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Mycoplasma: Its hidden role in rheumatoid arthritis and other clinical syndromes

-

Quantum Medicine Update by Jr. Yanick, Townsend Letter for Doctors and

Patients

Mycoplasma may serve as a cofactor that along with biological and/or chemical

stressors can induce cytokines and other immune abnormalities found in

rheumatoid

arthritis (RA). The overuse of antibiotics has caused widespread bacterial

resistance

to antibiotics and has contributed to the development of new strains of disease

that

are not treatable with current antibiotics. These stealth infections pose a

continuing

threat to human health and may be implicated as cofactors in a number of

clinical

syndromes.

Research has implicated certain mycoplasma species as a causative factor in

chronic fatigue syndrome (CFS), RA and gulf war syndrome (GWS) (1-4) In one

study, forty-nine of sixty RA subjects had evidence of mycoplasma infections

that

were documented by multiplex polymerase chain reaction (PCR). Unlike other lab

tests which fail to detect mycoplasma, multiplex PCR assays allow for unmatched

clinical accuracy in the identification and differentiation of mycoplasma

species.

Because RA, CFS, and GWS are characterized by overlapping symptoms,

researchers are gathering more and more evidence that points to stealth

infections

as the missing link to effective treatment in these disorders.

Rather than treating RA and other forms of arthritis with anti-inflammatory

drugs that

are poorly researched and not safe for long-term use, more clinical efforts need

to be

made in identifying stealth infections using multiplex PCR and quantum meridian

stress measurement (QMSM) techniques. Since mycoplasma infections are found

at significantly higher rates in RA, effective treatment should be aimed at

immunomodulation therapies that help the body uncover and destroy stealth

infections.

Members of the genus mycoplasma lack cell walls, and like viruses, are capable

of

self-replication. Microplasma fermentans colonizes human mucosal tissues and

under certain conditions can invade the host cells or fuse with CD4 cells,

inducing

the production of proinflammatory cytokines such as IL-6 and tumor necrosis

factor

alpha. (6-7) Indeed, the occurrence of mycoplasma and ureaplasma species in

joint

tissues of RA and other forms of arthritis provides convincing evidence that

mycoplasma is a significant cofactor, among other stressors, that precipitates

the

symptomatology of RA. (8-12) Yet, just as it took decades for medicine to

realize

that helicobacter pylori caused peptic ulcers, there continues to be a lag time

in the

acceptable treatment of mycoplasma and stealth infections in clinical cases of

arthritis. (13)

If infection is a causative agent in arthritis, what constitutes effective

treatment? In

two independent, randomized trials, RA patients were treated with lOOmg of oral

doxycycine or a placebo twice daily for 26 weeks. The results demonstrated a

greater than 75% improvement in arthritic symptoms in patients taking the drug

versus a placebo. (14,15)

While doxycycline or minocycline may be life-saving in advanced cases of

mycoplasma, solving the problem of infection requires more than the use of

antibiotics. Furthermore, many microbes now defy antibiotics that once killed

them.

And, the use of antibiotics causes resistant microbes, viruses, or fungal

infections

to replicate, grow and multiply. In many cases, the overuse of antibiotics has

resulted in the spread of hepatitis, cytomegalovirus, herpes viruses, parasites,

and

many other dreadful microbes.

The lifestyles of patients with mycoplasma infection lay the foundation for

opportunistic infections to flourish and conquer the body. Irradiated foods,

genetically-modified foods, radiation, and chemical stressors trigger clusters

of

mutations that interfere with DNA stability and reduce the function and

regeneratory

capacity of lymphocytes. QMSA techniques provide a window into the body to

quickly assess each individual's unique stress patterns between toxins,

parasites,

parasitic toxins, bacteria, mycoses, viruses and metabolism. Therefore, when

stress

is reduced, one may expect the immune system to function with greater

efficiency.

In RA patients, it is common for impregnated toxins and mutated proteins to

congest

and damage the body's lymph nodes causing a functional impairment in the immune

system. This deposition of toxins extends itself into the joints and allows

opportunistic infections to grow and flourish while continually damaging the

immune

system and extracellular matrix interface to the energetic system of healing. As

such, cold photon scalar therapies, advanced detoxification protocols and

immunomodulation therapies are necessary to reduce toxin and microbial stressors

and to help the body regain a greater range of immunological functions.

