multiple sclerosis

[Guest guest]

I recently read Steiners 1954 paper, in which he again used

silver impregnations to locate spirochetes intracellularly and

extracellularly at the periphery of fresh MS lesions in 4 patients. In

this one he tells about experiments he made to rule out certain

artifacts. Again, many more " fragments " were found than actual

spirochetes, and Steiner considers the fragments to probably be

inviable spirochetal degeneration products (actually that is very

unlikely).

It seems there are multiple animal transmission studies supporting the

role of this spirochete, tho I have not examined them, and dont know

if they were controlled, etc.

My impetus for posting is the information at this site:

http://home.earthlink.net/~webmedic4u/ms.html

A paper reproduced there from J Med Hypothesis addresses the dying out

of the line of spirochete investigations of Steiner and others (many

others). If everything asserted in that paper is true, its a damn

shame. This morning my mom walked 7 miles with some friends in a Walk

for MS fundraiser, the bulk of the money from which will I guess go

towards study of the mouse " model " , experimental autoimmune

encephalitis. Replicating (and beyond) the work of these 20th-century

investigators could easily be payed for by the MS society(ies), let

alone the NIH. Doing so is an immediate imperative, unless someone can

explain whats wrong with the work of these several early-20th-century

groups. Smart patients outside (and inside) the money-allocating

eschelon of the MS society(ies) need to read these papers and start

clamoring for their moneys worth.

The same page has a precis of a fascinating paper dealing with what is

said to be the first ever histological charecterization of a new

(day-old) MS lesion, which discovered apoptosis of myelin-producing

cells and microglial activation in the *absence* of infiltrating

leucocytes. Searching in data collections the authors were able to

uncover a few records of similar lesions in very early MS, suggesting

this may indeed the way all lesions evolve early on. Hopefully this

result, which is dissonant with the autoimmune theory of MS (*so*

often casually presented as fact even in learned literature), wont be

ignored. I will be reporting on full text of this ASAP. Abstract at:

http://www3.interscience.wiley.com/cgi-bin/abstract/107629227/ABSTRACT