Re: Microbe (fungus) killing by medium chain Fatty Acids

[Guest guest]

Hey you guys.........

Don't forget that there are natural agents that are excellent

at killing yeast (and they don't build in resistance to the microbe).

On my original Candida Panel (LabCorp) I had a 2.5 IgA titer

(< 0.8 is normal) and IgM and IgG were psuhing the normal limits.

I didn't use drugs to grt rids of it - I wanted to try enzymes or

natural products first (becuase I didn't want to create a

superinfection of resistant mutants).

I used Nystatin, Candex and Coconut oil(which is like 50% Lauric

Acid). I was using lauricidin previous to coconut oil- but swicthed

to the oil because I suspect it metabolizes easier. And the same oil

you ingest, you can put on your skin.

It took over 8 months for my titer dropped to normal- but that

might have been a reflection of how long it takes the titer to drop.

The other thing to remember about long term abx is frequent sore

throats: The last I looked at the stastics of people on long term

abx therapy for acne- showed those people getting sore thraots due to

the imbalance of flora in the throat.

So I think you HAVE to address fungal issues while on abx - or your

just trading one problem for another.

Barb

Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

In vitro killing of Candida albicans by fatty acids and

monoglycerides.

Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

Institute of Biology, University of Iceland, Reykjavik, Iceland.

gudmunb@...

The susceptibility of Candida albicans to several fatty acids and

their 1-monoglycerides was tested with a short inactivation time, and

ultrathin sections were studied by transmission electron microscopy

(TEM) after treatment with capric acid. The results show that capric

acid, a 10-carbon saturated fatty acid, causes the fastest and most

effective killing of all three strains of C. albicans tested, leaving

the cytoplasm disorganized and shrunken because of a disrupted or

disintegrated plasma membrane. Lauric acid, a 12-carbon saturated

fatty acid, was the most active at lower concentrations and after a

longer incubation time.

PMID: 11600381 [PubMed - indexed for MEDLINE]

____________________________________________________________________

Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

In vitro inactivation of Chlamydia trachomatis by fatty acids and

monoglycerides.

Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O, Thormar H.

Institute of Biology, University of Iceland, Reykjavik, Iceland.

The antichlamydial effects of several fatty acids and monoglycerides

were studied by incubating Chlamydia trachomatis bacteria with equal

volumes of lipid solutions for 10 min and measuring the reduction in

infectivity titer compared with that in a control solution without

lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin (12:0),

myristic acid (14:0), palmitoleic acid (16:1), monopalmitolein

(16:1), oleic acid (18:1), and monoolein (18:1) at concentrations of

20 mM (final concentration, 10 mM) had negligible effects on C.

trachomatis. In contrast, lauric acid (12:0), capric acid (10:0), and

monocaprin (10:0) caused a greater than 10,000-fold (>4-log10)

reduction in the infectivity titer. When the fatty acids and

monoglycerides were further compared at lower concentrations and with

shorter exposure times, lauric acid was more active than capric acid

and monocaprin was the most active, causing a greater than 100, 000-

fold (>5-log10) inactivation of C. trachomatis at a concentration of

5 mM for 5 min. The high levels of activity of capric and lauric

acids and particularly that of monocaprin are notable and suggest

that these lipids have specific antichlamydial effects. The mode of

action of monocaprin was further studied by removal of the lipid by

centrifugation before inoculation of Chlamydia onto host cells and by

electron microscopy. The results indicate that the bacteria are

killed by the lipid, possibly by disrupting the membrane(s) of the

elementary bodies. A 50% effective concentration of 30 microgram/ml

was found by incubation of Chlamydia with monocaprin for 2 h. The

rapid inactivation of large numbers of C. trachomatis organisms by

monocaprin suggests that it may be useful as a microbicidal agent for

the prevention of the sexual transmission of C. trachomatis.

PMID: 9736551 [PubMed - indexed for MEDLINE]

Or this page in PUBMED

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

[Guest guest]

> The other thing to remember about long term abx is frequent sore

> throats: The last I looked at the stastics of people on long term

> abx therapy for acne- showed those people getting sore thraots due

to

> the imbalance of flora in the throat.

My prof at GMU said Strep pyogenes can be recovered from about 10-20%

of healthy human throats. Its probably ready to party on a moments

notice.

[Guest guest]

My gut symptoms have been much better since starting to consume

coconut oil daily. Appetite is back, bowels more regular, much less

turbulence and discomfort.

*Bows in the general direction of B. Peck*

S.

>

> Hey you guys.........

>

> Don't forget that there are natural agents that are excellent

> at killing yeast (and they don't build in resistance to the

microbe).

>

> On my original Candida Panel (LabCorp) I had a 2.5 IgA titer

> (< 0.8 is normal) and IgM and IgG were psuhing the normal limits.

> I didn't use drugs to grt rids of it - I wanted to try enzymes or

> natural products first (becuase I didn't want to create a

> superinfection of resistant mutants).

>

> I used Nystatin, Candex and Coconut oil(which is like 50%

Lauric

> Acid). I was using lauricidin previous to coconut oil- but

swicthed

> to the oil because I suspect it metabolizes easier. And the same

oil

> you ingest, you can put on your skin.

>

> It took over 8 months for my titer dropped to normal- but that

> might have been a reflection of how long it takes the titer to

drop.

>

> The other thing to remember about long term abx is frequent sore

> throats: The last I looked at the stastics of people on long term

> abx therapy for acne- showed those people getting sore thraots due

to

> the imbalance of flora in the throat.

>

> So I think you HAVE to address fungal issues while on abx - or

your

> just trading one problem for another.

>

> Barb

>

>

>

> Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> In vitro killing of Candida albicans by fatty acids and

> monoglycerides.

>

> Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

>

> Institute of Biology, University of Iceland, Reykjavik, Iceland.

> gudmunb@h...

>

> The susceptibility of Candida albicans to several fatty acids and

> their 1-monoglycerides was tested with a short inactivation time,

and

> ultrathin sections were studied by transmission electron

microscopy

> (TEM) after treatment with capric acid. The results show that

capric

> acid, a 10-carbon saturated fatty acid, causes the fastest and

most

> effective killing of all three strains of C. albicans tested,

leaving

> the cytoplasm disorganized and shrunken because of a disrupted or

> disintegrated plasma membrane. Lauric acid, a 12-carbon saturated

> fatty acid, was the most active at lower concentrations and after

a

> longer incubation time.

>

> PMID: 11600381 [PubMed - indexed for MEDLINE]

>

____________________________________________________________________

>

> Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> In vitro inactivation of Chlamydia trachomatis by fatty acids and

> monoglycerides.

>

> Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O, Thormar H.

>

> Institute of Biology, University of Iceland, Reykjavik, Iceland.

>

> The antichlamydial effects of several fatty acids and

monoglycerides

> were studied by incubating Chlamydia trachomatis bacteria with

equal

> volumes of lipid solutions for 10 min and measuring the reduction

in

> infectivity titer compared with that in a control solution without

> lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin (12:0),

> myristic acid (14:0), palmitoleic acid (16:1), monopalmitolein

> (16:1), oleic acid (18:1), and monoolein (18:1) at concentrations

of

> 20 mM (final concentration, 10 mM) had negligible effects on C.

