Guest guest Posted May 6, 1999 Report Share Posted May 6, 1999 I found this article in the Great Smokies Diagnostic Lab newletter. Ithought it might be of interest. > >- HIGH C-REACTIVE PROTEIN TIED TO STROKE, CVD MORTALITY > >============== > >- SENSITIVE ACUTE PHASE REACTANT PROVIDES EARLY WARNING FOR SYSTEMIC >INFLAMMATION > >A growing body of scientific evidence is showing that chronic, low-grade >inflammation can be a hidden and deadly force driving the pathogenesis >of cardiovascular disease (CVD). Recently, researchers at the American >Heart Association's 39th Annual Conference on Cardiovascular Disease >Epidemiology and Prevention in Orlando presented new studies linking >C-reactive protein, a marker for low-grade systemic inflammation, with a >barrage of cardiovascular abnormalities. Among the dramatic findings: > >*Non-smokers with levels of C-reactive protein (CRP) in the highest >tertile had a 4.7-fold greater relative risk of suffering a sudden >stroke. The correlation between high CRP and stroke was strongest in >middle-aged men and non-smokers, suggesting CRP's pronounced ability to >identify individuals without visible risk factors early. > >*Elevated CRP was associated with significantly increased odds for >coronary heart disease and overall mortality, including cancer death, >CVD death, and non-CVD death in men, independent of age, smoking, lipid >profile, and other factors. > >*High CRP tended to run in families, with first-degree relatives showing >a marked tendency to " experience a similar systemic inflammatory >response because of shared genes or environmental factors. " > >Researchers called for a newer, more advanced approach to treating and >preventing cardiovascular disease using the latest modifiable markers. > > " The outlook for cardiovascular disease (CVD) - heart disease and stroke >- is bright " observed Dr. Kuller, University professor of Public >Health at the University of Pittsburgh. " The evolving understanding of >genetics will help identify a person's susceptibility to CVD and how >lifestyles, drug therapies, and environmental risk factors interact. " > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted June 30, 2001 Report Share Posted June 30, 2001 CRP is a non specific acute phase reactant and wouldn't help in diagnosing building related illness (IMO) and/or mold related reactions. We have seen that people with cornary heart disease and high CRPs are prone towards acute events (MIs). Organization: WEB-Presentations™ < > From: Barth <pbarthulster (DOT) net> Date sent: Sat, 30 Jun 2001 12:27:33 -0400 Send reply to: Subject: [] CRP Has anyone had high CRP blood levels from toxic mold? Barth SBS: MY STORY: www.presenting.net/sbs/sbs.html Terms of Service. Quote Link to comment Share on other sites More sharing options...
Guest guest Posted June 30, 2001 Report Share Posted June 30, 2001 As a physician who evaluates Sick Buildings and building related illness I would be interested in hearing on how your doctors made the correlation between PF from mold exposure (circumstances, tests, reasoning, literature cites) etc. Was this the result of a work related exposure ? Feel free to share what you wish privately or on the list. Alan Ross MD Date sent: Sat, 30 Jun 2001 16:54:56 -0400 From: Barth <pbarth@...> Send reply to: Barth <pbarth@...> Organization: WEB-Presentations™ " Alan Ross " <compchat@...> Subject: CRP > Thanks, Alan. Ever since I developed pulmonary fibrosis from mold, my > sed rate has been consistently high (around 45). Now the CRP level is > off the wall - 13. It is frightening, because I've been reading about > the link to heart attack and stroke. > > -- > Pat > NEW Mini CD-Rom Business Cards > WEB-Presentations™ www.presenting.net > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted June 30, 2001 Report Share Posted June 30, 2001 Dear Dr. Ross, I've shared some of this with the list already and will be doing some local and national media on this, so I don't mind answering your questions. Dr. Saxon, the pulmonary specialist I was seeing at first made the correlation between PF from mold exposure in the building in which I worked (United HealthCare - now Uniprise, ironically) because my oxygen level dipped drastically while I was there. What I didn't know until a few weeks ago was that he had just treated another victim of the the building - same thing - pulmonary fibrosis. This young man wasn't so fortunate. He died! I'm sure that Dr. Saxon consulted with the other Drs. in the group, as they are treating others from this building who faint frequently and have other symptoms. It was after this that he told me: " This thing is bigger than you or I " . No longer would he commit to saying that the building caused this. United HealthCare is their bread and butter combined with the fact that it supplies up to 600 jobs in this IBM deserted desolate rural area. For a more detailed account of my particular case, please review the story at the url provided below. In addition to that information, the building was in very bad repair with gushes of water coming in when it rained. We had to put wastebaskets under the leaks, but furniture upholstery was saturated anyway. Ceiling tiles presented water stains of different colors all over the building. Too much more to relate here. Hope this has helped. Barth SBS: MY STORY: www.presenting.net/sbs/sbsstory.html Quote Link to comment Share on other sites More sharing options...
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