Chronic Neurotoxins

[Guest guest]

Dear Carol,

I would like to have a copy of your summary of the book re organic

neurotoxins. It is very kind of you to summarize it and offer it.

Thank you, Chris

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[Guest guest]

a,

That's very interesting. Dr. Ritchie Shoemaker has identified quite

a few illnesses now that he classifies as chronic neurotoxin

mediated illnesses. I hadn't heard that C. pneumonia is one of

them, but maybe it is. He has identified dinoflagellate-related

illnesses such as ciguatera and pfiesteria, sick building syndrome

caused by a variety of molds, post-lyme disease, and coagulase-

negative staphylococcus infections in the nose. He has also found

that some CFS patients have neurotoxins, but apparently he is not

sure where they come from. They seem to be generated in the body.

One possibility is that a stealth virus has transferred DNA that

codes for these toxins into the DNA of the cells of the body. I

think this whole subject is a major development.

Rich

> > > This post is for Judith and for all on the

> > > Experimental list re: recent queries on Lyme.

> >

> >

> > This list is intended for patients to share personal experiences

with each

> other, not to give medical advice. If you are interested in any

treatment

> discussed here, please consult your doctor.

> >

> >

[Guest guest]

Rich, I went back and looked this up in my notes. Surprise, I had an error.

It was mycoplasmal pneumonia, not c. pneumoniae. But the concept was

correct. I am posting my overview and the reference if you want to look the

abstract up on Medline.

MYCOPLASMA PNEUMONIAE

R. D. Hardy from the University of Texas Southwestern Medical Center,

Dallas, TX 10described his study in which mice were infected with live

mycoplasma pneumoniae and UV killed mycoplasma. They were then treated with

clarithromycin. The mice with the live mycoplasma recovered. The mice with

the dead mycoplasma did not recover from the inflammation. Apparently dead

mycoplasma is still capable of triggering an autoimmune reaction. We have

been told that minocycline is anti-inflammatory. That is why it is effective

for autoimmune diseases such as rheumatoid arthritis. This study would

suggest that antibiotics, effective when the pathogen is still alive, are

not effective as anti-inflammatories.

10. Microbiologic and Immunologic Activity of Clarithromycin in a Murine

Model of Mycoplasma pneumoniae Pneumonia Category: B. Therapy in Animal

Models, Pathogenesis and Host Defenses R. D. HARDY, A. M. RIOS, S.

CHAVEZ-BUENO, H. S. JAFRI, J. HATFIELD, B. B. ROGERS, G. H. MCCRACKEN, O.

RAMILO; University of Texas Southwestern Medical Center, Dallas, TX.

Presentation Number: B-701 Poster

> a,

>

> That's very interesting. Dr. Ritchie Shoemaker has identified quite

> a few illnesses now that he classifies as chronic neurotoxin

> mediated illnesses. I hadn't heard that C. pneumonia is one of

> them, but maybe it is. He has identified dinoflagellate-related

> illnesses such as ciguatera and pfiesteria, sick building syndrome

> caused by a variety of molds, post-lyme disease, and coagulase-

> negative staphylococcus infections in the nose. He has also found

> that some CFS patients have neurotoxins, but apparently he is not

> sure where they come from. They seem to be generated in the body.

> One possibility is that a stealth virus has transferred DNA that

> codes for these toxins into the DNA of the cells of the body. I

> think this whole subject is a major development.

>

> Rich

>

>

> > Rich, one of the abtracts at ICAAC mentioned that c. pneumoniae

> could be

> > killed with antibiotics but that there were toxins which remained

> and the

> > patient was still sick. I found this very interesting and sounds

> like some

> > of the info on the website you gave us.

> > a

> >

> >

> > > Hope,

> > >

> > > Have you tried taking the Visual Contrast Sensitivity test at

> > > www.chronicneurotoxins.com?

> > >

> > > Rich

[Guest guest]

http://www.chronicneurotoxins.com

You'll need to join to explore this website . . . but

there's lot of info there! Rogene

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The role of biotoxins from mold in both saline and

silicone patients and this may be a major factor in

breast implant disease.

For many decades manufacturers have sold products,

such as silicone gel/saline, single-lumen (saline

mixed with silicone gel) and double-lumen adjustable

breast implants.

Saline Implants have leaky valves, shell defects

(rupture). Micro Organisms accumulate over many years.

As a result, with time, the closed space fills with a

complex mixture of bacteria, fungi, algae, and slimes.

Organisms Found In Such Environments Include:

Pseudomonas Aeruginosa, Pseudomonas Putida,

Streptococci, Spivarum, CoccidioidesImmitis, Papilloma

Viri, Herpes Simplex, Aspergillus Fumigatus,

Aspergillus Boufardi, Aspergillus Niger, Bacteroides

Fragillis, Curvularia, Staphylococci, Mycobacterium

Chelonei, Mycobacterium Fortuitum, Mycobacterium

Tuberculosis, Mycobacterium Avium, Alternaria Tenuis,

Rhodotorula Glutinis, Penicillium Notatum, Microsporum

Epidermophyton, Ricophyton, Candida Albicans, Proteus

Mirabillis, Propioni Bacterium Acne, Serratia

Marcescens as well as their metabolites and toxins.

A saline-filled breast implant has a silicone rubber

shell of polysiloxane(s), such as polydimethylsiloxane

and polydiphenylsiloxane, which is inflated with a

sterile isotonic saline. Silicone implants ALSO had

saline in some of them. The sterile saline used as a

filler is injection grade.

*Shelf- life and storage conditions for Intravenous

saline: 2 YEARS at a temperature NOT exceeding 25C