Re: No high fructose in V-8??

[Guest guest]

My advice is to say away from it ;=)Friend to everyone except for people like me, who, if we ingest tomatoes or carrots, will experience vomiting, bloating, and diarrhea (resulting in further potassium loss), and risk damage to our livers.Brief rundown of fructose intolerance here:http://www.bu.edu/aldolase/HFI/hfiinfoThis isn't a "weight loss" issue, but a serious health concern. If I ingest fructose, I become violently ill!> > >> > > > --- In hyperaldosteronism , Clarence Grim> > > > >> > > > > But if one is not ready for surgery because DASH and MCBs have> > > > failed> > > > > then I argue why take the risks (radiation) etc. and costs of an> > > > AVS.> > > >> > > > Dr Grim, I'm not sure if your response was meant for me or for > > others.> > > >> > > > If it was meant for me - I'm fully aware of your preference for > > DASH> > > > and MCBs before surgery. That's not working for me.> > > >> > > > I'm the original poster of this question, and we've been through> > > > this several times before - I can't take spiro, and due to > > inability> > > > to metabolize fructose and fructans (found in most fruits, many> > > > vegetables, and most grains) I can't eat most potassium-rich > > foods.> > > >> > > > - msmith1928> > > > 45, female, 5'3", 120 lbs, 1cm left adrenal nodule, aldosterone> > > > 42.3, renin 0.5, potassium <2.9 (when not taking supplements); > > 25mg> > > > spiro caused gynecomastia, no HTN meds; other meds are 20MEQ K 2x/> > > > day, singulair 10mg, norethindrone .35mg, and cyclobenzaprine > > 5mg as> > > > needed; low sodium, fructose- and grain-free diet> > > >> > > >> > > >> > >> >> >> >>

[Guest guest]

Fructose malabsorptionFrom Wikipedia, the free encyclopediaJump to: navigation,

search

Fructose Malabsorption

Classification and external resources

Fructose

ICD-10 E74.3

ICD-9 271

OMIM 138230

Fructose malabsorption, formerly named " dietary fructose intolerance, " is a

digestive disorder[1] in which absorption of fructose is impaired by deficient

fructose carriers in the small intestine's enterocytes. This results in an

increased concentration of fructose in the entire intestine.

Fructose malabsorption is found in up to 30% of the population of Western

countries.[2]

This condition is common in patients identified to be suffering symptoms of

irritable bowel syndrome, although occurrence in these patients is not higher

than occurrence in the normal population. Conversely, patients with fructose

malabsorption often fit the profile of those with irritable bowel syndrome.[3] A

small proportion of patients with both fructose malabsorption and lactose

intolerance also suffer from celiac disease.

Fructose malabsorption is not to be confused with hereditary fructose

intolerance, a potentially fatal condition in which the liver enzymes that break

up fructose are deficient.

Contents [hide]

1 Pathophysiology

2 Symptoms

3 Diagnosis

4 Treatment

4.1 Diet

4.1.1 Foods with high fructose content

4.1.2 Foods with high fructan content

4.1.3 Other problem foods

4.2 Dietary guidelines for the management of fructose malabsorption

4.2.1 Unfavorable foods (i.e. more fructose than glucose)

4.2.2 Favorable foods (i.e. fructose equal to or less than glucose)

5 United States Food-labeling laws

6 New research

7 See also

8 References

9 External links

PathophysiologyFructose is absorbed in the small intestine without help of

digestive enzymes. Even in healthy persons, however, only about 25-50g of

fructose per sitting can be properly absorbed. Persons with fructose

malabsorption may absorb less than 25g per sitting (NB: Amount is arbitrarily

determined according to investigation of fructose absorption in many

individuals).[4] In the large intestine, fructose that hasn't been adequately

absorbed osmotically reduces the absorption of water and is metabolized by

normal colonic bacteria to short chain fatty acids and the gases hydrogen,

carbon dioxide and methane. This abnormal increase in hydrogen is detectable

with the hydrogen breath test.

