RE: Diabetes Diet

[Guest guest]

Oops. Sorry.

Forgot to thank Wanita for posting those articles. We have to meet the

enemy before we can beat them.

Judith Alta

-----Original Message-----

" Any scientist who can figure out why Type 2 diabetics are insulin resistant

will probably be a candidate for a Nobel Prize. "

Dr. Atkins, Dr. Schwarzbein and the Drs. Eades, among others, have the

answer, but I'm not aware that any of them have received a Nobel Prize.

Anyone see anything wrong with these two articles?

Nowhere is it mentioned that our low nutrient, fake food, junk food diet is

to blame for diabetes.

Nowhere is it mentioned that eating whole, non-commercially processed food

will help to prevent diabetes.

The only good part was the bit about exercise.

I'm sure I'm not the only one to have noticed that these types of articles

never, or almost never, point their fingers at the garbage the government is

pressuring everyone to eat. Sure wouldn't want to make their advertisers and

supporters unhappy.

Billions of dollars are made each year from diabetes treatments and

paraphernalia. The people who rake in that money sure don't want a " cure " or

prevention unless it's everyone taking prescription drugs from birth to

death

Judith Alta

[Guest guest]

Wanita-

IMO this should really read " that ability seems to cause insulin resistance

when cheap, low-nutritional-value starch- and sugar-based foods are plentiful " .

>Unfortunately,

>that

>ability seems to cause insulin resistance when food is plentiful.

-

[Guest guest]

>Evolutionary biologists suspect that a predisposition toward diabetes

>developed

>among certain ethnic groups—such as Pima Indians or Americans of African

>descent—as a result of repeated, perhaps even recent, bouts of starvation.

>Those individuals who were better able to decrease energy expenditures

>during a

>famine survived and passed on the trait to their offspring.

Actually, I'd take it a step further. I'd say that Europeans adapted

to having food daily ... the " normal " state for humans was to

kill a big animal now and then, gorge, then sit around nibbling

on greens until the next big animal came along. Like lions

and most predators. If in fact we are from a line of meat

eaters, then the concept of constantly eating is a recent

development. (I'm exaggerating, but you get the idea).

Anyway, having dealt with one big animal lately, I just

can't see doing all that work daily! Even with a tribe of

helpers. Drying and fermenting probably came along later

in evolutionary history.

-- Heidi

[Guest guest]

Not to mention the food pyramid! When the midwives told me to " make sure I

ate 9-12 servings of whole grains " , my mouth dropped open. I couldn't

believe they were telling me that when I used to be diabetic - they diagnose

diabetes using blood sugar tests...when eating no cooked carbs(fruits,

veggies, raw honey only for carbs) my blood sugar remains in the normal

range for fasting and postprandial. A couple days eating even whole grain

products and I'm back to the danger borderline zone. Within a week, I'm full

blown diabetic. How has this been missed? It has been ignored to put money

into the pockets of the medical and drug companies. Makes me want to scream

when I look at the growing number of obese and diabetic children(not to

mention asthmatic) knowing it can all be prevented.

happy holidays

Michele

>From: " Judith Alta K " <jaltak@...>

>Reply-

>< >

>Subject: RE: Diabetes Diet

>Date: Fri, 19 Dec 2003 10:36:29 -0500

>

> " Any scientist who can figure out why Type 2 diabetics are insulin

>resistant

>will probably be a candidate for a Nobel Prize. "

>

>Dr. Atkins, Dr. Schwarzbein and the Drs. Eades, among others, have the

>answer, but I'm not aware that any of them have received a Nobel Prize.

>

>Anyone see anything wrong with these two articles?

>

>Nowhere is it mentioned that our low nutrient, fake food, junk food diet is

>to blame for diabetes.

>

>Nowhere is it mentioned that eating whole, non-commercially processed food

>will help to prevent diabetes.

>

>The only good part was the bit about exercise.

>

>I'm sure I'm not the only one to have noticed that these types of articles

>never, or almost never, point their fingers at the garbage the government

>is

>pressuring everyone to eat. Sure wouldn't want to make their advertisers

>and

>supporters unhappy.

>

>Billions of dollars are made each year from diabetes treatments and

>paraphernalia. The people who rake in that money sure don't want a " cure "

>or

>prevention unless it's everyone taking prescription drugs from birth to

>death

>

>Judith Alta

>

>-----Original Message-----

>

>Heidi,

>

>Thanks for gluten articles! There are two articles l was referring to it

>seems. Excerpt of starvation paragraph here. Doesn't say starvation gene in

>second article. Leaves me with 3 square meals a day isn't good when you

>look at

>ability to survive starvation and ethnicities most prone in first article.

>

>Evolutionary biologists suspect that a predisposition toward diabetes

>developed

>among certain ethnic groups-such as Pima Indians or Americans of African

>descent-as a result of repeated, perhaps even recent, bouts of starvation.

>Those individuals who were better able to decrease energy expenditures

>during a

>famine survived and passed on the trait to their offspring. Unfortunately,

>that

>ability seems to cause insulin resistance when food is plentiful. In other

>words, says Dr. ielsz, director of the Center for Women's

>Health

>Research at New York University, " you come out into the world-at least the

>developed world-and there is no shortage of cheap, fast food. Your thrifty

>metabolism has prepared you for the wrong future. "

>

>Wanita

>

>Sunday, Nov. 30, 2003

>Why So Many of Us Are Getting Diabetes

>Never have doctors known so much about how to prevent or control this

>disease,

>yet the epidemic keeps on raging. How you can protect yourself

>By CHRISTINE GORMAN

>Hillary Carroll knew something was amiss. she had spent Memorial Day

>happily

>frolicking in her grandmother's swimming pool, but by that evening she was

>doubling over in pain every time she went to the bathroom. Her mother

>figured

>it was probably an infection and the next day took Hillary, then 10, to the

>pediatrician. Instead of getting a prescription for an antibiotic, however,

>the

>220-lb. youngster was immediately admitted to the hospital. Lab tests

>showed

>that she had something far more serious-Type 2 diabetes.

