Alpha

[Guest guest]

I just had a one-day workshop with Jay Gunkelman, and I learned a lot!

Several points on alpha:

A. If alpha is being produced in the temporal lobes, it will show up in the ear lobe/mastoid.

A way to tease this out is: 1.Don't use linked ears (If you use linked ears, the alpha won't show, or will bleed to the other reference too and show up all over the head) 2. Use the contra-lateral ear for

reference if you want to measure whether a T site is producing lots of alpha.

B. Alpha should be coming from the source in the parietal lobe, the precuneus, or the occipital lobe, the cuneus.

BUT...alpha can be generated in the frontal cingulate gyrus...and this alpha is associated with anxiety.

It's hard to tease out the source of the alpha without a QEEG and the ability to re-montage and use LORETA...but it seems to me (a non-Q'er) that we can still be aware of these sources.

For the cingulate generated alpha, a supplement of GABA is recommended, plus inhibiting alpha at about Fz.

BTW, GABA is an inhibitory neurotransmitter, but is also capable of inhibiting the inhibitions...thus beer for instance, which is a great source of GABA!

C. (But wait! theres more!)

Remember that white matter damage from a TBI can show as Delta because delta is generated when the cortex is not getting input. If the axons and their white matter are damaged, the signals from the senses and the rest of the cortex don't get through, so the damaged area just keeps playing delta.

With grey matter damage, (I think of this as bruised neurons), we usually look for lower amplitude....but get this:

After one year, the area has healed, and starts producing alpha......think of those alpha towers we see on the histogram!...the alpha gets louder and louder...until that part of the brain is stimulated again (with NFB for instance) and gets back on line.

Jay specifically suggested training GAMMA UP at the high amplitude alpha areas. As the Gamma goes up the alpha should go down.

Gamma: Gamma is a product when areas of the brain link for a thought. The Gamma is seen in very short bursts. Areas are linked, and unlinked quickly. Think of the linkage as creating a temporary 'bell" and the gamma as the 'ringing' of that bell. If you see consistent gamma, think Parkinson's or dementia.

Rewarding Gamma doesn't have the same risks as rewarding hi-beta. ( I haven't done it yet, but I sure wondered about this!)

Hope this is helpful!

St. Clair

[Guest guest]

Rosemary,

I don't know. I meant to ask...especially when the alpha attenuates when EO.

Re: Alpha

,Thanks for the information. Are you referring to Eyes Closed, Eyes Open,or when alpha is high in both conditions?Rosemary>> I just had a one-day workshop with Jay Gunkelman, and I learned a lot!> > Several points on alpha:> A. If alpha is being produced in the temporal lobes, it will show up in the ear lobe/mastoid.> A way to tease this out is: 1.Don't use linked ears (If you use linked ears, the alpha won't show, or will bleed to the other reference too and show up all over the head) 2. Use the contra-lateral ear for> reference if you want to measure whether a T site is producing lots of alpha.> > B. Alpha should be coming from the source in the parietal lobe, the precuneus, or the occipital lobe, the cuneus. > BUT...alpha can be generated in the frontal cingulate gyrus...and this alpha is associated with anxiety.> It's hard to tease out the source of the alpha without a QEEG and the ability to re-montage and use LORETA...but it seems to me (a non-Q'er) that we can still be aware of these sources.> For the cingulate generated alpha, a supplement of GABA is recommended, plus inhibiting alpha at about Fz.> BTW, GABA is an inhibitory neurotransmitter, but is also capable of inhibiting the inhibitions...thus beer for instance, which is a great source of GABA!> > C. (But wait! theres more!)> Remember that white matter damage from a TBI can show as Delta because delta is generated when the cortex is not getting input. If the axons and their white matter are damaged, the signals from the senses and the rest of the cortex don't get through, so the damaged area just keeps playing delta.> With grey matter damage, (I think of this as bruised neurons), we usually look for lower amplitude....but get this:> After one year, the area has healed, and starts producing alpha......think of those alpha towers we see on the histogram!...the alpha gets louder and louder...until that part of the brain is stimulated again (with NFB for instance) and gets back on line.> Jay specifically suggested training GAMMA UP at the high amplitude alpha areas. As the Gamma goes up the alpha should go down.> > Gamma: Gamma is a product when areas of the brain link for a thought. The Gamma is seen in very short bursts. Areas are linked, and unlinked quickly. Think of the linkage as creating a temporary 'bell" and the gamma as the 'ringing' of that bell. If you see consistent gamma, think Parkinson's or dementia.> Rewarding Gamma doesn't have the same risks as rewarding hi-beta. ( I haven't done it yet, but I sure wondered about this!)> > Hope this is helpful!> St. Clair>

[Guest guest]

Rosemary,

I don't know. I meant to ask...especially when the alpha attenuates when EO.

