Our Deadly Diabetes Deception

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Our Deadly Diabetes Deception

Greed and dishonest science have promoted a lucrative worldwide

epidemic of diabetes that honesty and good science can quickly

reverse by naturally restoring the body's blood-sugar control

mechanism.

Extracted from Nexus Magazine, Volume 11, Number 4 (June-July 2004)

PO Box 30, Mapleton Qld 4560 Australia. editor@n...

Telephone: +61 (0)7 5442 9280; Fax: +61 (0)7 5442 9381

>From our web page at: www.nexusmagazine.com by © 2004

PO Box 7685

Loveland, CO 80537 USA

Email: <Valley@h...>

Website: http://www.Healingmatters.com

Introduction

The Diabetes Industry

Diabetes History

Cure versus Treatment

The Commercial Value of Symptoms

Epidemiological Lifestyle Link

The Nature of the Disease

Orthodox Medical Treatment

Alternative Medical Treatment

Recovery Time

About the Author

Endnotes

Introduction

If you are an American diabetic, your physician will never tell you

that most cases of diabetes are curable. In fact, if you even mention

the " cure " word around him, he will likely become upset and

irrational. His medical school training only allows him to respond to

the word " treatment " . For him, the " cure " word does not exist.

Diabetes, in its modern epidemic form, is a curable disease and has

been for at least 40 years. In 2001, the most recent year for which

US figures are posted, 934,550 Americans died from out-of-control

symptoms of this disease.1

Your physician will also never tell you that, at one time, strokes,

both ischaemic and haemorrhagic, heart failure due to neuropathy as

well as both ischaemic and haemorrhagic coronary events, obesity,

atherosclerosis, elevated blood pressure, elevated cholesterol,

elevated triglycerides, impotence, retinopathy, renal failure, liver

failure, polycystic ovary syndrome, elevated blood sugar, systemic

candida, impaired carbohydrate metabolism, poor wound healing,

impaired fat metabolism, peripheral neuropathy as well as many more

of today's disgraceful epidemic disorders were once well understood

often to be but symptoms of diabetes.

If you contract diabetes and depend upon orthodox medical treatment,

sooner or later you will experience one or more of its symptoms as

the disease rapidly worsens. It is now common practice to refer to

these symptoms as if they were separable, independent diseases with

separate, unrelated treatments provided by competing medical

specialists.

It is true that many of these symptoms can and sometimes do result

from other causes; however, it is also true that this fact has been

used to disguise the causative role of diabetes and to justify

expensive, ineffective treatments for these symptoms.

Epidemic Type II diabetes is curable. By the time you get to the end

of this article, you are going to know that. You're going to know why

it isn't routinely being cured. And, you're going to know how to cure

it. You are also probably going to be angry at what a handful of

greedy people have surreptitiously done to the entire orthodox

medical community and to its trusting patients.

The Diabetes Industry

Today's diabetes industry is a massive community that has grown step

by step from its dubious origins in the early 20th century. In the

last 80 years it has become enormously successful at shutting out

competitive voices that attempt to point out the fraud involved in

modern diabetes treatment. It has matured into a religion. And, like

all religions, it depends heavily upon the faith of the believer. So

successful has it become that it verges on blasphemy to suggest that,

in most cases, the kindly high priest with the stethoscope draped

prominently around his neck is a charlatan and a fraud. In the large

majority of cases, he has never cured a single case of diabetes in

his entire medical career.

The financial and political influence of this medical community has

almost totally subverted the original intent of our regulatory

agencies. They routinely approve death-dealing, ineffective drugs

with insufficient testing. Former commissioner of the FDA, Dr Herbert

Ley, in testimony before a US Senate hearing, commented: " People

think the FDA is protecting them. It isn't. What the FDA is doing and

what the public thinks it's doing are as different as night and

day. " 2

The financial and political influence of this medical community

dominates our entire medical insurance industry. Although this is

beginning to change, in America it is still difficult to find

employer group medical insurance to cover effective alternative

medical treatments. Orthodox coverage is standard in all states.