The physiological consequences of fructose malabsorption include increasing

osmotic load, providing substrate for rapid bacterial fermentation, changing

gastrointestinal motility, promoting mucosal biofilm and altering the profile of

bacteria. These effects are additive with other short-chain poorly absorbed

carbohydrates such as sorbitol. The clinical significance of these events

depends upon the response of the bowel to such changes; they have a higher

chance of inducing symptoms in people with functional gut disorders than

asymptomatic subjects. Some effects of fructose malabsorption are decreased

tryptophan, folic acid[5] and zinc in the blood.[6]

Restricting dietary intake of free fructose and/or fructans may provide symptom

relief in a high proportion of patients with functional gut disorders, but

quality evidence may be lacking.[7]

SymptomsBloating (from fermentation in the small and large intestine)

Diarrhea and/or constipation

Flatulence

Stomach pain (as a result of muscle spasms, the intensity of which can vary from

mild and chronic to acute but erratic)

Vomiting (if great quantities are consumed)

Early signs of mental depression[8]

DiagnosisThe diagnostic test, when used, is similar to that used to diagnose

lactose intolerance. It is called a hydrogen breath test and is the method

currently used for a clinical diagnosis.

TreatmentThere is no known cure, but an appropriate diet will help.

DietFoods that should be avoided by people with fructose malabsorption include:

Foods and beverages containing greater than 0.5g fructose in excess of glucose

per 100g and greater than 0.2g of fructans per serving should be avoided. Foods

with > 3 g per fructose per serving are termed a `high fructose load' and

possibly at-risk of inducing symptoms. However, the concept of a `high fructose

load' has not been evaluated in terms of its importance in the success of the

diet.[9]

Fructose consumed in large quantities

Foods with high fructose-to-glucose ratio (NB: Glucose enhances absorption of

fructose, so fructose from foods with fructose-to-glucose ratio <1, like

bananas, are readily absorbed, while foods with fructose-to-glucose ratio >1,

like apples and pears, are often problematic regardless of total amount of

fructose in the food[10]

Foods rich in fructans and other Fermentable Oligo-, Di- and Mono-saccharides

and Polyols (FODMAPs)

Foods rich in sorbitol

Foods such as high fructose corn syrup (HFCS) or honey

Foods with a high glucose content ingested with foods containing excess fructose

may help sufferers absorb the excess fructose.[11]

Foods with high fructose contentAccording to the USDA database,[12] foods with

more fructose than glucose include:

Food Fructose (grams / 100 grams) Glucose (grams / 100 grams)

Sucrose

(for reference) 50 50

Apples 5.9 2.4

Pears 6.2 2.8

Fruit juice

e.g. Apples,

Pears 5 to 7 2 to 3

Watermelon 3.4 1.6

Raisins 29.8 27.8

Honey 40.9 35.7

High fructose

corn syrup 55 to 90 45 to 10

The USDA food database reveals that many common fruits contain nearly equal

amounts of the fructose and glucose, and they do not present problems for those

individuals with fructose malabsorption.[13] Some fruits with a greater ratio of

fructose than glucose are apples, pears and watermelon, which contain more than

twice as much fructose as glucose. Fructose levels in grapes varies depending on

ripeness and variety, where unripe grapes contain more glucose.

Foods with high fructan contentChains of fructose molecules known as fructans

occur naturally in many foods. The following foods have a high fructan content:

Artichokes

Asparagus

Leeks

Onions including spring onion

Wheat including most beers, breads, cakes, biscuits, breakfast cereals, pies,

pastas, pizzas, and some noodles

The role that fructans play in fructose malabsorption is still under

investigation. However, it is recommended that fructan intake for fructose

malabsorbers should be kept to less than 0.5 grams/serving,[14] and supplements

with inulin and fructooligosaccharide (FOS), both fructans, should be

avoided.[14]

Other problem foodsIn addition, the following foods should also be avoided:

Sorbitol (present in some diet drinks and foods, and occurring naturally in some

stone fruits)

Xylitol present in some berries, and other polyols (sugar alcohols), such as

erythritol, mannitol, and other ingredients that end with -tol, commonly added

as artificial sweeteners in commercial foods.

