Re: Why does hypertrophy occur?

[Guest guest]

,

You wrote:

> You asked good questions I replied below to each. As far as reading

> goes, I suggest Supertraining & Facts & Fallacies by Mel Siff, Popular

> Trends in Modern Strength Training by Poliquin, and

> Science and Practice of Strength Training by Vladimir M. Zatsiorsky.

Thanks, those books are on my wish list :-)

> I have never read any conclusive evidence that showed 100% muscle

> recruitment at any time and this includes those done on professional

> powerlifters. It would be interesting to see his methods. Are they EMG

> studies? If so, there would be the answer. EMG will only show the

> activity of more superficial fibers but not all fibers (unless probes are

> inserted, but this will still only indicate the neural input in an

> incomplete # of motor units). Not only that, but I have never seen an EMG

> that I couldn't max out by just flexing pretty hard. This would suggest to

> me that the Typical EMG is not sensitive enough to show full neural

> abilities of highly trained individuals. (How can you assume that just

> because the instrument is maxed out that so is the individual, especially

> since we know that everyone has different neural capacities and abilities)

I also want to get Enoka's book, a friend sent me the quote when

discussing this. I querried a lot of people about this, and the more I

asked, the more I found it seemed to be the new accepted standard.

> Then explain your CNS hyper-aroused statement below. :-) See texts above

> for reading.

What I meant was this: Your CNS will adjust rate coding according to

arousal level. Possibly one could be training with less 'oomph' so they

are not 'using' their full 'rate coding' capabilities, then they become

more motivated and train a few times with this higher arousal level. It's

not that their CNS was able to increase it's rate coding, but that they

finally pushed it to the maximum.

> I have found that these new capacities are permanent (as long

> as you continue the exercise, which must be changed at some point,

> resulting in a small rollercoaster effect, This type of improvement leads me

> to believe that these gains are normal increases as apposed to just being

> jacked up (which is unusual for me unless I'm maxing anyway).

What makes me think this, is many studies I've read show a person reaches

tetany around 60 hz and without electrical stimulation, it appeared to be

the max frequency. It seems that once tetany is reached, since this is

simular to an always 'on' state, that more force wouldn't be shown by

increasing a bit more? (thinking this since actin-myosin have a certain

relaxation time period)

Thanks for the answers, this is an interesing discussion

Ron Sowers

Post Falls Id

USA

[Guest guest]

Ron Sowers wrote:

> What I meant was this: Your CNS will adjust rate coding according to

> arousal level. Possibly one could be training with less 'oomph' so they

> are not 'using' their full 'rate coding' capabilities, then they become

> more motivated and train a few times with this higher arousal level. It's

> not that their CNS was able to increase it's rate coding, but that they

> finally pushed it to the maximum.

>

> What makes me think this, is many studies I've read show a person reaches

> tetany around 60 hz and without electrical stimulation, it appeared to be

> the max frequency. It seems that once tetany is reached, since this is

> simular to an always 'on' state, that more force wouldn't be shown by

> increasing a bit more? (thinking this since actin-myosin have a certain

> relaxation time period)

Ok,

Yes, once tetanus is reached, that particular motor unit cannot contract any

harder. The problem is that the fast twitch fibers are harder to recruit

(especially the fastest, strongest ones), therefore tetanus is harder to

reach in these motor units. Yes arousal can increase this, but so can

training. With proper training one can more easily approach tetanus in these

motor units without being more aroused.

J s, MS, CSCS, USAW, NSCA-CPT

Assistant Strength & Conditioning Coach

College of ton

Strength Dept.

30 Str.

ton, SC 29424

[Guest guest]

J s wrote:

> Yes, once tetanus is reached, that particular motor unit cannot

> contract any harder. The problem is that the fast twitch fibers are harder to

> recruit (especially the fastest, strongest ones), therefore tetanus is

> harder to reach in these motor units. Yes arousal can increase this, but so

> can training. With proper training one can more easily approach tetanus

> in these motor units without being more aroused.

I agree, the only question that remains then, is how much proper

training will it take to reach this level.

Once reached, neural gains would be 'maxed' then.

Ron Sowers

Post Falls Id

USA

[Guest guest]

s <jjacobs24@h...> wrote:

> The problem is that the fast twitch fibers are harder to recruit

> (especially the fastest, strongest ones), therefore tetanus is

> harder to reach in these motor units. Yes arousal can increase this, but so

> can training. With proper training one can more easily approach tetanus

> in these motor units without being more aroused.

When I train my biceps, on the second and third sets of my exercises,

from reps 8-10, I build toward a cramp. By the completion of the

concentric squeeze of the 10th rep, my biceps are in a full " charlie-

horse " cramp that I must be immediately dissipate by eccentric

completion and loaded stretch because of the pain.

My question is: Is this cramp at the end of my sets the result of

fully achieved tetanus?

If so, then does a training program using progressive resistance in

this mode of training promote recruitment of Type IIb fibers? And

especially when compensatory acceleration type concentrics are used?

Thanks for your input,

Wayne Montierth

Sandy, Utah