CFS, the Immune System & Viral Infection

[Guest guest]

The authors below from London are speaking about:

*CFS as defined by recent criteria*

The only recent criteria, I can think of are the

terrible *unwellness criteria* by Reeves, ,

Unger, Vernon et al.

The CDC estimated that six to ten times more

people have this illness than their previous reports in

the US.

These figures were received with cheer on the

website of the US patient charity CAA by the director

Kim McCleary, who get a salary, which is the same as

that of the Minister President of The Netherlands.

Or do they mean the fraudulent Nice Guidelines from

London?

I'm too sick and brainfogged to think clearly, so

forgive me and blame it on ME, but one thing is for

sure, these scientists come with very old news.

~jan van roijen

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http://www.ncbi.nlm.nih.gov/pubmed/21756995

Brain Behav Immun. 2011 Jul 2. [Epub ahead of print]

Chronic fatigue syndrome, the

immune system and viral infection.

Bansal AS, Bradley AS, Bishop KN, Kiani S, Ford B.

Dept. of Immunology, Epsom and St. Helier University

Hospitals NHS Trust, Carshalton, Surrey, SM5 1AA and

Chronic Illness Research Team, Stratford Campus,

University of East London, London E15 4LZ, UK.

Abstract

The chronic fatigue syndrome (CFS), as defined by

recent criteria, is a heterogeneous disorder with a

common set of symptoms that often either follows a

viral infection or a period of stress.

Despite many years of intense investigation there is

little consensus on the presence, nature and degree

of immune dysfunction in this condition.

However, slightly increased parameters of

inflammation and pro-inflammatory cytokines such as

interleukin (IL) 1, IL6 and tumour necrosis factor

(TNF) alpha are likely present.

Additionally, impaired natural killer cell function

appears evident.

Alterations in T cell numbers have been described by

some and not others.

While the prevalence of positive serology for the

common herpes viruses appears no different from

healthy controls, there is some evidence of viral

persistence and inadequate containment of viral

replication.

The ability of certain herpes viruses to impair the

development of T cell memory may explain this viral

persistence and the continuation of symptoms.

New therapies based on this understanding are more

likely to produce benefit than current methods.

Copyright © 2011. Published by Elsevier Inc.

PMID: 21756995 [PubMed - as supplied by publisher]