Reestablishing the Superhighway in Our Genes

Genes send light/electrical signals to one another through a DNA information

" superhighway. " Biochemists are amazed at the distance over which these signals

can travel. (16,17) Toxin deposits seem to limit the distance these signals can

travel

and/or stops these signals from reaching their destination, thereby causing a

deficit

in biocommunication. Many physicists feel that these toxins are " light

scramblers " in

that they cause chaos in the DNA's signaling system which includes the

extracellular matrix and its interface with the human energy system.

In RA patients, enhancing the immune system's ability to patrol, enforce, and

attack

invading microbes with greater operational complexity requires understanding the

sub-molecular dynamics of DNA in the orchestration of the body's defense and

repair routines. In other words, while we understand the negative repercussions

of

oxidative stress at the molecular level, it's equally important to understand

how it

reduces the repair capacity of DNA at the quantum level. Therefore,

re-establishing

quantum coherence -- a state of immune competence with adequate DNA repair

enzymes -- may provide the best treatment in infectious disease.

The best way to understand quantum coherence and DNA is to look at how it works

in the salamander and pondworm. When a salamander loses a leg torn off by a

predator, it simply regrows a new one. When a pondworm is chopped in half, each

half regrows into a whole, complete worm. How does the worm's body know where to

reassemble new organs when half of them are missing? These miraculous powers of

healing are inherent in DNA.

For humans, these genetic cascades of DNA-generated biophoton energies are the

most powerful when we are embryos. Yet, as we grow older and enter the world our

immune systems immediately engage in warfare against pathogens, chemicals, and

other stressors. For some reason, over engaging our immune systems, has the

effect of repressing healing networks within our DNA. Plausible evidence exists

to

support the idea that defining and eliminating these stressors may be the first

step

in uncovering the missing links to feats of regeneration found in species like

the

salamander or pondworm.

These DNA-guided signals flow throughout the body with awe-inspiring speed.

Stately simply, the magnitude of DNA's coherence, determines the course of our

health. For example, when there is quantum coherence, the magnitude of inner

healing is so gigantic that it renders even the best of minds helpless for

exploring

the endless boundaries of its power. Can we " click on " the genetic switches that

power up our capacity to heal and regenerate? Unfortunately, more research is

needed to answer this question. The problem is that most current scientific

study of

the human body delves deeply into the chemistry and molecular nature of cells

with

very little emphasis on how healing functions can be nourished, how they can be

expanded, or how its faculties can be used more efficiently.

During the past 80 years of our existence these healing powers have been

shrouded

behind ignorance and myths, or principles held sacred by modern medicine or

assumed trivial by the medical branches of science. Yet a wide spectrum of non-

pharmaceutical/medical scientific research coupled with ancient wisdom,

documents

profound insights into our nature and ability to evoke self-healing and

regeneration.

From time to time modern medicine and science express opinions about the nature

of healing. None of these theories account for the true essence of healing.

Behind

the philosophical pronouncements on the essence of healing lies a chaotic jumble

of

fragmented and unexplored half-guesses about how the greatest healing power

within our bodies works or doesn't work. And, when medical experts observe

healing

from natural methods, they typically dismiss it as the " placebo effect. " Yet

when

this healing is observed on the very same patients who failed to respond to

their

best efforts, one has to question: where was the placebo effect in all their

patient-

doctor interactions? Obviously, when a patient who has consulted unsuccessfully

with six or more doctors for the same condition over a period of ten years,

suddenly

experiences healing after visiting an alternative practitioner, there's more

than a

placebo effect involved.

Buffeted by the whims of political and industrial economics, these researchers

hesitate to probe the natural healing dimension of the body. This has been an

unfortunate barrier to understanding how to nurture, strengthen, and fulfill the

body's

optimal potential for healing.

