Guest guest Posted March 7, 2011 Report Share Posted March 7, 2011 How many students?You could assign each one a separate report on different aspects of Conn's.1. Classical PA. From the first case to today at Yahoo.2. The history of PA with a detailed review of the first case. She was in the Hosp for nearly a year for study. 3. GRA familial Conn's 4. AME-5. How does licorice cause HTN?6. DM and Conn's-why is glucose metabolism messed up in Conn's 7. Salt and Conn's8. Drug resistant HTN and Conn's9. Why do Conn's pee so much at night?10. Why do Conn's get paralyzed?11. The evolution of testing for Conn's. 1954-present.12. The nurses role in Conn's syndrome.13. WWII research on heat adaptation and Conn's Syndrome.14. Normokalemic Conn's 15. Conn's is it rare or common?16. The first case of Conn's in India-if you have any Indian students.17. Liddle's Syndrome18. DOCA HTN in animals and man.19. Cushing's Syndrome or Conn's how to separate them.20. Pheochromocytoma21. Renal artery stenosis22. Salt and Hypertension23. How does salt and aldo cause kidney and heart fibrosis?24. Genetics of Conn's syndrome. 25. Why is hypertension more common and severe in the African Diaspora?26. Why are captive great apes dying of heart failure around the world?27. Why is HTN and DM more common in the Indian Diaspora in Trinidad.28. Differences in HTN in Guyana immigrants to Holland by ethnicity. 29. Why is HTN more common in survivors of the Siege of Leningrad during WWII.30. What happens to BP during starvation? To mention a few. By the time you have reviewed and critiqued you will be an expert yourself.Let me know how I can help. Might work better if I call it the right thing uh? Ok medscape you're off the hook for now.As if God (call it Karma if you so desire) doesn't have enough to do, he decided this month I need a serious refresher lesson so I am teaching, not too in depth mind you, but deep enough to learn something new, a renal anatomy and disorder class to a bunch of nursing students (I teach right now, mostly health care administration, and am going back into family practice FT in a few months). I couldn't be more happy really. BTW they get extra credit if they do a report on Conn's Syndrome. Completely serious! I'm also interested in the actual stats on 'much less'. If the recommendation of the AAES (if that's how they're known) is to skip AVS if you're under 40, I presume they base this on some data that supports that (my yet-to-be-supported 97%?)? Especially as over 40 they clearly recommend AVS first so it's not that they're anti-AVS. And is it even more näive to imagine that my position is currently a lifetime of drugs vs. surgery that may or may not lead to cure. And if it doesn't lead to cure, then I have a lifetime of drugs (either immediately or whenever I get a recurrence on the other side). If surgery is not successful, does that mean a higher drug requirement than if I just opt for drugs straight off? Or is it a question of life expectancy? What downside have I missed?I get the clear risk that the growth on one side could be benign and co-incidentally the other, apparently clear, side could be the trouble maker (or it could be bilateral) but if I can get a justification for 97%, that's a 3% risk I'm willing to take (topside risk given that even AVS will not necessarily give a clear picture). Especially if the only downside of the risk is a lifetime of drugs. I'm not ignoring DASH and all that but I see that as supplementary in every scenario - it's not going to effect a cure for me.Hester> > > > > > > >> > > > > > > > My doctor just called with the results of last week's > > > aldosterone suppresion test/oral sodium loading test. As expected, > > > my aldo was off the charts high, both serum and urine. (I can post > > > numbers when I have a hard copy in front of me, if anyone is > > > interested.) The interesting thing my doctor observed was that I did > > > not excrete a lot of sodium - he believes it's because my diet is > > > very low sodium. (I believe it's still trapped in my body in the > > > form of the extra 2 pounds I've been carrying around since the test!)> > > > > > > >> > > > > > > > This gives me the go-ahead for AVS, or, interestingly, he > > > said I could skip the AVS and have the adrenalectomy. I want to be > > > good and certain that the culprit is my left adrenal before surgery, > > > so I think I'm going through with the AVS first. I'm aware of the > > > high risk of inconclusive results, and the risks of the procedure in > > > general.> > > > > > > >> > > > > > > > This brings up two questions: how common is it to skip the > > > AVS entirely? I only heard of that recently from another member > > > here; I had been under the impression that standard protocol was AVS > > > before surgery.> > > > > > > >> > > > > > > > And, long shot I know, but - anybody here have their AVS > > > done at UCLA? Or anywhere in the greater Los Angeles area? If you > > > have an interventional radiologist in the area that you'd recommend, > > > please let me know. Thanks!> > > > > > > >> > > > > > > >> > > > > > > > - msmith1928> > > > > > > > 45, female, 5'3", 120 lbs, 1cm left adrenal nodule, > > > aldosterone 42.3, renin 0.5, potassium <2.9 (when not taking > > > supplements); 25mg spiro caused gynecomastia, no meds currently > > > except 20MEQ K 2x/day; low sodium, fructose- and grain-free diet> > > > > > > >> > > > > > >> > > > > >> > > > >> > > >> > >> > >> > >> >> Quote Link to comment Share on other sites More sharing options...
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