Guest guest Posted March 20, 2009 Report Share Posted March 20, 2009 it could be that attacking one virus "awakens" the others, but reading up on HIV (and now fully convinced that something similar at play in autism - HIV-3 anyone??) believe the following scenario is more than likely, especially when kids are doing great on antivirals, and you stop them and then gains are gone: suppose the new regression is not because of new pathogen, but because one and the same virus goes through cycles. if you suppose for a moment that some type of provirus/retrovirus is involved that is a very very likely scenario. antivirals target viruses mostly while it is in its active stages - while it is outside the cell, in the form of virion (the round ball), or some antivirals they stop the reassembly of new virions etc... so while that is happening we would be seeing good things - reduced viral load, less neurotoxicity etc. BUT the problem here would be that the virus has already left its genetic print in the human gene, waiting to be reactivated - so once antivirals are stopped, this viral mother-gene recognises/receives signals that the viral colonies have been reduced, and starts to send new proteins out for new viruses to be produced (sort of like MOTHER BEE !) saying this 'cos this is exactly what happens with HIV retrovirus (been immersed in it deeply for too long, as you can see , and why decades of most powerful antivirals, used in combination, are not able to erradicate it from human cells - it simply lies in the cell nucleus, inserted snuggly within human DNA, and reactivates when triggered - making new viruses/virions that go on to infect more cells. there are many possible triggers, suddenly stopping antiviral therapy could be one - the virus feels under threat and decides to reactivate in order to colonise more cells. sort of when you stop dieting your body makes more fat! Note also that the strong immue system wouldn't help that much here – it cannot attack those retroviruses while they are in resting stage, within our DNA/genes. A strong immune system would help to stop the reactivated virus spreading to other cells, BUT NOT IF THIS IS HAPPENING IN THE BRAIN! (or not if it is happening within immune cells themselves, which is the `genius' of HIV). Those immune cells that `catch' viruses cannot enter the nervous system, that would not be good news... The most poor brain immune system can do is to send inflammation into overdrive, to stop the virus spreading to neurons. So nowdays there is more and more talk by HIV experts of the need to establish good methylation – methylation would mean that this mother-bee virus that lies in our genes cannot send signals for new colonies to be produced and spread. Sorry so long. Here is a nice illustration of the retroviral cell cycle – scroll down to the last/bottom illustration, the blue bit in the purple centre of the cell, called Provirus is that mother-bee that cannot be destroyed, only silenced through methylation. https://eapbiofield.wikispaces.com/Ch+19+Viruses+LEW and sorry if this scares someone, didn't mean to. About 40% of human genes is composed of retroviruses. they can lie there, inactive, for many generations. Natasa x > >> > Hi > > > > We did a viral protocol starting at the end of August last year and ending at Xmas. We saw huge gains, by the end my son was working in class for 3 weeks up to the Xmas holidays without any support (mainstream). We used prescription anti virals (prescribed by Dr Magdalena at the Breakspear) of X -tra cell thymus factor, immuniovir and acyclovir. They cost a lot, I think the 3 together were about £270.> > > > However we then have seen a huge regression, so much so that school have excluded him twice in the last 7 weeks and have now moved him into another class!> Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 20, 2009 Report Share Posted March 20, 2009 I'm just gob smacked at your knowledge, all of you! x > > > > > > Hi > > > > > > We did a viral protocol starting at the end of August last year and > ending at Xmas. We saw huge gains, by the end my son was working in > class for 3 weeks up to the Xmas holidays without any support > (mainstream). We used prescription anti virals (prescribed by Dr > Magdalena at the Breakspear) of X -tra cell thymus factor, immuniovir > and acyclovir. They cost a lot, I think the 3 together were about > £270. > > > > > > However we then have seen a huge regression, so much so that school > have excluded him twice in the last 7 weeks and have now moved him into > another class! > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 20, 2009 Report Share Posted March 20, 2009 I'm a bit foggy on this now: will teh enzymes be able to target viruses even if they're not active? Not the retroviruses, but other kinds. >>I don;t think there is any evidence that enzymes have an antiviral effect at all. The Virastop thing is solely based on feedback from people given enough of them to try the higher dose protocol which is what we did. Started Valtrex today....................still on LDM-100, doing high dose C and planning to add back in Zeolites soon which gave us I think biggest viral die off yet Mandi x Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 20, 2009 Report Share Posted March 20, 2009 That's fascinating Natasa, Sara natasa778 wrote: > > it could be that attacking one virus " awakens " the others, but reading > up on HIV (and now fully convinced that something similar at play in > autism - HIV-3 anyone??) believe the following scenario is more than > likely, especially when kids are doing great on antivirals, and you > stop them and then gains are gone: > > suppose the new regression is not because of new pathogen, but because > one and the same virus goes through cycles. if you suppose for a > moment that some type of provirus/retrovirus is involved that is a > very very likely scenario. > > antivirals target viruses mostly while it is in its active stages - > while it is outside