How Interferon Ruins Your Brain

June 6, 2005

The mechanism of this injury to the brain appears to involve the brain's

special immune cell called the microglia. Normally, these cells remain dormant

in the brain. That is, they are sleeping. Microglia cells can be activated

by numerous factors, including mercury, aluminum, iron, overvaccination, and

brain trauma, strokes, infections (viruses, bacteria, rickettsia) and

cytokines such as interferons.

Once activated, microglia can move about the brain secreting very toxic

compounds, which include two excitotoxins (glutamate and quinolinic acid).

These excitotoxins dramatically increase free radical generation in the brain

as

well as oxidation of lipids (called lipid peroxidation). These radicals

damage synaptic connections, interfere with neurotransmitters and can even

kill

neurons. In addition, these activated microglia generate other toxic

compounds such as prostaglandins (PGE2), which increase brain inflammation.

If the microglia activation is short lived, the damage to the brain is

minimal and recovery takes place. Yet, should the activation continue,

which would occur with high-dose and long-term use of interferons, the damage

could be substantial and irreversible. Protecting the brain with high-dose and

varied antioxidants as well as certain metabolic stimulants can

substantially reduce this damage. Certain nutrients, such as malate, pyruvate,

DHA,

ascorbate, magnesium and methylcobalamin inhibit excitotoxicity.

Physicians Frequently Miss Side Effects

Physicians often ignore patient complaints of neurological difficulties

during interferon treatments, assuming they are benign and reversible. As

stated in the beginning, natural alternatives have been shown to be much more

effective and dramatically safer than interferon treatments.

No information in this letter should be construed as medical advice.

This information is for educational purposes only.

Jeff el

10360 Pine Lakes Blvd

North Fort Myers, Fl 33903

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