Alternative medicine is not. For example, there are only 1,400

licensed naturopaths in 11 states compared to over 3.4 million

orthodox licensees in 50 states.3 Generally, only approved treatments

from licensed, credentialed practitioners are insurable. This, in

effect, neatly creates a special kind of money that can only be spent

within the orthodox medical and drug industry. No other industry in

the world has been able to manage the politics of convincing people

to accept so large a part of their pay in a form that often does not

allow them to spend it as they see fit.

The financial and political influence of this medical community

completely controls virtually every diabetes publication in the

country. Many diabetes publications are subsidised by ads for

diabetes supplies. No diabetes editor is going to allow the truth to

be printed in his magazine. This is why the diabetic only pays about

one-quarter to one-third of the cost of printing the magazine he

depends upon for accurate information. The rest is subsidised by

diabetes manufacturers with a vested commercial interest in

preventing diabetics from curing their diabetes. When looking for a

magazine that tells the truth about diabetes, look first to see if it

is full of ads for diabetes supplies.

And then there are the various associations that solicit annual

donations to find a cure for their proprietary disease. Every year

they promise that a cure is just around the corner money! Some of these very

same associations have been clearly

implicated in providing advice that promotes the progress of diabetes

in their trusting supporters. For example, for years they heavily

promoted exchange diets,4 which are in fact scientifically worthless<

as anyone who has ever tried to use them quickly finds out. They

ridiculed the use of glycaemic tables, which are actually very

helpful to the diabetic. They promoted the use of margarine as heart

healthy, long after it was well understood that margarine causes

diabetes and promotes heart failure.5

If people ever wake up to the cure for diabetes that has been

suppressed for 40 years, these associations will soon be out of

business. But until then, they nonetheless continue to need our

support.

For 40 years, medical research has consistently shown with increasing

clarity that diabetes is a degenerative disease directly caused by an

engineered food supply that is focused on profit instead of health.

Although the diligent can readily glean this information from a

wealth of medical research literature, it is generally otherwise

unavailable. Certainly this information has been, and remains,

largely unavailable in the medical schools that train our retail

doctors.

Prominent among the causative agents in our modern diabetes epidemic

are the engineered fats and oils that are sold in today's

supermarkets.

The first step to curing diabetes is to stop believing the lie that

the disease is incurable.

Diabetes History

In 1922, three Canadian Nobel Prize winners, Banting, Best and

Macleod, were successful in saving the life of a fourteen-year-old

diabetic girl in Toronto General Hospital with injectable insulin.6

Eli Lilly was licensed to manufacture this new wonder drug, and the

medical community basked in the glory of a job well done.

It wasn't until 1933 that rumours about a new rogue form of diabetes

surfaced. This was in a paper presented by Joslyn, Dublin and Marks

and printed in the American Journal of Medical Sciences. This

paper, " Studies on Diabetes Mellitus " ,7 discussed the emergence of a

major epidemic of a disease which looked very much like the diabetes

of the early 1920s, only it did not respond to the wonder drug,

insulin. Even worse, sometimes insulin treatment killed the patient.

This new disease became known as " insulin-resistant diabetes " because

it had the elevated blood sugar symptom of diabetes but responded

poorly to insulin therapy. Many physicians had considerable success

in treating this disease through diet. A great deal was learned about

the relationship between diet and diabetes in the 1930s and 1940s.

Diabetes, which had a per-capita incidence of 0.0028% at the turn of

the century, had by 1933 zoomed 1,000% in the United States to become

a disease seen by many doctors.8 This disease, under a variety of

aliases, was destined to go on to wreck the health of over half the

American population and incapacitate almost 20% by the 1990s.9

In 1950, the medical community became able to perform serum insulin

assays. These assays quickly revealed that this new disease wasn't

classic diabetes; it was characterised by sufficient, often

excessive, blood insulin levels.

The problem was that the insulin was ineffective; it did not reduce

blood sugar. But since the disease had been known as diabetes for

almost 20 years, it was renamed Type II diabetes. This was to

distinguish it from the earlier Type I diabetes, caused by

insufficient insulin production by the pancreas.

Had the dietary insights of the previous 20 years dominated the

medical scene from this point and into the late 1960s, diabetes would

have become widely recognised as curable instead of merely treatable.

