Re: Pitocin & mercury: Could someone please verify this?

[Guest guest]

,

One of the mechanisms that might explain this assoication is the change

produced by TNF which Dr. Waring found experimentally. This cytokine,

released during infection (perhaps chorionic infection, too, although she

didn't measure it there), reroutes the sulfur chemistry, apparently

shutting down sulfoxidation, which is the route to make sulfate. It looks

like one reason for this is to put more emphasis on metallothionein as a

route of supplying cysteine. What this switch does, though, is make it

hard to make sulfate locally. Sulfate is needed to flip a switch on the

hormone cholecystokinin which allows this hormone to tell oxytocin-making

neurons to make oxytocin.

The way I learned about this is the hard way. I did have labor that

petered out half-way, and was induced by pitocin, but they removed the drip

after my daughter was born. However, this was premature, because I needed

the oxytocin to make my uterus clamp down, and it didn't. I started to

hemorrhage, and we had us a medical emergency, so they started beating on

my stomach to get the uterus to clamp down, leaving me black and

blue. After that, another oxytocin-requiring process failed...my milk

coming in. Using one of those big milking machines, it still took two

weeks before I was making adequate milk, but then it came in

gang-busters. I think it took that long for my sulfur chemistry to recover.

You may forward this to the mom if she is not on this list.

At 05:26 AM 11/2/2004 -0700, you wrote:f

>I was asked a similar question recently. Here is my reply:

>

>To a parent activist,

>

>I don't know if pitocin contains thimerosal. I think I would have

>learned that by now if it were so, tho' genetically stubborn <g>, I'm

>not too late to learn whether or not Pitocin contains thimerosal and

>would appreciate authoritative cites. A medline search {pitocin AND

>thimerosal}generated only one citation (1), and that citation appears to

>be based upon a study of how thimerosal interfered with oxytocin and

>contractility.

>

>Pitocin is often credited/blamed by autism moms, but in each specific

>case other factors related to the decision to use pitocin need to be

>considered. For instance, chorioamnionitis (which can be silent, ie,

>unnoticed) can exacerbate the weaking of muscles used in parturition and

>can diminish the sensitivity of oxytocin receptors in those muscles.

>Thus, a mother beginning labor in the context of recent or current

>chorioamnionitis (even if mild, silent) may not develop or sustain

>enough push for a successful delivery. As that situation develops, many

>gynecologists will realize that additional help is needed (eg, C-section

>or pitocin).

>

>The coalescing of these several processes (including pitocin use) - in

>many and perhaps most cases - may be the tip of an iceberg:

>chorioamnitotis and/or intraamniotic infection. Recent studies document

>that chorioamnionitis can cause a fetal cytokines reaction and that

>those cytokines can affect vascular development.

>

>A mother who had pitocin use ought scrutinize her medical records for

>that pregnancy and that delivery. Did she have vaginal bleeding,

>intra-amniotic infection, or chorioamnionitis (though it can be silent)?

>How long was her labor? Does she remember becoming exhausted during

>labor? The last question is very subjective. Tired of the pain, surely.

>Tired, perhaps, maybe even probably. But exhausted in regard to her

>ability to " push " adequately?

>

>The last birth at which I was a midwife's assistant involved a

>vegetarian mom-to-be. She tried and tried, pushed and pushed - but as

>that went on too long (in the experienced midwife's opinion - more than

>400 births midwifed), the midwife said, No more, You're going to the

>hospital. The Mom was disappointed but cooperated. 'Twas obvious she was

>exhausted after so many hours of labor. I didn't accompany into the

>hospital. Fortunately, the doc allowed the midwife to attend the

>delivery. The son is now a young man, healthy, tall, strong. The

>anecdote provides an example of prolonged labor and the exhaustion of

>the Mom-to-be, including the exhaustion of the muscles needed for delivery.

>

>Several citations from my Physicians' Training presentation are hereinbelow.

>

>

>

>1: Biochem Biophys Res Commun. 1995 Jun 6;211(1):1-6.

>

>The effects of thimerosal, a sulfhydryl reagent, on phasic myometrial

>contractions.

>

>pe M.

>

>Department of Obstetrics & Gynecology, University of Chicago 60637, USA.

>

>Thimerosal inhibits calcium uptake and IP3-induced calcium release from

>IP3-sensitive endoplasmic reticulum; this study sought to evaluate the

>effects

>of thimerosal on agonist-stimulated phasic myometrial contractions.

>Thimerosal

>was found to significantly inhibit phasic contractions stimulated by

>oxytocin,

>aluminum fluoride, potassium chloride, ionomycin, and Bay K 8644. These

>observations provide support for the hypothesis that calcium uptake and

>IP3-induced calcium release are important events during agonist-stimulated

>phasic myometrial contractions.

>

>PMID: 7539999 [PubMed - indexed for MEDLINE]

>

>

>

>

> > Do you have any information on pitocin/mercury. As I understand it,

> > pitocin has mercury...I talk to so many mom who think their baby's

> > autism has nothing to do with mercury because... " they had a problem

> > immediately after birth. " I would think that could be attributed to

> > many things but pitocin seems to be a likely culprit..

> >

> > Your opinion and any info would be appreciated.

> >

> > Thanks

> > [A parent]

> >

>

>Chorioamnionitis, fetal, placental

>

>135. Abruptio placentae and chorioamnionitis-microbiological and

>histologic correlation. Acta Obstet Gynecol Scand. 1999 May;78(5):363-6

>PMID 10326877

> " Conclusion: The incidence of silent chorioamnionitis

>(placental membrane culture positivity) is higher in the abruptio

>placentae. "

>

>136. Clinical chorioamnionitis, elevated cytokines, and brain injury in

>term infants.

>Pediatrics. 2002 Oct;110(4):673-80 PMID 12359779

>

>137. Fetal endothelial cells express vascular cell adhesion molecule in

>the setting of chorioamnionitis. Am J Reprod Immunol 2000 43(5):259-63

>PMID 12359779

>

>138. Chorioamnionitis and uterine function. Obstet Gynecol 2000;

>95:909-12 PMID: 10831982

> " Several small studies have suggested that chorioamnionitis

>has an inhibitory effect upon labor, characterized by decreased uterine

>contractility, decreased sensitivity to oxytocin stimulation, and

>subnormal cervical dilation. " [3 cites]

>

>139. Effect of amniotic fluid bacteria on the course of labor in

>nulliparous women at term

>Obstet Gynecol 68:587-592 1986 PMID: 3763067

>[Nulliparous - no prior live births]

> " Patients with intraamniotic infection have an increased

>rate of cesarean delivery... These results support a causal relationship

>between high-virulence bacteria in the amniotic fluid and poor cervical

>dilation response to oxytocin... "

> " Friedman... studied nulliparous patients with 'amniotic

>infection syndrome' and found that 70.5% had labor dysfunction. More

>recent reports have confirmed this association and have also identified

>an increased frequency of cesarean delivery among these women. "

> " Koh et al... reported a 43% cesarean section rate in 140

>patients with clinical 'chorioamnionitis.' "

> " Two-thirds of the cesarean sections were performed because

>of poor progress in labor despite the use of oxytocin... "

>

>

>mom2deeda wrote:

>

> >Does anyone know if the infamous drug that is used to induce babies

> >everyday in this country (pitocin?)contains mercury?I have heard

> >this,but I don't know for sure.If so,babies are definately getting a

> >good dose of mercury before they are born.How terrible!Thanks in

> >advance.

> >

> >DeAnn,mom to ,4 yr.asd,pitocin induced delivery

> >

> >

> >

>

>

>