>Hillary is not the first overweight child to learn she has this form of

>diabetes, a chronic metabolic disorder that used to be called adult onset

>but

>was renamed in part because so many kids Hillary's age were getting it. As

>doctors have repeatedly warned, the U.S. is experiencing a diabetes

>epidemic.

>Some 18 million Americans suffer from one form or another, with 1.3 million

>new

>cases diagnosed last year-up from 878,000 in 1997. And although Type 2

>diabetes

>still tends to strike people in their fifth or sixth decade, more children

>are

>getting it, a fact of grave concern to health officials. Not only are these

>kids likely to face a lifetime of problems-including higher risks of

>blindness,

>heart disease and stroke-they are also a warning sign that something in our

>way

>of life has gone terribly wrong.

>And yet scientists in just the past decade have learned that the most

>devastating complications of diabetes-and in some cases the disease

>itself-are

>almost entirely preventable. There are better techniques for monitoring

>diabetes and more effective drugs for treating it, and a major study

>published

>last year shows that by making only modest changes in diet and exercise,

>people

>at high risk of Type 2 diabetes can stave off the disease for at least

>three

>years and perhaps a lot longer (more on Type 1 in just a bit).

>It's a puzzle. Never have physicians known so much about Type 2 diabetes

>and

>how to control it, yet the number of cases is expected to rise at an

>alarming

>rate. Epidemiologists predict that by 2025 the incidence in the U.S. will

>double. Annual treatment costs are projected to rise, from $132 billion to

>$192

>billion in 2020-not counting inflation. Hardest hit will be certain ethnic

>groups-including African Americans and Native Americans, Hispanics and

>Asians-that for complicated reasons are more prone to the disorder.

>To the dismay of health experts, diabetes is becoming a global problem. In

>the

>next couple of decades, the prevalence of diabetes is expected to triple in

>Africa, the eastern Mediterranean, the Middle East and Southeast Asia, to

>double in the western Pacific and to nearly double in Europe. With an

>estimated

>33 million cases, India has the most people with diabetes; China has 23

>million.

>Lots of doctors will tell you that the reason for the explosion is obvious:

>Americans are eating too much and exercising too little, and much of the

>world

>is following our bad example. There is no question that excessive weight

>increases your risk of becoming diabetic. But that explains only part of

>the

>problem. Diabetes has a strong genetic component, and scientists are

>beginning

>to suspect that certain evolutionary factors, as well as your mother's

>metabolic or nutritional status during pregnancy, may predispose you to

>develop

>diabetes. That may explain why Indians from the subcontinent often become

>diabetic after gaining just a few pounds and why the consequences of

>obesity

>seem far more devastating for Americans of African, as opposed to European,

>descent.

>A complex picture is emerging that is changing the way we think about what

>was

>already a complex disease. It turns out that patients are not as helpless

>against its ravages as was once thought, especially if they are warned at

>the

>disease's very earliest stages. Changes in lifestyle and diet can, in the

>vast

>majority of cases, make a big difference. The future for anyone with

>diabetes

>has never been brighter, provided he or she has access to the right

>treatments.

>But the consequences of inaction have never been more broadly devastating.

>This

>year more than 200,000 Americans with diabetes will die from its

>complications.

>

>What Is Diabetes?

>To understand the latest insights into the disease, it helps to know a

>little

>more about two key molecules-glucose and insulin-and the roles they play in

>the

>conditions doctors call Type 1 and Type 2 diabetes. We'll start with

>glucose,

>the sugar molecule that is a major source of fuel for the body. You can get

>your glucose levels tested at a doctor's office or at home with a device

>called

>a glucometer. What you're looking for is a reading measured in milligrams

>of

>glucose per deciliter of blood (or, on some glucometers, in millimoles per

>liter). Anyone whose glucose level before breakfast-the fasting level-is

>126

>mg/dL (7 mmol/L) or higher is considered diabetic. A normal fasting level

>runs

>anywhere from 65 mg/dL to just under 100 mg/dL (3.6 mmol/L to 5.6 mmol/L).

>Insulin is a hormone made by specialized cells in the pancreas, whose job

>is to

>push glucose out of the blood into various cells in the body.

>Whenever the amount of glucose in the blood starts to rise, which happens

>just

>about whenever you eat, the pancreas pumps out more insulin to keep sugar

>levels stable.

>Here's where the difference between Type 1 and Type 2 is clearest. Type 1

>diabetics have high glucose levels because their pancreas can no longer

>make

>insulin. By definition, Type 1 diabetics must eventually take insulin shots

>to

>get their diabetes under control. Type 2 diabetics can still make their own

>insulin, but their bodies don't respond as well to it-a situation called

>insulin resistance.

>Any scientist who can figure out why Type 2 diabetics are insulin resistant

>will probably be a candidate for a Nobel Prize. It's not a simple

>consequence

>of being overweight. Many obese people are not insulin resistant, and not

>everyone who is insulin resistant is overweight. Researchers at the Salk

>Institute in La Jolla, Calif., believe that at least part of the answer

>lies

>not in the pancreas but in the liver. In a study of mice published in the

>Nov.

>13 issue of Nature, scientists identified a protein that tells the liver to

>favor the metabolism of fat over that of glucose. The result is a buildup

>of

>glucose levels in the blood, a hallmark of insulin resistance.

>Evolutionary biologists suspect that a predisposition toward diabetes

>developed

>among certain ethnic groups-such as Pima Indians or Americans of African

>descent-as a result of repeated, perhaps even recent, bouts of starvation.