The complete eradication of mycoplasmas from the body requires an intact,

functional immune system working in concert with a DNA repair system and the

human energy system. In these cases, it is of utmost importance to support Phase

1,11 and III liver detoxification systems and the kidneys using immunomodulation

techniques to augment the body's army of immune cells that identify, tag,

poison,

blast, and consume microbes that invade the body's biological turf. Indeed, the

augmentation of lymphocytes with herbal medicine is well known. (18)

Clinical Research with Mycoplasma Infections

Our preliminary observations with mycoplasma reveals that it usually underlies

sensory nerve root inflammations and allied conditions caused by herpes-type

viruses. It appears to be the deepest infection in the nervous system in

chronically

ill patients and the underlying cause or stressor in recurrent herpes-type viral

infections. Yet, despite its wide prevalence, clinicians have ignored a possible

role

for mycoplasma in a wide spectrum of clinical syndromes. It seems that the

thread

that ties mycoplasma to RA and other degenerative diseases is stressed body

defenses caused by a reduction in DNA-guided repair mechanisms and

immunological defenses. Moreover, hidden parasite infections and parasitic

toxins

may be able to switch off the host's immune system, allowing mycoplasma to

spread rapidly throughout the body.

It appears that mycoplasma, like the herpes virus, harbors itself in the

tube-like

fibers of the nervous system which serves as a hidden tunnel for this infection

to

spread throughout the body without being detected by immune surveillance. With

QMSM methods of electrodermal biofeedback, a practitioner can quickly identify

and

observe how these microbes stress the body and where they are predominantly

located. Moreover, the sequence or layering of these immune-stressing microbes

can be observed with a reasonable degree of accuracy (depending on the test

system and practitioner) and confirmed by lab tests. In an overwhelming majority

of

clinical cases, we can trace these stressors back to old, unresolved dental foci

such

as septic root canals or ostitis in old tooth extraction sites.

Our clinical efforts against mycoplasma have included the use of a blend of

hyssop

(whole herb concentrate; not extract) containing the full spectrum of naturally-

occurring caravacrol, thymol, and other phytochemicals in synergistic

combinations

with other herbs and nutrients. The antimicrobial, anti-viral and nervine

properties

and cleansing effect of hyssop are extraordinary when combined with appropriate

cofactors and transporters. (19-21) Most importantly, the oral use of the

essential

oils of oregano, rosemary, clove, DL limonene, and fermented adaptogenic herbs

and citrus seed extracts may provide powerful phytomedicines against stealth

infections, like mycoplasma. However, since mycoplasma commonly underlies viral

infections, immunomodulation strategies against viral pathogens or hidden

parasites

may need to be employed for best results. These immunomodulation therapies are

designed to capitalize on the immune system's vastly superior qualities to fight

viral

infections and account for the web-like int eraction and interpenetration of the

immune system with the human energy system. In previous columns, I have

discussed the components of such an immune enhancement program in detail. (22-

23)

Reestablishing the extraordinary complexity of the immune system at the quantum

level of biophoton light communication is of primary importance in helping to

alleviate the suffering of RA patients. While the anecdotal and scientific

evidence

discussed in this article are astute and insightful, detailed,

carefully-controlled

studies that uncover the clinical manifestations of mycoplasma and its

interaction

with parasites and other emerging microbes are warranted. (24) Since these

studies

reveal that mycoplasma is a cofactor, it should be assessed and treated before

anti-

inflammatory and immunosuppressive drugs are employed. Clearly, eliminating the

pain and inflammation of arthritis requires a better understanding of how

multiple

biological and chemical stressors interact with mycoplasma and with the immune

system to induce inflammation. The major therapeutic advances will be with

phytonutrients and mycelial mushrooms that augment immune functions in a

multidirectional manner while chemical stressors that indu ce DNA instability

are

properly detoxified from the body.

Finally, The Brucellosis Triangle documents how biological warfare research

between 1942 to the present time has resulted in more deadly and infectious

forms

of mycoplasma. (25-27) Because of its affinity with the nervous system,

detecting

and properly treating mycoplasma will require clinical approaches that target

and

cleanse the nervous system. Toxicity of the nervous system, especially with

heavy

metals, serves as an impetus for mycoplasma to withdraw energy from the neurons

and paralyze the immune system. Since ordinary blood and tissue tests do not

reveal the presence of mycoplasma infection, physicians should employ multiplex

PCR to detect this infectious agent and make some attempt to categorize the

strata

of microbial stressors that interfere with immunological functions in

chronically ill

patients. (29)

References

(1.) Vojdani A et al Detection of Mycoplasma genus and Mycoplasma fermentans by

PCR in patients with chronic fatigue syndrome. FEMS Immunology & Medical

Microbiology 1998 22355-65.

(2.) Schaeverbeke T et al Systematic detection of mycoplasma by culture and PCR

in 209 synovial fluid samples. British J Rheumat 1997; 36:310-14.

