Guest guest Posted May 4, 2005 Report Share Posted May 4, 2005 Hey, thanks for that info... if anyones looking at the vid it might be good to also scope the last 1/4 (or more) of this lecture by Dr , which addresses this kinda stuff: http://www.lymediseaseaction.org.uk/conference/t_2004_7.htm Tansy, what I am confused about is this... if such a large proportion of blood cells are infected, isnt it a giant revolution in borreliosis pathogenesis? As it exists in the literature, borreliosis is a big mystery because electron microscopy, as well as light microscopy, usually find so very few borrelia in inflamed tissues, including within white cells that enter inflamed tissues. There arent " enough " borrelia. (E.g., Nanagara 1995 for EM, Steeres early studies for light microscopy) Thats why some investigators favor an autoimmune pathogenesis - and why Ive been focused on the possibility of very very small cytoplasmic variant forms like those photographed by the Wirostko/ group, because such forms are so subtle they can be missed even by electron microscopy (and are totally invisible to the light microscope). But if blood cells are crawling with borrelia, things are very different - different from anything thats been published in lit. Except for Lida Mattmans book, which touches on this picture, but not with much elaboration. Thats why Im kinda surprised theres not more discussion about it? Do you know if the big, roundish, red-cell-sized borrelial forms are cleared from the blood when abx are used? If so, might that suggest that the part of the infection that can be abx-refractory is within blood cells? Wouldnt that suggest we should trying to design drugs to penetrate those cells? Do you know why Dr Krouns talk found here deal so much with the concept of relapse? I ask because it seems like the majority of people with lyme cant get fully well in the first place: http://lymerick.ulmarweb.dk/York2003/York2003.ppt > > Hey, who has watched this vid by Drs. & Kroun - link found > at > > in the spirochetes section of the homepage of LymeRICK? > > http://lymerick.ulmarweb.dk/ > > > > Im surprised there hasnt been more shock about this. > > > > I think this may be blood left out on the slide for many hours. If > > so it would be different from what youd see on a straight smear, > but > > given the ~12h ideal generation time of Bb, it would not be too > much > > more biomass than exists in circulating blood. What do you guys > > think about the idea that the red cell may be the prime sanctuary > of > > Bb - an idea I saw in one place once. That source said the red cell > > was poorly penetrated by abx. As for the immunological policing of > > the red cell, it may be poorly understood - if not I dont quite > > understand it. Unlike those of mice, ours express no MHC-I. > > > > Someone should use fluorescent Ab to make video like this and show > > it to the world. > > > > ====================================== > > Andy , Bolton UK – a collection of several videoclips of > > moving filamentous and granular structures filmed in blood of > > chronically ill UK patients diagnosed with CFS and/or chronic Lyme > > disease (52 Mb) – A MUST SEE ! Quote Link to comment Share on other sites More sharing options...
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