> trachomatis. In contrast, lauric acid (12:0), capric acid (10:0),

and

> monocaprin (10:0) caused a greater than 10,000-fold (>4-log10)

> reduction in the infectivity titer. When the fatty acids and

> monoglycerides were further compared at lower concentrations and

with

> shorter exposure times, lauric acid was more active than capric

acid

> and monocaprin was the most active, causing a greater than 100,

000-

> fold (>5-log10) inactivation of C. trachomatis at a concentration

of

> 5 mM for 5 min. The high levels of activity of capric and lauric

> acids and particularly that of monocaprin are notable and suggest

> that these lipids have specific antichlamydial effects. The mode

of

> action of monocaprin was further studied by removal of the lipid

by

> centrifugation before inoculation of Chlamydia onto host cells and

by

> electron microscopy. The results indicate that the bacteria are

> killed by the lipid, possibly by disrupting the membrane(s) of the

> elementary bodies. A 50% effective concentration of 30

microgram/ml

> was found by incubation of Chlamydia with monocaprin for 2 h. The

> rapid inactivation of large numbers of C. trachomatis organisms by

> monocaprin suggests that it may be useful as a microbicidal agent

for

> the prevention of the sexual transmission of C. trachomatis.

>

> PMID: 9736551 [PubMed - indexed for MEDLINE]

>

>

> Or this page in PUBMED

>

> http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

>

[Guest guest]

wow! I forgot about putting coconut oil ON the skin! Good idea! I've

been dieting and sort of backed off the coconut oil, for the short

term, but that is a really good idea.

thanks, Barb.

>

> Hey you guys.........

>

> Don't forget that there are natural agents that are excellent

> at killing yeast (and they don't build in resistance to the

microbe).

>

> On my original Candida Panel (LabCorp) I had a 2.5 IgA titer

> (< 0.8 is normal) and IgM and IgG were psuhing the normal limits.

> I didn't use drugs to grt rids of it - I wanted to try enzymes or

> natural products first (becuase I didn't want to create a

> superinfection of resistant mutants).

>

> I used Nystatin, Candex and Coconut oil(which is like 50%

Lauric

> Acid). I was using lauricidin previous to coconut oil- but

swicthed

> to the oil because I suspect it metabolizes easier. And the same

oil

> you ingest, you can put on your skin.

>

> It took over 8 months for my titer dropped to normal- but that

> might have been a reflection of how long it takes the titer to

drop.

>

> The other thing to remember about long term abx is frequent sore

> throats: The last I looked at the stastics of people on long term

> abx therapy for acne- showed those people getting sore thraots due

to

> the imbalance of flora in the throat.

>

> So I think you HAVE to address fungal issues while on abx - or

your

> just trading one problem for another.

>

> Barb

>

>

>

> Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> In vitro killing of Candida albicans by fatty acids and

> monoglycerides.

>

> Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

>

> Institute of Biology, University of Iceland, Reykjavik, Iceland.

> gudmunb@h...

>

> The susceptibility of Candida albicans to several fatty acids and

> their 1-monoglycerides was tested with a short inactivation time,

and

> ultrathin sections were studied by transmission electron

microscopy

> (TEM) after treatment with capric acid. The results show that

capric

> acid, a 10-carbon saturated fatty acid, causes the fastest and

most

> effective killing of all three strains of C. albicans tested,

leaving

> the cytoplasm disorganized and shrunken because of a disrupted or

> disintegrated plasma membrane. Lauric acid, a 12-carbon saturated

> fatty acid, was the most active at lower concentrations and after

a

> longer incubation time.

>

> PMID: 11600381 [PubMed - indexed for MEDLINE]

>

____________________________________________________________________

>

> Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> In vitro inactivation of Chlamydia trachomatis by fatty acids and

> monoglycerides.

>

> Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O, Thormar H.

>

> Institute of Biology, University of Iceland, Reykjavik, Iceland.

>

> The antichlamydial effects of several fatty acids and

monoglycerides

> were studied by incubating Chlamydia trachomatis bacteria with

equal

> volumes of lipid solutions for 10 min and measuring the reduction

in

> infectivity titer compared with that in a control solution without

> lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin (12:0),

> myristic acid (14:0), palmitoleic acid (16:1), monopalmitolein

> (16:1), oleic acid (18:1), and monoolein (18:1) at concentrations

of

> 20 mM (final concentration, 10 mM) had negligible effects on C.

> trachomatis. In contrast, lauric acid (12:0), capric acid (10:0),

and

> monocaprin (10:0) caused a greater than 10,000-fold (>4-log10)

> reduction in the infectivity titer. When the fatty acids and

> monoglycerides were further compared at lower concentrations and

with

> shorter exposure times, lauric acid was more active than capric

acid

> and monocaprin was the most active, causing a greater than 100,

000-

> fold (>5-log10) inactivation of C. trachomatis at a concentration

of

> 5 mM for 5 min. The high levels of activity of capric and lauric

> acids and particularly that of monocaprin are notable and suggest

> that these lipids have specific antichlamydial effects. The mode

of

> action of monocaprin was further studied by removal of the lipid

by

> centrifugation before inoculation of Chlamydia onto host cells and

by

> electron microscopy. The results indicate that the bacteria are

> killed by the lipid, possibly by disrupting the membrane(s) of the

> elementary bodies. A 50% effective concentration of 30

microgram/ml

> was found by incubation of Chlamydia with monocaprin for 2 h. The

> rapid inactivation of large numbers of C. trachomatis organisms by

> monocaprin suggests that it may be useful as a microbicidal agent

for

> the prevention of the sexual transmission of C. trachomatis.

>

> PMID: 9736551 [PubMed - indexed for MEDLINE]

>

>

> Or this page in PUBMED

>

> http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

>

[Guest guest]

I can't really answer your question - I used Nystatin initially for

about 30 + days on s recommendation- I look to him as the expert

on Yeasts. The dose was quite high, but I don't remember off hand

what it was (it's measured in bacterial units or something).

ANd then I took Candex and the monolaurin for months and months after

that. And I was also pumping in Yogurt (and DanActive when I could

get that shipped up here - that's the best probiotic IMO) and plus -

I was trying to eat more fermented foods.

I would have and will use the fungal drugs if I have to - but just

wanted to wait and see if I could get rid of Candida without the

drugs. (I'm not against the drugs)

Barb

PS... I'm not sure if anything will totally control yeast while your

on abx.. but maybe you can hold even with the board if you use anti-

fungal drugs and the natural products (i.e. kill some- then grow

more, kill 'em - grow more, and so on)

> >    Don't forget that there are natural agents that are excellent

> > at killing yeast (and they don't build in resistance to the

microbe).

> > ...

> >    I used Nystatin, Candex and Coconut oil(which is like 50%

Lauric

> > Acid).  I was using lauricidin previous to coconut oil- but

swicthed

> > to the oil because I suspect it metabolizes easier. And the same

oil

> > you ingest, you can put on your skin.

>

> Barb, I've been taking Lauricidin and meaning to order Candex

(always a

> pain to have stuff sent to Canada). I'll definitely use these

things,

> but have been in the situation of needing some immediate relief.

>

> With the Nystatin, should it be used regularly, or just brought in

when

> the natural stuff isn't able to hold down the fort?

