Re: Sue: Microbe (fungus) killing by medium chain Fatty Acids

[Guest guest]

Yes- it looks like it's approximately a tablespoon full (coconut

oil) that I put on toast if I take it every day- if I'm doing it less

often, I think I take a higher amount.

Ok- some of you should stop reading here - because here's a

typical " fat " sandwich that I have eaten VERY often.. a little less

often since I'm feeling so well, becuase I don't crave fat as much

now as I did over the years of feeling crappy. For some reason dairy

fat made me feel much better.

I'll smear about a tbs. coconut oil on some toast- cover that with

the same amount of butter, then cover that with fresh ground peanut

butter, and sometimes I'll put either Mayo or Jam on the other piece

of toast- then wash that all down with 8 Oz. of Half and Hhalf...I can

just hear people saying omigod- and I thought she was a smart

person... But ya know- there is something in that dairy fat - and I

think it might be the lauric acid (it's in butter too) that would

make me feel better - it would even lessen my almost constant head

ache.. And for some odd reason I'd crave it.

Like I said, the craving is mostly gone - but I can still slab

off butter and eat it like cheese.

It's what my husband says is a " quirk " . I have low

triglicerides, and low total cholesterol, and high HDL - so where's

the stuff going???????? It's a mystery to me.

Barb

> > > > >

> > > > > Hey you guys.........

> > > > >

> > > > > Don't forget that there are natural agents that are

> excellent

> > > > > at killing yeast (and they don't build in resistance to the

> > > > microbe).

> > > > >

> > > > > On my original Candida Panel (LabCorp) I had a 2.5 IgA

> titer

> > > > > (< 0.8 is normal) and IgM and IgG were psuhing the normal

> > limits.

> > > > > I didn't use drugs to grt rids of it - I wanted to try

> enzymes

> > > or

> > > > > natural products first (becuase I didn't want to create a

> > > > > superinfection of resistant mutants).

> > > > >

> > > > > I used Nystatin, Candex and Coconut oil(which is like

50%

> > > > Lauric

> > > > > Acid). I was using lauricidin previous to coconut oil- but

> > > > swicthed

> > > > > to the oil because I suspect it metabolizes easier. And the

> > same

> > > > oil

> > > > > you ingest, you can put on your skin.

> > > > >

> > > > > It took over 8 months for my titer dropped to normal- but

> > that

> > > > > might have been a reflection of how long it takes the titer

> to

> > > > drop.

> > > > >

> > > > > The other thing to remember about long term abx is

> frequent

> > > sore

> > > > > throats: The last I looked at the stastics of people on

> long

> > > term

> > > > > abx therapy for acne- showed those people getting sore

> thraots

> > > due

> > > > to

> > > > > the imbalance of flora in the throat.

> > > > >

> > > > > So I think you HAVE to address fungal issues while on abx -

> or

> > > > your

> > > > > just trading one problem for another.

> > > > >

> > > > > Barb

> > > > >

> > > > >

> > > > >

> > > > > Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> > > > > In vitro killing of Candida albicans by fatty acids and

> > > > > monoglycerides.

> > > > >

> > > > > Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

> > > > >

> > > > > Institute of Biology, University of Iceland, Reykjavik,

> > Iceland.

> > > > > gudmunb@h...

> > > > >

> > > > > The susceptibility of Candida albicans to several fatty

> acids

> > > and

> > > > > their 1-monoglycerides was tested with a short inactivation

> > > time,

> > > > and

> > > > > ultrathin sections were studied by transmission electron

> > > > microscopy

> > > > > (TEM) after treatment with capric acid. The results show

> that

> > > > capric

> > > > > acid, a 10-carbon saturated fatty acid, causes the fastest

> and

> > > > most

> > > > > effective killing of all three strains of C. albicans

> tested,

> > > > leaving

> > > > > the cytoplasm disorganized and shrunken because of a

> disrupted

> > > or

> > > > > disintegrated plasma membrane. Lauric acid, a 12-carbon

> > > saturated

> > > > > fatty acid, was the most active at lower concentrations and

> > > after

> > > > a

> > > > > longer incubation time.