Instead, in 1950, a search was launched for another wonder drug to

deal with the Type II diabetes problem.

Cure versus Treatment

This new, ideal, wonder drug would be effective, like insulin, in

remitting obvious adverse symptoms of the disease but not effective

in curing the underlying disease. Thus it would be needed continually

for the remaining life of the patient. It would have to be

patentable; that is, it could not be a natural medication because

these are non-patentable. Like insulin, it would have to be highly

profitable to manufacture and distribute. Mandatory government

approvals would be required to stimulate physicians to prescribe it

as a prescription drug. Testing required for these approvals would

have to be enormously expensive to prevent other, unapproved,

medications from becoming competitive.

This is the origin of the classic medical protocol of " treating the

symptoms " . By doing this, both the drug company and the doctor could

prosper in business, and the patient, while not being cured of his

disease, was sometimes temporarily relieved of some of his symptoms.

Additionally, natural medications that actually cured disease would

have to be suppressed. The more effective they were, the more they

would need to be suppressed and their proponents jailed as quacks.

After all, it wouldn't do to have some cheap, effective, natural

medication cure disease in a capital-intensive monopoly market

specifically designed to treat symptoms without curing disease.

Often the natural substance really did cure disease. This is why the

force of law has been and is being used to drive the natural, often

superior, medicines from the marketplace, to remove the " cure " word

from the medical vocabulary and to undermine totally the very concept

of a free marketplace in the medical business.

Now it is clear why the " cure " word is so vigorously suppressed by

law. The FDA has extensive Orwellian regulations that prohibit the

use of the " cure " word to describe any competing medicine or natural

substance. It is precisely because many natural substances do

actually both cure and prevent disease that this word has become so

frightening to the drug and orthodox medical community.

The Commercial Value of Symptoms

After the drug development policy was redesigned to focus on

ameliorating symptoms rather than curing disease, it became necessary

to reinvent the way drugs were marketed. This was done in 1949 in the

midst of a major epidemic of insulin-resistant diabetes.

So, in 1949, the US medical community reclassified the symptoms of

diabetes10 along with many other disease symptoms into diseases in

their own right. With this reclassification as the new basis for

diagnosis, competing medical speciality groups quickly seized upon

related groups of symptoms as their own proprietary symptoms set.

Thus the heart specialist, endocrinologist, allergist, kidney

specialist and many others started to treat the symptoms for which

they felt responsible. As the underlying cause of the disease was

widely ignored, all focus on actually curing anything was completely

lost.

Heart failure, for example, which had previously been understood

often to be but a symptom of diabetes, now became a disease not

directly connected to diabetes. It became fashionable to think that

diabetes " increased cardiovascular risk " . The causal role of a failed

blood-sugar control system in heart failure became obscured.

Consistent with the new medical paradigm, none of the treatments

offered by the heart specialist actually cures, or is even intended

to cure, their proprietary disease. For example, the three-year

survival rate for bypass surgery is almost exactly the same as if no

surgery was undertaken.11

Today, over half of the people in America suffer from one or more

symptoms of this disease. In its beginnings, it became well known to

physicians as Type II diabetes, insulin-resistant diabetes, insulin

resistance, adult-onset diabetes or, more rarely, hyperinsulinaemia.

According to the American Heart Association, almost 50% of Americans

suffer from one or more symptoms of this disease. One third of the US

population is morbidly obese; half of the population is overweight.

Type II diabetes, also called adult-onset diabetes, now appears

routinely in six-year-old children.

Many degenerative diseases can be traced to a massive failure of the

endocrine system. This was well known to the physicians of the 1930s

as insulin-resistant diabetes. This basic underlying disorder is

known to be a derangement of the blood-sugar control system by badly

engineered fats and oils. It is exacerbated and complicated by the

widespread lack of other essential nutrition that the body needs to

cope with the metabolic consequences of these poisons.

All fats and oils are not equal. Some are healthy and beneficial;

many, commonly available in the supermarket, are poisonous. The

health distinction is not between saturated and unsaturated, as the

fats and oils industry would have us believe. Many saturated oils and

fats are highly beneficial; many unsaturated oils are highly

poisonous. The important health distinction is between natural and

engineered.