>

> - Kate

>

[Guest guest]

, Barb:

How much and how do you eat coconut oil daily?

I have been using coconut oil (EVCO) for quite some time - it is the

best for both cooking and skin, but I have never thought of taking

it therapeutically - how do you do it?

*S*

> >

> > Hey you guys.........

> >

> > Don't forget that there are natural agents that are excellent

> > at killing yeast (and they don't build in resistance to the

> microbe).

> >

> > On my original Candida Panel (LabCorp) I had a 2.5 IgA titer

> > (< 0.8 is normal) and IgM and IgG were psuhing the normal limits.

> > I didn't use drugs to grt rids of it - I wanted to try enzymes

or

> > natural products first (becuase I didn't want to create a

> > superinfection of resistant mutants).

> >

> > I used Nystatin, Candex and Coconut oil(which is like 50%

> Lauric

> > Acid). I was using lauricidin previous to coconut oil- but

> swicthed

> > to the oil because I suspect it metabolizes easier. And the same

> oil

> > you ingest, you can put on your skin.

> >

> > It took over 8 months for my titer dropped to normal- but that

> > might have been a reflection of how long it takes the titer to

> drop.

> >

> > The other thing to remember about long term abx is frequent

sore

> > throats: The last I looked at the stastics of people on long

term

> > abx therapy for acne- showed those people getting sore thraots

due

> to

> > the imbalance of flora in the throat.

> >

> > So I think you HAVE to address fungal issues while on abx - or

> your

> > just trading one problem for another.

> >

> > Barb

> >

> >

> >

> > Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> > In vitro killing of Candida albicans by fatty acids and

> > monoglycerides.

> >

> > Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

> >

> > Institute of Biology, University of Iceland, Reykjavik, Iceland.

> > gudmunb@h...

> >

> > The susceptibility of Candida albicans to several fatty acids

and

> > their 1-monoglycerides was tested with a short inactivation

time,

> and

> > ultrathin sections were studied by transmission electron

> microscopy

> > (TEM) after treatment with capric acid. The results show that

> capric

> > acid, a 10-carbon saturated fatty acid, causes the fastest and

> most

> > effective killing of all three strains of C. albicans tested,

> leaving

> > the cytoplasm disorganized and shrunken because of a disrupted

or

> > disintegrated plasma membrane. Lauric acid, a 12-carbon

saturated

> > fatty acid, was the most active at lower concentrations and

after

> a

> > longer incubation time.

> >

> > PMID: 11600381 [PubMed - indexed for MEDLINE]

> >

>

____________________________________________________________________

> >

> > Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> > In vitro inactivation of Chlamydia trachomatis by fatty acids

and

> > monoglycerides.

> >

> > Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O, Thormar

H.

> >

> > Institute of Biology, University of Iceland, Reykjavik, Iceland.

> >

> > The antichlamydial effects of several fatty acids and

> monoglycerides

> > were studied by incubating Chlamydia trachomatis bacteria with

> equal

> > volumes of lipid solutions for 10 min and measuring the

reduction

> in

> > infectivity titer compared with that in a control solution

without

> > lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin

(12:0),

> > myristic acid (14:0), palmitoleic acid (16:1), monopalmitolein

> > (16:1), oleic acid (18:1), and monoolein (18:1) at

concentrations

> of

> > 20 mM (final concentration, 10 mM) had negligible effects on C.

> > trachomatis. In contrast, lauric acid (12:0), capric acid

(10:0),

> and

> > monocaprin (10:0) caused a greater than 10,000-fold (>4-log10)

> > reduction in the infectivity titer. When the fatty acids and

> > monoglycerides were further compared at lower concentrations and

> with

> > shorter exposure times, lauric acid was more active than capric

> acid

> > and monocaprin was the most active, causing a greater than 100,

> 000-

> > fold (>5-log10) inactivation of C. trachomatis at a

concentration

> of

> > 5 mM for 5 min. The high levels of activity of capric and lauric

> > acids and particularly that of monocaprin are notable and

suggest

> > that these lipids have specific antichlamydial effects. The mode

> of

> > action of monocaprin was further studied by removal of the lipid

> by

> > centrifugation before inoculation of Chlamydia onto host cells

and

> by

> > electron microscopy. The results indicate that the bacteria are

> > killed by the lipid, possibly by disrupting the membrane(s) of

the

> > elementary bodies. A 50% effective concentration of 30

> microgram/ml

> > was found by incubation of Chlamydia with monocaprin for 2 h.

The

> > rapid inactivation of large numbers of C. trachomatis organisms

by

> > monocaprin suggests that it may be useful as a microbicidal

agent

> for

> > the prevention of the sexual transmission of C. trachomatis.

> >

> > PMID: 9736551 [PubMed - indexed for MEDLINE]

> >

> >

> > Or this page in PUBMED

> >

> > http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> > db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

> >

>

[Guest guest]

It should be cold pressed extra virgin - if you're going to ingest it.

http://tropicaltraditions.com/faq.htm

is where I buy it- and this is their Q & A page and they'll answer

the questions about amount.

If you've normally been a " low fat " kinda person with your diet, you

should ease into this.

I've always been a person who ingested very high amounts of

saturated fats (mostly dairy - but some plant oils too) and I know I

metabolize them well -. SO my total comsumption of dairy fat and oil

fats is way higher than most people - it's got to be a genetic thing

with me.

SO- I am not a good person to compare to.

I keep the oil in solid form, and shave it off onto warm toast -

that's the way I like to take it best-

Barb

> > >

> > > Hey you guys.........

> > >

> > > Don't forget that there are natural agents that are excellent

> > > at killing yeast (and they don't build in resistance to the

> > microbe).

> > >

> > > On my original Candida Panel (LabCorp) I had a 2.5 IgA titer

> > > (< 0.8 is normal) and IgM and IgG were psuhing the normal

limits.

> > > I didn't use drugs to grt rids of it - I wanted to try enzymes

> or

> > > natural products first (becuase I didn't want to create a

> > > superinfection of resistant mutants).

> > >

> > > I used Nystatin, Candex and Coconut oil(which is like 50%

> > Lauric

> > > Acid). I was using lauricidin previous to coconut oil- but

> > swicthed

> > > to the oil because I suspect it metabolizes easier. And the

same

> > oil

> > > you ingest, you can put on your skin.

> > >

> > > It took over 8 months for my titer dropped to normal- but

that

> > > might have been a reflection of how long it takes the titer to

> > drop.

> > >

> > > The other thing to remember about long term abx is frequent

> sore

> > > throats: The last I looked at the stastics of people on long

> term

> > > abx therapy for acne- showed those people getting sore thraots

> due

> > to

> > > the imbalance of flora in the throat.

> > >

> > > So I think you HAVE to address fungal issues while on abx - or

> > your

> > > just trading one problem for another.

> > >

> > > Barb

> > >

> > >

> > >

> > > Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> > > In vitro killing of Candida albicans by fatty acids and

> > > monoglycerides.

> > >

> > > Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

> > >

> > > Institute of Biology, University of Iceland, Reykjavik,

Iceland.

> > > gudmunb@h...