Any processed foods or foods prepared by others containing the foods listed

above

For example:

Sodas and other beverages containing high fructose corn syrup (HFCS)

Dried fruit (including " health " bars containing dried fruit)

Tinned fruit in " natural " juice (often, this is pear juice)

Sweet wines

Dietary guidelines for the management of fructose malabsorptionDietary

guidelines[14] have been developed for managing fructose malabsorption

particularly for individuals with IBS.

Unfavorable foods (i.e. more fructose than glucose)Fruit — apple, pear, guava,

honeydew melon, nashi fruit, pawpaw, papaya, quince, star fruit, watermelon

Dried fruit - apple, currant, date, fig, pear, raisin, sultana

Honey

Fortified wines

High Fructose Corn Syrup - many processed products contain this

Corn syrup solids

Fruit juice concentrates

Agave nectar[not in citation given]

Tomato products (some individuals can tolerate small quantities of ripe

tomatoes)

Brown rice[not in citation given]

Squash[not in citation given]

sweetened cereal, wheat cereal, bran cereal (plain corn cereal may be tolerated)

Lettuce

Some wheat products

Vinegar[not in citation given] (balsamic, red wine vinegar, apple cider vinegar)

-- distilled vinegar may be tolerated

Favorable foods (i.e. fructose equal to or less than glucose)Stone fruit:

apricot, nectarine, peach, plum (caution - these fruits contain sorbitol)

Berry fruit: blueberry, blackberry, boysenberry, cranberry, raspberry,

strawberry, loganberry

Citrus fruit: kumquat, grapefruit, lemon, lime, mandarin, orange, tangelo

Other fruits: ripe banana, jackfruit, kiwi fruit, passion fruit, pineapple,

rhubarb, tamarillo

Vegetables: celery,[not in citation given] spinach,[not in citation given]

potatoes (white)[not in citation given]

Grains: Rye bread, corn tortillas, grits, oatmeal, unsweetened corn cereal (corn

puffs), rice

Meat[not in citation given] - any meat is favorable[not in citation given]

Dairy[not in citation given] - unsweetened dairy (milk, cottage cheese,

cheese)[not in citation given] is favorable in those who can digest lactose

Nuts[not in citation given], especially pistachios[not in citation given] (other

nuts are also well tolerated in most individuals)

United States Food-labeling lawsProducers of processed food in the USA are not

currently required by law to mark foods containing " fructose in excess of

glucose. " This can cause some surprises and pitfalls for fructose malabsorbers.

Foods (such as bread) marked " gluten-free " are usually suitable for fructose

malabsorbers, though sufferers need to be careful of gluten-free foods that

contain dried fruit or high fructose corn syrup or fructose itself in sugar

form. However, fructose malabsorbers do not need to avoid gluten, as those with

celiac disease must.

Many fructose malabsorbers can eat breads made from rye and corn flour. However,

these may contain wheat unless marked " wheat-free " (or " gluten-free " ) (Note, rye

bread is NOT gluten-free). Although often assumed to be an acceptable

alternative to wheat, spelt flour is not suitable for sufferers of fructose

malabsorption[citation needed], just as it is not appropriate for those with

wheat allergies or celiac disease. However, some fructose malabsorbers do not

have difficulty with fructans from wheat products while they may have problems

with foods that contain excess free fructose.[7]

There are many breads on the market that boast having no High Fructose Corn

Syrup. In lieu of high fructose corn syrup, however, one may find the production

of special breads with a high inulin content, where inulin is a replacement in

the baking process for the following: high fructose corn syrup, flour and fat.

Because of the caloric reduction, lower fat content, dramatic fiber increase and

prebiotic tendencies of the replacement inulin, these breads are considered a

healthier alternative to traditionally prepared leavening breads. Though the

touted health benefits may exist, sufferers of fructose malabsorption will

likely find no difference between these new breads and traditionally prepared

breads in alleviating their symptoms because inulin is a fructan, and, again,

consumption of fructans should be reduced dramatically in those with fructose

malabsorption in an effort to appease symptoms.