(3.) Nicholson GL at al Diagnosis and treatment of Mycoplasmal infections in

Persian Gulf war Illness-CHIDS Patients. Int; J Occupational Medicine,

Immunology, and Toxicology 1996;5(1):69-78.

(4.) Vojdani A Franco AR Multiplex PCR for the detection of Mycoplasma

fermentans, M. hominis, and M. penetrans in Patients with Chronic Fatigue

Syndrome, Fibromyalgia, Rheumatoid Arthritis, and Gulf Syndrome. Microbiology

199; 187-99.

(5.) Gerard HC et al Frequency of apolipoprotien E allele types in patients with

Chlamydia-associated arthritis and other arthritides, 1998; Microbial

Pathogenesis

26: 35-43.

(6.) Dimitrov DS et al Mycoplasma fermentans cells are able to fuse with T-

lymphocytes. Clin Inf Dis 1993: 17(1):305-08.

(7.) Mulhradt PF et al Mycoplasma fermentans leads to in vitro IL-1, IL-6, tumor

necrosis factor and prostaglandin product and is pyrogenic in rabbits. Inf Immun

1991 59:3969-74.

(8.) MH Recovery of mycoplasma from rheumatoid synovial fluid. In Third

Pfizer Symposium on Rheumotic Diseases, 1966: Edinburgh University Press, 161-

89.

(9.) Washburn, LR et al Chronic arthritis of rabbits induced by mycoplasma.

Clinical,

microbiologic, and histological features. Arthritis Rheum 1960 23:825-36.

(10.) Burdge DR et al Septic arthritis due to dual infections with Mycoplasma

hominis and Ureaplasma urealyticym. J of Rheu 1988 15: 366-71.

(11.) Posnett DN et al Interaction of Mycoplasma arthritidis superantigen with

human

T-cells. Clin Inf Dis 1993 17:170-75.

(12.) Behar SM et al Mechanisms of autoimmune disease induction: the role of the

immune response to microbial pathogens. Arth & Rheu 1995 38:458-76.

(13.) Ewald, PW Plaque Time 2000; The Free Press, NY.

(14.) - D Mycoplasma in rheumatoid arthritis and other human

arthritides. J Clin Path 1996: 49:762-82.

(15.) Cohen MA et al In vitro susceptibilities of Mycoplasma pneumoniae, bominis

and ureaplasma urealyticum to clinafloxacin, PD 131628, ciprofloxacin and

comparator drugs. J Antimicrob Chemother 1997: 40:308-9.

(16.) Barton J et al Chem & Biolol 1999, 2:85.

(17.) Coghlan A Electric DNA. New Scientist 1999: 2: 19.

(18.) Greunwald J et al PDR for Herbal Medicines, 1998, Medical Economics

Company, New Jersey.

(19.) Yanick P. Meridian/Organ Nutraceutic Resonant Complexes: New Hope for

Chronically-Sick Individuals. 2000, May Townsend Letter for-Doctors & Patients;

136-39.

(20.) Yanick, P. Boosting Nutrient Uptake in Chronic Illness, Dec 2000, Townsend

Letter for Doctors & Patients.

(21.) Yanick, P. Food Supplement Benefits and Risks in Carcinogenesis: Part I,

Oct

2001. Townsend Letter for Doctors & Patients.

(22.) Yanick, P Immune System Protection against Bioterrorism. Dec 2001.

Townsend Letter for Doctors & Patients.

(23.) Yanick, P Novel Antiviral Strategies against Biotorrorist Attacks.

Feb-March

2002. Townsend Letter for Doctors & Patients.

(24.) Ewald W Guarding against the most dangerous emerging pathegens: Insights

from evolutionary biology. 1996. Emerging Infectious Diseases 2:245-57.

(25.) DW & WLC The Brucellasis Triangle 1998: The Chelmsford

Publishers, Canada.

(26.) Pathogenic Mycoplasma. US Patent No. 5, 242,820 issued September 7, 1993.

(27.) Howell at al Acute Brucellosis among laboratory workers. 1948 New England

Journal of Medicine 236:741.

(28.) Colmonero et al Complications associated with brucellosis melitensis

infection:

A study of 530 cases, 1996; Medicine 75:4.

(29.) Vojdani, A. Immunosciences Lab Manual, 2000.

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