There exists great dishonesty in advertising in the fats and oils

industry. It is aimed at creating a market for cheap junk oils such

as soy, cottonseed and rapeseed oils.

With an informed and aware public, these oils would have no market at

all, and the USA diabetes.

Epidemiological Lifestyle Link

As early as 1901, efforts had been made to manufacture and sell food

products by the use of automated factory machinery because of the

immense profits that were possible. Most of the early efforts failed

because people were inherently suspicious of food that wasn't farm

fresh and because the technology was poor. As long as people were

prosperous, suspicious food products made little headway. Crisco,12

the artificial shortening, was once given away free in 21 & #8260;2 lb

cans

in an unsuccessful effort to influence American housewives to trust

and buy the product in preference to lard.

Margarine was introduced and was bitterly opposed by the dairy states

in the USA. With the advent of the Depression of the 1930s,

margarine, Crisco and a host of other refined and hydrogenated

products began to make significant penetration into the food markets

of America. Support for dairy opposition to margarine faded during

World War II because there wasn't enough butter for the needs of both

the civilian population and the military.13 At this point, the dairy

industry, having lost much support, simply accepted a diluted market

share and concentrated on supplying the military.

Flax oils and fish oils, which were common in the stores and

considered dietary staples before the American population became

diseased, have disappeared from the shelf. The last supplier of flax

oil to the major distribution chains was Archer s Midland, and

it stopped producing and supplying the product in 1950.

More recently, one of the most important of the remaining, genuinely

beneficial, fats was subjected to a massive media disinformation

campaign that portrayed it as a saturated fat that causes heart

failure. As a result, it has virtually disappeared from the

supermarket shelves. Thus was coconut oil removed from the food chain

and replaced with soy oil, cottonseed oil and rapeseed oil.14 Our

parents and grandparents would never have swapped a fine, healthy oil

like coconut oil for these cheap, junk oils. It was shortly after

this successful media blitz that the US populace lost its war on fat.

For many years, coconut oil had been our most effective dietary

weight-control agent.

The history of the engineered adulteration of our once-clean food

supply exactly parallels the rise of the epidemic of diabetes and

hyperinsulinaemia now sweeping the United States as well as much of

the rest of the world.

The second step to a cure for this disease epidemic is to stop

believing the lie that our food supply is safe and nutritious.

The Nature of the Disease

Diabetes is classically diagnosed as a failure of the body to

metabolise carbohydrates properly. Its defining symptom is a high

blood-glucose level. Type I diabetes results from insufficient

insulin production by the pancreas. Type II diabetes results from

ineffective insulin. In both types, the blood-glucose level remains

elevated. Neither insufficient insulin nor ineffective insulin can

limit post-prandial (after-eating) blood sugar to the normal range.

In established cases of Type II diabetes, these elevated blood sugar

levels are often preceded and accompanied by chronically elevated

insulin levels and by serious distortions of other endocrine hormonal

markers.

The ineffective insulin is no different from effective insulin. Its

ineffectiveness lies in the failure of the cell population to respond

to it. It is not the result of any biochemical defect in the insulin

itself. Therefore, it is appropriate to note that this is a disease

that affects almost every cell in the 70 trillion or so cells of the

body. All of these cells are dependent upon the food that we eat for

the raw materials they need for self repair and maintenance.

The classification of diabetes as a failure to metabolise

carbohydrates is a traditional classification that originated in the

early 19th century when little was known about metabolic diseases or

processes.15 Today, with our increased knowledge of these processes,

it would appear quite appropriate to define Type II diabetes more

fundamentally as a failure of the body to metabolise fats and oils

properly. This failure results in a loss of effectiveness of insulin

and in the consequent failure to metabolise carbohydrates.

Unfortunately, much medical insight into this matter, except at the

research level, remains hampered by its 19th-century legacy.

Thus Type II diabetes and its early hyperinsulinaemic symptoms are

whole-body symptoms of this basic cellular failure to metabolise

glucose properly. Each cell of the body, for reasons which are

becoming clearer, finds itself unable to transport glucose from the

bloodstream to its interior. The glucose then remains in the

bloodstream, or is stored as body fat or as glycogen, or is otherwise

disposed of in urine.