>Those individuals who were better able to decrease energy expenditures

>during a

>famine survived and passed on the trait to their offspring. Unfortunately,

>that

>ability seems to cause insulin resistance when food is plentiful. In other

>words, says Dr. ielsz, director of the Center for Women's

>Health

>Research at New York University, " you come out into the world-at least the

>developed world-and there is no shortage of cheap, fast food. Your thrifty

>metabolism has prepared you for the wrong future. "

>The Sugar Blues

>What's so bad about being insulin resistant and having too much glucose in

>your

>blood? For reasons that researchers are still trying to figure out, having

>diabetes greatly increases your risk of suffering a heart attack or a

>stroke. A

>man with diabetes appears to have the same risk of cardiovascular problems

>as a

>nondiabetic who has had a heart attack. A woman who develops diabetes loses

>the

>cardioprotective benefits of being female. And kids with Type 2 diabetes

>are

>more likely to develop heart disease in their 20s and 30s.

>The condition also damages small blood vessels throughout the

>body-particularly

>those in the eyes and kidneys. As many as 24,000 diabetics in the U.S.

>become

>blind each year, more than 100,000 require dialysis or kidney

>transplantation,

>and 82,000 need to have a toe, foot or leg amputated. Diabetics are twice

>as

>likely as nondiabetics to suffer from depression.

>It doesn't have to be this way. Back in 1993 doctors proved that Type 1

>diabetics could greatly reduce their risk of complications by intensively

>managing their glucose levels to keep them as close to normal as possible

>(using a glucometer to measure the level of sugar in a pinprick of blood

>and an

>insulin shot when necessary to bring the level down). Similar results have

>since been seen with Type 2 diabetics.

>But most Type 2 diabetics don't have to resort to insulin shots to manage

>their

>condition. Because the fundamental problem in Type 2 diabetes is insulin

>resistance-not the inability to produce insulin as in Type 1-other options

>are

>available. Your physician may first give you pills that can either

>sensitize

>your body to insulin's effects or help your body produce more of the

>hormone.

>But some of your best allies in this struggle are your muscles.

>Building them up and using them regularly in such pursuits as walking or

>dancing draw more glucose out of the bloodstream and increase insulin's

>efficiency. It also pays to avoid easily digested foods-like chips, nondiet

>soda and other junk food-which require large amounts of insulin to

>metabolize.

>Finally, losing a little weight usually makes insulin's job a lot easier.

>Donna Black Bradley, 52, of Los Angeles is living proof. Bradley was

>driving

>home from work one evening when she suddenly was unable to read the freeway

>signs. When her doctor diagnosed diabetes, she felt paralyzed. " Then I said

>O.K., I got something I got to do here, " Bradley says. " I got to change. "

>And

>change she did. The 5-ft. 7-in. mother and grandmother started eating

>better

>and working out on a treadmill several times a week. Her weight dropped

>from

>272 lbs. to 210 lbs., and her fasting glucose fell from 300 mg/dL to 103

>mg/dL.

> " It's amazing how your cravings diminish when you're eating the right food

>groups, " Bradley says. Her vision problems have disappeared, and her doctor

>believes she will no longer need to take insulin-sensitizing drugs if she

>can

>get her weight under 200 lbs.-something she's determined to do, both for

>herself and for her grandson Isaiah. Says she: " I want to be around for

>that

>2-year old. "

>Small Steps, Big Rewards

>The more scientists learn about diabetes and the complications it causes,

>the

>more they find themselves looking at the conditions that precede it. " The

>big

>question now is, When does the diabetes clock start ticking? " says Dr.

>

>Vinicor, director of the diabetes program at the Centers for Disease

>Control.

> " For eye or small-vessel disease, we think the clock starts ticking when

>blood

>sugar starts to go up. But for heart disease, we think the clock may start

>ticking much, much earlier, even before the first sign of blood sugar going

>up. "

>That's why researchers are focusing on a precursor condition called

>prediabetes, in which glucose levels are only slightly elevated but which

>may

>put at risk an additional 20 million Americans. In 2002 researchers

>published

>the results of the Diabetes Prevention Program (D.P.P.), one of the

>largest,

>most rigorous clinical trials ever conducted on the subject. More than

>3,000

>people from all over the U.S. participated. All were overweight, and blood

>tests indicated that their bodies were having trouble handling glucose,

>though

>none had yet developed diabetes. Half were from ethnic communities that are

>at

>higher risk. Participants in the D.P.P. were divided into three groups. One

>was

>given the diabetes drug metformin, one was given a dummy pill, and one was

>enrolled in a nutrition and exercise program with the goal for participants

>to

>lose 7% of their initial weight and to exercise 30 minutes a day, five

>times

>a

>week.

>The D.P.P. trial was halted a full year early " because the results were

>just so

>remarkable, " says Dr. , director of the Diabetes Center at

>Massachusetts General Hospital in Boston, who chaired the study. During the

>three years of the study, nearly 30% of the placebo group developed

>diabetes.

>For the metformin group the figure was 22%. The subjects who exercised

>moderately and lost weight had the lowest incidence of all-just 14%. (The

>first

>two groups were given diet and exercise information but did not lose as

>much

>weight as the last group.) The study is continuing to see if lifestyle

>changes

>can prevent diabetes for life or merely delay its occurrence. At the very

>least, the trial proves that the point of no return is not as early or as

>fixed

>as was once feared.

>Diabetes researchers have been so impressed by the D.P.P. results that many

>would like to lower the thresholds that determine who is prediabetic. In

>January the American Diabetes Association plans to officially recommend

>that

>physicians consider treating anyone with a fasting glucose of 100 mg/dL or

>higher, down from 110 mg/dL or higher in the previous guidelines. " If your

>fasting blood sugar is below 100, your chances of getting diabetes are

>quite

>low, " says Dr. Rizza, an endocrinologist at the Mayo Clinic in

>Rochester, Minn., and a vice president of the American Diabetes

>Association.