> > >

> > > The susceptibility of Candida albicans to several fatty acids

> and

> > > their 1-monoglycerides was tested with a short inactivation

> time,

> > and

> > > ultrathin sections were studied by transmission electron

> > microscopy

> > > (TEM) after treatment with capric acid. The results show that

> > capric

> > > acid, a 10-carbon saturated fatty acid, causes the fastest and

> > most

> > > effective killing of all three strains of C. albicans tested,

> > leaving

> > > the cytoplasm disorganized and shrunken because of a disrupted

> or

> > > disintegrated plasma membrane. Lauric acid, a 12-carbon

> saturated

> > > fatty acid, was the most active at lower concentrations and

> after

> > a

> > > longer incubation time.

> > >

> > > PMID: 11600381 [PubMed - indexed for MEDLINE]

> > >

> >

> ____________________________________________________________________

> > >

> > > Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> > > In vitro inactivation of Chlamydia trachomatis by fatty acids

> and

> > > monoglycerides.

> > >

> > > Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O,

Thormar

> H.

> > >

> > > Institute of Biology, University of Iceland, Reykjavik, Iceland.

> > >

> > > The antichlamydial effects of several fatty acids and

> > monoglycerides

> > > were studied by incubating Chlamydia trachomatis bacteria with

> > equal

> > > volumes of lipid solutions for 10 min and measuring the

> reduction

> > in

> > > infectivity titer compared with that in a control solution

> without

> > > lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin

> (12:0),

> > > myristic acid (14:0), palmitoleic acid (16:1), monopalmitolein

> > > (16:1), oleic acid (18:1), and monoolein (18:1) at

> concentrations

> > of

> > > 20 mM (final concentration, 10 mM) had negligible effects on C.

> > > trachomatis. In contrast, lauric acid (12:0), capric acid

> (10:0),

> > and

> > > monocaprin (10:0) caused a greater than 10,000-fold (>4-log10)

> > > reduction in the infectivity titer. When the fatty acids and

> > > monoglycerides were further compared at lower concentrations

and

> > with

> > > shorter exposure times, lauric acid was more active than capric

> > acid

> > > and monocaprin was the most active, causing a greater than 100,

> > 000-

> > > fold (>5-log10) inactivation of C. trachomatis at a

> concentration

> > of

> > > 5 mM for 5 min. The high levels of activity of capric and

lauric

> > > acids and particularly that of monocaprin are notable and

> suggest

> > > that these lipids have specific antichlamydial effects. The

mode

> > of

> > > action of monocaprin was further studied by removal of the

lipid

> > by

> > > centrifugation before inoculation of Chlamydia onto host cells

> and

> > by

> > > electron microscopy. The results indicate that the bacteria are

> > > killed by the lipid, possibly by disrupting the membrane(s) of

> the

> > > elementary bodies. A 50% effective concentration of 30

> > microgram/ml

> > > was found by incubation of Chlamydia with monocaprin for 2 h.

> The

> > > rapid inactivation of large numbers of C. trachomatis organisms

> by

> > > monocaprin suggests that it may be useful as a microbicidal

> agent

> > for

> > > the prevention of the sexual transmission of C. trachomatis.

> > >

> > > PMID: 9736551 [PubMed - indexed for MEDLINE]

> > >

> > >

> > > Or this page in PUBMED

> > >

> > > http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> > > db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

> > >

> >

>

[Guest guest]

Thanks , and glad it's been a positive addition for you.

Barb

> >

> > Hey you guys.........

> >

> > Don't forget that there are natural agents that are excellent

> > at killing yeast (and they don't build in resistance to the

> microbe).

> >

> > On my original Candida Panel (LabCorp) I had a 2.5 IgA titer

> > (< 0.8 is normal) and IgM and IgG were psuhing the normal limits.

> > I didn't use drugs to grt rids of it - I wanted to try enzymes or

> > natural products first (becuase I didn't want to create a

> > superinfection of resistant mutants).

> >

> > I used Nystatin, Candex and Coconut oil(which is like 50%

> Lauric

> > Acid). I was using lauricidin previous to coconut oil- but

> swicthed

> > to the oil because I suspect it metabolizes easier. And the same

> oil

> > you ingest, you can put on your skin.

> >

> > It took over 8 months for my titer dropped to normal- but that

> > might have been a reflection of how long it takes the titer to

> drop.

> >

> > The other thing to remember about long term abx is frequent

sore

> > throats: The last I looked at the stastics of people on long

term

> > abx therapy for acne- showed those people getting sore thraots

due

> to

> > the imbalance of flora in the throat.

> >

> > So I think you HAVE to address fungal issues while on abx - or

> your

> > just trading one problem for another.

> >

> > Barb

> >

> >

> >

> > Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> > In vitro killing of Candida albicans by fatty acids and

> > monoglycerides.

> >

> > Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

> >

> > Institute of Biology, University of Iceland, Reykjavik, Iceland.

> > gudmunb@h...

> >

> > The susceptibility of Candida albicans to several fatty acids and

> > their 1-monoglycerides was tested with a short inactivation time,

> and

> > ultrathin sections were studied by transmission electron

> microscopy

> > (TEM) after treatment with capric acid. The results show that

> capric

> > acid, a 10-carbon saturated fatty acid, causes the fastest and

> most

> > effective killing of all three strains of C. albicans tested,

> leaving

> > the cytoplasm disorganized and shrunken because of a disrupted or

> > disintegrated plasma membrane. Lauric acid, a 12-carbon saturated

> > fatty acid, was the most active at lower concentrations and after

> a

> > longer incubation time.

> >

> > PMID: 11600381 [PubMed - indexed for MEDLINE]

> >

> ____________________________________________________________________

> >

> > Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> > In vitro inactivation of Chlamydia trachomatis by fatty acids and

> > monoglycerides.

> >

> > Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O, Thormar

H.

> >

> > Institute of Biology, University of Iceland, Reykjavik, Iceland.

> >

> > The antichlamydial effects of several fatty acids and

> monoglycerides

> > were studied by incubating Chlamydia trachomatis bacteria with

> equal

> > volumes of lipid solutions for 10 min and measuring the reduction

> in

> > infectivity titer compared with that in a control solution

without

> > lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin

(12:0),

> > myristic acid (14:0), palmitoleic acid (16:1), monopalmitolein

> > (16:1), oleic acid (18:1), and monoolein (18:1) at concentrations

> of

> > 20 mM (final concentration, 10 mM) had negligible effects on C.

> > trachomatis. In contrast, lauric acid (12:0), capric acid (10:0),

> and

> > monocaprin (10:0) caused a greater than 10,000-fold (>4-log10)

> > reduction in the infectivity titer. When the fatty acids and

> > monoglycerides were further compared at lower concentrations and

> with

> > shorter exposure times, lauric acid was more active than capric

> acid

> > and monocaprin was the most active, causing a greater than 100,

> 000-

> > fold (>5-log10) inactivation of C. trachomatis at a concentration

> of

> > 5 mM for 5 min. The high levels of activity of capric and lauric

> > acids and particularly that of monocaprin are notable and suggest

> > that these lipids have specific antichlamydial effects. The mode

> of

> > action of monocaprin was further studied by removal of the lipid

> by

> > centrifugation before inoculation of Chlamydia onto host cells

and

> by

> > electron microscopy. The results indicate that the bacteria are

> > killed by the lipid, possibly by disrupting the membrane(s) of

the

> > elementary bodies. A 50% effective concentration of 30

> microgram/ml

> > was found by incubation of Chlamydia with monocaprin for 2 h. The

> > rapid inactivation of large numbers of C. trachomatis organisms

by

> > monocaprin suggests that it may be useful as a microbicidal agent

> for

> > the prevention of the sexual transmission of C. trachomatis.