It appears that when insulin binds to a cell membrane receptor, it

initiates a complex cascade of biochemical reactions inside the cell.

This causes a class of glucose transporters known as GLUT4 molecules

to leave their parking area inside the cell and travel to the inside

surface of the plasma cell membrane.

When in the membrane, they migrate to special areas of the membrane

called caveolae areas.16 There, by another series of biochemical

reactions, they identify and hook up with glucose molecules and

transport them into the interior of the cell by a process called

endocytosis. Within the cell's interior, this glucose is then burned

as fuel by the mitochondria to produce energy to power cellular

activity. Thus these GLUT4 transporters lower glucose in the

bloodstream by transporting it out of the bloodstream into all the

cells of the body.

Many of the molecules involved in these glucose- and insulin-mediated

pathways are lipids; that is, they are fatty acids. A healthy plasma

cell membrane, now known to be an active player in the glucose

scenario, contains a complement of cis-type w=3 unsaturated fatty

acids.17 This makes the membrane relatively fluid and slippery. When

these cis- fatty acids are chronically unavailable because of our

diet, trans- fatty acids and short- and medium-chain saturated fatty

acids are substituted in the cell membrane. These substitutions make

the cellular membrane stiffer and more sticky, and inhibit the

glucose transport mechanism.18

Thus, in the absence of sufficient cis omega 3 fatty acids in our

diet, these fatty acid substitutions take place, the mobility of the

GLUT4 transporters is diminished, the interior biochemistry of the

cell is changed and glucose remains elevated in the bloodstream.

Elsewhere in the body, the pancreas secretes excess insulin, the

liver manufactures fat from the excess sugar, the adipose cells store

excess fat, the body goes into a high urinary mode, insufficient

cellular energy is available for bodily activity and the entire

endocrine system becomes distorted. Eventually, pancreatic failure

occurs, body weight plummets and a diabetic crisis is precipitated.

Although there remains much work to be done to elucidate fully all of

the steps in all of these pathways, this clearly marks the beginning

of a biochemical explanation for the known epidemiological

relationship between cheap, engineered dietary fats and oils and the

onset of Type II diabetes.

Orthodox Medical Treatment

After the diagnosis of diabetes, modern orthodox medical treatment

consists of either oral hypoglycaemic agents or insulin.

? Oral hypoglycaemic agents

In 1955, oral hypoglycaemic drugs were introduced. Currently

available oral hypoglycaemic agents fall into five classifications

according to their biophysical mode of action.19 These classes are:

biguanides; glucosidase inhibitors; meglitinides; sulphonylureas; and

thiazolidinediones.

The biguanides lower blood sugar in three ways. They inhibit the

normal release by the liver of its glucose stores, they interfere

with intestinal absorption of glucose from ingested carbohydrates,

and they are said to increase peripheral uptake of glucose.

The glucosidase inhibitors are designed to inhibit the amylase

enzymes produced by the pancreas and which are essential to the

digestion of carbohydrates. The theory is that if the digestion of

carbohydrates is inhibited, the blood sugar level cannot be elevated.

The meglitinides are designed to stimulate the pancreas to produce

insulin in a patient that likely already has an elevated level of

insulin in their bloodstream. Only rarely does the doctor even

measure the insulin level. Indeed, these drugs are frequently

prescribed without any knowledge of the pre-existing insulin level.

The fact that an elevated insulin level is almost as damaging as an

elevated glucose level is widely ignored.

The sulphonylureas are another pancreatic stimulant class designed to

stimulate the production of insulin. Serum insulin determinations are

rarely made by the doctor before he prescribes these drugs. They are

often prescribed for Type II diabetics, many of whom already have

elevated ineffective insulin. These drugs are notorious for causing

hypoglycaemia as a side effect.

The thiazolidinediones are famous for causing liver cancer. One of

them, Rezulin, was approved in the USA through devious political

infighting, but failed to get approval in the UK because it was known

to cause liver cancer. The doctor who had responsibility to approve

it at the FDA refused to do so. It was only after he was replaced by

a more compliant official that Rezulin gained approval by the FDA. It

went on to kill well over 100 diabetes patients and cripple many

others before the fight to get it off the market was finally won.