New researchA low FODMAP diet is now sufficiently evidenced for efficacy, and

its widespread application in conditions such as IBS and IBD is recommended.[15]

Restriction of Fermentable Oligo-, Di- and Mono-saccharides and Polyols

globally, rather than individually, controls the symptoms of functional gut

disorders (e.g. IBS), and the majority of IBD patients respond just as well. For

those who suffer from fructose malabsorption, a low FODMAP diet may be more

successful than restricting only fructose and fructans, as is currently

recommended.[16] Compliance to the diet is high.

See alsoGastroenterology

Hydrogen breath test

Invisible disability

Food intolerance

Irritable bowel syndrome

Malabsorption

References1.^ MayoClinic.com

2.^ Born: Carbohydrate malabsorption in patients with non-specific

abdominal complaints World Journal of Gastroenterology, 2007, ISSN 1007-9327.

3.^ Ledochowski M et al.: Fruktosemalabsorption. Journal für Ernährungsmedizin,

2001 (German)

4.^ http://www.uihealthcare.com/kxic/2008/06/fructose.html

5.^ Maximilian Ledochowski, Florian Überall, Theresia Propst, and Dietmar Fuchs

Fructose Malabsorption Is Associated with Lower Plasma Folic Acid Concentrations

in Middle-Aged Subjects, Clin. Chem., Nov 1999; 45: 2013 - 2014.

6.^ Ledochowski M, Uberall F, Propst T, Fuchs D (1999). " Fructose malabsorption

is associated with lower plasma folic acid concentrations in middle-aged

subjects " . Clin. Chem. 45 (11): 2013–4. PMID 10545075.

http://www.clinchem.org/cgi/content/full/45/11/2013.

7.^ a b Gibson PR, Newnham E, Barrett JS, Shepherd SJ, Muir JG (2007). " Review

article: fructose malabsorption and the bigger picture " . Aliment. Pharmacol.

Ther. 25 (4): 349–63. doi:10.1111/j.1365-2036.2006.03186.x. PMID 17217453.

8.^ Ledochowski M, Sperner-Unterweger B, Widner B, Fuchs D. (2010). Fructose

malabsorption is associated with early signs of mental depression..

http://www.ncbi.nlm.nih.gov/pubmed/9620891.

9.^ Gibson PR, Shepherd SJ (2010). " Evidence-based dietary management of

functional gastrointestinal symptoms: The FODMAP approach " . Advances in Clinical

Practice 25 (2): 252–258. doi:10.1111/j.1440-1746.2009.06149.x. PMID 20136989.

http://www3.interscience.wiley.com/journal/122650565/abstract.

10.^

http://www.healthsystem.virginia.edu/internet/digestive-health/nutrition/Barrett\

Article.pdf

11.^ Skoog SM, Bharucha AE (2004). " Dietary fructose and gastrointestinal

symptoms: a review " . Am. J. Gastroenterol. 99 (10): 2046–50.

doi:10.1111/j.1572-0241.2004.40266.x. PMID 15447771.

http://www.bashaar.org.il/files/101022005111814.pdf.

12.^ USDA National Nutrient Database Release 20, September 2007

13.^ Sugar Content of Selected Foods: Individual and Total Sugars Ruth H.

s, Pamela R. Pehrsson, and Mojgan Farhat-Sabet, (1987) U.S.D.A.

14.^ a b c Shepherd SJ, Gibson PR (2006). " Fructose malabsorption and symptoms

of irritable bowel syndrome: guidelines for effective dietary management " .

Journal of the American Dietetic Association 106 (10): 1631–9.

doi:10.1016/j.jada.2006.07.010. PMID 17000196.

