data support your argument that the moisture lost during baking

increases the percent of ascorbic acid, the potato data contradict it. What

do you think the explanation is?

>

>________

>The regular potato data

>contradicts your explanation, which I agree is the most obvious one. But

>it's only based on 3(!) DATA POINTS. Any 3 potatoes in the United States

>can't possibly represent the nutrient profile of the millions of potatoes

>grown in this country.

>_______

>

>~~~~~> Why do they even bother using three? It's my understanding

>that the

>USDA's positions is that nutrient composition is dependent on species and

>breed, and not affected by soil. So shouldn't *one* sample be

>sufficient? If they

>implicitly admit the need for a variety of samples, you'd think

>they'd use a

>lot more than three.

It varies widely. I think the *raw* potato data that I just posted was based

on 141 data points. The boiled sweet potato was based on 4 data points. You

have an interesting point about them implicitly admitting the need for more

data points. I'm pretty sure they KNOW that these data are largely based on

soil health, they just don't want US to know that. That's probably why they

went for decades without providing any NEW data, but just re-printed OLD

data and put a new date on it. The declines in nutrient content is

astounding when you look back about 60 years. Unless they change the way

they measure nutrients, it's an indication that our soil sucks, which we

already KNOW is the case, so the declining nutrient values of the USDA data

just bolster a fact we already know.

>_______

>This is why I think it may be even MORE confusing to

>use USDA nutrient data to try to learn anything meaningful about nutrient

>content of produce. A possible exception might be the drastic decline in

>nutrients over several decades.

>______

>

>~~~~~> I think that would be useless as well, because there are unresolved

>possible alternative explanations for the decline in nutrient

>value that is

>shown in successive USDA figures, mentioned in an article you

>posted a couple

>months ago, such as changes in the definition of " edible portion "

>over time.

While that may be so, I invoke your earlier argument about the reliability

of the study you posted in which you argued that *logic* supported the

conclusions of the study. In this case, I'd say logic AND *fact* support the

declining nutrient values of produce as published by the USDA. It's

absolutely no mystery that our soil health is rapidly declining. Therefore,

it's fairly logical to deduce that the declining nutrient values of the USDA

database reflect this, even though their data may also be influenced by

things such as changes to the definition of " edible portion. "

__

>The article she posted didn't say. I think it did say though that these

>potatoes only contained *soluble* oxalates. Perhaps they are more prone to

>destruction by heat or drying or fermentation, as someone else mentioned?

>_____

>

>~~~~~> If they are heat-labile, then being in wet heat would

>probably have a

>greater rate of destruction than dry heat. But since I still

>haven't seen any

>evidence that oxalates are destroyed by heat, and since I really

>don't think

>sun-drying would generate all that much heat anyway,

Have you ever been to southern Florida??? LOL. I used to live there and I

can assure you that even several *minutes* of sun exposure during certain

times of the day and year can involve an *intense* amount of heat. Having

said that, I think the temp. in the Andes may vary widely, so I don't know

if it ever got " southern Florida " hot where these tribes sun-dried their

potatoes or not.

Suze Fisher

Lapdog Design, Inc.

Web Design & Development

http://members.bellatlantic.net/~vze3shjg

Weston A. Price Foundation Chapter Leader, Mid Coast Maine

http://www.westonaprice.org

----------------------------

" The diet-heart idea (the idea that saturated fats and cholesterol cause

heart disease) is the greatest scientific deception of our times. " --

Mann, MD, former Professor of Medicine and Biochemistry at Vanderbilt

University, Tennessee; heart disease researcher.

The International Network of Cholesterol Skeptics

<http://www.thincs.org>

----------------------------

>

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In a message dated 10/20/04 9:19:09 AM Eastern Daylight Time,

s.fisher22@... writes:

> Oops! I don't know where I got that figure from. It's not correct. I think I

> scanned the entries too quickly and selected *just* skin, assuming it meant

> WITH skin. Here are the correct data for ascorbic acid content per 100

> grams.

>

> raw: 19.7 mgs

> baked: 9.6 mgs (w/salt)

> boiled: 13 mgs (w/salt)

> boiled: 13 mgs (without/salt)

>

> So *baking* a potato (according to these data) decreases the ascorbic acid

> content approx. 20% more than boiling does in *potatoes*. Would salt

> interfere with ascorbic acid loss in baking? Apparently it doesn't in

> boiling.