Rezulin was designed to stimulate the uptake of glucose from the

bloodstream by the peripheral cells and to inhibit the normal

secretion of glucose by the liver. The politics of why this drug ever

came onto market, and then remained in the market for such an

unexplainable length of time with regulatory agency approval, is not

clear.20 As of April 2000, lawsuits commenced to clarify this

situation.21

? Insulin

Today, insulin is prescribed for both the Type I and Type II

diabetics. Injectable insulin substitutes for the insulin that the

body no longer produces. Of course, this treatment, while necessary

for preserving the life of the Type I diabetic, is highly

questionable when applied to the Type II diabetic.

It is important to note that neither insulin nor any of these oral

hypoglycaemic agents exerts any curative action whatsoever on any

type of diabetes. None of these medical strategies is designed to

normalise the cellular uptake of glucose by the cells that need it to

power their activity.

The prognosis with this orthodox treatment is increasing disability

and early death from heart or kidney failure or the failure of some

other vital organ.

Alternative Medical Treatment

The third step to a cure for this disease is to become informed and

to apply an alternative methodology that is soundly based upon good

science.

Effective alternative treatment that directly leads to a cure is

available today for some Type I and for many Type II diabetics. About

5% of the diabetic population suffers from Type I diabetes; about 95%

has Type II diabetes.22 Gestational diabetes is simply ordinary

diabetes contracted by a woman who is pregnant.

For the Type I diabetic, an alternative methodology for the treatment

of Type I diabetes is now available. It was developed in modern

hospitals in Madras, India, and subjected to rigorous double-blind

studies to prove its efficacy.23 It operates to restore normal

pancreatic beta cell function so that the pancreas can again produce

insulin as it should. This approach apparently was capable of curing

Type I diabetes in over 60% of the patients on whom it was tested.

The major complication lies in whether the antigens that originally

led to the autoimmune destruction of these beta cells have

disappeared from or remain in the body. If they remain, a cure is

less likely; if they have disappeared, the cure is more likely. For

reasons already discussed, this methodology is not likely to appear

in the United States any time soon, and certainly not in the American

orthodox medical community.

The goal of any effective alternative program is to repair and

restore the body's own blood-sugar control mechanism. It is the

malfunctioning of this mechanism that, over time, directly causes all

of the many debilitating symptoms that make orthodox treatment so

financially rewarding for the diabetes industry. For Type II

diabetes, the steps in the program are:24

? Repair the faulty blood sugar control system. This is done simply

by substituting clean, healthy, beneficial fats and oils in the diet

for the pristine-looking but toxic trans-isomer mix found in

attractive plastic containers on supermarket shelves. Consume only

flax oil, fish oil and occasionally cod liver oil until blood sugar

starts to stabilise. Then add back healthy oils such as butter,

coconut oil, olive oil and clean animal fat. Read labels; refuse to

consume cheap junk oils when they appear in processed food or on

restaurant menus. Diabetics are chronically short of minerals; they

need to add a good-quality, broad-spectrum mineral supplement to the

diet.

? Control blood sugar manually during the recovery cycle. Under

medical supervision, gradually discontinue all oral hypoglycaemic

agents along with any additional drugs given to counteract their side

effects. Develop natural blood-sugar control by the use of glycaemic

tables, by consuming frequent small meals (including fibre-rich

foods), by regular post-prandial exercise, and by the complete

avoidance of all sugars along with the judicious use of only non-

toxic sweeteners.25 Avoid alcohol until blood sugar stabilises in the

normal range. Keep score by using a pinprick-type glucose meter. Keep

track of everything you do with a medical diary.

? Restore a proper balance of healthy fats and oils when the blood

sugar controller again works. Permanently remove from the diet all

cheap, toxic, junk fats and oils as well as the processed and

restaurant foods that contain them. When the blood sugar controller

again starts to work correctly, gradually introduce additional

healthy foods to the diet. Test the effect of these added foods by

monitoring blood sugar levels with the pinprick-type blood sugar

monitor. Be sure to include the results of these tests in your diary

also.

? Continue the program until normal insulin values are also restored

after blood sugar levels begin to stabilise in the normal region.

