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Re: Schroeder/Fiber Type

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> " A

> >noticeable feature of Jay's programs is that everything is either very

> >heavy or very fast- nothing in-between. There really is no repeated effort

> >method at all, although perhaps he would use this for an athlete needing to

> >get bigger. "

>

> The following study shows an increase in IIB fiber % with weight training

> using high speed eccentrics. If one examines the role of the faster

> contracting fibers (IIB,C,X, etc), it would seem one way to maximize their

> availability would be to expose the body to both very high forces and very

> fast forces at the same time. If the body can get the job done with IIA it

> will, but perhaps if exposed to supramaximal forces over a period of time

> (as can be done with plyometric training), the only logical adaptation is to

> a faster and stronger fiber. I think its important to look at what signals

> the training is sending to the nervous and metabolic system. When

> performing submaximal eccentric accentuated training, the load is such that

> it can be dealt with easily enough, so the metabolic adaptation favors the

> more efficient IIA fiber. When performing supramaximal eccentrics or

> plyometrics, the load is either more then the body can handle or occurs with

> lots of simultaneous force and speed, both which favor a faster and stronger

> fiber, the IIB.

***Remember though that MHC expression in a fiber is not necessarily

indicative of it's hypertrophic capacities. MHC expression, IIRC, is

more related to contraction velocity as opposed to maximal force

output (as in, MHC IIa and IIx both have identical force outputs,

but IIx is " faster " ).

Nothing has been directly done academically to support this (that

I'm aware of), but my thoughts on the matter are that the MHC IIx

isoform is the " default " state of a given population of high-

threshold motor units. The observation that most strength athletes

show a predominance of IIa fibers is a result of form following

function-- strength training doesn't require IIx fibers, and the IIa

state is somehow more beneficial to maximal and/or repeated efforts.

On the other hand we have explosive strength/RFD and reactive

ability, as in the study you listed (they used isokinetic equipment

if that's the same study I'm thinking of, but for the sake of this

thought experiment I'll consider it valid for movements without

fixed velocity), showing that IIx expression becomes more dominant

with exposure to faster movements. It also seems to me that

weightlifters and sprinters, groups that both regularly engage in

movements with high RFD, showed similar characteristics.

Just extrapolating from those facts, it seems that individuals

involved in pure strength training via maximal and repeated efforts

show a conversion from the " default " IIx expression of higher

threshold MU's to a more efficient IIa. Those involved in training

that involves training for a high RFD and for reversal strength show

a greater dominance of IIx fibers.

The IIx " overshoot " phenomenon observed with periods of detraining

could simply be those fibers returning to their " default " states.

The question is, does the MHC conversion provide a physiological

reason for the observed loss of speed-strength characteristics after

periods of heavy strength training? I'd be willing to bet it is at

least related. Interestingly enough, this idea fits in nicely with

Verkhoshansky's concentration of loading periodization model, where

speed-strength characteristics are enhanced sharply after periods of

high-volume strength trainig.

Now, as I said above this is all hypothesizing on my part so far as

I'm aware, but it is consistent with the literature. If it is the

case, it provides even more evidence for the concept of training for

goals, not for fibers, but also for structuring periods of strength

training with periods of speed-strength training.

If one is concerned with simple hypertrophy (yes, I finally got to

the original question), then it's really hard to go wrong assuming

the proper intensity, volume, density, and quality of eccentric

action is present. So yeah, maximal and ballistic-type movements

could potentially be very useful for hypertrophy.

man

Birmingham, AL

www.wannabebig.com

www.fortifiediron.com

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> " A

> >noticeable feature of Jay's programs is that everything is either very

> >heavy or very fast- nothing in-between. There really is no repeated effort

> >method at all, although perhaps he would use this for an athlete needing to

> >get bigger. "

>

> The following study shows an increase in IIB fiber % with weight training

> using high speed eccentrics. If one examines the role of the faster

> contracting fibers (IIB,C,X, etc), it would seem one way to maximize their

> availability would be to expose the body to both very high forces and very

> fast forces at the same time. If the body can get the job done with IIA it

> will, but perhaps if exposed to supramaximal forces over a period of time

> (as can be done with plyometric training), the only logical adaptation is to

> a faster and stronger fiber. I think its important to look at what signals

> the training is sending to the nervous and metabolic system. When

> performing submaximal eccentric accentuated training, the load is such that

> it can be dealt with easily enough, so the metabolic adaptation favors the

> more efficient IIA fiber. When performing supramaximal eccentrics or

> plyometrics, the load is either more then the body can handle or occurs with

> lots of simultaneous force and speed, both which favor a faster and stronger

> fiber, the IIB.