> " But if your fasting glucose is over 100, you have a 10% to 15% chance of

>getting diabetes in the next seven years. "

>You don't have to convince Tom Marinello, 51, of Carson, Calif., that

>prediabetes should be taken seriously. After his father-in-law died of

>complications from diabetes, Marinello, unaware that he was at risk,

>enrolled

>in the Diabetes Prevention Program at the University of California, Los

>Angeles, hoping to help others with the disease. He was surprised to learn

>that

>he was prediabetic. By paying closer attention to what he eats and by

>making

>sure to walk nearly every day, Marinello dropped 28 lbs. from his 260-lb.,

>6-ft. 1-in. frame, and his fasting-glucose level is down to 110 mg/dL. " I

>may

>still be at risk, but I'm not diabetic, " Marinello says. " I'm kind of proud

>of

>that. "

>Beyond Apples and Pears

>Is it possible to turn the clock back even further to find who is at

>greatest

>risk of developing prediabetes and lower their risk of future health

>problems?

>That turns out to be much more complicated.

>Cardiologists have long known that if you carry extra weight around your

>waist,

>which they liken to being shaped like an apple, you are at greater risk of

>heart disease. The other configuration, being shaped like a pear, with

>excess

>weight around the hips, doesn't eliminate your risk but seems to lessen it.

>Over the years it has become clear that apple-shaped folks have a certain

>kind

>of metabolism: they are more likely to be resistant to insulin, have high

>amounts of triglycerides (one of the fatty molecules you don't want too

>much of

>in your blood) and have low levels of hdl (the " good " cholesterol). They

>also

>tend to have high blood pressure.

>Coincidence? Probably not, which is why physicians have lumped all these

>symptoms together in one condition that they now call metabolic syndrome.

>They

>believe that anyone with metabolic syndrome is at much greater risk of

>developing not just heart disease but diabetes as well. They're not sure

>whether there is a primary trigger for metabolic syndrome-say, obesity or

>insulin resistance-or if several biological pathways are involved.

>Whatever the case, says Dr. Grundy, a leading expert on cholesterol

>who

>chaired the American Heart Association's first clinical conference on

>metabolic

>syndrome in September, " right now there's no single drug that can treat the

>whole metabolic syndrome. " Individual symptoms like high blood pressure

>still

>have to be treated separately. But your best bet for an overall solution is

>to

>eat better, lose weight and get more exercise.

>An Inflammatory Question

>As central as insulin resistance has become to understanding Type 2

>diabetes,

>scientists are starting to wonder whether another factor, the inflammatory

>response, may also play a key role. Inflammation is a complex biological

>process the immune system uses to limit the damage caused by various

>injuries.

>(Ever notice how a turned ankle swells or a sunburn feels warm to the

>touch?

>That's inflammation in action.) But when inflammation becomes chronic, it

>no

>longer limits damage. In fact, it starts to do harm to the body.

>Over the past five years, researchers have shown that inflammation is at

>least

>as important as high cholesterol levels in causing heart disease. (High

>levels

>in the blood of certain molecules, such as C-reactive protein (CRP),

>indicate a

>runaway inflammatory process and are better predictors of heart attacks

>than

>cholesterol.) Could the same be true for diabetes? " In 2001, when we

>published

>our first paper on inflammation and diabetes, everybody thought we were

>just

>wrong, " recalls Dr. Ridker, a cardiologist at Brigham & Women's

>Hospital

>in Boston. " Now there are half a dozen studies confirming that if you

>measure

>markers of inflammation, and CRP in particular, you can do a good job of

>predicting who's going to get diabetes. "

>If these results are confirmed and inflammation turns out to be as

>important as

>insulin resistance in triggering diabetes, then it should become much

>easier to

>identify incipient problems in metabolism before they get out of control.

>Why?

>Because inflammation is much easier to measure than insulin resistance; all

>that is required is a single blood test. (Abnormal glucose levels only

>suggest

>the possibility of insulin resistance; they don't prove it. Insulin

>resistance

>is difficult to measure directly.) It might also mean that

>anti-inflammatory

>agents like aspirin may be particularly effective in diabetics.

>An Ounce of Prevention

>Advances in diabetes research over the past few years have been swift and

>wide

>ranging. Scientists are beginning to identify the genetic and environmental

>factors that predispose some people to insulin resistance and increase

>their

>risk of diabetes. They are looking beyond glucose levels to gauge patients'

>health and progress. They have identified other pathways that may play a

>role

>in triggering diabetes. Every new insight into Type 2 diabetes, from its

>biochemistry to its metabolic roots, makes clear that it can be avoided-and

>that the earlier you intervene the better.

>The real question is whether we as a society are up to the challenge. " Our

>health-care system is currently set up to deliver care for acute disease, "

>says

>Ann Albright, chief of the California Diabetes Prevention and Control

>Program.

> " It's get in, get your shot, and away you go. " Diabetes, however, is a

>chronic

>disorder that demands constant attention. You have to change your eating

>habits

>and incorporate physical exercise into each day's activities. You need to

>monitor your glucose levels several times a day to see how well you're

>doing.

>These prevention measures pay off in the long run in fewer heart attacks,

>strokes, amputations and cases of blindness and kidney failure. But very

>few

>insurance programs focus on them-or pay for health professionals who can

>teach

>folks how best to incorporate them into their lives.

>Comprehensive prevention programs aren't cheap, but the cost of doing

>nothing

>is far greater. " If we don't take care of this issue now, we will have huge

>numbers of Type 2 diabetics, and we will be paying for them with our tax

>dollars, " says Dr. Phyllis Preciado, an internist who runs a diabetes

>clinic in

>California's farming-rich Central Valley. As the U.S. loses productive

>members

>of the work force, she notes, more people will turn to public assistance

>for

>treatment. And the increased toll in human suffering will be staggering.

>There are ways to keep costs down. It doesn't take a physician to teach a

>patient the principles of better nutrition or how to use a glucometer.

>Nurses,

>nutritionists, diabetes educators and other non-M.D.s can play a key role.

