Re: Post-op TPLO with occasional limping

[Guest guest]

Diane,

All dogs with CCL disease, even with surgical correction, will develop

osteoarthritis. The use of disease modifying osteoarthritic drugs, Adequan is

my recommendation, and Omega 3 fatty acids, ( I recommend by way of diet ) will

help slow down the development and additionally manage the effects.

If the implant is the problem then there should be pain localized to that site,

radiographic evidence of implant loosening, fistulazation and/or infection.

A medial meniscal injury is always a consideration and if so pain should be

localized to the joint and you may appreciate some crepitus on manipulation.

I think Krista's comments concerning DM are of importance due to the breed and

perhaps should always be a consideration, however I would not expect it to be

the cause of the obvious weight bearing lameness in the surgical limb but

individual cases can always vary!

I am all behind Jean's comments to look at the other areas that are asked to

compensate for the stifle dysfunction, especially the hip flexors (m. sartorius,

m. tensor fascia latae, m. rectus femoris and m. iliopsoas {m. psoas major and

m. iliacus} and also the the other stifle extensors that I refer to as a group -

vastus group. Problems in these areas can prolong recovery by limiting joint

rom of the coxofemoral and stifle joint and adding to joint dysfunction and

pain. They prolong the stifle dysfunction brought about by the primary injury

and aggravated by the trauma from surgery.

Your history of improvement with your therapy supports a possible myofascial,

caused by and complicating the existing joint dysfunction.

Examine joints and muscles for pain and let us know what you find.

Rick Wall

[Guest guest]

Not disagreeing...but if ALL dogs with CCL disease develop OA, why do we charge

folks thousands of dollars for surgery?

[Guest guest]

Please take me off your e mail list stephen fletcher

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- Re: Post-op TPLO with occasional limping

Not disagreeing...but if ALL dogs with CCL disease develop OA, why do we charge

folks thousands of dollars for surgery?

[Guest guest]

1) surgery slows down and can even stop the progression-not all dogs get OA-most have it long before we arrive on the scene as surgeons- Freedman VMD CCRT

 

Not disagreeing...but if ALL dogs with CCL disease develop OA, why do we charge folks thousands of dollars for surgery?

[Guest guest]

lizzypowpow,

It sounds as if you are disagreeing!

Since CCL problems in the canine are much more of a long standing progressive

disease, often by the time they present, significant damage has already been

done to the articular cartilage. This damage will result in osteoarthritiis.

In the patient with a totally normal CCL who acutely ruptures (no preexisting

disease) then immediate stabilization can possibly prevent intraarticular damage

and subsequent OA would be minimal.

It would be helpful if you would provide a signature at the end of your comments

so that we may address you professionally. I look forward to your response and

suggestion of alternative therapies that are not as costly!

Rick Wall, DVM

Certified Canine Rehabilitation Practitioner

Diplomate, American Academy of Pain Management

Certified Myofascial Trigger Point Therapist

Center for Veterinary Pain Management and Rehabilitation

The Woodlands, TX

www.vetrehabcenter.com

[Guest guest]

Hi there,

A follow-up on Grizzly, my limping TPLO patient:

I did find a painful sartorius, worked on that with laser and massage, continued

his home exercise program. The knot in his sartorius went away, but he still

didn't improve all that much so we finally had the plate removed.

The surgeon said the plate had a small black corroded-looking area on it. It was

cultured and grew serratia.

Grizzly is doing much better now.

Thanks for all the suggestions,

Diane

>

> Diane,

>

> All dogs with CCL disease, even with surgical correction, will develop

osteoarthritis. The use of disease modifying osteoarthritic drugs, Adequan is

my recommendation, and Omega 3 fatty acids, ( I recommend by way of diet ) will

help slow down the development and additionally manage the effects.

>

> If the implant is the problem then there should be pain localized to that

site, radiographic evidence of implant loosening, fistulazation and/or

infection.

>

> A medial meniscal injury is always a consideration and if so pain should be

localized to the joint and you may appreciate some crepitus on manipulation.

>

> I think Krista's comments concerning DM are of importance due to the breed and

perhaps should always be a consideration, however I would not expect it to be

the cause of the obvious weight bearing lameness in the surgical limb but

individual cases can always vary!

>

> I am all behind Jean's comments to look at the other areas that are asked to

compensate for the stifle dysfunction, especially the hip flexors (m. sartorius,

m. tensor fascia latae, m. rectus femoris and m. iliopsoas {m. psoas major and

m. iliacus} and also the the other stifle extensors that I refer to as a group -

vastus group. Problems in these areas can prolong recovery by limiting joint

rom of the coxofemoral and stifle joint and adding to joint dysfunction and

pain. They prolong the stifle dysfunction brought about by the primary injury

and aggravated by the trauma from surgery.

>

> Your history of improvement with your therapy supports a possible myofascial,

caused by and complicating the existing joint dysfunction.

>

> Examine joints and muscles for pain and let us know what you find.

>

> Rick Wall

>