> >

> > PMID: 9736551 [PubMed - indexed for MEDLINE]

> >

> >

> > Or this page in PUBMED

> >

> > http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> > db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

> >

>

[Guest guest]

Penny:

The right fats won't make you fat.

Barb

> >

> > Hey you guys.........

> >

> > Don't forget that there are natural agents that are excellent

> > at killing yeast (and they don't build in resistance to the

> microbe).

> >

> > On my original Candida Panel (LabCorp) I had a 2.5 IgA titer

> > (< 0.8 is normal) and IgM and IgG were psuhing the normal limits.

> > I didn't use drugs to grt rids of it - I wanted to try enzymes or

> > natural products first (becuase I didn't want to create a

> > superinfection of resistant mutants).

> >

> > I used Nystatin, Candex and Coconut oil(which is like 50%

> Lauric

> > Acid). I was using lauricidin previous to coconut oil- but

> swicthed

> > to the oil because I suspect it metabolizes easier. And the same

> oil

> > you ingest, you can put on your skin.

> >

> > It took over 8 months for my titer dropped to normal- but that

> > might have been a reflection of how long it takes the titer to

> drop.

> >

> > The other thing to remember about long term abx is frequent

sore

> > throats: The last I looked at the stastics of people on long

term

> > abx therapy for acne- showed those people getting sore thraots

due

> to

> > the imbalance of flora in the throat.

> >

> > So I think you HAVE to address fungal issues while on abx - or

> your

> > just trading one problem for another.

> >

> > Barb

> >

> >

> >

> > Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> > In vitro killing of Candida albicans by fatty acids and

> > monoglycerides.

> >

> > Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

> >

> > Institute of Biology, University of Iceland, Reykjavik, Iceland.

> > gudmunb@h...

> >

> > The susceptibility of Candida albicans to several fatty acids and

> > their 1-monoglycerides was tested with a short inactivation time,

> and

> > ultrathin sections were studied by transmission electron

> microscopy

> > (TEM) after treatment with capric acid. The results show that

> capric

> > acid, a 10-carbon saturated fatty acid, causes the fastest and

> most

> > effective killing of all three strains of C. albicans tested,

> leaving

> > the cytoplasm disorganized and shrunken because of a disrupted or

> > disintegrated plasma membrane. Lauric acid, a 12-carbon saturated

> > fatty acid, was the most active at lower concentrations and after

> a

> > longer incubation time.

> >

> > PMID: 11600381 [PubMed - indexed for MEDLINE]

> >

> ____________________________________________________________________

> >

> > Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> > In vitro inactivation of Chlamydia trachomatis by fatty acids and

> > monoglycerides.

> >

> > Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O, Thormar

H.

> >

> > Institute of Biology, University of Iceland, Reykjavik, Iceland.

> >

> > The antichlamydial effects of several fatty acids and

> monoglycerides

> > were studied by incubating Chlamydia trachomatis bacteria with

> equal

> > volumes of lipid solutions for 10 min and measuring the reduction

> in

> > infectivity titer compared with that in a control solution

without

> > lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin

(12:0),

> > myristic acid (14:0), palmitoleic acid (16:1), monopalmitolein

> > (16:1), oleic acid (18:1), and monoolein (18:1) at concentrations

> of

> > 20 mM (final concentration, 10 mM) had negligible effects on C.

> > trachomatis. In contrast, lauric acid (12:0), capric acid (10:0),

> and

> > monocaprin (10:0) caused a greater than 10,000-fold (>4-log10)

> > reduction in the infectivity titer. When the fatty acids and

> > monoglycerides were further compared at lower concentrations and

> with

> > shorter exposure times, lauric acid was more active than capric

> acid

> > and monocaprin was the most active, causing a greater than 100,

> 000-

> > fold (>5-log10) inactivation of C. trachomatis at a concentration

> of

> > 5 mM for 5 min. The high levels of activity of capric and lauric

> > acids and particularly that of monocaprin are notable and suggest

> > that these lipids have specific antichlamydial effects. The mode

> of

> > action of monocaprin was further studied by removal of the lipid

> by

> > centrifugation before inoculation of Chlamydia onto host cells

and

> by

> > electron microscopy. The results indicate that the bacteria are

> > killed by the lipid, possibly by disrupting the membrane(s) of

the

> > elementary bodies. A 50% effective concentration of 30

> microgram/ml

> > was found by incubation of Chlamydia with monocaprin for 2 h. The

> > rapid inactivation of large numbers of C. trachomatis organisms

by

> > monocaprin suggests that it may be useful as a microbicidal agent

> for

> > the prevention of the sexual transmission of C. trachomatis.

> >

> > PMID: 9736551 [PubMed - indexed for MEDLINE]

> >

> >

> > Or this page in PUBMED

> >

> > http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> > db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

> >

>

[Guest guest]

I put extra virgin coconut oil on my body after the shower every

morning. It's great, smooth, not greasy, AND it has no methylparabens

and other crap.

> >

> > Hey you guys.........

> >

> > Don't forget that there are natural agents that are excellent

> > at killing yeast (and they don't build in resistance to the

> microbe).

> >

> > On my original Candida Panel (LabCorp) I had a 2.5 IgA titer

> > (< 0.8 is normal) and IgM and IgG were psuhing the normal limits.

> > I didn't use drugs to grt rids of it - I wanted to try enzymes or

> > natural products first (becuase I didn't want to create a

> > superinfection of resistant mutants).

> >

> > I used Nystatin, Candex and Coconut oil(which is like 50%

> Lauric

> > Acid). I was using lauricidin previous to coconut oil- but

> swicthed

> > to the oil because I suspect it metabolizes easier. And the same

> oil

> > you ingest, you can put on your skin.

> >

> > It took over 8 months for my titer dropped to normal- but that

> > might have been a reflection of how long it takes the titer to

> drop.

> >

> > The other thing to remember about long term abx is frequent

sore

> > throats: The last I looked at the stastics of people on long

term

> > abx therapy for acne- showed those people getting sore thraots

due

> to

> > the imbalance of flora in the throat.

> >

> > So I think you HAVE to address fungal issues while on abx - or

> your

> > just trading one problem for another.

> >

> > Barb

> >

> >

> >

> > Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> > In vitro killing of Candida albicans by fatty acids and

> > monoglycerides.

> >

> > Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

> >

> > Institute of Biology, University of Iceland, Reykjavik, Iceland.

> > gudmunb@h...

> >

> > The susceptibility of Candida albicans to several fatty acids and

> > their 1-monoglycerides was tested with a short inactivation time,

> and

> > ultrathin sections were studied by transmission electron

> microscopy

> > (TEM) after treatment with capric acid. The results show that

> capric

> > acid, a 10-carbon saturated fatty acid, causes the fastest and

> most

> > effective killing of all three strains of C. albicans tested,

> leaving

> > the cytoplasm disorganized and shrunken because of a disrupted or

> > disintegrated plasma membrane. Lauric acid, a 12-carbon saturated

> > fatty acid, was the most active at lower concentrations and after

> a

> > longer incubation time.