15.^ Gibson PR, Shepherd SJ. (Feb 2010). " Evidence-based dietary management of

functional gastrointestinal symptoms: The FODMAP approach. " . J Gastroenterol

Hepatol. 25 (2): 252–8.. doi:10.1111/j.1440-1746.2009.06149.x. PMID 20136989.

16.^ Fructose Malabsorption

External linksFructose Malabsorption at the food intolerance network -- Society

for Public Health

Fructose Malabsorption -- Practical Gastroenterology

Low FODMAP Diet

Ledochowski M, Widner B, Bair H, Probst T, Fuchs D (2000). " Fructose- and

sorbitol-reduced diet improves mood and gastrointestinal disturbances in

fructose malabsorbers " . Scand. J. Gastroenterol. 35 (10): 1048–52.

doi:10.1080/003655200451162. PMID 11099057.

Ledochowski M, Uberall F, Propst T, Fuchs D (1999). " Fructose malabsorption is

associated with lower plasma folic acid concentrations in middle-aged subjects " .

Clin. Chem. 45 (11): 2013–4. PMID 10545075.

http://www.clinchem.org/cgi/content/full/45/11/2013.

[show]v · d · eDigestive system · Digestive disease · Gastroenterology

(primarily K20–K93, 530–579)

Upper GI tract Esophagus Esophagitis (Candidal, Herpetiform) · rupture

(Boerhaave syndrome, Mallory-Weiss syndrome) · UES (Zenker's diverticulum) · LES

(Barrett's esophagus) · Esophageal motility disorder (Nutcracker esophagus,

Achalasia, Diffuse esophageal spasm, Gastroesophageal reflux disease (GERD)) ·

Laryngopharyngeal reflux (LPR) · Esophageal stricture · Megaesophagus

Stomach Gastritis (Atrophic, Ménétrier's disease, Gastroenteritis) · Peptic

(gastric) ulcer (Cushing ulcer, Dieulafoy's lesion) · Dyspepsia · Pyloric

stenosis · Achlorhydria · Gastroparesis · Gastroptosis · Portal hypertensive

gastropathy · Gastric antral vascular ectasia · Gastric dumping syndrome ·

Gastric volvulus

Lower GI tract:

Intestinal/

enteropathy Small intestine/

(duodenum/jejunum/ileum) Enteritis (Duodenitis, Jejunitis, Ileitis) — Peptic

(duodenal) ulcer (Curling's ulcer) — Malabsorption: Coeliac · Tropical sprue ·

Blind loop syndrome · Whipple's · Short bowel syndrome · Steatorrhea · Milroy

disease

Large intestine

(appendix/colon) Appendicitis · Colitis (Pseudomembranous, Ulcerative, Ischemic,

Microscopic, Collagenous, Lymphocytic) · Functional colonic disease (IBS,

Intestinal pseudoobstruction/Ogilvie syndrome) — Megacolon/Toxic megacolon ·

Diverticulitis/Diverticulosis

Large and/or small Enterocolitis (Necrotizing) · IBD (Crohn's disease) —

vascular: Abdominal angina · Mesenteric ischemia · Angiodysplasia — Bowel

obstruction: Ileus · Intussusception · Volvulus · Fecal impaction — Constipation

· Diarrhea (Infectious) · Intestinal adhesions

Rectum Proctitis (Radiation proctitis) · Proctalgia fugax · Rectal prolapse ·

Anismus

Anal canal Anal fissure/Anal fistula · Anal abscess · Anal dysplasia ·

Pruritus ani

GI bleeding/BIS Upper (Hematemesis, Melena) · Lower (Hematochezia)