> > > > >

> > > > > PMID: 11600381 [PubMed - indexed for MEDLINE]

> > > > >

> > > >

> > >

> ____________________________________________________________________

> > > > >

> > > > > Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> > > > > In vitro inactivation of Chlamydia trachomatis by fatty

> acids

> > > and

> > > > > monoglycerides.

> > > > >

> > > > > Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O,

> > Thormar

> > > H.

> > > > >

> > > > > Institute of Biology, University of Iceland, Reykjavik,

> Iceland.

> > > > >

> > > > > The antichlamydial effects of several fatty acids and

> > > > monoglycerides

> > > > > were studied by incubating Chlamydia trachomatis bacteria

> with

> > > > equal

> > > > > volumes of lipid solutions for 10 min and measuring the

> > > reduction

> > > > in

> > > > > infectivity titer compared with that in a control solution

> > > without

> > > > > lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin

> > > (12:0),

> > > > > myristic acid (14:0), palmitoleic acid (16:1),

> monopalmitolein

> > > > > (16:1), oleic acid (18:1), and monoolein (18:1) at

> > > concentrations

> > > > of

> > > > > 20 mM (final concentration, 10 mM) had negligible effects

on

> C.

> > > > > trachomatis. In contrast, lauric acid (12:0), capric acid

> > > (10:0),

> > > > and

> > > > > monocaprin (10:0) caused a greater than 10,000-fold (>4-

> log10)

> > > > > reduction in the infectivity titer. When the fatty acids

and

> > > > > monoglycerides were further compared at lower

concentrations

> > and

> > > > with

> > > > > shorter exposure times, lauric acid was more active than

> capric

> > > > acid

> > > > > and monocaprin was the most active, causing a greater than

> 100,

> > > > 000-

> > > > > fold (>5-log10) inactivation of C. trachomatis at a

> > > concentration

> > > > of

> > > > > 5 mM for 5 min. The high levels of activity of capric and

> > lauric

> > > > > acids and particularly that of monocaprin are notable and

> > > suggest

> > > > > that these lipids have specific antichlamydial effects. The

> > mode

> > > > of

> > > > > action of monocaprin was further studied by removal of the

> > lipid

> > > > by

> > > > > centrifugation before inoculation of Chlamydia onto host

> cells

> > > and

> > > > by

> > > > > electron microscopy. The results indicate that the bacteria

> are

> > > > > killed by the lipid, possibly by disrupting the membrane(s)

> of

> > > the

> > > > > elementary bodies. A 50% effective concentration of 30

> > > > microgram/ml

> > > > > was found by incubation of Chlamydia with monocaprin for 2

> h.

> > > The

> > > > > rapid inactivation of large numbers of C. trachomatis

> organisms

> > > by

> > > > > monocaprin suggests that it may be useful as a microbicidal

> > > agent

> > > > for

> > > > > the prevention of the sexual transmission of C. trachomatis.

> > > > >

> > > > > PMID: 9736551 [PubMed - indexed for MEDLINE]

> > > > >

> > > > >

> > > > > Or this page in PUBMED

> > > > >

> > > > > http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> > > > > db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

> > > > >

> > > >

> > >

> >

>

[Guest guest]

I eat an extremely high fat diet but very low sugar. I think its the

combo thats bad, or rather, sugar is bad.

I eat a lot of unsweetened chocolate, olive oil, fish oil, fish,

meat, butter, nuts, nut butters, you name it. There was just an

article on Eureka Alert about fats being anti inflammatory in the gut.

Mayo--I don't like that because of the sugar.

> > > > > >

> > > > > > Hey you guys.........

> > > > > >

> > > > > > Don't forget that there are natural agents that are

> > excellent

> > > > > > at killing yeast (and they don't build in resistance to

the

> > > > > microbe).

> > > > > >

> > > > > > On my original Candida Panel (LabCorp) I had a 2.5 IgA

> > titer

> > > > > > (< 0.8 is normal) and IgM and IgG were psuhing the normal

> > > limits.

> > > > > > I didn't use drugs to grt rids of it - I wanted to try

> > enzymes

> > > > or

> > > > > > natural products first (becuase I didn't want to create a

> > > > > > superinfection of resistant mutants).