> >

> > PMID: 11600381 [PubMed - indexed for MEDLINE]

> >

> ____________________________________________________________________

> >

> > Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> > In vitro inactivation of Chlamydia trachomatis by fatty acids and

> > monoglycerides.

> >

> > Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O, Thormar

H.

> >

> > Institute of Biology, University of Iceland, Reykjavik, Iceland.

> >

> > The antichlamydial effects of several fatty acids and

> monoglycerides

> > were studied by incubating Chlamydia trachomatis bacteria with

> equal

> > volumes of lipid solutions for 10 min and measuring the reduction

> in

> > infectivity titer compared with that in a control solution

without

> > lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin

(12:0),

> > myristic acid (14:0), palmitoleic acid (16:1), monopalmitolein

> > (16:1), oleic acid (18:1), and monoolein (18:1) at concentrations

> of

> > 20 mM (final concentration, 10 mM) had negligible effects on C.

> > trachomatis. In contrast, lauric acid (12:0), capric acid (10:0),

> and

> > monocaprin (10:0) caused a greater than 10,000-fold (>4-log10)

> > reduction in the infectivity titer. When the fatty acids and

> > monoglycerides were further compared at lower concentrations and

> with

> > shorter exposure times, lauric acid was more active than capric

> acid

> > and monocaprin was the most active, causing a greater than 100,

> 000-

> > fold (>5-log10) inactivation of C. trachomatis at a concentration

> of

> > 5 mM for 5 min. The high levels of activity of capric and lauric

> > acids and particularly that of monocaprin are notable and suggest

> > that these lipids have specific antichlamydial effects. The mode

> of

> > action of monocaprin was further studied by removal of the lipid

> by

> > centrifugation before inoculation of Chlamydia onto host cells

and

> by

> > electron microscopy. The results indicate that the bacteria are

> > killed by the lipid, possibly by disrupting the membrane(s) of

the

> > elementary bodies. A 50% effective concentration of 30

> microgram/ml

> > was found by incubation of Chlamydia with monocaprin for 2 h. The

> > rapid inactivation of large numbers of C. trachomatis organisms

by

> > monocaprin suggests that it may be useful as a microbicidal agent

> for

> > the prevention of the sexual transmission of C. trachomatis.

> >

> > PMID: 9736551 [PubMed - indexed for MEDLINE]

> >

> >

> > Or this page in PUBMED

> >

> > http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> > db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

> >

>

[Guest guest]

Yeah, yeah, yeah. I know. :-) I'm actually pro-fat, but I also have

a very specific way of losing weight and unfortunately, the fat

doesn't fit. :-) It's only a short term thing. :-)

> > >

> > > Hey you guys.........

> > >

> > > Don't forget that there are natural agents that are

excellent

> > > at killing yeast (and they don't build in resistance to the

> > microbe).

> > >

> > > On my original Candida Panel (LabCorp) I had a 2.5 IgA titer

> > > (< 0.8 is normal) and IgM and IgG were psuhing the normal

limits.

> > > I didn't use drugs to grt rids of it - I wanted to try enzymes

or

> > > natural products first (becuase I didn't want to create a

> > > superinfection of resistant mutants).

> > >

> > > I used Nystatin, Candex and Coconut oil(which is like 50%

> > Lauric

> > > Acid). I was using lauricidin previous to coconut oil- but

> > swicthed

> > > to the oil because I suspect it metabolizes easier. And the

same

> > oil

> > > you ingest, you can put on your skin.

> > >

> > > It took over 8 months for my titer dropped to normal- but

that

> > > might have been a reflection of how long it takes the titer to

> > drop.

> > >

> > > The other thing to remember about long term abx is frequent

> sore

> > > throats: The last I looked at the stastics of people on long

> term

> > > abx therapy for acne- showed those people getting sore thraots

> due

> > to

> > > the imbalance of flora in the throat.

> > >

> > > So I think you HAVE to address fungal issues while on abx - or

> > your

> > > just trading one problem for another.

> > >

> > > Barb

> > >

> > >

> > >

> > > Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> > > In vitro killing of Candida albicans by fatty acids and

> > > monoglycerides.

> > >

> > > Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

> > >

> > > Institute of Biology, University of Iceland, Reykjavik,

Iceland.

> > > gudmunb@h...

> > >

> > > The susceptibility of Candida albicans to several fatty acids

and

> > > their 1-monoglycerides was tested with a short inactivation

time,

> > and

> > > ultrathin sections were studied by transmission electron

> > microscopy

> > > (TEM) after treatment with capric acid. The results show that

> > capric

> > > acid, a 10-carbon saturated fatty acid, causes the fastest and

> > most

> > > effective killing of all three strains of C. albicans tested,

> > leaving

> > > the cytoplasm disorganized and shrunken because of a disrupted

or

> > > disintegrated plasma membrane. Lauric acid, a 12-carbon

saturated

> > > fatty acid, was the most active at lower concentrations and

after

> > a

> > > longer incubation time.

> > >

> > > PMID: 11600381 [PubMed - indexed for MEDLINE]

> > >

> >

____________________________________________________________________

> > >

> > > Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> > > In vitro inactivation of Chlamydia trachomatis by fatty acids

and

> > > monoglycerides.

> > >

> > > Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O,

Thormar

> H.

> > >

> > > Institute of Biology, University of Iceland, Reykjavik,

Iceland.

> > >

> > > The antichlamydial effects of several fatty acids and

> > monoglycerides

> > > were studied by incubating Chlamydia trachomatis bacteria with

> > equal

> > > volumes of lipid solutions for 10 min and measuring the

reduction

> > in

> > > infectivity titer compared with that in a control solution

> without

> > > lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin

> (12:0),

> > > myristic acid (14:0), palmitoleic acid (16:1), monopalmitolein

> > > (16:1), oleic acid (18:1), and monoolein (18:1) at

concentrations

> > of

> > > 20 mM (final concentration, 10 mM) had negligible effects on

C.

> > > trachomatis. In contrast, lauric acid (12:0), capric acid

(10:0),

> > and

> > > monocaprin (10:0) caused a greater than 10,000-fold (>4-log10)

> > > reduction in the infectivity titer. When the fatty acids and

> > > monoglycerides were further compared at lower concentrations

and

> > with

> > > shorter exposure times, lauric acid was more active than

capric

> > acid

> > > and monocaprin was the most active, causing a greater than

100,

> > 000-

> > > fold (>5-log10) inactivation of C. trachomatis at a

concentration

> > of

> > > 5 mM for 5 min. The high levels of activity of capric and

lauric

> > > acids and particularly that of monocaprin are notable and

suggest

> > > that these lipids have specific antichlamydial effects. The

mode

> > of

> > > action of monocaprin was further studied by removal of the

lipid

> > by

> > > centrifugation before inoculation of Chlamydia onto host cells

> and

> > by

> > > electron microscopy. The results indicate that the bacteria

are

> > > killed by the lipid, possibly by disrupting the membrane(s) of

> the

> > > elementary bodies. A 50% effective concentration of 30

> > microgram/ml

> > > was found by incubation of Chlamydia with monocaprin for 2 h.