>Experts say it's important to reach the communities that are hardest hit by

>diabetes-American Indians, for example-all the while taking cultural

>differences into account. " You can't give everybody the same diet to solve

>the

>problem, " says Albright, a registered dietitian. " People obviously eat the

>foods they've grown up with. So you have to try to help them get as much of

>those things that they like into their eating plan but also make the

>changes

>that will help lower the fat or moderate the carbohydrates. "

>Hillary Carroll and her family have taken those lessons to heart. While

>still

>in the hospital, Hillary went through a kind of diabetes boot camp in which

>she

>learned how to monitor her blood-sugar levels, change the way she eats and

>boost the amount of physical activity in her life. Her parents bought her a

>turquoise BMX bike, which she rides at least an hour every day. She has

>lost 24

>lbs. so far, and her glucose levels have stabilized. " Before she was

>diagnosed,

>she was happy to sit here by herself, playing with her Barbies, " says Tammy

>Carroll, Hillary's mother. " I thought she was so calm then, but now that we

>have her sugar under control, she's more active. " Hillary, now 11, is

>taking

>charge of her life. To get control of the diabetes epidemic, the rest of us

>may

>have to do the same.

>-Reported by Bjerklie and Alice Park/New York, Dan Cray/ Los Angeles

>and

> Randall/ Cincinnati

>http://www.time.com/time/magazine/printout/0,8816,552059,00.html

>

>

>

>Diabetes Genes Stacking Up

>Gabe Romain

>Betterhumans Staff

>Tuesday, December 02, 2003, 4:58:33 PM CT

>The linkage of eight genes to type 2 diabetes has provided support for the

>theory that an accumulation of many small genetic variations makes people

>susceptible to the disease.

>Researcher Inês Barroso and colleagues at the University of Cambridge in

>the UK

>found the genes while correlating genetic variations with metabolic changes

>characteristic of diabetes.

>The researchers analyzed 71 genes in more than 2,000 people and found that

>eight of the 71 were linked to type 2 diabetes.

>Major health problem

>Diabetes is a disease that impairs the body's ability to use glucose, a

>sugar

>that is a cell's main source of energy.

>Also known as adult onset diabetes, type 2 diabetes is the most common form

>of

>the disease.

>In type 2 diabetes, either the pancreas doesn't produce enough insulin-a

>hormone that regulates levels of glucose in the blood-or tissues become

>resistant to the action of the hormone.

>Type 2 diabetes is a major health problem affecting more than 100 million

>people worldwide.

>While the influence of environmental factors such as obesity and lifestyle

>on

>the disease's development has been well documented, genetic components have

>proven more elusive.

>Understanding such components could lead to more effective prevention and

>treatment.

>Multiple factors

>The researchers report that most of the genes they linked to type 2

>diabetes

>are related to B cells-cells that are responsible for the production of

>antibodies-and not to insulin function.

>This may suggest that dysfunctions associated with type 2 diabetes are

>affected

>differently by environmental and genetic factors, with insulin resistance

>related more to environmental factors and problems with insulin secretion

>related more to genetic factors.

>The findings also suggest that large-scale gene screening studies are a

>good

>way to identify a multitude of disease-associated genes that individually

>exert

>weak effects.

>The research is reported in the journal PLoS Biology (read full text).

>http://www.betterhumans.com/News/news.aspx?articleID=2003-12-02-2

>

>

>

>

>

>

>

[Guest guest]

,

Its pretty much a run of the mill article other than the starvation

reference. Not daring to go anywhere other than to implicating individual

defect. Certainly not going outside the box to anything that is part of

societal structure. Sugar and diabetes is simple thought but saying carbs

are sugar too hits an even larger part of the food pyramid. It's Time

Magazine. If the article did say what you've added can't imagine why they

wouldn't have gone along considering Taube's recent New York Times

articles for example. There's been little in this country's news about

people consuming less bread, U.S. meeting in the U.K. about it or the

article Dedy sent through few months back saying fast food manufacturers

knew their food makes you want more and the latest anti Atkins too. Looks

like word is spreading through friendly networks without media/medical

saying it.

>

>IMO this should really read " that ability seems to cause insulin resistance

>when cheap, low-nutritional-value starch- and sugar-based foods are

plentiful " .

>

>>Unfortunately,

>>that

>>ability seems to cause insulin resistance when food is plentiful.

Wanita

[Guest guest]

Agreed. Those that couldn't survive famine to feast died off. Pretty sure

thats the theory with Neanderthal when others moved into area. Starved

instead of adapting. Fermenting seems to have originated in southern

latitudes to protect from heat spoilage then spread north to provide for

winter. Drying seems more suited to northern survival. Even with getting

food in season for later use like salmon runs or berry, nut harvests it

involves a lot of work in a short time period and then less work for a

while. Another feast, famine, more,less process. Somewhere along the way,

likely along with agriculture and land ownership came work all the time in

order to eat all the time.

Wanita

>Actually, I'd take it a step further. I'd say that Europeans adapted

>to having food daily ... the " normal " state for humans was to

>kill a big animal now and then, gorge, then sit around nibbling

>on greens until the next big animal came along. Like lions

>and most predators. If in fact we are from a line of meat

>eaters, then the concept of constantly eating is a recent

>development. (I'm exaggerating, but you get the idea).

>

>Anyway, having dealt with one big animal lately, I just

>can't see doing all that work daily! Even with a tribe of

>helpers. Drying and fermenting probably came along later

>in evolutionary history.

>

>-- Heidi

[Guest guest]

>. Somewhere along the way,

>likely along with agriculture and land ownership came work all the time in

>order to eat all the time.

Wanita:

That's the part I hate. I like the " work alot then play alot " paradigm!

One psychologist was saying that humans (at least in the north) used

to be snowed in half the year, probably telling stories, doing

needlework, or whatever, and that that " down time " might be really

important for our brain development/happiness/social structure.

Also in that structure a woman could take a week off after

having a baby and just hang out with the baby, or stop to

nurse every few hours without the boss telling her to

get back to work ...