the cell, in the form of virion (the round ball), > or some antivirals they stop the reassembly of new virions etc... so > while that is happening we would be seeing good things - reduced viral > load, less neurotoxicity etc. > > BUT the problem here would be that the virus has already left its > genetic print in the human gene, waiting to be reactivated - so once > antivirals are stopped, this viral mother-gene recognises/receives > signals that the viral colonies have been reduced, and starts to send > new proteins out for new viruses to be produced (sort of like MOTHER > BEE !) > > saying this 'cos this is exactly what happens with HIV retrovirus > (been immersed in it deeply for too long, as you can see , and why > decades of most powerful antivirals, used in combination, are not able > to erradicate it from human cells - it simply lies in the cell > nucleus, inserted snuggly within human DNA, and reactivates when > triggered - making new viruses/virions that go on to infect more > cells. there are many possible triggers, suddenly stopping antiviral > therapy could be one - the virus feels under threat and decides to > reactivate in order to colonise more cells. sort of when you stop > dieting your body makes more fat! > > Note also that the strong immue system wouldn't help that much here – > it cannot attack those retroviruses while they are in resting stage, > within our DNA/genes. A strong immune system would help to stop the > reactivated virus spreading to other cells, BUT NOT IF THIS IS > HAPPENING IN THE BRAIN! (or not if it is happening within immune cells > themselves, which is the `genius' of HIV). Those immune cells that > `catch' viruses cannot enter the nervous system, that would not be > good news... The most poor brain immune system can do is to send > inflammation into overdrive, to stop the virus spreading to neurons. > > So nowdays there is more and more talk by HIV experts of the need to > establish good methylation – methylation would mean that this > mother-bee virus that lies in our genes cannot send signals for new > colonies to be produced and spread. > > Sorry so long. > > Here is a nice illustration of the retroviral cell cycle – scroll down > to the last/bottom illustration, the blue bit in the purple centre of > the cell, called Provirus is that mother-bee that cannot be destroyed, > only silenced through methylation. > > https://eapbiofield.wikispaces.com/Ch+19+Viruses+LEW > <https://eapbiofield.wikispaces.com/Ch+19+Viruses+LEW> > > and sorry if this scares someone, didn't mean to. About 40% of human > genes is composed of retroviruses. they can lie there, inactive, for > many generations. > > Natasa x > > > > > > > > > > > > > > > > > > > > > > Hi > > > > > > We did a viral protocol starting at the end of August last year > and ending at Xmas. We saw huge gains, by the end my son was working > in class for 3 weeks up to the Xmas holidays without any support > (mainstream). We used prescription anti virals (prescribed by Dr > Magdalena at the Breakspear) of X -tra cell thymus factor, immuniovir > and acyclovir. They cost a lot, I think the 3 together were about £270. > > > > > > However we then have seen a huge regression, so much so that > school have excluded him twice in the last 7 weeks and have now moved > him into another class! > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 20, 2009 Report Share Posted March 20, 2009 Don't apologize for long explanations, Natasa. I do think that the retroviral analogy may be very accurate. But in terms of the immune system, there are case studies of kids who have eradicated HIV with powerful surges of their immune system. I've not read about this stuff since doing the viral file, so much may have changed, but these kids--in fact all people infected with HIV--have these very strong immune system surges which try to eradicate the HIV. They are insufficient and each subsequent one a bit weaker and therefore even less likely to work. But researchers seemed to think that the few kids who did have HIV but then didn't upon later testing had actually hit strong enough the first time. I try to use that model in my protocol decisions, balancing that need for strength with the fact that its all happening inside a six year old body. Methylation is also crucial, I agree (you know my favourite animal is my much beloved agouti mouse ;-)). The problem for many of our mercury toxic kids is the fact that methylation, even if supported, must IMO be somewhat erratic, given the mercury's effect. So, we're stuck in that " where to begin? " problem. If you wait for methylation to be ideal before attacking viruses, if mercury is a problem, you'll wait for a very long time. At the same time, I do have that worry I wrote about in doing too many viral protocols that aren't entirely effective (that is, there is regression at some point after). I'm a bit foggy on this now: will teh enzymes be able to target viruses even if they're not active? Not the retroviruses, but other kinds. Seems to me I thought they could--but I may have been wrong about that, or maybe not remembering properly. I think lauricidin might be able to work that way also? It has a similar sort of action, not suppressing duplication, but digesting of a sort. This leads me to the brain. I asked deFelice about enzymes working systemically. We know they do, but in the brain is what I wonder. As you say, the brain is pretty defenseless once something is past the BBB. I'm going to look one day at the studies in her book that reference this stuff. It's a bit unnerving, to say the least, when you start reading about viruses. We have so many in us and they've lasted forever, historically speaking. Anita > > > > > > Hi > > > > > > We did a viral protocol starting at the end of August last year and > ending at Xmas. We saw huge gains, by the end my son was working in > class for 3 weeks up to the Xmas holidays without any support > (mainstream). We used prescription anti virals (prescribed