> > > > > >

> > > > > > I used Nystatin, Candex and Coconut oil(which is like

> 50%

> > > > > Lauric

> > > > > > Acid). I was using lauricidin previous to coconut oil-

but

> > > > > swicthed

> > > > > > to the oil because I suspect it metabolizes easier. And

the

> > > same

> > > > > oil

> > > > > > you ingest, you can put on your skin.

> > > > > >

> > > > > > It took over 8 months for my titer dropped to normal-

but

> > > that

> > > > > > might have been a reflection of how long it takes the

titer

> > to

> > > > > drop.

> > > > > >

> > > > > > The other thing to remember about long term abx is

> > frequent

> > > > sore

> > > > > > throats: The last I looked at the stastics of people on

> > long

> > > > term

> > > > > > abx therapy for acne- showed those people getting sore

> > thraots

> > > > due

> > > > > to

> > > > > > the imbalance of flora in the throat.

> > > > > >

> > > > > > So I think you HAVE to address fungal issues while on

abx -

> > or

> > > > > your

> > > > > > just trading one problem for another.

> > > > > >

> > > > > > Barb

> > > > > >

> > > > > >

> > > > > >

> > > > > > Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> > > > > > In vitro killing of Candida albicans by fatty acids and

> > > > > > monoglycerides.

> > > > > >

> > > > > > Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

> > > > > >

> > > > > > Institute of Biology, University of Iceland, Reykjavik,

> > > Iceland.

> > > > > > gudmunb@h...

> > > > > >

> > > > > > The susceptibility of Candida albicans to several fatty

> > acids

> > > > and

> > > > > > their 1-monoglycerides was tested with a short

inactivation

> > > > time,

> > > > > and

> > > > > > ultrathin sections were studied by transmission electron

> > > > > microscopy

> > > > > > (TEM) after treatment with capric acid. The results show

> > that

> > > > > capric

> > > > > > acid, a 10-carbon saturated fatty acid, causes the

fastest

> > and

> > > > > most

> > > > > > effective killing of all three strains of C. albicans

> > tested,

> > > > > leaving

> > > > > > the cytoplasm disorganized and shrunken because of a

> > disrupted

> > > > or

> > > > > > disintegrated plasma membrane. Lauric acid, a 12-carbon

> > > > saturated

> > > > > > fatty acid, was the most active at lower concentrations

and

> > > > after

> > > > > a

> > > > > > longer incubation time.

> > > > > >

> > > > > > PMID: 11600381 [PubMed - indexed for MEDLINE]

> > > > > >

> > > > >

> > > >

> >

____________________________________________________________________

> > > > > >

> > > > > > Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> > > > > > In vitro inactivation of Chlamydia trachomatis by fatty

> > acids

> > > > and

> > > > > > monoglycerides.

> > > > > >

> > > > > > Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O,

> > > Thormar

> > > > H.

> > > > > >

> > > > > > Institute of Biology, University of Iceland, Reykjavik,

> > Iceland.

> > > > > >

> > > > > > The antichlamydial effects of several fatty acids and

> > > > > monoglycerides

> > > > > > were studied by incubating Chlamydia trachomatis bacteria

> > with

> > > > > equal

> > > > > > volumes of lipid solutions for 10 min and measuring the

> > > > reduction

> > > > > in

> > > > > > infectivity titer compared with that in a control

solution

> > > > without

> > > > > > lipid. Caprylic acid (8:0), monocaprylin (8:0),

monolaurin

> > > > (12:0),

> > > > > > myristic acid (14:0), palmitoleic acid (16:1),

> > monopalmitolein

> > > > > > (16:1), oleic acid (18:1), and monoolein (18:1) at

> > > > concentrations

> > > > > of

> > > > > > 20 mM (final concentration, 10 mM) had negligible effects

> on

> > C.

> > > > > > trachomatis. In contrast, lauric acid (12:0), capric acid

> > > > (10:0),

> > > > > and

> > > > > > monocaprin (10:0) caused a greater than 10,000-fold (>4-

> > log10)

> > > > > > reduction in the infectivity titer. When the fatty acids

> and

> > > > > > monoglycerides were further compared at lower

> concentrations

> > > and

> > > > > with

> > > > > > shorter exposure times, lauric acid was more active than

> > capric

> > > > > acid

> > > > > > and monocaprin was the most active, causing a greater

than

> > 100,

> > > > > 000-

> > > > > > fold (>5-log10) inactivation of C. trachomatis at a

> > > > concentration

> > > > > of

> > > > > > 5 mM for 5 min. The high levels of activity of capric and

> > > lauric

> > > > > > acids and particularly that of monocaprin are notable and

> > > > suggest

> > > > > > that these lipids have specific antichlamydial effects.