Accessory Liver Hepatitis (Viral hepatitis, Autoimmune hepatitis, Alcoholic

hepatitis) · Cirrhosis (PBC) · Fatty liver (NASH) · vascular (Budd-Chiari

syndrome, Hepatic veno-occlusive disease, Portal hypertension, Nutmeg liver) ·

Alcoholic liver disease · Liver failure (Hepatic encephalopathy, Acute liver

failure) · Liver abscess (Pyogenic, Amoebic) · Hepatorenal syndrome · Peliosis

hepatis

Gallbladder Cholecystitis · Gallstones/Cholecystolithiasis · Cholesterolosis ·

Rokitansky-Aschoff sinuses · Postcholecystectomy syndrome · Porcelain

gallbladder

Bile duct/

other biliary tree Cholangitis (PSC, Secondary sclerosing cholangitis,

Ascending) · Cholestasis/Mirizzi's syndrome · Biliary fistula · Haemobilia ·

Gallstones/Cholelithiasiscommon bile duct (Choledocholithiasis, Biliary

dyskinesia) · Sphincter of Oddi dysfunction

Pancreatic Pancreatitis (Acute, Chronic, Hereditary, Pancreatic abscess) ·

Pancreatic pseudocyst · Exocrine pancreatic insufficiency · Pancreatic fistula

Abdominopelvic Hernia Diaphragmatic (Congenital) · HiatusInguinal (Indirect,

Direct) · Umbilical · Femoral · Obturator · Spigelianlumbar (Petit's,

Grynfeltt-Lesshaft)undefined location (Incisional · Internal hernia)

Peritoneal Peritonitis (Spontaneous bacterial peritonitis) · Hemoperitoneum ·

Pneumoperitoneum

M: DIG

anat(t, g, p)/phys/devp/cell/enzy

noco/cong/tumr, sysi/epon

proc, drug(A2A/2B/3/4/5/6/7/14/16), blte

[show]v · d · eGenetic disorder, membrane: Solute carrier disorders

1-10 SLC1A3 (Episodic ataxia 6) · SLC2A1 (De Vivo disease) · SLC2A5 (Fructose

malabsorption) · SLC2A10 (Arterial tortuosity syndrome) · SLC3A1 (Cystinuria) ·

SLC4A1 (Hereditary spherocytosis 4/Hereditary elliptocytosis 4) · SLC4A11

(Congenital endothelial dystrophy type 2, Fuchs' dystrophy 4) · SLC5A1

(Glucose-galactose malabsorption) · SLC5A2 (Renal glycosuria) · SLC5A5 (Thyroid

dyshormonogenesis type 1) · SLC6A19 (Hartnup disease) · SLC7A7 (Lysinuric

protein intolerance) · SLC7A9 (Cystinuria)

11-20 SLC11A1 (Crohn's disease) · SLC12A3 (Gitelman syndrome) · SLC16A1 (HHF7) ·

SLC16A2 (Allan–Herndon–Dudley syndrome) · SLC17A5 (Salla disease) · SLC17A8

(DFNA25)

21-40 SLC26A2 (Multiple epiphyseal dysplasia 4, Achondrogenesis type 1B,

Recessive multiple epiphyseal dysplasia, Atelosteogenesis, type II, Diastrophic

dysplasia) · SLC26A4 (Pendred syndrome) · SLC35C1 (CDOG 2C) · SLC39A4

(Acrodermatitis enteropathica) · SLC40A1 (African iron overload)

see also solute carrier family

B structural (perx, skel, cili, mito, nucl, sclr) · DNA/RNA/protein synthesis

(drep, trfc, tscr, tltn) · membrane (icha, slcr, atpa, abct, othr) ·

transduction (iter, csrc, itra), trfk

[show]v · d · eInborn error of carbohydrate metabolism: monosaccharide

metabolism disorders (including glycogen storage diseases) (E73–E74, 271)

Sucrose, transport

(extracellular) Disaccharide catabolism Lactose intolerance · Sucrose

intolerance

Monosaccharide transport Glucose-galactose malabsorption · Inborn errors of

renal tubular transport (Renal glycosuria) · Fructose malabsorption

Hexose & #8594; glucose Monosaccharide catabolism fructose: Essential fructosuria