by Dr > Magdalena at the Breakspear) of X -tra cell thymus factor, immuniovir > and acyclovir. They cost a lot, I think the 3 together were about > £270. > > > > > > However we then have seen a huge regression, so much so that school > have excluded him twice in the last 7 weeks and have now moved him into > another class! > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 20, 2009 Report Share Posted March 20, 2009 I took part in a Virastop trial (free pills) and so I was able to give as many as I liked. At one point I was giving 20 a day. I saw no response at all over about three months. The theory is that the enzymes break down the protein coat around the virus and enable them to be disposed of more easily by the immune system or whatever Sally Mum231ASD@... wrote: > > In a message dated 20/03/2009 18:31:07 GMT Standard Time, > mysuperteach@... writes: > > I'm a bit foggy on this now: will teh enzymes be able to target > viruses even if they're not active? Not the retroviruses, but > other kinds. > > >>I don;t think there is any evidence that enzymes have an antiviral > effect at all. The Virastop thing is solely based on feedback from > people given enough of them to try the higher dose protocol which is > what we did. > > Started Valtrex today....................still on LDM-100, doing high > dose C and planning to add back in Zeolites soon which gave us I think > biggest viral die off yet > > Mandi x > > > ------------------------------------------------------------------------ > > > No virus found in this incoming message. > Checked by AVG - www.avg.com > Version: 8.0.238 / Virus Database: 270.11.21/2014 - Release Date: 03/20/09 06:59:00 > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 20, 2009 Report Share Posted March 20, 2009 yes, it is also possible that in those kids that irradicate hiv, their cells, the ones that hiv infects, change/adapt in ways the virus cannot damage them any longer and has nowhere to go. epigenetics maybe? but you are right, viruses have been around so longer, and they are more intelligent than we are. I would not be suprised if HIV is soon found to have mutated and adapted for life in humans as to not be deadly any more at all. actually a new type of it has been discovered recently, HIV-2, and it is not nearly as dangerous to humans as HIV-1. it is not in viruse's best interest to have a host that drops dead, so really we can expect much more about this in future. re enzymes, good question, don't know the answer. natasa > > > > > > > > Hi > > > > > > > > We did a viral protocol starting at the end of August last year and > > ending at Xmas. We saw huge gains, by the end my son was working in > > class for 3 weeks up to the Xmas holidays without any support > > (mainstream). We used prescription anti virals (prescribed by Dr > > Magdalena at the Breakspear) of X -tra cell thymus factor, immuniovir > > and acyclovir. They cost a lot, I think the 3 together were about > > £270. > > > > > > > > However we then have seen a huge regression, so much so that school > > have excluded him twice in the last 7 weeks and have now moved him into > > another class! > > > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 20, 2009 Report Share Posted March 20, 2009 > > yes, it is also possible that in those kids that irradicate hiv, their > cells, the ones that hiv infects, change/adapt in ways the virus cannot > damage them any longer and has nowhere to go. epigenetics maybe? hmm, that's a really good point. I've only read about a handful of kids that this has happened to. Perhaps there are more, but if not, this phenomenon might say more about some very rare snps or genes being expressed than it says about immune systems in general. > > but you are right, viruses have been around so longer, and they are more > intelligent than we are. I would not be suprised if HIV is soon found to > have mutated and adapted for life in humans as to not be deadly any > more at all. actually a new type of it has been discovered recently, > HIV-2, and it is not nearly as dangerous to humans as HIV-1. it is not > in viruse's best interest to have a host that drops dead, so really we > can expect much more about this in future. we can also probably expect that the vaccine to eradicate hiv will be introduced at around this same time, and given full credit--just like for other previously deadly viruses. Anita Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 20, 2009 Report Share Posted March 20, 2009 > > yes, it is also possible that in those kids that irradicate hiv, their > cells, the ones that hiv infects, change/adapt in ways the virus cannot > damage them any longer and has nowhere to go. epigenetics maybe? hmm, that's a really good point. I've only read about a handful of kids that this has happened to. Perhaps there are more, but if not, this phenomenon might say more about some very rare snps or genes being expressed than it says about immune systems in general. -------------- human SNPs related to the immune system are very diverse for exactly that reason – to preserve humankind in cases of epidemics. So that even with most deadly viruses some individuals can be spared, and carry on. > > but you are right, viruses have been around so longer, and they are more > intelligent than we are. I would not be suprised if HIV is soon found to > have mutated and adapted for life in humans as to not be deadly any > more at all. actually a new type of it has been discovered recently, > HIV-2, and it is not nearly as dangerous to humans as HIV-1. it is not > in viruse's best interest to have a host that drops dead, so really we > can expect much more about this in future. we can also probably expect that the vaccine to eradicate hiv will be introduced at around this same time, and given full credit--just like for other previously deadly viruses. -------------- bingo!! Anita Quote Link to comment Share on other sites More sharing options...
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