-- Heidi

[Guest guest]

At 10:07 AM 12/20/2003 -0800, you wrote:

>

>>. Somewhere along the way,

>>likely along with agriculture and land ownership came work all the time in

>>order to eat all the time.

>

>Wanita:

>

>That's the part I hate. I like the " work alot then play alot " paradigm!

>One psychologist was saying that humans (at least in the north) used

>to be snowed in half the year, probably telling stories, doing

>needlework, or whatever, and that that " down time " might be really

>important for our brain development/happiness/social structure.

>Also in that structure a woman could take a week off after

>having a baby and just hang out with the baby, or stop to

>nurse every few hours without the boss telling her to

>get back to work ...

>

>-- Heidi

Serotonin levels too! Everytime l have to clean the car off at 6:30 AM

after a night snowstorm, scrape windshield or get nervous on not cleared

yet roads, am not a happy camper wondering what's wrong with this picture.

Humans are supposed to be in at least semi hibernation in the north anyway.

Come home to a cool house because both of us worked all day and the

woodstove didn't get fed.

Wanita

[Guest guest]

At 10:24 AM 12/19/2003 -0800, you wrote:

>

>>Evolutionary biologists suspect that a predisposition toward diabetes

>>developed

>>among certain ethnic groups—such as Pima Indians or Americans of African

>>descent—as a result of repeated, perhaps even recent, bouts of starvation.

>>Those individuals who were better able to decrease energy expenditures

>>during a

>>famine survived and passed on the trait to their offspring.

>

>Actually, I'd take it a step further. I'd say that Europeans adapted

>to having food daily ...

>

>-- Heidi

That adaptation seems to have held well until the last few generations of

Europeans here. With the mass commercialization, processing and nutritional

decrease of food in the last 50 years especially, type ll diabetes and

obesity has soared as well in non ethnic population. Children being

diagnosed type ll should be a serious warning for chances of future

survival. Is insulin the next ritalin?

Wanita

[Guest guest]

Judith,

Didn't catch the no salt in TSP. Gave up salting food 30 years ago

retaining water with first pregnancy. Tried French sea salt before sending

away for Celtic last year. Salt no matter what it is obviously sets my

potassium/sodium balance off.

Wanita

>For some very good reading on this get a copy of Dr. Schwartzbein's

>book, " The Schwartzbein Principle. " It's intended as a weight loss book, but

>she explains exactly why the PC (politically correct) diet is killing us.

>And what she learned when she first went into practice.

>

>She differs from Atkins in two major ways that I disagree with. One, she

>says no one should ever be in ketosis. And, Two, no one should ever eat

>salt.

>

>Judith Alta

[Guest guest]

>Serotonin levels too! Everytime l have to clean the car off at 6:30 AM

>after a night snowstorm, scrape windshield or get nervous on not cleared

>yet roads, am not a happy camper wondering what's wrong with this picture.

ACK! I remember the early morning commute. I'm lucky I actually

survived it ... I was always semi-asleep at the wheel.

>Humans are supposed to be in at least semi hibernation in the north anyway.

>Come home to a cool house because both of us worked all day and the

>woodstove didn't get fed.

We did come up with a semi-solution to winter doldrums ... we hung

a " super grow light " (the ones that are of sun-strength, more or less)

in the garage, and crank up the woodstove, and it's like a nice summer day ...

-- Heidi

[Guest guest]

Nothing like wood heat. Puts me to sleep early on the couch many nights.

There's a full spectrum light bulb at computer, northernmost and darkest

room in house. Whole house negative ionizer also.

Wanita

>We did come up with a semi-solution to winter doldrums ... we hung

>a " super grow light " (the ones that are of sun-strength, more or less)

>in the garage, and crank up the woodstove, and it's like a nice summer day

....

>

>

>-- Heidi

[Guest guest]

No two people are alike. One person's best food is another's poison.

And I guess the only way we can find out which foods are best for each of us

is to try them.

Judith Alta

-----Original Message-----

Judith,

Didn't catch the no salt in TSP. Gave up salting food 30 years ago

retaining water with first pregnancy. Tried French sea salt before sending

away for Celtic last year. Salt no matter what it is obviously sets my

potassium/sodium balance off.

Wanita

>For some very good reading on this get a copy of Dr. Schwartzbein's

>book, " The Schwartzbein Principle. " It's intended as a weight loss book,

but

>she explains exactly why the PC (politically correct) diet is killing us.

>And what she learned when she first went into practice.

>

>She differs from Atkins in two major ways that I disagree with. One, she

>says no one should ever be in ketosis. And, Two, no one should ever eat

>salt.

>

>Judith Alta

[Guest guest]

In a message dated 12/19/03 10:46:22 AM Eastern Standard Time,

jaltak@... writes:

> I'm sure I'm not the only one to have noticed that these types of articles

> never, or almost never, point their fingers at the garbage the government is

> pressuring everyone to eat. Sure wouldn't want to make their advertisers and

> supporters unhappy.

I don't think Atkins et al have unravelled the mystery at all, but rather

barely cracked the tip of the iceberg.

While junk food certainly contributes, there's certainly more to it than

that. Rats fet high-sugar diet develop T2, but if they are fed fish oil, they

don't. Why not?

Chris

[Guest guest]

In a message dated 12/19/03 6:25:26 PM Eastern Standard Time,

heidis@... writes:

> Anyway, having dealt with one big animal lately, I just

> can't see doing all that work daily! Even with a tribe of

> helpers. Drying and fermenting probably came along later

> in evolutionary history.

>

Plus sharing work. You get the big animal and share it with me for a couple

days. Then I get a big animal and share it with you a couple days. Then our

other neighbor gets one, etc. That's how it worked in ol' New England.

In a tribe, it only takes *some* of the men to hunt a giraffe. The other

days, you don't necessarily go meatless, but you get some squirrels or turtles

or

something in between giraffe hunts. You get plenty of food all the time, and

spend about 2 hours a day with food, average.