______

~~~~> Probably not, but moreover, I'd think that the salt would be added

after boiling rather than before.

______

> >(although if it

> >didn't, it wouldn't mean the data were useless, it would seem to

> >suggest that there

> >is a large heat-induced loss of vitamin C).

>

> That's what it looks like, n'est pas?

______

~~~~~>It does, assuming that the skin is present in all samples. If the raw

potato includes the skin, and the other two have the skin discarded, that

would be a problem. Unfortunately, it doesn't appear to indicate one or the

other.

______

> Here's the sweet potato data:

> raw: 2.4

> baked: 19.6 (without salt)

> boiled: 12.8 mgs (without SKIN). no data for boiled WITH skin.)

>

> So we're right back to what I was fussing about re the usefuleness of these

> data earlier - these data totally contradict each other. While the sweet

> potato data support your argument that the moisture lost during baking

> increases the percent of ascorbic acid, the potato data contradict it. What

> do you think the explanation is?

_______

~~~~~> I don't have one except to conclude, as you'd suggested, that the USDA

are useless for this kind of analysis.

_______

> It varies widely. I think the *raw* potato data that I just posted was

based

> on 141 data points. The boiled sweet potato was based on 4 data points. You

> have an interesting point about them implicitly admitting the need for more

> data points. I'm pretty sure they KNOW that these data are largely based on

> soil health, they just don't want US to know that. That's probably why they

> went for decades without providing any NEW data, but just re-printed OLD

> data and put a new date on it.

________

~~~~~> I think that that would be pretty consistent with their claim that the

nutrition of a plant is dependent entirely on its genes. If the nutritive

value wouldn't change, why bother taking new data?

________

> While that may be so, I invoke your earlier argument about the reliability

> of the study you posted in which you argued that *logic* supported the

> conclusions of the study. In this case, I'd say logic AND *fact* support

the

> declining nutrient values of produce as published by the USDA. It's

> absolutely no mystery that our soil health is rapidly declining. Therefore,

> it's fairly logical to deduce that the declining nutrient values of the

USDA

> database reflect this, even though their data may also be influenced by

> things such as changes to the definition of " edible portion. "

_____

~~~~> Good point.

Chris

____

" What can one say of a soul, of a heart, filled with compassion? It is a

heart which burns with love for every creature: for human beings, birds, and

animals, for serpents and for demons. The thought of them and the sight of them

make the tears of the saint flow. And this immense and intense compassion,

which flows from the heart of the saints, makes them unable to bear the sight of

the smallest, most insignificant wound in any creature. Thus they pray

ceaselessly, with tears, even for animals, for enemies of the truth, and for

those

who do them wrong. "

--Saint Isaac the Syrian

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> Re: OXALATES

>

>

>

>> >(although if it

>> >didn't, it wouldn't mean the data were useless, it would seem to

>> >suggest that there

>> >is a large heat-induced loss of vitamin C).

>>

>> That's what it looks like, n'est pas?

>______

>

>~~~~~>It does, assuming that the skin is present in all samples.

>If the raw

>potato includes the skin, and the other two have the skin discarded, that

>would be a problem. Unfortunately, it doesn't appear to indicate

>one or the other.

>______

>

They all included the skin. I just didn't write it down. They had separate

data for skinless pototoes.

Suze Fisher

Lapdog Design, Inc.

Web Design & Development

http://members.bellatlantic.net/~vze3shjg

Weston A. Price Foundation Chapter Leader, Mid Coast Maine

http://www.westonaprice.org

----------------------------

" The diet-heart idea (the idea that saturated fats and cholesterol cause

heart disease) is the greatest scientific deception of our times. " --

Mann, MD, former Professor of Medicine and Biochemistry at Vanderbilt

University, Tennessee; heart disease researcher.

The International Network of Cholesterol Skeptics

<http://www.thincs.org>

----------------------------

>

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  • 1 year later...
Guest guest

I just read in Dr. Kaayla s' outstanding book, The Whole Soy Story,

that cooking does not neutralise oxalates nearly to the extent that was

previously believed.

~*~*~*~*~*~*~*~*~*~

Canfield

~We always grow in the direction in which we express ourselves.~

" The law of the Lord is perfect, converting the soul: the testimony of the

Lord is sure, making wise the simple. " Psalm 19:7

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