Re: SSRI genital anesthesia

[Guest guest]

I asked Dr. Lin for his explanation and here is his response:

It destroys dopaminergic nervous cells and synapes for deficiency of oxytocin

responsible for arterial dilation and orgasm, and outgrow serotoninergic cells

and synapses by inhibiting the serotonin reputaking in the synpases for

addiction while poisoning the liver and disabling serotonin syntheses in the

nervous terminals.

You should read this link-

http://www.actionlove.com/extra/ssris.htm#SSRIs (or SSRIs/SNRIs) anti-depression

drugs

SSRIs (or SSRIs/SNRIs) anti-depression drugs cause chronic excessive prolactin

production (called HyperProlactimia, a pituitary functional disorder) to block

the brain's dopamine nervous function in the hypothalamus-pituitary axis

responsible for emotional and orgasm hormone oxytocin release, and interfere

with other neurotransmitter systems and syntheses and hormonal production by

overloading the liver P45 detoxification system, and blocks the spinal nervous

transmission in the synapses with a flooding of serotonin due to the re-uptaking

inhabitation action. They alternate the gene expression of the dopamine D2

receptors, acetylcholine receptors, and 5-HT (Serotonin) 2C receptors.

Blocking the dopamine and acetylcholine transduction in the synapses and dumping

high level of prolactin in the bloodstream disable the erectile nervous function

in the penis/clitoris and G-spot, the seminal production or vaginal lubrication

and de-sensitizes the nerves in the penis, prostate, seminal vesicles,

clitoris, G-spot, vagina, cervix and uterus for no more sexual orgasm. The most

critical problem is that SSRIs drugs also block the hypothalamic/adrenal

dopamine-norepinephrine conversion and the pituitary oxytocin release, essential

to activation of the orgasmic contraction via the sympathetic nerves L1 and L2

in the prostate, seminal vesicles, penis, uterus, cervix, vagina and clitoris.

Thus, you will never achieve orgasm or produce semen with a a high level of

prolactin and a low level of oxytocin in your blood stream and with the

desensitized sensory and blocked parasympathetic and sympathetic nerves.

SSRIs will kill your sexual function naturally via the destruction of the

pituitary and liver function. SSRIS drugs only inhibit the re-uptaking of

serotonin in the nervous synapse, but actually decrease the serotonin synthesis

in the nervous terminals due to their destruction effects on the liver P450.

Taking SSRIs drugs may reduce the serotonin nervous action on the neuromsucular

ending receptors in the guts, bladder, urethra, prostate and anus for

incontinence. By the way, excessive prolactin in the bloodstream reduced the

testosterone and DHT release from the testicles and ovaries, resulting in bone

and muscle weakness, in addition to disabling sexual functions and responses and

seminal production.

SSRIs antidepressants cause neuroplasticity (deformation of synapses and

outgrowth of serotonin neurons) for withdrawal symptom and addiction. They

inhibit the reuptaking of serotonin in the nervous synapse by blocking

serotonin transporter, so that the serotonin neurotransmitter can continue

acting its effects on the postsynaptic neurons and possible outgrow the

postsynaptic neurons (positive neurplasticity) under a long-term use . However,

the SSRIs won't increase (actually decrease) the neurotransitter synthesis due

to its intoxification effects on the liver Thus, SSRIs stimulates more

serotonin neurons, but cuts down natural serotonin release from the nervous

terminals. The SSRIs drug can also alter the temporal and spatial relationship

between dopamine and serotionin signaling in the striatum which receives rich

dopaminergic and more moderate serotonergic innervation. Under the high level of

extracellular serotonin in the innervation synapses, elevated by exogenous

application or by using antidepressants to inhibit the serotonin transporters,

the extremely dense striatal Dopamine Transporters can uptake serotonin into

dopamine terminals. This can destroy the dopamine nervous function responsible

for pituitary oxytocin, LH and FSH release associated with sexual and testicular

functions. A weak dopamine nervous function also results in a high pituitary

prolactin release for disable sexual function.

http://www.ncbi.nlm.nih.gov/pubmed/15820694?dopt=Abstract & holding=f1000,f1000m,i\

srctn

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> I wonder if there is any Docter, Neuroscientist etc that know the

reason/theory why these drugs cause the awful loss of sensation ''numbness'' in

the genitals and skin, what is the mechanism of action. Can low testosterone

cause numbness in the genitals and in other areas of the body. I don't think so

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[Guest guest]

Thanks. His English is a bit " off " sometimes, but his scientific understanding

appears to be accurate.

