Bacteria and Immune System Race

[Guest guest]

Here's the famous " slime " Tony's always harping about.

This really explains what we're dealing with. You can replace

the " non-living " prothesis in the article with any necrotic region

(dead bone, dead tissue, etc.) or any place that does not receive

good blood flow (Immune system). You can also replace this " non-

living prothesis " , with root canals or dental implants. That's why

none of us should be having these procedures done, and most

especially if we're already ill and colonized.

Race for surface theory

The total joint prosthesis is a non-living object with no

protection of its own against the colonization by bacteria. When a

total joint prosthesis is placed into the human body, the body's

cells hurry to get hold on the surface of the prosthesis.

Once the body cells colonize the surface of the total joint

prosthesis they can protect this non living surface against the

colonization attempts of the bacteria.

If any bacteria are present in the operation wound, and most often

these bacteria are the skin bacteria, they hurry to get hold on the

prosthesis surface too. Thus there develops a race - who will be

first to colonize the surface, bacteria or body's cells.

By the long biological development, the bacteria evolved the

capability to adhere to surfaces for their survival, not only on

the human body, but everywhere in the nature. Bacteria have thus

biological advantage against the body's cells in this race.

Once the bacteria attached themselves to the surface of the

prosthesis they form a slime envelope that protects them against the

action of antibiotics and the immune defense of the body. Moreover,

adherence to a surface also changes the characteristics of the

adhering bacteria, among others their resistance to antibiotics

increases 100 to 1000 times.

[Guest guest]

'they form a slime envelope that protects them against the

action of antibiotics and the immune defense of the body.'

This is the best EXPLANATION of why our immune system fails- 'the

tricks' AKA-ANTIBIOTIC RESISTANCE IN BACTERIA, is why we get into

trouble, the 'same mecahanisms' are at play to beat our IMMUNE

SYSTEM IMO.And funny enough this is a problem with people in the

general community, not just the chronically ill like us.

tony

>

> Here's the famous " slime " Tony's always harping about.

>

> This really explains what we're dealing with. You can replace

> the " non-living " prothesis in the article with any necrotic region

> (dead bone, dead tissue, etc.) or any place that does not receive

> good blood flow (Immune system). You can also replace this " non-

> living prothesis " , with root canals or dental implants. That's why

> none of us should be having these procedures done, and most

> especially if we're already ill and colonized.

>

>

> Race for surface theory

>

> The total joint prosthesis is a non-living object with no

> protection of its own against the colonization by bacteria. When a

> total joint prosthesis is placed into the human body, the body's

> cells hurry to get hold on the surface of the prosthesis.

>

> Once the body cells colonize the surface of the total joint

> prosthesis they can protect this non living surface against the

> colonization attempts of the bacteria.

>

> If any bacteria are present in the operation wound, and most often

> these bacteria are the skin bacteria, they hurry to get hold on

the

> prosthesis surface too. Thus there develops a race - who will be

> first to colonize the surface, bacteria or body's cells.

>

> By the long biological development, the bacteria evolved the

> capability to adhere to surfaces for their survival, not only on

> the human body, but everywhere in the nature. Bacteria have thus

> biological advantage against the body's cells in this race.

>

> Once the bacteria attached themselves to the surface of the

> prosthesis they form a slime envelope that protects them against

the

> action of antibiotics and the immune defense of the body.

Moreover,

> adherence to a surface also changes the characteristics of the

> adhering bacteria, among others their resistance to antibiotics

> increases 100 to 1000 times.

>