Once blood sugar levels fall into the normal range, the pancreas will

gradually stop overproducing insulin. This process will typically

take a little longer and can be tested by having your physician send

a sample of your blood to a lab for a serum insulin determination. A

good idea is to wait a couple of months after blood sugar control is

restored and then have your physician check your insulin level. It's

nice to have blood sugar in the normal range; it's even nicer to have

this accomplished without excess insulin in the bloodstream.

? Separately repair the collateral damage done by the disease.

Vascular problems caused by a chronically elevated glucose level will

normally reverse themselves without conscious effort. The effects of

retinopathy and of peripheral neuropathy, for example, will usually

self repair. However, when the fine capillaries in the basement

membranes of the kidneys begin to leak due to chronic high blood

glucose, the kidneys compensate by laying down scar tissue to prevent

the leakage. This scar tissue remains even after the diabetes is

cured, and is the reason why the kidney damage is not believed to

self repair.

A word of warningS When retinopathy develops, there may be a

temptation to have the damage repaired by laser surgery. This laser

technique stops the retinal bleeding by creating scar tissue where

the leaks have developed. This scar tissue will prevent normal

healing of the fine capillaries in the eye when the diabetes is

reversed. By reversing the diabetes instead of opting for laser

surgery, there is an excellent chance that the eye will heal

completely. However, if laser surgery is done, this healing will

always be complicated by the scar tissue left by the laser.

The arterial and vascular damage done by years of elevated sugar and

insulin and by the proliferation of systemic candida will slowly

reverse due to improved diet. However, it takes many years to clean

out the arteries by this form of oral chelation. Arterial damage can

be reversed much more quickly by using intravenous chelation

therapy.26 What would normally take many years through diet alone can

often be done in six months with intravenous therapy. This is reputed

to be effective over 80% of the time. For obvious reasons, don't

expect your doctor to approve of this, particularly if he's a heart

specialist.

Recovery Time

The prognosis is usually swift recovery from the disease and

restoration of normal health and energy levels in a few months to a

year or more. The length of time that it takes to effect a cure

depends upon how long the disease was allowed to develop.

For those who work quickly to reverse the disease after early

discovery, the time is usually a few months or less. For those who

have had the disease for many years, this recovery time may lengthen

to a year or more. Thus, there is good reason to get busy reversing

this disease as soon as it becomes clearly identified.

By the time you get to this point in this article, and if we've done

a good job of explaining our diabetes epidemic, you should know what

causes it, what orthodox medical treatment is all about, and why

diabetes has become a national and international disgrace.

Of even greater importance, you have become acquainted with a self-

help program that has demonstrated great potential to actually cure

this disease. & #8734;

About the Author:

is a reluctant medical investigator, having been forced

into curing his own diabetes because it was obvious that his doctor

would not or could not cure it.

He has published the results of his successful diabetes investigation

in his self-help manual, Insulin: Our Silent Killer, written for the

layperson but also widely valued by the medical practitioner. This

manual details the steps required to reverse Type II diabetes and

references the work being done with Type I diabetes. The book may be

purchased from the author at PO Box 7685, Loveland, Colorado 80537,

USA (North American residents send $US25.00; overseas residents

should contact the author for payment and shipping instructions).

has also posted a great deal of useful information about

diabetes on his website, http://www.Healingmatters. com. He can be

contacted by telephone at +1 (970) 669 9176 and by email at

valley@h...

Endnotes:

1. National Center for Health Statistics, " Fast Stats " ,

Deaths/Mortality Preliminary 2001 data

2. Dr Herbert Ley, in response to a question from Senator Long

about the FDA during US Senate hearings in 1965

3. Eisenberg, M., MD, " Credentialing complementary and

alternative medical providers " , ls of Internal Medicine 137

(12):968 (December 17, 2002)

4. American Diabetes Association and the American Dietetic

Association, The Official Pocket Guide to Diabetic Exchanges, McGraw-

Hill/Contemporary Distributed Products, newly updated March 1, 1998

5. American Heart Association, " How Do I Follow a Healthy Diet? " ,

American Heart Association

National Center (7272 Greenville Avenue, Dallas, Texas 75231-4596,

USA), http://www.americanheart.org

6. Brown., J.A.C., Pears Medical Encyclopedia Illustrated, 1971, p.

250

7. Joslyn, E.P., Dublin, L.I., Marks, H.H., " Studies on Diabetes

Mellitus " , American Journal of Medical Sciences 186:753-773 (1933)

8. " Diabetes Mellitus " , Encyclopedia Americana, Library Edition, vol.