Re: Alpha

,Thanks for the information. Are you referring to Eyes Closed, Eyes Open,or when alpha is high in both conditions?Rosemary>> I just had a one-day workshop with Jay Gunkelman, and I learned a lot!> > Several points on alpha:> A. If alpha is being produced in the temporal lobes, it will show up in the ear lobe/mastoid.> A way to tease this out is: 1.Don't use linked ears (If you use linked ears, the alpha won't show, or will bleed to the other reference too and show up all over the head) 2. Use the contra-lateral ear for> reference if you want to measure whether a T site is producing lots of alpha.> > B. Alpha should be coming from the source in the parietal lobe, the precuneus, or the occipital lobe, the cuneus. > BUT...alpha can be generated in the frontal cingulate gyrus...and this alpha is associated with anxiety.> It's hard to tease out the source of the alpha without a QEEG and the ability to re-montage and use LORETA...but it seems to me (a non-Q'er) that we can still be aware of these sources.> For the cingulate generated alpha, a supplement of GABA is recommended, plus inhibiting alpha at about Fz.> BTW, GABA is an inhibitory neurotransmitter, but is also capable of inhibiting the inhibitions...thus beer for instance, which is a great source of GABA!> > C. (But wait! theres more!)> Remember that white matter damage from a TBI can show as Delta because delta is generated when the cortex is not getting input. If the axons and their white matter are damaged, the signals from the senses and the rest of the cortex don't get through, so the damaged area just keeps playing delta.> With grey matter damage, (I think of this as bruised neurons), we usually look for lower amplitude....but get this:> After one year, the area has healed, and starts producing alpha......think of those alpha towers we see on the histogram!...the alpha gets louder and louder...until that part of the brain is stimulated again (with NFB for instance) and gets back on line.> Jay specifically suggested training GAMMA UP at the high amplitude alpha areas. As the Gamma goes up the alpha should go down.> > Gamma: Gamma is a product when areas of the brain link for a thought. The Gamma is seen in very short bursts. Areas are linked, and unlinked quickly. Think of the linkage as creating a temporary 'bell" and the gamma as the 'ringing' of that bell. If you see consistent gamma, think Parkinson's or dementia.> Rewarding Gamma doesn't have the same risks as rewarding hi-beta. ( I haven't done it yet, but I sure wondered about this!)> > Hope this is helpful!> St. Clair>

[Guest guest]

Was that the workshop in Clare, Michigan?

Francois

De :

braintrainer [mailto:braintrainer ] De la part de St. Clair

Envoyé : 5 octobre 2008 13:09

À :

braintrainer

Objet : Alpha

I just had a one-day workshop with Jay Gunkelman, and I

learned a lot!

Several points on alpha:

A. If alpha is being produced in the temporal lobes,

it will show up in the ear lobe/mastoid.

A way to tease this out is: 1.Don't

use linked ears (If you use linked ears, the alpha won't show, or will

bleed to the other reference too and show up all over the head) 2. Use the

contra-lateral ear for

reference if you want to measure whether a T site is

producing lots of alpha.

B. Alpha should be coming from the source in the

parietal lobe, the precuneus, or the occipital lobe, the cuneus.

BUT...alpha can be generated in the

frontal cingulate gyrus...and this alpha is associated with anxiety.

It's hard to tease out the source of the

alpha without a QEEG and the ability to re-montage and use LORETA...but it

seems to me (a non-Q'er) that we can still be aware of these sources.

For the cingulate generated alpha, a

supplement of GABA is recommended, plus inhibiting alpha at about Fz.

BTW, GABA is an inhibitory

neurotransmitter, but is also capable of inhibiting the inhibitions...thus

beer for instance, which is a great source of GABA!

C. (But wait! theres more!)

Remember that white matter damage from a

TBI can show as Delta because delta is generated when the cortex is not getting

input. If the axons and their white matter are damaged, the signals from

the senses and the rest of the cortex don't get through, so the damaged area

just keeps playing delta.

With grey matter damage, (I think

of this as bruised neurons), we usually look for lower amplitude....but

get this:

After one year, the area has healed, and

starts producing alpha......think of those alpha towers we see on the

histogram!...the alpha gets louder and louder...until that part of the

brain is stimulated again (with NFB for instance) and gets back on line.

Jay specifically suggested training GAMMA

UP at the high amplitude alpha areas. As the Gamma goes up the alpha

should go down.

Gamma: Gamma is a product when areas of the

brain link for a thought. The Gamma is seen in very short bursts.

Areas are linked, and unlinked quickly. Think of the linkage as

creating a temporary 'bell " and the gamma as the 'ringing' of that

bell. If you see consistent gamma, think Parkinson's or dementia.

Rewarding Gamma doesn't have the same risks as rewarding

hi-beta. ( I haven't done it yet, but I sure wondered about this!)

Hope this is helpful!

St. Clair