Chris

[Guest guest]

In a message dated 12/23/03 12:34:28 AM Eastern Standard Time,

jaltak@... writes:

> Fish oil in addition to the sugar, or in place of it?

>

> Fish oil contains needed nutrients. White sugar has no nutrients. Not

> surprising that the rats develop problems on it.

The fish oil was not given in place of the sugar, it was given to the rats on

the high-sugar diet. Also a certain drug. It's from a study done at the

University of New England College of Osteopathic Medicine.

There is clearly far more to insulin resistance than too many carbs or too

many processed carbs. Nutrient deficiencies are part of the picture, magnesium

and omega-3s especially, activity levels also seem very important. Eating

patterns have a decisive effect on insulin sensitivity too.

Chris

[Guest guest]

In a message dated 12/23/03 12:50:16 AM Eastern Standard Time,

jaltak@... writes:

> Precisely. And the low-fat diet is notoriously low in all nutrients. The

> human body simply cannot sustain itself on such poor fuel.

>

> Here is an example of a day's worth of " healthy " eating. It's no wonder

> people have a hard time staying on it. There's very little there worth

> eating.

>

> ==========================================

> Breakfast: 1 poached egg, 1 slice dry toast, 1 cup skim milk, 1/2

> grapefruit, 1 large wedge of melon

> Lunch: 1 ounce Swiss cheese, 1 baked potato with skin, 1 cup skim milk, 1/2

> cup sliced raw cucumber, 1 medium apple, 2 slices dry bread

> Dinner: 1 cup salad greens (no dressing), 3 ounces lean beef, 3 cups cooked

> spaghetti, 1 cup cooked broccoli, 3/4 cup tomato juice, 2 slices dry bread

> ==========================================

I agree this is a horrible diet, but there are plenty of populations, afaik,

that eat pretty low-fat but don't get diabetes. Granted, they aren't eating

skim milk and spaghetti.

Chris

[Guest guest]

Fish oil in addition to the sugar, or in place of it?

Fish oil contains needed nutrients. White sugar has no nutrients. Not

surprising that the rats develop problems on it.

Judith Alta

-----Original Message-----

In a message dated 12/19/03 10:46:22 AM Eastern Standard Time,

jaltak@... writes:

> I'm sure I'm not the only one to have noticed that these types of articles

> never, or almost never, point their fingers at the garbage the government

is

> pressuring everyone to eat. Sure wouldn't want to make their advertisers

and

> supporters unhappy.

I don't think Atkins et al have unravelled the mystery at all, but rather

barely cracked the tip of the iceberg.

While junk food certainly contributes, there's certainly more to it than

that. Rats fet high-sugar diet develop T2, but if they are fed fish oil,

they

don't. Why not?

Chris

[Guest guest]

Precisely. And the low-fat diet is notoriously low in all nutrients. The

human body simply cannot sustain itself on such poor fuel.

Here is an example of a day's worth of " healthy " eating. It's no wonder

people have a hard time staying on it. There's very little there worth

eating.

==========================================

Breakfast: 1 poached egg, 1 slice dry toast, 1 cup skim milk, 1/2

grapefruit, 1 large wedge of melon

Lunch: 1 ounce Swiss cheese, 1 baked potato with skin, 1 cup skim milk, 1/2

cup sliced raw cucumber, 1 medium apple, 2 slices dry bread

Dinner: 1 cup salad greens (no dressing), 3 ounces lean beef, 3 cups cooked

spaghetti, 1 cup cooked broccoli, 3/4 cup tomato juice, 2 slices dry bread

==========================================

Judith Alta

-----Original Message-----

In a message dated 12/23/03 12:34:28 AM Eastern Standard Time,

jaltak@... writes:

> Fish oil in addition to the sugar, or in place of it?

>

> Fish oil contains needed nutrients. White sugar has no nutrients. Not

> surprising that the rats develop problems on it.

The fish oil was not given in place of the sugar, it was given to the rats

on

the high-sugar diet. Also a certain drug. It's from a study done at the

University of New England College of Osteopathic Medicine.

There is clearly far more to insulin resistance than too many carbs or too

many processed carbs. Nutrient deficiencies are part of the picture,

magnesium

and omega-3s especially, activity levels also seem very important. Eating

patterns have a decisive effect on insulin sensitivity too.

Chris

[Guest guest]

on our recent tv show called ACA.

they were talking abotu diets ect ect and how we are fatter and to my

suprise

they are looking at ways to literally take out ALL fat from our major

products.

which means going to the butcher soon will entail cuts of meat that have

been lipo suctioned.

so there be no fat at all. ( as it is its hard to buy a hcicken breast with

skin on it )

_____

From: Judith Alta K [mailto:jaltak@...]

Sent: Tuesday, 23 December 2003 3:48 PM

Subject: RE: Diabetes Diet

Precisely. And the low-fat diet is notoriously low in all nutrients. The

human body simply cannot sustain itself on such poor fuel.

Here is an example of a day's worth of " healthy " eating. It's no wonder

people have a hard time staying on it. There's very little there worth

eating.

==========================================

Breakfast: 1 poached egg, 1 slice dry toast, 1 cup skim milk, 1/2

grapefruit, 1 large wedge of melon

Lunch: 1 ounce Swiss cheese, 1 baked potato with skin, 1 cup skim milk, 1/2

cup sliced raw cucumber, 1 medium apple, 2 slices dry bread

Dinner: 1 cup salad greens (no dressing), 3 ounces lean beef, 3 cups cooked

spaghetti, 1 cup cooked broccoli, 3/4 cup tomato juice, 2 slices dry bread

==========================================

Judith Alta

-----Original Message-----

In a message dated 12/23/03 12:34:28 AM Eastern Standard Time,

jaltak@... writes:

> Fish oil in addition to the sugar, or in place of it?