The

> > > mode

> > > > > of

> > > > > > action of monocaprin was further studied by removal of

the

> > > lipid

> > > > > by

> > > > > > centrifugation before inoculation of Chlamydia onto host

> > cells

> > > > and

> > > > > by

> > > > > > electron microscopy. The results indicate that the

bacteria

> > are

> > > > > > killed by the lipid, possibly by disrupting the membrane

(s)

> > of

> > > > the

> > > > > > elementary bodies. A 50% effective concentration of 30

> > > > > microgram/ml

> > > > > > was found by incubation of Chlamydia with monocaprin for

2

> > h.

> > > > The

> > > > > > rapid inactivation of large numbers of C. trachomatis

> > organisms

> > > > by

> > > > > > monocaprin suggests that it may be useful as a

microbicidal

> > > > agent

> > > > > for

> > > > > > the prevention of the sexual transmission of C.

trachomatis.

> > > > > >

> > > > > > PMID: 9736551 [PubMed - indexed for MEDLINE]

> > > > > >

> > > > > >

> > > > > > Or this page in PUBMED

> > > > > >

> > > > > > http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> > > > > > db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

> > > > > >

> > > > >

> > > >

> > >

> >

>

[Guest guest]

I could do that fat sandwich...

I haven't done this since I was a kid, but back when I was a pre-

teen, my mom would kill me because I would take a piece of bread,

butter both sides then fry it.

*S*

> > > > > >

> > > > > > Hey you guys.........

> > > > > >

> > > > > > Don't forget that there are natural agents that are

> > excellent

> > > > > > at killing yeast (and they don't build in resistance to

the

> > > > > microbe).

> > > > > >

> > > > > > On my original Candida Panel (LabCorp) I had a 2.5 IgA

> > titer

> > > > > > (< 0.8 is normal) and IgM and IgG were psuhing the normal

> > > limits.

> > > > > > I didn't use drugs to grt rids of it - I wanted to try

> > enzymes

> > > > or

> > > > > > natural products first (becuase I didn't want to create a

> > > > > > superinfection of resistant mutants).

> > > > > >

> > > > > > I used Nystatin, Candex and Coconut oil(which is like

> 50%

> > > > > Lauric

> > > > > > Acid). I was using lauricidin previous to coconut oil-

but

> > > > > swicthed

> > > > > > to the oil because I suspect it metabolizes easier. And

the

> > > same

> > > > > oil

> > > > > > you ingest, you can put on your skin.

> > > > > >

> > > > > > It took over 8 months for my titer dropped to normal-

but

> > > that

> > > > > > might have been a reflection of how long it takes the

titer

> > to

> > > > > drop.

> > > > > >

> > > > > > The other thing to remember about long term abx is

> > frequent

> > > > sore

> > > > > > throats: The last I looked at the stastics of people on

> > long

> > > > term

> > > > > > abx therapy for acne- showed those people getting sore

> > thraots

> > > > due

> > > > > to

> > > > > > the imbalance of flora in the throat.

> > > > > >

> > > > > > So I think you HAVE to address fungal issues while on

abx -

> > or

> > > > > your

> > > > > > just trading one problem for another.

> > > > > >

> > > > > > Barb

> > > > > >

> > > > > >

> > > > > >

> > > > > > Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> > > > > > In vitro killing of Candida albicans by fatty acids and

> > > > > > monoglycerides.

> > > > > >

> > > > > > Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

> > > > > >

> > > > > > Institute of Biology, University of Iceland, Reykjavik,

> > > Iceland.

> > > > > > gudmunb@h...