***Remember though that MHC expression in a fiber is not necessarily

indicative of it's hypertrophic capacities. MHC expression, IIRC, is

more related to contraction velocity as opposed to maximal force

output (as in, MHC IIa and IIx both have identical force outputs,

but IIx is " faster " ).

Nothing has been directly done academically to support this (that

I'm aware of), but my thoughts on the matter are that the MHC IIx

isoform is the " default " state of a given population of high-

threshold motor units. The observation that most strength athletes

show a predominance of IIa fibers is a result of form following

function-- strength training doesn't require IIx fibers, and the IIa

state is somehow more beneficial to maximal and/or repeated efforts.

On the other hand we have explosive strength/RFD and reactive

ability, as in the study you listed (they used isokinetic equipment

if that's the same study I'm thinking of, but for the sake of this

thought experiment I'll consider it valid for movements without

fixed velocity), showing that IIx expression becomes more dominant

with exposure to faster movements. It also seems to me that

weightlifters and sprinters, groups that both regularly engage in

movements with high RFD, showed similar characteristics.

Just extrapolating from those facts, it seems that individuals

involved in pure strength training via maximal and repeated efforts

show a conversion from the " default " IIx expression of higher

threshold MU's to a more efficient IIa. Those involved in training

that involves training for a high RFD and for reversal strength show

a greater dominance of IIx fibers.

The IIx " overshoot " phenomenon observed with periods of detraining

could simply be those fibers returning to their " default " states.

The question is, does the MHC conversion provide a physiological

reason for the observed loss of speed-strength characteristics after

periods of heavy strength training? I'd be willing to bet it is at

least related. Interestingly enough, this idea fits in nicely with

Verkhoshansky's concentration of loading periodization model, where

speed-strength characteristics are enhanced sharply after periods of

high-volume strength trainig.

Now, as I said above this is all hypothesizing on my part so far as

I'm aware, but it is consistent with the literature. If it is the

case, it provides even more evidence for the concept of training for

goals, not for fibers, but also for structuring periods of strength

training with periods of speed-strength training.

If one is concerned with simple hypertrophy (yes, I finally got to

the original question), then it's really hard to go wrong assuming

the proper intensity, volume, density, and quality of eccentric

action is present. So yeah, maximal and ballistic-type movements

could potentially be very useful for hypertrophy.

man

Birmingham, AL

www.wannabebig.com

www.fortifiediron.com

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Share on other sites

***Remember though that MHC expression in a fiber is not necessarily

indicative of it's hypertrophic capacities. MHC expression, IIRC, is

more related to contraction velocity as opposed to maximal force

output (as in, MHC IIa and IIx both have identical force outputs,

but IIx is " faster " ).

The original statement was that Schroeder used contraction speeds that were

very fast and loads that were very high and nothing in between and I was

trying to give some reasons why. Speed is obviously always going to be

important for any athlete but for example, if one needs hypertrophy, there

are several different ways of training for that. One could train like a

bodybuilder with lots of pump sets and put on plenty of muscle, but what is

that going to do to contraction speeds? There are different means of

arriving at the same goal and I think what Schroeder does is keep both the

short term and long term goals, signals, and adaptations in mind.

Baggett

on,AR

USA

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***Remember though that MHC expression in a fiber is not necessarily

indicative of it's hypertrophic capacities. MHC expression, IIRC, is

more related to contraction velocity as opposed to maximal force

output (as in, MHC IIa and IIx both have identical force outputs,

but IIx is " faster " ).

The original statement was that Schroeder used contraction speeds that were

very fast and loads that were very high and nothing in between and I was

trying to give some reasons why. Speed is obviously always going to be

important for any athlete but for example, if one needs hypertrophy, there

are several different ways of training for that. One could train like a

bodybuilder with lots of pump sets and put on plenty of muscle, but what is

that going to do to contraction speeds? There are different means of

arriving at the same goal and I think what Schroeder does is keep both the

short term and long term goals, signals, and adaptations in mind.

