Guest guest Posted October 24, 2010 Report Share Posted October 24, 2010 I am needing a little help on a case. 70 yo male dxd with corticobasal syndrome (movement disorder 8 yrs ago). He was a coal miner and farmer all his life therefore he has many occupational exposures and head injuries of his past. 35pack yr smoker and quit 5 yrs ago. He owned a coal mine and sold it 3 yrs ago. Onset of dz was noticed during a stressful event when he was being sued by a former worker. Sxs exacerbated when his wife passed of breast CA in May 2009. He was also dxd with DM 5yrs ago and hypercholesterolemia. He is still able to swallow but with increases of Vitamin E (more than 400IU) begins having issues with bruising. He gets outside daily with his medical team on an ATV riding slowly throughout his farm. He loves being outside and our goal is to assist with QOL. He is well cared for but speech is down to two words. All 5 caregivers were present for his home visits with me. He responds extremely well to cranial sacral and massage where his speed of gait and agility are well increased that lasts for approximately 4-5 days. His sleep is great, loves to eat, and a true delight to be around. He is on a bunch of meds which we are addressing appropriately over time (zantac- now weaned off, simvastatin- weaning off currently, namenda, carbidopa/levadopa, synthroid, trilipix- will be weaning off during December, metformin 1000mg BID). Blood sugar is about 170 at am daily most likely to lack of mobility and use of Metformin for 5 yrs. However, his ferritin is 535. His RBCs are still WNL but on the way down (where he will be anemic in 2-3 mos) and his MCV is 87. According to Fishbach and Baker his current drugs are not know to increase the ferritin. So my question is ....can corticobasal syndrome cause the ferritin to increase like this? Is this due to his lack of mobility where his iron stores can not be utilized? Or should I be looking for something else (like hemochromatosis, CA, other/current chronic dz)? Regardless, we have to get the ferritin down due to potential of harm or increased harm. I remember hearing tx could be with phlebotomy if it is hemochromatosis. I need some guidance as to cause in order to make sure I am not missing something. Additionally, I will do what is needed to decrease ferritin but would like to use drainage via gemmos. Would tamarix gallica be indicated for something like this along with 1, 20, and 243 at the same time or will the 1, 20 and 243 take the iron out without him being able to utilize the iron once mobilized? I have put him also on Magnlvrs, B-complex with extra B12 and folate into his regimen as well as 1200mg of CoQ10 daily and probiotic. Any advice on cause or treatment would be greatly appreciated.Thanks,Leah Hollon, MPH, NDVansant, VA 24656 Quote Link to comment Share on other sites More sharing options...
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