[CoMeD] PR: LANDMARK STUDY FINDS: THIMEROSAL CAUSES AUTISM BRAIN PATHOLOGY

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All,

Attached is a Press Release, in pdf & doc formats,

for a recent research paper,

>Mitochondrial Dysfunction, Impaired Oxidative-

>Reduction Activity, Degeneration, and Death in

>Human Neuronal and Fetal Cells Induced by Low-

>Level Exposure to Thimerosal and Other Metal

>Compounds,

which clearly establishes that low (sub-ppm)

levels of Thimerosal can cause pathology in human

neuronal cell systems that is like that found in

the studies of brain tissue from those who have

been diagnosed with an Autism Spectrum Disorder.

For those who do not receive attachments, the

rough text of this PR states:

>LANDMARK STUDY FINDS: THIMEROSAL CAUSES AUTISM

> BRAIN PATHOLOGY

>

>PRESS RELEASE CONTACTS:

>For Immediate Release CoMeD President [Rev. K.

> Sykes (Richmond, VA)

> ]

>July 9, 2009 CoMeD Sci. Advisor [Dr. King

> (Lake Hiawatha, NJ)

> ]

>

>WASHINGTON, DC - A new study, " A Mitochondrial

>Dysfunction, Impaired Oxidative-Reduction Activity,

>Degeneration, and Death in Human Neuronal and Fetal

>Cells Induced by Low-Level Exposure to Thimerosal

>and Other Metal Compounds " , published in the most

>recent issue of the peer-reviewed journal of

>Toxicology & Environmental Toxicology [1], confirms

>a causal connection between Thimerosal and the brain

>pathology found in patients diagnosed with an autism

>spectrum disorder (ASD).

> [1] Geier DA, King PG, Geier MR. Mitochondrial

> Dysfunction, Impaired Oxidative-Reduction

> Activity, Degeneration, and Death in Human

> Neuronal and Fetal Cells Induced by Low-Level

> Exposure to Thimerosal and Other Metal Compounds.

> Toxicological & Environmental Chemistry 2009; 91:

> 735-749. [access to articles full-text available at:

>http://www.informaworld.com/smpp/content~db=all~content=a910652305 ]

>

>The research in this article [2] was undertaken to

>investigate cellular damage in three in vitro human

>neuronal and fetal-cell model systems. The potential

>damage induced by Thimerosal and other metal compounds,

>including aluminum sulfate, methylmercury hydroxide,

>lead acetate, and mercuric chloride was assessed using

>cell vitality assays and microscope-based digital image

>capture techniques.

> [2] Researchers with extensive backgrounds in medicine,

> chemistry, genetics, and biochemistry, from the

> Institute of Chronic Illnesses, Inc., CoMeD, Inc.,

> and the Genetic Centers of America collaborated on

> the study.

>

>This study showed Thimerosal-induced cellular damage in

>human neuronal and fetal-cell model systems in a

>concentration- and time- dependent fashion using

>Thimerosal at low nanomolar (parts-per-billion) concen-

>trations. These concentrations are comparable to those

>found in fetal and early infant exposure to mercury from

>Thimerosal-containing biologics and vaccines in the 1990s

>and, in some instances, today. These levels induced

>significant cellular toxicity in the human neuronal and

>fetal cells studied. The Thimerosal-induced cellular

>damage was consistent with that found in pathophysio-

>logical studies of patients diagnosed with an ASD. In

>both instances, the studies found significant mito-

>chondrial dysfunction, reduced cellular oxidative-

>reduction activity, cell degeneration, and cell death.

>

>The present study also revealed that Thimerosal is

>significantly more toxic than the other metal compounds

>studied (e.g., aluminum sulfate, methylmercury hydroxide,

>lead acetate, and mercuric chloride). The explanation

>for Thimerosal's greater toxicity than even methylmercury

>hydroxide (MeHgOH) appears to be the fact that Thimerosal

>was chemically engineered in the 1920s to be a more

>highly toxic alkylmercury compound, whose biological

>transport and intracellular delivery properties were

>enhanced. Compared to MeHgOH, Thimerosal has: 1) higher

>aqueous solubility (i.e. ability to dissolve in water

>and water-based systems); 2) higher solubility in cell

>membranes (i.e. ability to dissolve in cell membranes);

>and 3) higher intracellular toxicity (i.e. ability to

>inactivate essential cell processes) and mercury

>retention.

>

>The non-profit CoMeD, Inc., and, through a grant from

>the Brenen Hornstein Autism Research & Education (BHARE)

>Foundation, the non-profit Institute of Chronic Illnesses,

>Inc. funded this research study.

>

>Today, any parent, physician, or healthcare provider

>can easily confirm whether or not a non-chelated child

>diagnosed with an ASD is mercury poisoned by having

>urinary porphyrin profile analysis (UPPA) testing run at

>LabCorp (CLIA-certified, test# 120980) or Laboratoire

>Philippe Auguste (ISO-certified, 119 Philippe Auguste

>Avenue, Paris, France 75011). Please, visit CoMeD's web

>site, http://www.Mercury-freeDrugs.org for information

>on how to order UPPA tests and full copies of published

>papers validating the UPPA test.

>

>Your generous tax-free donations will help us fund

>additional research, similar to the present study, to

>examine mercury's links to autism and other illnesses,

>define the causal roles of mercury in the linked child-

>hood and adult illnesses, and find appropriate curative

>therapies.

>_______________________________________________________

>To support the ongoing efforts of CoMeD, Inc. with your

>tax-deductible contributions, please use the PayPal link

>on CoMeD's Internet website,

> http://www.Mercury-freeDrugs.org. CoMeD, Inc. is a

>not-for-profit 501©(3) corporation that is actively

>engaged in legal, educational and scientific efforts to

>stop all use of mercury in medicine, and to ban the use

>of all mercury-containing medicines.

>

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Hopefully, after reading this PR and the article,

you will better understand how Thimerosal damages

and kills brain cells and distorts the brain's

function.

Respectfully,

Dr. King

http://www.dr-king.com

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