The

> > > rapid inactivation of large numbers of C. trachomatis

organisms

> by

> > > monocaprin suggests that it may be useful as a microbicidal

agent

> > for

> > > the prevention of the sexual transmission of C. trachomatis.

> > >

> > > PMID: 9736551 [PubMed - indexed for MEDLINE]

> > >

> > >

> > > Or this page in PUBMED

> > >

> > > http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> > > db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

> > >

> >

>

[Guest guest]

Thanks Barb - you and I are often similar. I am a WI girl and love

my dairy fats and cow.

I have Spectrum Naturals - I will check and see if it is cold

pressed and also the info page you suggest on TT. Thanks.

If you eat it on toast, I am guessing a couple teaspoons? I can do a

tablespoon of good butter on toast, easy.

Cheers and thanks,

*S*

> > > >

> > > > Hey you guys.........

> > > >

> > > > Don't forget that there are natural agents that are

excellent

> > > > at killing yeast (and they don't build in resistance to the

> > > microbe).

> > > >

> > > > On my original Candida Panel (LabCorp) I had a 2.5 IgA

titer

> > > > (< 0.8 is normal) and IgM and IgG were psuhing the normal

> limits.

> > > > I didn't use drugs to grt rids of it - I wanted to try

enzymes

> > or

> > > > natural products first (becuase I didn't want to create a

> > > > superinfection of resistant mutants).

> > > >

> > > > I used Nystatin, Candex and Coconut oil(which is like 50%

> > > Lauric

> > > > Acid). I was using lauricidin previous to coconut oil- but

> > > swicthed

> > > > to the oil because I suspect it metabolizes easier. And the

> same

> > > oil

> > > > you ingest, you can put on your skin.

> > > >

> > > > It took over 8 months for my titer dropped to normal- but

> that

> > > > might have been a reflection of how long it takes the titer

to

> > > drop.

> > > >

> > > > The other thing to remember about long term abx is

frequent

> > sore

> > > > throats: The last I looked at the stastics of people on

long

> > term

> > > > abx therapy for acne- showed those people getting sore

thraots

> > due

> > > to

> > > > the imbalance of flora in the throat.

> > > >

> > > > So I think you HAVE to address fungal issues while on abx -

or

> > > your

> > > > just trading one problem for another.

> > > >

> > > > Barb

> > > >

> > > >

> > > >

> > > > Antimicrob Agents Chemother. 2001 Nov;45(11):3209-12.

> > > > In vitro killing of Candida albicans by fatty acids and

> > > > monoglycerides.

> > > >

> > > > Bergsson G, Arnfinnsson J, Steingrimsson O, Thormar H.

> > > >

> > > > Institute of Biology, University of Iceland, Reykjavik,

> Iceland.

> > > > gudmunb@h...

> > > >

> > > > The susceptibility of Candida albicans to several fatty

acids

> > and

> > > > their 1-monoglycerides was tested with a short inactivation

> > time,

> > > and

> > > > ultrathin sections were studied by transmission electron

> > > microscopy

> > > > (TEM) after treatment with capric acid. The results show

that

> > > capric

> > > > acid, a 10-carbon saturated fatty acid, causes the fastest

and

> > > most

> > > > effective killing of all three strains of C. albicans

tested,

> > > leaving

> > > > the cytoplasm disorganized and shrunken because of a

disrupted

> > or

> > > > disintegrated plasma membrane. Lauric acid, a 12-carbon

> > saturated

> > > > fatty acid, was the most active at lower concentrations and

> > after

> > > a

> > > > longer incubation time.

> > > >

> > > > PMID: 11600381 [PubMed - indexed for MEDLINE]

> > > >

> > >

> >

____________________________________________________________________

> > > >

> > > > Antimicrob Agents Chemother. 1998 Sep;42(9):2290-4.

> > > > In vitro inactivation of Chlamydia trachomatis by fatty

acids

> > and

> > > > monoglycerides.

> > > >

> > > > Bergsson G, Arnfinnsson J, Karlsson SM, Steingrimsson O,

> Thormar

> > H.

> > > >

> > > > Institute of Biology, University of Iceland, Reykjavik,

Iceland.

> > > >

> > > > The antichlamydial effects of several fatty acids and

> > > monoglycerides

> > > > were studied by incubating Chlamydia trachomatis bacteria

with

> > > equal

> > > > volumes of lipid solutions for 10 min and measuring the

> > reduction

> > > in

> > > > infectivity titer compared with that in a control solution

> > without

> > > > lipid. Caprylic acid (8:0), monocaprylin (8:0), monolaurin

> > (12:0),

> > > > myristic acid (14:0), palmitoleic acid (16:1),

monopalmitolein

> > > > (16:1), oleic acid (18:1), and monoolein (18:1) at

> > concentrations

> > > of

> > > > 20 mM (final concentration, 10 mM) had negligible effects on

C.

> > > > trachomatis. In contrast, lauric acid (12:0), capric acid

> > (10:0),

> > > and

> > > > monocaprin (10:0) caused a greater than 10,000-fold (>4-

log10)

> > > > reduction in the infectivity titer. When the fatty acids and

> > > > monoglycerides were further compared at lower concentrations

> and

> > > with

> > > > shorter exposure times, lauric acid was more active than

capric

> > > acid

> > > > and monocaprin was the most active, causing a greater than

100,

> > > 000-

> > > > fold (>5-log10) inactivation of C. trachomatis at a

> > concentration

> > > of

> > > > 5 mM for 5 min. The high levels of activity of capric and

> lauric

> > > > acids and particularly that of monocaprin are notable and

> > suggest

> > > > that these lipids have specific antichlamydial effects. The

> mode

> > > of

> > > > action of monocaprin was further studied by removal of the

> lipid

> > > by

> > > > centrifugation before inoculation of Chlamydia onto host

cells

> > and

> > > by

> > > > electron microscopy. The results indicate that the bacteria

are

> > > > killed by the lipid, possibly by disrupting the membrane(s)

of

> > the

> > > > elementary bodies. A 50% effective concentration of 30

> > > microgram/ml

> > > > was found by incubation of Chlamydia with monocaprin for 2

h.

> > The

> > > > rapid inactivation of large numbers of C. trachomatis

organisms

> > by

> > > > monocaprin suggests that it may be useful as a microbicidal

> > agent

> > > for

> > > > the prevention of the sexual transmission of C. trachomatis.

> > > >

> > > > PMID: 9736551 [PubMed - indexed for MEDLINE]

> > > >

> > > >

> > > > Or this page in PUBMED

> > > >

> > > > http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?

> > > > db=pubmed & cmd=Display & dopt=pubmed_pubmed & from_uid=11600381

> > > >

> > >

> >

>

[Guest guest]

Hi S.,

> My gut symptoms have been much better since starting to consume

> coconut oil daily.

Do you use a measured amount or just tuck it casually into your food?

Or since it's warmer on the west coast maybe you can just slosh it on?

Sue ,

Upstate New York

[Guest guest]

Interesting, Nelly, as a child I had almost zero sugar in my diet.