>

> Fish oil contains needed nutrients. White sugar has no nutrients. Not

> surprising that the rats develop problems on it.

The fish oil was not given in place of the sugar, it was given to the rats

on

the high-sugar diet. Also a certain drug. It's from a study done at the

University of New England College of Osteopathic Medicine.

There is clearly far more to insulin resistance than too many carbs or too

many processed carbs. Nutrient deficiencies are part of the picture,

magnesium

and omega-3s especially, activity levels also seem very important. Eating

patterns have a decisive effect on insulin sensitivity too.

Chris

[Guest guest]

>In a tribe, it only takes *some* of the men to hunt a giraffe. The other

>days, you don't necessarily go meatless, but you get some squirrels or turtles

or

>something in between giraffe hunts. You get plenty of food all the time, and

>spend about 2 hours a day with food, average.

>

>Chris

What do you mean by " 2 hours a day with food " ? They eat one main

meal or it takes that long to prepare it? I've heard the same about

the hunts ... they ate a lot of small animals between hunts, and the

hunts weren't always successful.

I have a freezer and a sink and running water and sharp knives AND

kitchen help a lot of the time, and meals still take a couple of hours

at least! I REALLY like the one-meal-a-day mentality ... in those

old novels where they had the big breakfasts, they always seemed

to have a cook and servants.

-- Heidi

[Guest guest]

mmmmmm

maybe with our NT enlightenment we should have doctors as our cooking

servents

and teach them the ways of old hehehehe

_____

From: Heidi Schuppenhauer [mailto:heidis@...]

Sent: Tuesday, 23 December 2003 5:12 PM

Subject: Re: Diabetes Diet

>In a tribe, it only takes *some* of the men to hunt a giraffe. The other

>days, you don't necessarily go meatless, but you get some squirrels or

turtles or

>something in between giraffe hunts. You get plenty of food all the time,

and

>spend about 2 hours a day with food, average.

>

>Chris

What do you mean by " 2 hours a day with food " ? They eat one main

meal or it takes that long to prepare it? I've heard the same about

the hunts ... they ate a lot of small animals between hunts, and the

hunts weren't always successful.

I have a freezer and a sink and running water and sharp knives AND

kitchen help a lot of the time, and meals still take a couple of hours

at least! I REALLY like the one-meal-a-day mentality ... in those

old novels where they had the big breakfasts, they always seemed

to have a cook and servants.

-- Heidi

[Guest guest]

In a message dated 12/23/03 2:19:33 AM Eastern Standard Time,

heidis@... writes:

> What do you mean by " 2 hours a day with food " ? They eat one main

> meal or it takes that long to prepare it? I've heard the same about

> the hunts ... they ate a lot of small animals between hunts, and the

> hunts weren't always successful.

I meant hunting and gathering, not eating. That was the average for the San,

and I think other h & g's were pretty similar. I think big game hunts weren't

always successful, but hunting smaller animals is easier.

Chris

[Guest guest]

I would get really fat if I ate like that!

Michele

>From: " Judith Alta K " <jaltak@...>

>Reply-

>< >

>Subject: RE: Diabetes Diet

>Date: Tue, 23 Dec 2003 00:48:24 -0500

>

>Precisely. And the low-fat diet is notoriously low in all nutrients. The

>human body simply cannot sustain itself on such poor fuel.

>

>Here is an example of a day's worth of " healthy " eating. It's no wonder

>people have a hard time staying on it. There's very little there worth

>eating.

>

>==========================================

>Breakfast: 1 poached egg, 1 slice dry toast, 1 cup skim milk, 1/2

>grapefruit, 1 large wedge of melon

>Lunch: 1 ounce Swiss cheese, 1 baked potato with skin, 1 cup skim milk, 1/2

>cup sliced raw cucumber, 1 medium apple, 2 slices dry bread

>Dinner: 1 cup salad greens (no dressing), 3 ounces lean beef, 3 cups cooked

>spaghetti, 1 cup cooked broccoli, 3/4 cup tomato juice, 2 slices dry bread

>==========================================

>

>

>Judith Alta

>

>-----Original Message-----

>

>In a message dated 12/23/03 12:34:28 AM Eastern Standard Time,

>jaltak@... writes:

>

> > Fish oil in addition to the sugar, or in place of it?

> >

> > Fish oil contains needed nutrients. White sugar has no nutrients. Not

> > surprising that the rats develop problems on it.

>

>The fish oil was not given in place of the sugar, it was given to the rats

>on

>the high-sugar diet. Also a certain drug. It's from a study done at the

>University of New England College of Osteopathic Medicine.

>

>There is clearly far more to insulin resistance than too many carbs or too

>many processed carbs. Nutrient deficiencies are part of the picture,

>magnesium

>and omega-3s especially, activity levels also seem very important. Eating

>patterns have a decisive effect on insulin sensitivity too.

>

>Chris

>

>

>

[Guest guest]

Exactly!

And that's why this country is so overweight. Not because people overeat,

but because of what they eat.

Judith Alta

RE: Diabetes Diet

>Date: Tue, 23 Dec 2003 00:48:24 -0500

>

>Precisely. And the low-fat diet is notoriously low in all nutrients. The

>human body simply cannot sustain itself on such poor fuel.

>

>Here is an example of a day's worth of " healthy " eating. It's no wonder

>people have a hard time staying on it. There's very little there worth

>eating.

>

>==========================================

>Breakfast: 1 poached egg, 1 slice dry toast, 1 cup skim milk, 1/2

>grapefruit, 1 large wedge of melon

>Lunch: 1 ounce Swiss cheese, 1 baked potato with skin, 1 cup skim milk, 1/2

>cup sliced raw cucumber, 1 medium apple, 2 slices dry bread

>Dinner: 1 cup salad greens (no dressing), 3 ounces lean beef, 3 cups cooked

>spaghetti, 1 cup cooked broccoli, 3/4 cup tomato juice, 2 slices dry bread

>==========================================

>

>