· Fructose intolerancegalactose/galactosemia : GALK deficiency · GALT

deficiency/GALE deficiency

Glucose & #8644; glycogen Glycogenesis GSD type 0, glycogen synthase · GSD type

IV, Andersen's, branching

Glycogenolysis extralysosomal: GSD type V, McArdle, muscle glycogen

phosphorylase/GSD type VI, Hers', liver glycogen phosphorylase · GSD type III,

Cori's, debranchinglysosomal/LSD: GSD type II, Pompe's, glucosidase

Glucose & #8644; CAC Glycolysis MODY 2/HHF3 · GSD type VII, Tarui's,

phosphofructokinase · Triosephosphate isomerase deficiency · Pyruvate kinase

deficiency

Gluconeogenesis PCD · Fructose bisphosphatase deficiency · GSD type I, von

Gierke, glucose 6-phosphatase

Pentose phosphate pathway Glucose-6-phosphate dehydrogenase deficiency

Other Hyperoxaluria (Primary hyperoxaluria) · Pentosuria

M: MET

mt, k, c/g/r/p/y/i, f/h/s/l/o/e, a/u, n, h

k, cgrp/y/i, f/h/s/l/o, au, n, h, epon

m(A16, C10),i(k, c/g/r/p/y/i, f/h/s/o/e, a/u, n, h)

Retrieved from " http://en.wikipedia.org/wiki/Fructose_malabsorption "

Categories: Membrane transport protein disorders | Metabolic disorders

Hidden categories: All articles with unsourced statements | Articles with

unsourced statements from March 2011 | Articles with unsourced statements from

October 2010 | Use dmy dates from October 2010Personal tools

Log in / create accountNamespaces

ArticleDiscussionVariantsViews

ReadView sourceView historyActions

Search

Navigation

Main pageContentsFeatured contentCurrent eventsRandom articleDonate to

WikipediaInteractionHelpAbout WikipediaCommunity portalRecent changesContact

WikipediaToolboxWhat links hereRelated changesUpload fileSpecial pagesPermanent

linkCite this page

Print/exportCreate a bookDownload as PDFPrintable version

Languages & #1575; & #1604; & #1593; & #1585; & #1576; & #1610; & #1577;DeutschFrançaisNederla\

ndsThis page was last modified on 20 March 2011 at 01:31.

Text is available under the Creative Commons Attribution-ShareAlike License;

additional terms may apply. See Terms of Use for details.

Wikipedia® is a registered trademark of the Wikimedia Foundation, Inc., a

non-profit organization.

Contact us

Privacy policyAbout WikipediaDisclaimers

> > > > > >

> > > > > > > --- In hyperaldosteronism , Clarence Grim

> > > > > > > >

> > > > > > > > But if one is not ready for surgery because DASH and MCBs

> > > have

> > > > > > > failed

> > > > > > > > then I argue why take the risks (radiation) etc. and costs

> > > of an

> > > > > > > AVS.

> > > > > > >

> > > > > > > Dr Grim, I'm not sure if your response was meant for me or for

> > > > > others.

> > > > > > >

> > > > > > > If it was meant for me - I'm fully aware of your preference

> > > for

> > > > > DASH

> > > > > > > and MCBs before surgery. That's not working for me.

> > > > > > >

> > > > > > > I'm the original poster of this question, and we've been

> > > through

> > > > > > > this several times before - I can't take spiro, and due to

> > > > > inability

> > > > > > > to metabolize fructose and fructans (found in most fruits,

> > > many

> > > > > > > vegetables, and most grains) I can't eat most potassium-rich

> > > > > foods.

> > > > > > >

> > > > > > > - msmith1928

> > > > > > > 45, female, 5'3 " , 120 lbs, 1cm left adrenal nodule,

> > > aldosterone

> > > > > > > 42.3, renin 0.5, potassium <2.9 (when not taking supplements);

> > > > > 25mg

> > > > > > > spiro caused gynecomastia, no HTN meds; other meds are 20MEQ

> > > K 2x/

> > > > > > > day, singulair 10mg, norethindrone .35mg, and cyclobenzaprine

> > > > > 5mg as

> > > > > > > needed; low sodium, fructose- and grain-free diet

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > >

> > > > >

> > > > >

> > > > >

> > > >

> > >

> > >

> > >

> >

>