Re: Immune modulators...Ken F.

[Guest guest]

Hi Ken,

Please feel free to post any actual references either you or

possess, and I'll reply to them.

Meanwhile, if I understand you correctly, you're conceding that ARBs

impair macrophage migratory capability and phagocytic activity, but

claiming that some other mechanism will rid the body of the bugs and

their toxins.

Can you tell me what that other mechanism would be?

Thanks,

S.

> S. said:

> " ABSs do modulate the immune system, but not in a way that would

shut

> down cytokine cascades, which are mainly triggered by tlrs, not AII

> type 1 receptors, which are primarily involved in getting

> macrophages to infection sites and putting them to work cleaning up

> the mess. "

>

> AT1 receptors not involved in inflammation? Sorry , not buying

> that one. Check s paper and references, the science is

> against you on this one.

>

> TLRs can also lead to inflammation, but this is because they get

> triggered by bacterial LPS, i.e: a direct response to bacteria.

>

> The overall result is that if you block the macrophage AT1

> receptors, you reduce the inflammation while still allowing an IS

> response to bacterial presence through the TLR. Yes, you reduce

the

> IS ability to respond through the AT1 pathway, but that has not

been

> shown to be a major response pathway to either bacteriological or

> viral infection.

>

> If your reading of these studies is correct, then I would expect a

> large percentage of the hundreds of people on high doses of

Benicar

> to be dropping due to out of control staph (and other)

infections.

> Yet this has not been the case.

>

> Ken