Baggett

on,AR

USA

_________________________________________________________________

Worried about inbox overload? Get MSN Extra Storage now!

http://join.msn.com/?PAGE=features/es

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> Just extrapolating from those facts, it seems that individuals

> involved in pure strength training via maximal and repeated efforts

> show a conversion from the " default " IIx expression of higher

> threshold MU's to a more efficient IIa. Those involved in training

> that involves training for a high RFD and for reversal strength show

> a greater dominance of IIx fibers.

IIB/X fibers are the so-called " couch potato " fibers. The overwhelming majority

of the research is showing a decrease in the expression of MHC IIb/x protein and

an increase in MHC IIa proteins. In strength and power sports, this occurs

regardless if it is a more " strength-oriented " or " power-oriented " - see Fry et

al. in the last two issues of Journal of Strength and Conditioning Research -

one article on biopsies in powerlifters, the other in weightlifters. In

general, a shift of MHC IIb/x to MHC IIa occurs within two weeks of initition of

training in untrained individuals (Staron et al. JAP 1994?)

> The IIx " overshoot " phenomenon observed with periods of detraining

> could simply be those fibers returning to their " default " states.

> The question is, does the MHC conversion provide a physiological

> reason for the observed loss of speed-strength characteristics after

> periods of heavy strength training?

Unlikely. Research we presented (while I was at The University of Memphis) at

the 2002 NSCA conference indicated that decreases in speed and power related

variables during high relative intensity overtraining were unrelated to changes

in MHC expression. The abstracts are available online for NSCA members (Fry et

al. and Schilling et al.).

Also see Chiu and in the latest Strength and Conditioning Journal (yeah,

I'm plugging my own article :). Myosin heavy chain expression appears to become

fairly stable following a prolonged period of training, and is thus a general

fitness characteristic. Changes in speed and power related variables tend to

occur as an acute response or short-term adaptation making these a function of

fitness and/or fatigue after-effects.

--

Loren Chiu, M.S., C.S.C.S.

Department of Biokinesiology and Physical Therapy

University of Southern California LA

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> Just extrapolating from those facts, it seems that individuals

> involved in pure strength training via maximal and repeated efforts

> show a conversion from the " default " IIx expression of higher

> threshold MU's to a more efficient IIa. Those involved in training

> that involves training for a high RFD and for reversal strength show

> a greater dominance of IIx fibers.

IIB/X fibers are the so-called " couch potato " fibers. The overwhelming majority

of the research is showing a decrease in the expression of MHC IIb/x protein and

an increase in MHC IIa proteins. In strength and power sports, this occurs

regardless if it is a more " strength-oriented " or " power-oriented " - see Fry et

al. in the last two issues of Journal of Strength and Conditioning Research -

one article on biopsies in powerlifters, the other in weightlifters. In

general, a shift of MHC IIb/x to MHC IIa occurs within two weeks of initition of

training in untrained individuals (Staron et al. JAP 1994?)

> The IIx " overshoot " phenomenon observed with periods of detraining

> could simply be those fibers returning to their " default " states.

> The question is, does the MHC conversion provide a physiological

> reason for the observed loss of speed-strength characteristics after

> periods of heavy strength training?

Unlikely. Research we presented (while I was at The University of Memphis) at

the 2002 NSCA conference indicated that decreases in speed and power related

variables during high relative intensity overtraining were unrelated to changes

in MHC expression. The abstracts are available online for NSCA members (Fry et

al. and Schilling et al.).

Also see Chiu and in the latest Strength and Conditioning Journal (yeah,

I'm plugging my own article :). Myosin heavy chain expression appears to become

fairly stable following a prolonged period of training, and is thus a general

fitness characteristic. Changes in speed and power related variables tend to

occur as an acute response or short-term adaptation making these a function of

fitness and/or fatigue after-effects.

--

Loren Chiu, M.S., C.S.C.S.

Department of Biokinesiology and Physical Therapy

University of Southern California LA

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Also see Chiu and in the latest Strength and Conditioning Journal

(yeah, I'm plugging my own article :).

***Let me also plug Chiu and " The Fitness-Fatigue Model Revisited:

Implications for Planning Short- and Long-Term Training. " It is an excellent

piece of work!

Kenny Croxdale

Rio Rancho, NM

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