My father was diabetic so my mother cooked for Dad, and that

was " meat, two vegetables and one " starch " " . I hated sweets. For

birthday cake it was angel food cake or nothin'. It wasn't until a

year or two ago that my sweet tooth kicked in and I think it was

because it was my body's way to try to cope with the fatigue.

>

> ,

>

> Are you saying we, in the western world are sick/not coping with

this or that because of sugar consumptiom by the individual?

>

> If so you'll have to think again because I hardly ever ate sugar,

or anything sweet, I didn't even like jams and/or chocolate as a

kid. Never ate cereals, never ate lollies, ate unsweetened yogurt

and ALL the right things. Yet I had all these weird repeated extra

febrile bouts with vomiting, massive headaches, diarrhea, pains in

legs etc + catching all kinds of VIRAL respiratory infections which

develop into bronchitis etc etc.

>

> My guess is that bugs do cause major chronic illness, and that if

I had done any of the things that other kids, and other adults did,

I would be dead (I never drank any alcohol, not even wine or beer,

never smoked anything, and I don't even eat any flour).So I agree

that you can kill yourself with bad diet but good, EXTRA good diet

won't insure good health

>

> Nelly

> ----- Original Message -----

[Guest guest]

Hiya Sue,

My delivery system of choice is toasted yeast-free spelt bread,

which has a subtle nutty flavor and despite being pretty sturdy is

very easy to digest.

I spread about 3/4 tablespoon per slice, the toast can take it

without getting soggy and it melts into it deliciously.

*Looks at kitchen, decides to demonstrate.*

Back.

First meal of the day is a smoothie with about half a cup of whole

coconut milk (not reduced fat), yogurt, half a banana, and a quarter

cup or so of fresh blueberries. I read on the FAQ page for Barb's

brand that people use the oil itself in smoothies, so I'm going to

try tossing in another tablespoon there.

You can make outrageously good baked potatoes by rubbing coconut oil

into the skin and then splitting them just enough to wedge in a bit

more plus a whole clove of garlic.

When we have fish, it's usually lightly pan-fried over low heat in

coconut oil and curry or basil with a bit of coconut milk added,

kind of Thai-style.

>

> Hi S.,

>

> > My gut symptoms have been much better since starting to consume

> > coconut oil daily.

>

> Do you use a measured amount or just tuck it casually into your

food?

> Or since it's warmer on the west coast maybe you can just slosh it

on?

>

> Sue ,

> Upstate New York

>

[Guest guest]

I just grabbed coconut oil today. I am a serious culinary philistine

so its too sophisticated for me to put on food, I therefore melted

some in the microwave in some water and threw in a pat of butter and a

tablespoon of fish oil to make a cocktail which I call the Barb Peck.

Just kidding - Im not sure Barb digs vitamin D containing oils. Also

Im not sure the fatty acids like to be microwaved, so tomorrow I will

probably not nuke em like that.

If nothing else it should help get the doctors off my back about being

underweight.

People with cardiovascular disease potential might want to consider

the high saturated fat content of cononut oil (but then they might

also want to consider that cardiovascular disease might possibly be

contributed to by a microbe sensitive to coconut oil; personally I

wouldnt know).

[Guest guest]

Hah!- I really don't know what microwaves will do to FFAs..

Oh god Gag me if you mixed it with water and fish oil- You are hard

core if you can swallow that mixture!!!!!!.

As far as D goes

I'm sure I get plenty of vit D as I'm outside alot, and I eat eggs

(we have chickens).

When it MIGHT have helped me to avoid D - it was the late 70's and

the nameless protocol hadn't been born yet, becuase I was very sun

sensitive during alot of those years.. but that was then - and this

is now. ( I would have run that D deprivation experiment on my self

then if I'd of know about it).

Barb

>

> I just grabbed coconut oil today. I am a serious culinary

philistine

> so its too sophisticated for me to put on food, I therefore melted

> some in the microwave in some water and threw in a pat of butter

and a

> tablespoon of fish oil to make a cocktail which I call the Barb

Peck.

>

> Just kidding - Im not sure Barb digs vitamin D containing oils.

Also

> Im not sure the fatty acids like to be microwaved, so tomorrow I

will

> probably not nuke em like that.

>

> If nothing else it should help get the doctors off my back about

being

> underweight.

>

> People with cardiovascular disease potential might want to consider

> the high saturated fat content of cononut oil (but then they might

> also want to consider that cardiovascular disease might possibly be

> contributed to by a microbe sensitive to coconut oil; personally I

> wouldnt know).

>

[Guest guest]

This place has me hungry lately. I'm going to try those coconut

dunked garlic baked potatoes soon.

As an aside about chickens, I now get my organic eggs at the farmer's

market whenever I can as the big one is righ tnear my doc's office

and their egg yolks are so nice and orangey-deep yellow. As opposed

to these pale yellow imitation from horizon organic eggs at the

supermarket. so the guy told me their chickens eat not only high

qulaity organic feed but whatever they want outside. So the organic

supermarket eggs have low quality organic feed and they're stlil

cooped up inside.

> >

> > I just grabbed coconut oil today. I am a serious culinary

> philistine

> > so its too sophisticated for me to put on food, I therefore

melted

> > some in the microwave in some water and threw in a pat of butter

> and a

> > tablespoon of fish oil to make a cocktail which I call the Barb

> Peck.

> >

> > Just kidding - Im not sure Barb digs vitamin D containing oils.

> Also

> > Im not sure the fatty acids like to be microwaved, so tomorrow I

> will

> > probably not nuke em like that.

> >

> > If nothing else it should help get the doctors off my back about

> being

> > underweight.

> >

> > People with cardiovascular disease potential might want to

consider

> > the high saturated fat content of cononut oil (but then they

might

> > also want to consider that cardiovascular disease might possibly

be

> > contributed to by a microbe sensitive to coconut oil; personally

I

> > wouldnt know).

> >

>

[Guest guest]

Barb

You'd sign a petition for the unnamed protocol to get a noble peace

prize wouldn't you?

> >

> > I just grabbed coconut oil today. I am a serious culinary

> philistine

> > so its too sophisticated for me to put on food, I therefore

melted

> > some in the microwave in some water and threw in a pat of butter

> and a

> > tablespoon of fish oil to make a cocktail which I call the Barb

> Peck.

> >

> > Just kidding - Im not sure Barb digs vitamin D containing oils.

> Also

> > Im not sure the fatty acids like to be microwaved, so tomorrow I

> will

> > probably not nuke em like that.

> >

> > If nothing else it should help get the doctors off my back about

> being

> > underweight.

> >

> > People with cardiovascular disease potential might want to

consider

> > the high saturated fat content of cononut oil (but then they

might

> > also want to consider that cardiovascular disease might possibly

be

> > contributed to by a microbe sensitive to coconut oil; personally

I

> > wouldnt know).

> >

>

[Guest guest]

>

> Hah!- I really don't know what microwaves will do to FFAs..

> Oh god Gag me if you mixed it with water and fish oil- You are hard

> core if you can swallow that mixture!!!!!!.

Fish oil is not an appealing concept (I cant even bring myself to eat

fish) but for me it really has no taste.

I just ate some coconut oil in its semisolid state. Not too bad. Those

fatty acids are probably some of the more stable organic molecules you

can find, but I still hate to microwave em without having data.