> > > > > >

> > > > > > The susceptibility of Candida albicans to several fatty

> > acids

> > > > and

> > > > > > their 1-monoglycerides was tested with a short

inactivation

> > > > time,

> > > > > and

> > > > > > ultrathin sections were studied by transmission electron

> > > > > microscopy

> > > > > > (TEM) after treatment with capric acid. The results show

> > that

> > > > > capric

> > > > > > acid, a 10-carbon saturated fatty acid, causes the

fastest

> > and

> > > > > most

> > > > > > effective killing of all three strains of C. albicans

> > tested,

> > > > > leaving

> > > > > > the cytoplasm disorganized and shrunken because of a

> > disrupted

> > > > or

> > > > > > disintegrated plasma membrane. Lauric acid, a 12-carbon

> > > > saturated

> > > > > > fatty acid, was the most active at lower concentrations

and

> > > > after

> > > > > a

> > > > > > longer incubation time.

> > > > > >

> > > > > > PMID: 11600381 [PubMed - indexed for MEDLINE]

> > > > > >

> > > > >

> > > >

> >

____________________________________________________________________

> > > > > >

> > > > > > Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> > > > > > In vitro inactivation of Chlamydia trachomatis by fatty

> > acids

> > > > and

> > > > > > monoglycerides.

> > > > > >

> > > > > > Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O,

> > > Thormar

> > > > H.

> > > > > >

> > > > > > Institute of Biology, University of Iceland, Reykjavik,

> > Iceland.

> > > > > >

> > > > > > The antichlamydial effects of several fatty acids and

> > > > > monoglycerides

> > > > > > were studied by incubating Chlamydia trachomatis bacteria

> > with

> > > > > equal

> > > > > > volumes of lipid solutions for 10 min and measuring the

> > > > reduction

> > > > > in

> > > > > > infectivity titer compared with that in a control

solution

> > > > without

> > > > > > lipid. Caprylic acid (8:0), monocaprylin (8:0),

monolaurin

> > > > (12:0),

> > > > > > myristic acid (14:0), palmitoleic acid (16:1),

> > monopalmitolein

> > > > > > (16:1), oleic acid (18:1), and monoolein (18:1) at

> > > > concentrations

> > > > > of

> > > > > > 20 mM (final concentration, 10 mM) had negligible effects

> on

> > C.

> > > > > > trachomatis. In contrast, lauric acid (12:0), capric acid

> > > > (10:0),

> > > > > and

> > > > > > monocaprin (10:0) caused a greater than 10,000-fold (>4-

> > log10)

> > > > > > reduction in the infectivity titer. When the fatty acids

> and

> > > > > > monoglycerides were further compared at lower

> concentrations

> > > and

> > > > > with

> > > > > > shorter exposure times, lauric acid was more active than

> > capric

> > > > > acid

> > > > > > and monocaprin was the most active, causing a greater

than

> > 100,

> > > > > 000-

> > > > > > fold (>5-log10) inactivation of C. trachomatis at a

> > > > concentration

> > > > > of

> > > > > > 5 mM for 5 min. The high levels of activity of capric and

> > > lauric

> > > > > > acids and particularly that of monocaprin are notable and

> > > > suggest

> > > > > > that these lipids have specific antichlamydial effects.

The

> > > mode

> > > > > of

> > > > > > action of monocaprin was further studied by removal of

the

> > > lipid

> > > > > by

> > > > > > centrifugation before inoculation of Chlamydia onto host

> > cells

> > > > and

> > > > > by

> > > > > > electron microscopy. The results indicate that the

bacteria

> > are

> > > > > > killed by the lipid, possibly by disrupting the membrane

(s)

> > of

> > > > the

> > > > > > elementary bodies. A 50% effective concentration of 30

> > > > > microgram/ml

> > > > > > was found by incubation of Chlamydia with monocaprin for

2

> > h.

> > > > The

> > > > > > rapid inactivation of large numbers of C. trachomatis

> > organisms

> > > > by

> > > > > > monocaprin suggests that it may be useful as a

microbicidal

> > > > agent

> > > > > for

> > > > > > the prevention of the sexual transmission of C.

trachomatis.

> > > > > >

> > > > > > PMID: 9736551 [PubMed - indexed for MEDLINE]

> > > > > >

> > > > > >

> > > > > > Or this page in PUBMED

> > > > > >

> > > > > > http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> > > > > > db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

> > > > > >

> > > > >

> > > >

> > >

> >

>