V

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> > I wonder if there is any Docter, Neuroscientist etc that know the

reason/theory why these drugs cause the awful loss of sensation ''numbness'' in

the genitals and skin, what is the mechanism of action. Can low testosterone

cause numbness in the genitals and in other areas of the body. I don't think so

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[Guest guest]

My lot feels like a piece of polystyrene and I have never got used to

this feeling when I go to the toilet. It is always a very unpleasant

reminder of the damage these drugs have done to me.

Kv

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> > I can't say I have no sensation at all. I can feel an ejaculation

but it's not pleasurable. Also, when I urinate it feels different. It's

hard to explain, but even during urination it feels different. I can

feel the urine coming up from my bladder through the tubes and I never

noticed this feeling before. It's too weird of a feeling to explain. The

best way I can describe it is a " hollow " feeling during urination and

ejaculation, like someone else is getting the pleasure. It makes me feel

weird.

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> > Also, I don't get an erection when I have to urinate badly anymore

no matter how bad I have to go. I used to get erections when I had to

urinate. The only way I know I have to urinate now is the pressure in my

abdomen and bladder.

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[Guest guest]

I agree however I think the sert transporter is to high, it's high serotonin that causes sexual dysfunction and numbs anxiety, I don't know about other but my anxiety is way way less. It feels like I am still on SSRs. I don`t think you need high sertonin or normal serotonin for sexual function, serotonin is anti sex from what I have read . Please correct me if I am wrong. To: SSRIsex Sent: Tuesday, December 6, 2011 5:39:55 PM Subject: Re: SSRI genital anesthesia

What's depressing, ? The multitude of changes? I think the primary change is probably a decrease in SERT transporter gene expression with chronic SSRI use, and the other changes are secondary.

V

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> > > > I wonder if there is any Docter, Neuroscientist etc that know the reason/theory why these drugs cause the awful loss of sensation ''numbness'' in the genitals and skin, what is the mechanism of action. Can low testosterone cause numbness in the genitals and in other areas of the body. I don't think so .

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[Guest guest]

The serotonin transporter (SERT) removes serotonin from the synapse. So if SERT

is low, serotonin will remain high. That's why it feels like you're still on an

SSRI.

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> > > > > I wonder if there is any Docter, Neuroscientist etc that know the

reason/theory why these drugs cause the awful loss of sensation ''numbness'' in

the genitals and skin, what is the mechanism of action. Can low testosterone

cause numbness in the genitals and in other areas of the body. I don't think so

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[Guest guest]

Hi Does Inositol help resensatize the sert, how can we get in back to normal. To: SSRIsex Sent: Wednesday, December 7, 2011 12:58:18 AM Subject: Re: SSRI genital anesthesia

The serotonin transporter (SERT) removes serotonin from the synapse. So if SERT is low, serotonin will remain high. That's why it feels like you're still on an SSRI.

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[Guest guest]

I'm not trying to sell anyone on Dr. Lin, just presenting my experience with him

and some more of my thoughts on him and why I'm taking a chance on his products.

Dr. Lin has his Ph D. which earns him the right to be called " doctor. "

Nowhere in his information does he claim to be a medical doctor.

His theories seem to match up with what I have learned from others in this group

during the past year including his thoughts on prolactin. My prolactin also has

been tested, and my results were normal. I have read that blood tests are not

reliable for measuring prolactin, so I still suspect that I have high prolactin

even though the blood test said otherwise.

The ingredients in his supplements match up with what I've read others in the

group have had positive experiences with. And I feel that my recovery is a

direct result of being on his products for 7 months.

There is the possibility that had you stayed on his products for longer you

would've seen results. I was told that it would take at least 6 months for

improvement and it took exactly that time for me.

If you read his information, he explains that all the damage that SSRIs have

done CAN be reversed, that the damage is not permanent especially when you have

the right nutrients for recovery. This matches up with many other things I have

read online about good nutrition and megadoses of vitamins playing key roles in

the body being able to heal from many conditions.

His advice and explanations have been accurate about 75% of the time - just a

rough estimation - of the numerous things I have asked him about in the past 7

months. I agree with you that sometimes he gives answers that don't make sense

or that he should say that he needs more information to give an answer.

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> > > > > > > I wonder if there is any Docter, Neuroscientist etc that

> know the reason/theory why these drugs cause the awful loss of sensation

> ''numbness'' in the genitals and skin, what is the mechanism of action.

> Can low testosterone cause numbness in the genitals and in other areas

> of the body. I don't think so .

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