9, 1966, pp. 54-56

9. American Heart Association, " Stroke (Brain Attack) " , August 28,

1998, http://www.amhrt.org/ScientificHStats98/05stroke.html;

American Heart Association, " Cardiovascular Disease Statistics " ,

August 28, 1998,

http://www.amhrt.org/Heart_and_Stroke_A_Z_Guide/cvds.html;

" Statistics related to overweight and obesity " ,

http://niddk.nih.gov/health/nutrit/pubs/statobes.htm ;

http://www.winltdusa.com/about/infocenter/healthnews/articles/obesest

a

ts.htm

10. " Diabetes Mellitus " , Encyclopedia Americana, ibid., pp. 54-55

11. The Veterans Administration Coronary Artery Bypass Co-operative

Study Group, " Eleven-year survival in the Veterans Administration

randomized trial of coronary bypass surgery for stable angina " , New

Eng. J. Med. 311:1333-1339 (1984); Coronary Artery Surgery Study

(CASS), " A randomized trial of coronary artery bypass surgery:

quality of life in patients randomly assigned to treatment groups " ,

Circulation 68(5):951-960 (1983)

12. Trager, J., The Food Chronology, Henry Holt & Company, New York,

1995 (items listed by date)

13. " Margarine " , Encyclopedia Americana, Library Edition, vol. 9,

1966, pp. 279-280

14. Fallon, S., Connolly, P., Enig, M.C., Nourishing Traditions,

Promotion Publishing, 1995;

Enig, M.C., " Coconut: In Support of Good Health in the 21st Century " ,

http://www.livecoconutoil.com/maryenig.htm

15. Houssay, Bernardo, A., MD, et al., Human Physiology, McGraw-Hill

Book Company, 1955, pp. 400-421

16. Gustavson, J., et al., " Insulin-stimulated glucose uptake

involves the transition of glucose transporters to a caveolae-rich

fraction within the plasma cell membrane: implications for type II

diabetes " , Mol. Med. 2(3):367-372 (May 1996)

17. Ganong, F., MD, Review of Medical Physiology, 19th

edition, 1999, p. 9, pp. 26-33

18. Pan, D.A. et al., " Skeletal muscle membrane lipid composition is

related to adiposity and insulin action " , J. Clin. Invest. 96

(6):2802-

2808 (December 1995)

19. Physicians' Desk Reference, 53rd edition, 1999

20. , , Insulin: Our Silent Killer, ,

Loveland, Colorado, revised 2nd

edition, July 2000, p. 20

21. Law Offices of H. & Associates (telephone 1 800

535 5727, toll free in North America)

22. American Heart Association, " Diabetes Mellitus Statistics " ,

http://www.amhrt.org

23. Shanmugasundaram, E.R.B. et al. (Dr Ambedkar Institute of

Diabetes, Kilpauk Medical College Hospital, Madras, India), " Possible

regeneration of the Islets of Langerhans in Streptozotocin-diabetic

rats given Gymnema sylvestre leaf extract " , J. Ethnopharmacology

30:265-279 (1990);

Shanmugasundaram, E.R.B. et al., " Use of Gemnema sylvestre leaf

extract in the control of blood glucose in insulin-dependent diabetes

mellitus " , J. Ethnopharmacology 30:281-294 (1990)

24. , ibid., pp. 97-123

25. Many popular artificial sweeteners on sale in the supermarket are

extremely poisonous and dangerous to the diabetic; indeed, many of

them are worse than the sugar the diabetic is trying to avoid; see,

for example, , ibid., pp. 53-58.

26. , Morton, MD, and Shah, Hitendra, MD, Chelation Therapy,

Keats Publishing, Inc., New Canaan, Connecticut, 1997, ISBN 0-87983-

730-6

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